ライフサイエンスコーパス: amacrine

1 te to postsynaptic currents evoked in either amacrine.

2 and GluN2A-containing NMDA receptors on A17 amacrines.

3 of intracellular Ca(2+) in dendrites of both amacrines.

4 receptors can drive release of GABA from A17 amacrines.

5 The neurites of the retinal ganglion, amacrine and bipolar cell subtypes that form synapses in

6 he Ascl1-expressing Muller glia give rise to amacrine and bipolar cells and photoreceptors.

7 s of horizontal cells and reduced numbers of amacrine and bipolar cells, while the number of Muller g

8 nti-VEGF-treated WT mice also presented mild amacrine and ganglion cell death, but no overt abnormali

9 eurons that form microcircuits with bipolar, amacrine and ganglion cells to process visual informatio

10 y-stage retinal progenitors, differentiating amacrine and ganglion cells, and late-stage progenitors

11 essed strongly in BP cells and a fraction of amacrine and ganglion cells.

12 ory feedforward drive from photoreceptors to amacrine and ganglion cells.

13 nducible and proangiogenic factors, and that amacrine and horizontal cell dysfunction induces alterat

14 sities of early-born retinal ganglion cells, amacrine and horizontal cells, as well as cone photorece

15 d that a subset of retinal interneurons, the amacrine and horizontal cells, form neurovascular units

16 rgic cells and their postsynaptic cells, AII amacrine and melanopsin-containing retinal ganglion cell

17 sruptions of synaptic structures and loss of amacrine and retinal ganglion cells in anti-VEGF treated

18 the inner nuclear layer as well as widefield amacrine and small bistratified ganglion cells in the ga

19 us studies have suggested the involvement of amacrine and/or horizontal cells.

20 tered organization of NMDA receptors on both amacrines and a close spatial association between GluN2B

21 established Dbn1 as a new marker enriched in amacrines and Fmnl3 as a marker for subsets of alphaRGCs

22 ound GluN2B-containing NMDA receptors on AII amacrines and GluN2A-containing NMDA receptors on A17 am

23 to multiple cell types, including ganglion, amacrine, and bipolar cells and photoreceptors, but not

24 o spatially map individual GCs to underlying amacrine, bipolar, horizontal, photoreceptor, and retina

25 ence of impaired vesicular GABA release from amacrine but not horizontal interneurons.

26 ipolar cell (BC) classes inhibit rod BCs via amacrine cell (AC) motifs (C1-6); that all cone BC class

27 Although no change was detected in total amacrine cell (AC) numbers, increased PRKCA(+) and choli

28 The central neuron in this pathway, the AII amacrine cell (AC), exhibits a spatially tuned receptive

29 it in the vertebrate retina, whereby the AII amacrine cell (AII AC) provides inhibition onto cone bip

30 The AII amacrine cell (AII) is a key information hub in the reti

31 ized type of amacrine cell, the dopaminergic amacrine cell (DAC).

32 cine and glutamate, by the vGluT3-expressing amacrine cell (GAC) in the mouse retina.

33 rientation-selective, wide-field, polyaxonal amacrine cell (PAC) in the rabbit retina and demonstrate

34 individual dendritic sectors of a starburst amacrine cell (SAC) are preferentially activated by diff

35 es light-dependent spiking primarily through amacrine cell activation.

36 The AII amacrine cell also makes direct glycinergic synapses wit

37 cted ON-OFF segregation within a small-field amacrine cell arose from local synaptic processing, medi

38 of other ON parasol cells recorded near the amacrine cell axonal projections.

39 p to further our understanding of how single amacrine cell circuits act together to help decompose th

40 Studies investigating individual amacrine cell circuits like the starburst or A17 circuit

41 of postsynaptic plasticity in a subgroup of amacrine cell dendrites.

42 neurotransmitter release sites on starburst amacrine cell dendrites: the excitatory input distributi

43 The amacrine cell density ranged from 30,000 cells/mm(2) at

44 cadherin Fat3 acts during multiple stages of amacrine cell development in mice to orient overall chan

45 Light-evoked depolarizations of the AII-amacrine cell elicited exocytosis that was graded to lig

46 e cells are a recently characterized type of amacrine cell exhibiting local dendritic autonomy.

47 Finally, Sox2, Sox8, and Sox9 promoted amacrine cell formation in Lhx2-deficient cells, but not

48 ct the competence of OTX2+ cells, preventing amacrine cell formation.

49 The starburst amacrine cell in the mouse retina presents an opportunit

50 e from the presynaptic interneuron starburst amacrine cell in the mouse retina.

51 our understanding of how general features of amacrine cell inhibition lead to general features of com

52 vity of a retinal interneuron called the AII amacrine cell is responsible for anti-correlated spiking

53 This S-cone amacrine cell makes highly selective inhibitory synapses

54 e cells immunolabeled for an RGC marker, not amacrine cell markers, suggesting that they are dopamine

55 the periphery, via the electrically coupled amacrine cell network.

56 We focused on amacrine cell number control, identifying three downstre

57 yses placed Pten downstream of TgfbetaRII in amacrine cell number control.

58 we show that Pten is a critical regulator of amacrine cell number in the retina, acting via multiple

59 In PD, dopaminergic amacrine cell number was reduced between 58% and 26% in

60 s cholinergic activity or reducing starburst amacrine cell numbers prevents invasion of endothelial c

61 s sufficient to recapitulate the Fat3 mutant amacrine cell phenotype.

62 We identify a type of polyaxonal amacrine cell physiologically via its distinctive electr

63 Cpne4 expression is restricted to one amacrine cell population of the INL, but is specifically

64 istry and major cell types of a VIP-ires-Cre amacrine cell population.

65 etinal explants phenocopies the reduction in amacrine cell production observed in Pten cKOs.

66 Pten is thus a positive regulator of amacrine cell production, acting via multiple downstream

67 ts, which is a negative feedback pathway for amacrine cell production.

68 Identification of the S-cone amacrine cell provides the missing component of an evolu

69 l subtypes, retinal ganglion cells, and some amacrine cell subtypes but not significantly in Muller c

70 levels to promote the differentiation of all amacrine cell subtypes, which are each reduced in number

71 urotransmitter release at bipolar neuron/AII amacrine cell synapses and rendered spontaneous miniatur

72 Our results suggest that AII-amacrine cell synapses are capable of providing both pha

73 Bipolar but not amacrine cell synapses have presynaptic ribbon-like stru

74 aptic transmission at rod bipolar neuron-AII amacrine cell synapses in acute mouse retina slices as a

75 isolates a specific pathway through the AII amacrine cell that does not require iGluRs: cone->ON con

76 isolates a specific pathway through the AII amacrine cell that does not require iGluRs: cone-->ON co

77 that enables a small-field, dual-transmitter amacrine cell to process diverse dendritic functions in

78 el cholinergic, non-GABAergic, non-starburst amacrine cell type described for the first time in teleo

79 se and test a model for the function of this amacrine cell type, in which the extra-classical recepti

80 The VIP-ires-Cre amacrine cell types were identified in VIP-Brainbow2.1 r

81 s some other subsets of retinal ganglion and amacrine cell types, along with horizontal cells, while

82 function of most of the approximately 30-40 amacrine cell types, each of which synapses onto a subse

83 es and intercellular connections of specific amacrine cell types.

84 The AII amacrine cell uses sign-conserving electrical synapses t

85 One presynaptic amacrine cell was identified as semilunar type 2, a poly

86 terminals in the IPL, as well as a putative amacrine cell with their cell bodies in the inner nuclea

87 s for one type of rod pathway interneuron (A amacrine cell) in the retina of some but not all mammali

88 revealing a novel inhibitory interneuron, an amacrine cell, receiving excitatory glutamatergic input

89 he exception of the rod pathway-specific AII amacrine cell, the connectivity of glycinergic small-fie

90 esized and released by a specialized type of amacrine cell, the dopaminergic amacrine cell (DAC).

91 uire iGluRs: cone->ON cone bipolar cell->AII amacrine cell->RGC.

92 re iGluRs: cone-->ON cone bipolar cell-->AII amacrine cell-->RGC.

93 mmalian retina, gap junctions within the Aii amacrine cell-ON cone bipolar cell (CBC) network are ess

94 inhibitory input from an undiscovered S-cone amacrine cell.

95 ized circuit: rods-->rod bipolar cells-->AII amacrine cell.

96 ating direction selectivity in the starburst amacrine cell.

97 ipolar cells as the rod pathway-specific AII amacrine cell.

98 d connectivity pattern of the small-field A8 amacrine cell.

99 alized circuit: rods->rod bipolar cells->AII amacrine cell.

100 Amacrine cells (ACs) are a diverse class of interneurons

101 glutamate transporter 3 (VGluT3)-expressing amacrine cells (ACs) to a broad set of visual stimuli.

102 We genetically identify a class of amacrine cells (ACs) with elongated dendritic arbors tha

103 GCs), followed by horizontal cells (HCs) and amacrine cells (ACs), beginning with the early stages of

104 r the structural and functional integrity of amacrine cells (ACs), the largest cohort of neurons in t

105 from bipolar cells and inhibitory input from amacrine cells (ACs).

106 ypes, horizontal cells, as well as GABAergic amacrine cells (ACs).

107 onses of individual bipolar cells (BCs), AII amacrine cells (AIIACs), and ON and sustained OFF alpha-

108 ced glial activation and loss of function of amacrine cells (brain nitric oxide synthetase/tyrosine h

109 in the number of synaptic contacts with AII amacrine cells (by 60%) and melanopsin cells (by 35%).

110 Dopaminergic amacrine cells (DACs) release dopamine in response to li

111 electrophysiological inputs to dopaminergic amacrine cells (DACs), we show here that the release of

112 ar layer amacrine cells (iACs) and displaced amacrine cells (dACs)--reach their specific laminar posi

113 e source of retinal dopamine is dopaminergic amacrine cells (DACs).

114 make connections with upstream dopaminergic amacrine cells (DACs): (1) ipRGC signaling to DACs is bl

115 e found that vGluT3-expressing glutamatergic amacrine cells (GACs) generate ON-OFF somatic responses

116 -horizontal cells (HCs), inner nuclear layer amacrine cells (iACs) and displaced amacrine cells (dACs

117 ian retina, inhibitory inputs onto starburst amacrine cells (SACs) are required for robust direction

118 The dendrites of starburst amacrine cells (SACs) in the mammalian retina are prefer

119 inhibition arising from GABAergic starburst amacrine cells (SACs) strongly contributes to direction

120 inhibitory neurotransmission from starburst amacrine cells (SACs) to direction selective ganglion ce

121 acetylcholine (ACh) and GABA from starburst amacrine cells (SACs) to direction-selective ganglion ce

122 nglion cells (DSGCs) and GABAergic starburst amacrine cells (SACs), and the SACs then provide FF inhi

123 In this study, we used retinal starburst amacrine cells (SACs), critical components of a directio

124 rodents, retinal waves initiate in starburst amacrine cells (SACs), propagating across retinal gangli

125 Here, we focused on one such pair, starburst amacrine cells (SACs), to explore how closely related ne

126 rs (beta2-nAChRs) selectively from starburst amacrine cells (SACs), we show that mutual excitation am

127 hibition, which is provided by OFF starburst amacrine cells (SACs).

128 ulated cholinergic excitation from starburst amacrine cells (SACs).

129 Directional GABA release from starburst amacrine cells (SBACs) is critical for generating direct

130 release from the dendritic tips of starburst amacrine cells (SBACs).

131 sitive and shows the morphology of widefield amacrine cells (stellate, semilunar, and thorny amacrine

132 study visual processing in VGluT3-expressing amacrine cells (VG3-ACs) in the mouse retina.

133 s to the recruitment of GABAergic wide-field amacrine cells (WACs) endowing the DS circuit with an ad

134 re, our results reveal a class of wide-field amacrine cells (WACs) with straight, unbranching dendrit

135 typical network formed by different types of amacrine cells across the inner plexiform layer prompts

136 ce that general functions of the ensemble of amacrine cells across types are critical for establishin

137 of glutamate (or AMPA) onto the dendrites of amacrine cells also significantly potentiated evoked cur

138 sity of parvalbumin- and calretinin-positive amacrine cells and a loss of ganglion cells was detected

139 ied input to the melanopsin-ir RGCs from AII amacrine cells and directly from rod bipolar cells via r

140 ity in the shape and function of the studied amacrine cells and elucidate their connections with spec

141 plexiform layer (IPL) and from dopaminergic amacrine cells and GABAergic processes in the outermost

142 identified in photoreceptors, bipolar cells, amacrine cells and ganglion cells, but have not been con

143 ns, including photoreceptors, bipolar cells, amacrine cells and ganglion cells, but they have not bee

144 DA amacrine cells and M1 ipRGCs express the SRIF receptor s

145 circuit composed of dopamine (DA)-containing amacrine cells and melanopsin-containing, intrinsically

146 , it has all cell types and layers including amacrine cells and Muller glia.

147 Retinae derived from f-iPSCs had fewer amacrine cells and other inner nuclear layer cells.

148 c "retinal waves" are initiated in starburst amacrine cells and propagate to retinal ganglion cells a

149 and bipolar cells were next most central and amacrine cells and retinal ganglion cells were on the ou

150 (GCL) and inner nuclear layer (INL) in both amacrine cells and RGCs.

151 med to analyze the state of the dopaminergic amacrine cells and some of their main postsynaptic neuro

152 ndene-1,4'-piperidine]-1'-carboxamide) in DA amacrine cells and the selective sst4 agonist L-803,087

153 Starburst amacrine cells and their synaptic partners, ON-OFF direc

154 Dopamine is released by retinal dopaminergic amacrine cells and transmits signaling either by convent

155 that melanopsin cells were tracer coupled to amacrine cells and would be applicable to electrophysiol

156 Amacrine cells are a heterogeneous group of interneurons

157 Vesicular glutamate transporter 3 (VGluT3) amacrine cells are a recently characterized type of amac

158 VIP-1 amacrine cells are bistratified, wide-field cells that r

159 AII-amacrine cells are interneurons in the retina that exhib

160 apse imaging assay, we found that developing amacrine cells are less directed towards the IPL in the

161 ramify in strata 1, 4, and 5, VIP-2A and 2B amacrine cells are medium-field cells that mainly ramify

162 amify in strata 3 and 4, and VIP-3 displaced amacrine cells are medium-field cells that ramify in str

163 acetyltransferase (ChAT) expressing retinal amacrine cells are present across vertebrates.

164 Amacrine cells are thought to be a major locus for mecha

165 eceiving direct photoreceptor input, whereas amacrine cells are usually monopolar inhibitory interneu

166 Most of these amacrine cells are wide field, although some are medium

167 A labels astrocytes on the day of birth, AII amacrine cells at postnatal (P) day 5, and Muller glia b

168 ased the generation of mislocalized ganglion/amacrine cells at the expense of rod and cone photorecep

169 However, AII amacrine cells avoid making synapses with numerous RGC t

170 Amacrine cells comprise approximately 30 morphological t

171 We find that miR-181a is expressed in amacrine cells during development and in adult retinas,

172 Developing AII amacrine cells exhibit accumulating DNER labeling at the

173 VIP-ires-Cre amacrine cells form a neuropeptide-expressing cell popul

174 x of Ca(2+) in dendritic varicosities of A17 amacrine cells from diabetic compared with normal animal

175 the connectivity of glycinergic small-field amacrine cells has not been investigated in the mouse re

176 mma-aminobutyric acid (GABA)ergic wide-field amacrine cells have recently been studied; however, with

177 ctrical coupling between RGCs and polyaxonal amacrine cells in mouse retina forms the synaptic mechan

178 ciated virus-mediated technique to label AII amacrine cells in mouse retina, we observed diminished d

179 he full complement of rod photoreceptors and amacrine cells in mouse.

180 rved diminished dopaminergic contacts to AII amacrine cells in RD mice.

181 amine is released by a specialized subset of amacrine cells in response to light and has a potent inf

182 tically connected rod bipolar and AII or A17 amacrine cells in retinal slices from female rats, we fo

183 d with targeted patch-clamp recordings of DA amacrine cells in TH-RFP mice and M1 ipRGCs in OPN4-EGFP

184 ear total RGC loss with a marked increase in amacrine cells in the ganglion cell layer.

185 against calretinin can be used to identify A amacrine cells in the inner nuclear layer as well as wid

186 ns, receive synaptic inputs from bipolar and amacrine cells in the inner plexiform layer (IPL) and se

187 bipolar cells are presynaptic to AII and A17 amacrine cells in the inner plexiform layer.

188 fia), that are each heavily expressed in AII amacrine cells in the mature mouse retina, and which con

189 edianus magnocellularis, the spinal cord and amacrine cells in the retina.

190 ateral inhibition onto Off SACs from non-SAC amacrine cells is required for optimal direction selecti

191 The amacrine cells labeled in Tg(mglur6b:EGFP)zh1 constitute

192 e from its presynaptic arrays of bipolar and amacrine cells less efficiently than the OFF cell does.

193 These recordings show that AII amacrine cells make direct synapses with OFF Alpha, OFF

194 Because Ca(2+)-permeable receptors in A17 amacrine cells mediate synaptic release of GABA, the red

195 For instance, retinal amacrine cells migrate towards the inner plexiform layer

196 the inner plexiform layer, where inhibitory amacrine cells modulate the excitatory signal of bipolar

197 terneuron in the mouse retina that resembles amacrine cells morphologically but is glutamatergic and,

198 Amacrine cells of the retina are conspicuously variable

199 esynaptic inhibition is generated by spiking amacrine cells on a larger spatial scale covering severa

200 NER, by contrast, is present in ganglion and amacrine cells on P1, also labeling the horizontal cells

201 se of gamma-aminobutyric acid from starburst amacrine cells onto direction-selective ganglion cells (

202 Silencing amacrine cells or pharmacologically blocking inhibitory

203 KEY POINTS: Bipolar and amacrine cells presynaptic to the ON sustained alpha cel

204 However, unlike AII cells, A8 amacrine cells provide glycinergic input to ON A-type ga

205 Our data suggest that A8 amacrine cells receive glutamatergic input from both OFF

206 ic to gamma-aminobutyric acid (GABA)ergic to amacrine cells remained relatively constant.

207 Wide-field amacrine cells span large segments of the retina, making

208 Here we report a population of retinal amacrine cells that do not develop dendritic arbors in r

209 sion largely restricted to a small subset of amacrine cells that express disabled-1 (Dab1) but lack e

210 Coupling causes the amacrine cells to fire spikes that propagate radially ov

211 ynapses and/or glycinergic synapses from AII amacrine cells to OFF ganglion cells) is sufficient for

212 This allows the actions of SRIF on DA amacrine cells to proceed with adjusting retinal DA leve

213 ChAT amacrine cells typically comprise two spatially segregat

214 Instead, NMDA receptors on both amacrine cells were activated by ambient glutamate, and

215 bipolar cells and conventional synapses from amacrine cells were identified in electron microscopic i

216 The presynaptic wide-field amacrine cells were reconstructed, and two types were id

217 These are GABAergic amacrine cells with long, relatively straight dendrites,

218 ion of M1 ipRGCs and DA amacrine cells, SRIF amacrine cells would provide inhibitory modulation to bo

219 hes targeting neurons upstream of RGCs, DAA (amacrine cells) and DAD (bipolar cells) suppress the fre

220 crine cells (stellate, semilunar, and thorny amacrine cells).

221 r of the retina derives from the activity of amacrine cells, a large and diverse group of GABAergic a

222 ons exhibit aberrant activity, driven by AII amacrine cells, a primary target of the retinal dopamine

223 ase in the number of retinal ganglion cells, amacrine cells, and an increase in the number of the lat

224 SRIF amacrine cells, DA amacrine cells, and M1 ipRGCs form numerous contacts.

225 ds, bipolar cells, amacrine cells, displaced amacrine cells, and Muller glia were generated between F

226 d by recurrent connectivity within starburst amacrine cells, and retinal ganglion cells act as "reado

227 teraction is reciprocal: M1 ipRGCs excite DA amacrine cells, and these, in turn, feed inhibition back

228 es dendrite targeting in type 2 dopaminergic amacrine cells, by restricting the stratum in which expl

229 SRIF amacrine cells, DA amacrine cells, and M1 ipRGCs form nu

230 Rods, bipolar cells, amacrine cells, displaced amacrine cells, and Muller gli

231 RGCs, derived from electrical coupling with amacrine cells, encodes information critical to global o

232 ic for bipolar cells, and therefore resemble amacrine cells, excite inner retinal circuits using glut

233 heterogeneous class of retinal interneurons, amacrine cells, identifying 63 distinct types.

234 Lin28 expression was required only in amacrine cells, inhibitory neurons that innervate RGCs.

235 sponses in DACs, which are mediated by other amacrine cells, likely driven by type 1 and type 2/3a OF

236 The majority of their input was from amacrine cells, local circuit neurons of the inner retin

237 points, such as the neurites of cholinergic amacrine cells, or to define a number of bins into which

238 ere, we demonstrate that retinal AII and A17 amacrine cells, postsynaptic partners at rod bipolar dya

239 photoreceptors, Muller glia, bipolar cells, amacrine cells, retinal ganglion cells, horizontal cells

240 idirectional interaction of M1 ipRGCs and DA amacrine cells, SRIF amacrine cells would provide inhibi

241 another subpopulation of upstream GABAergic amacrine cells, thereby sustaining the GABAC receptor ac

242 ecordings between bipolar cell terminals and amacrine cells, we have simultaneously measured presynap

243 Although PRDM13 is expressed in the amacrine cells, we have yet to identify an abnormality s

244 dendrites of GABAergic/cholinergic starburst amacrine cells, where it is first observed.

245 ss in the Ndufs4 KO is the loss of starburst amacrine cells, which may be an important target in the

246 this study was on the presynaptic wide-field amacrine cells, which provided 17% of the input to ON pa

247 crush pathologically upregulated activity in amacrine cells, which reduced RGC electrical activity an

248 is provided by a subpopulation of wide-field amacrine cells, which stimulate the GABAC receptors at r

249 equency signals was regulated by glycinergic amacrine cells, while GABAergic inhibition regulated the

250 ion mediated by wide-field spiking GABAergic amacrine cells.

251 originating from a population of wide-field amacrine cells.

252 is present in both GABAergic and glycinergic amacrine cells.

253 for each gene in the differentiation of AII amacrine cells.

254 ticipating in the development of retinal AII amacrine cells.

255 orny ganglion cells, 14 cells were displaced amacrine cells.

256 ic/cholinergic signals mediated by starburst amacrine cells.

257 er are ganglion cells, and 20% are displaced amacrine cells.

258 DACs, impairing functional regulation of AII amacrine cells.

259 d inputs from axon terminals of dopaminergic amacrine cells.

260 tentiated evoked EPSCs in a subpopulation of amacrine cells.

261 bition from the ON pathway through GABAergic amacrine cells.

262 cells, but also in a subset of ganglion and amacrine cells.

263 is propagated to an atypical subtype of AII amacrine cells.

264 g horizontal cells and a subset (25%) of AII amacrine cells.

265 ter 1, and shows the typical morphology of A amacrine cells.

266 on between retinal rod bipolar cells and AII amacrine cells.

267 of M1 ipRGCs caused by SRIF inhibition of DA amacrine cells.

268 erminals, ganglion cell dendrites, and other amacrine cells.

269 recursive bistratified ganglion cells and A1 amacrine cells.

270 s the synaptic receptors on A17, but not AII amacrine cells.

271 with inhibitory interneurons: horizontal and amacrine cells.

272 ter plexiform layers and a greater number of amacrine cells.

273 BCs, and eliminate synapses with wide-field amacrine cells.

274 d GABA-immunoreactivity indicating they were amacrine cells.

275 Both conditions generated excess amacrine cells.

276 cal varieties of such ipRGC-driven displaced amacrine cells: (1) monostratified cells with dendrites

277 t from rod bipolar cells to both AII and A17 amacrines, diabetes changes the synaptic receptors on A1

278 Our results suggest that AII and A17 amacrines express clustered, extrasynaptic NMDA receptor

279 Recent evidence suggests that both amacrines express NMDA receptors, raising questions conc

280 AII amacrines forward their signals to ON- and OFF-cone bipo

281 Here, we investigated functional AII amacrine->RGC synaptic connections in the retina of the

282 Here, we investigated functional AII amacrine-->RGC synaptic connections in the retina of the

283 ever, showed the opposite effect of removing amacrine input, which was a decrease in SNR.

284 glion cell layer of rat retinas, all spiking amacrine interneurons with sustained ON photoresponses r

285 mation about many of the retina's inhibitory amacrine interneurons.

286 k required for generating the horizontal and amacrine lineages.

287 When the amacrine-modulated conductance was removed, the ON cell'

288 The fractions of rod bipolar, cone bipolar, amacrine, Muller, and horizontal cells of all cells in t

289 l surface protein and are presynaptic to Dm8 amacrine neurons (yDm8) that express Dpr11's binding par

290 eptors described in horizontal, OFF-bipolar, amacrine or ganglion cells, which could not be fully blo

291 the brain, but the interneuron (bipolar and amacrine) populations providing input to ganglion cells

292 ls to ON- and OFF-cone bipolar cells and A17 amacrines provide GABAergic feedback inhibition to rod b

293 classes: photoreceptor, horizontal, bipolar, amacrine, retinal ganglion and non-neuronal cells.

294 d for RB and type 2 OFF-CB cell survival and amacrine subtype identity, and they present PRDM8 as a c

295 c ACs were seen, suggesting an alteration in amacrine subtype identity.

296 tween GluN2B subunits and connexin 36 on AII amacrines, suggesting that NMDA receptor modulation of g

297 der retina, we show that a decrease in tonic amacrine transmission is necessary for and is correlated

298 Furthermore, introducing a decrease in amacrine transmission is sufficient to sensitize nearby

299 formed between rod bipolar cells (RBCs) and amacrine type-2 (AII) cells in the mouse retina but dram

300 A second amacrine was identified as wiry type 2, a type known to