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1 ntification of some of these likely sites of aminergic action will allow for directed studies of thei
2  observed potencies of the effector lines in aminergic and cholinergic neurons assessed here may help
3   Based on data showing brainstem changes in aminergic and inflammatory signaling during carotid body
4 , pinpointing locations in the network where aminergic and neuropeptide signalling modulate synaptic
5 a new topological plan for understanding how aminergic and peptidergic modulation of behaviour is ach
6 ) of adult lobsters and crabs receives dense aminergic and peptidergic projections.
7 ronal networks are endogenously modulated by aminergic and peptidergic substances.
8                 In comparison with the other aminergic and the cholinergic projection systems, which
9 he aminergic systems is similar, and several aminergic cells have locations and morphologies that str
10 at strongly suggest homology with identified aminergic cells in other crustaceans.
11 es that increase the presynaptic function of aminergic cells may provide neuroprotection in Parkinson
12 m LDCVs is critical for the function of some aminergic circuits.
13 gent potentiates the presynaptic function of aminergic circuits.
14 etaining ancestral Hebbian-like networks and aminergic connections.
15                            Moreover, several aminergic drugs either show off-target binding to alphaA
16                              To identify new aminergic drugs in vivo, we used a mutation in the Droso
17 ve ligands and may guide the optimization of aminergic drugs to prevent off-target binding to alphaAR
18                                          The aminergic family of G protein-coupled receptors (GPCRs)
19                                   Descending aminergic fibers, revealed by antibodies to tyrosine hyd
20 ectivity for allosteric binding sites across aminergic G protein-coupled receptor.
21                                              Aminergic G protein-coupled receptors (GPCRs) have been
22 xtracellular domain was described at several aminergic G protein-coupled receptors, including muscari
23 e privileged structural motifs recognized by aminergic G protein-coupled receptors.
24 e virtual library but is well suited to many aminergic G-protein-coupled receptors.
25                       Applying this model to aminergic GPCR sequences, we first validate the general
26 ystems to minimize drug promiscuity at other aminergic GPCR sites.
27  analysis of 6692 mutation data points on 34 aminergic GPCR subtypes, covering the chemical space of
28  the established secondary binding pocket in aminergic GPCRs and could potentially be used to design
29                                     Although aminergic GPCRs are the target for ~25% of approved drug
30 inding site in the D2R and probably in other aminergic GPCRs as well.
31 sponding G-protein-coupled receptors (termed aminergic GPCRs) belong to the class of cell membrane re
32 e pharmacologically highly relevant class of aminergic GPCRs, we here present the development of cova
33 nanomolar affinity fluorescent probes for 14 aminergic GPCRs.
34 es, an observation with few precedents among aminergic GPCRs.
35 ns of these helices in different families of aminergic GPCRs.
36  major determinant of subtype selectivity in aminergic GPCRs.
37  of information regarding patterns of spinal aminergic innervation at early stages, when the fish are
38                     Despite depletion of the aminergic innervation on the ipsilateral side, shock-ind
39 lomeruli-like complexes, adult neurogenesis, aminergic innervation, and elevated expression of protei
40 uence of the alterations in, and decline of, aminergic inputs to both autonomic and somatic spinal nu
41                               Specificity of aminergic involvement was tested by using intra-PVH admi
42 , which is activated by both cholinergic and aminergic ligands.
43  diverse ligands including acetylcholine and aminergic ligands.
44 2-null mice have lower-than-normal levels of aminergic metabolites and content.
45 ry learning; however, our knowledge of other aminergic modulation lags behind.
46                                              Aminergic modulation of glycinergic transmission may thu
47 at C. elegans is a useful model to study the aminergic modulation of sensory-mediated locomotory beha
48 ining alpha6 subunits are typically found at aminergic nerve endings where they play important roles
49  and showed an endocytic deficit specific to aminergic nerve terminals.
50 n to influence sleep homeostasis and receive aminergic neuromodulator input critical to sleep-wake sw
51 known synergistic actions of NA with another aminergic neuromodulator, serotonin, and raise the possi
52    The proximity of Ebony-containing glia to aminergic neurons and genetic interaction results sugges
53 ter understanding of the functional roles of aminergic neurons and how they influence behavior.
54                 We report the development of aminergic neurons from 0-10 days postfertilization (dpf)
55 of Lewy bodies, neurofibrillary tangles, and aminergic neurons in the locus ceruleus, dorsal raphe nu
56 i) footshock-induced activation of medullary aminergic neurons is a secondary consequence of stress,
57  (ii) stress-induced activation of medullary aminergic neurons is not necessarily predictive of an in
58                    Alternatively, individual aminergic neurons might selectively modulate the animals
59 ial reduction of Fos expression in medullary aminergic neurons on the ipsilateral side.
60                                              Aminergic neurons that modulate the synapse may have ver
61 y expression of DVMAT in a single subtype of aminergic neurons, but required at least two systems, su
62 me that converts tyramine into octopamine in aminergic neurons, is increased by food deprivation, thu
63 al properties and expression postsynaptic to aminergic neurons.
64 om SV to LDCV fractions was also enhanced in aminergic neurons.
65  mushroom body by differential contacts with aminergic neurons.
66 erve the relative trajectories of individual aminergic neurons.
67 ulation of rate-limiting enzymes involved in aminergic neurotransmitter production.
68 P2-dependent mechanism for the regulation of aminergic neurotransmitter synthesis contributing to uni
69 ch asks for detailed characterization of the aminergic neurotransmitter systems in this model.
70 isease states associated with alterations in aminergic neurotransmitters, we investigated the contrib
71        Orexin neurons heavily innervate many aminergic nuclei that promote wakefulness and inhibit RE
72 n systems in goldfish barely innervate these aminergic populations related to the regulation of sleep
73                    Our findings suggest that aminergic populations with descending processes are amon
74 ols this decision via top-down extrasynaptic aminergic potentiation of the primary osmosensory neuron
75                In summary, we postulate that aminergic presynaptic taste cells synthesize only 5-HT,
76 ism, rats bearing unilateral transections of aminergic projections were challenged with intravenous I
77 rograde labeling, that the sources of spinal aminergic reactivity include the posterior tuberculum (d
78                     We find that each of the aminergic receptor genes is expressed in restricted regi
79                                  We also use aminergic receptor gfp reporter fusions as tools to visu
80 mologous 5-HT(1F)R, we screened a library of aminergic receptor ligands at both receptors and observe
81 g mutation data to guide the design of novel aminergic receptor ligands.
82 entally determined structures of 52 distinct aminergic receptor-ligand complexes.
83 ct, with the TAARs being most similar to the aminergic receptors such as those activated by adrenalin
84 ectivity of this scaffold for D1R over other aminergic receptors, and sheds light on the mechanism fo
85 e the expression pattern of several of these aminergic receptors, including two serotonin receptors (
86  expression caused alterations in epidermis, aminergic, sensory, and motor neurons.
87  adaptive responses to reduced or eliminated aminergic signaling and will be useful to identify the u
88                    Despite the importance of aminergic signaling for regulating locomotion and other
89  of animals carrying a null mutation for all aminergic signaling is sufficient to restore odor-tracki
90                                              Aminergic signaling modulates associative learning and m
91 ected the relative contribution of different aminergic signalling pathways to biological processes es
92         Structure determination of all major aminergic subfamilies has enabled structure-based ligand
93    We found increased apoptosis in the three aminergic systems analyzed when compared with animals ma
94           Our results suggest that the major aminergic systems described in adults are in place short
95   Our previous study tracked the ontogeny of aminergic systems in zebrafish (Danio rerio).
96 evertheless, the general organization of the aminergic systems is similar, and several aminergic cell
97  this model will help determine how multiple aminergic systems may contribute to the regulation of ot
98                       Our data indicate that aminergic systems may interact at all levels of the sens
99 AT activity in either individual or multiple aminergic systems, using transgenic rescue techniques.
100 nation of the potential synaptic contacts of aminergic systems.
101 fferences between, and evolution of biogenic aminergic systems.
102 hich are heavily biased toward "traditional" aminergic targets and commonly described as small lipoph
103 higher median property values than those for aminergic targets, such as monoamine transporters and GP
104 results begin to sort out how purinergic and aminergic transmitters function within the taste bud to

 
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