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1 the most differentially upregulated genes in ameloblastoma.
2 antly upregulated in KCOT when compared with ameloblastoma.
3 roduced biopsy material that proved to be an ameloblastoma.
4  the interaction of stromal osteoblasts with ameloblastoma.
5 e diagnostic classification and treatment of ameloblastomas.
6 enic behaviors of enamel epithelium cells in ameloblastomas.
7 ttractive target for pre-surgical imaging of ameloblastomas.
8 ss tumor margins for the surgical removal of ameloblastomas.
9                                              Ameloblastoma (AB) is an odontogenic tumor that arises f
10  is the most common mutation in conventional ameloblastoma (AM) of the mandible.
11  and molecular features in common with human ameloblastomas (AM), spontaneous CAA can serve as a usef
12 ) is considered a less aggressive variant of ameloblastoma, amenable to more conservative treatment,
13 udy was to characterize the transcriptome of ameloblastoma and identify relevant genes and molecular
14 racterize the molecular relationship between ameloblastoma and keratocystic odontogenic tumor (KCOT)
15 ng patient-derived xenograft (PDX) models of ameloblastoma and positron emission tomography/computed
16 rly targeted therapies for the management of ameloblastoma and related odontogenic neoplasms.
17 samples of 15 solid/multicystic intraosseous ameloblastomas and 12 sporadic KCOTs was hybridized on A
18            Hierarchical clustering separated ameloblastomas and KCOTs into 2 distinct groups.
19  dental genes showed a similar separation of ameloblastomas and KCOTs.
20 n several tumors, including medulloblastoma, ameloblastoma, and basal cell carcinoma.
21                                              Ameloblastomas are assumed to derive from aberrant lamin
22                                              Ameloblastomas are odontogenic tumors that are rare in p
23                 Because canine acanthomatous ameloblastomas (CAA) have clinicopathologic and molecula
24 fused with other pathologic entities such as ameloblastomas, carcinomas, and fibromas and clinically
25       Multivariate gene analysis showed that ameloblastoma cells downregulate bone formation genes su
26                         In a similar manner, ameloblastoma cells harboring an activating BRAF mutatio
27                                              Ameloblastoma cells increased expression of MMP-2 and -9
28                                    In PDX of ameloblastomas from four patients (AB-36, AB-37, AB-39 A
29  provide clear evidence for the diagnosis of ameloblastoma in Telmatosaurus.
30 nd ISL1 were differentially overexpressed in ameloblastoma, indicating its dental identity.
31 tion between bone forming 3D bone stroma and ameloblastoma introduced 3D bone stroma.
32                                              Ameloblastoma is a benign neoplasic tumour with a strong
33                                              Ameloblastoma is a benign, epithelial cancer of the jawb
34                                              Ameloblastoma is a locally invasive benign neoplasm deri
35 rotein kinase (MAPK) pathways in over 80% of ameloblastomas, locally destructive odontogenic tumors o
36 enetic data from the shark dental lamina and ameloblastoma may lead to the development of novel metho
37    We hypothesize that expression of EGFR in ameloblastomas may be used to specifically visualize tum
38            Gene expression profiles of human ameloblastoma microdissected cells were characterized wi
39               In addition, recognition of an ameloblastoma neoplasm in a taxon lying close to the ori
40 report for the first time the presence of an ameloblastoma neoplasm in the lower jaw of a specimen re
41 SMO (encoding Smoothened, SMO) are common in ameloblastomas of the maxilla, whereas BRAF mutations ar
42 beled anti-EGFR demonstrates specificity for ameloblastoma PDX tumor xenografts, we believe (89)Zr-pa
43 ken together, the gene expression profile of ameloblastoma reflects differentiation from dental lamin
44                                              Ameloblastoma separated into 2 distinct molecular cluste
45 lying inter-tumor molecular heterogeneity of ameloblastoma sub-types and have implications for the us
46  intense in both the epithelial component of ameloblastomas, the most common epithelial odontogenic t
47              We also assessed cases of human ameloblastoma to characterise further the proliferative
48                                  Introducing ameloblastoma to the bone stroma, completely inhibited b
49 a comprehensive bone stroma, we show that an ameloblastoma tumour mass prevents osteoblasts from form
50                                    Unicystic ameloblastoma (UAM) is considered a less aggressive vari
51 d, decalcified, paraffin-embedded samples of ameloblastoma were subjected to laser capture microdisse
52  forming stroma, to probe the interaction of ameloblastoma with its native tumour bone microenvironme
53                           The interaction of ameloblastoma with its tumour stroma drives invasion and