1 c-expressing lung DCs, preferentially in the alveolar septa.

2 g the alveolar walls, and projected into the alveolar septa.

3 were frequently in the interior of thickened alveolar septa.

4 , was primarily found at the free margins of alveolar septa.

5               IM density was greatest in the alveolar septa.

6 nd the second phase involves thinning of the alveolar septa.

7      Of the IMs, 78% were located within the alveolar septa, 14% around small vessels, and 7% around

8 chyma shows hypercellularity, with thickened alveolar septa and an increase in the number of endothel

9 issue) shows hypercellularity with thickened alveolar septa and an increase in the number of vascular

10 he only vessels present were the ones in the alveolar septa, and their presence highlighted, through

11 malformed, dilated, muscular arteries within alveolar septa; congested capillaries within alveolar wa

12 everely reduced capillary density, thickened alveolar septa containing centrally located dilated capi

13 displacement, destruction, or replacement of alveolar septa, distal airways, and small vessels within

14 ormation of the elastin ECM, thereby driving alveolar septa formation to increase the gas-exchange su

15 e nucleic acid (microg) was increased in the alveolar septa of emphysema lungs (14.2 +/- 2.0/microg,

16 wed marked inflammation and edema within the alveolar septa of SNV-infected hamsters, results which a

17 ed to correlate changes in components of the alveolar septa surrounding enlarged airspaces in human e

18  The local density of IMs was greater in the alveolar septa than in the connective tissue surrounding

19 haracterized by the formation of millions of alveolar septa that constitute the vast gas-exchange sur

20 lveolar epithelial type I and II cells, form alveolar septa, the functional units in the lung.

21 nent lymphocyte infiltrates that thicken the alveolar septa with poorly formed granulomas or giant ce