ライフサイエンスコーパス: aminobutyric acid

1 ment of glutamate, glutamine and GABA (gamma-aminobutyric acid).

2 ajor inhibitory neurotransmitter GABA (gamma-aminobutyric acid).

3 as a receptor of the priming activator beta-aminobutyric acid.

4 ant on the inhibitory neurotransmitter gamma-aminobutyric acid.

5 press galanin and the neurotransmitter gamma-aminobutyric acid.

6 fatty acid compositions and levels of gamma-aminobutyric acid (54.9 mg/100 g), free phenolics (507.4

7 lateral infusions into the RMTg of the gamma-aminobutyric acid A (GABAA) agonist, muscimol, indeed ac

8 Nlrp3 inflammasome were independent of gamma-aminobutyric acid A receptor activation or N-methyl-d-as

9 ve action on the omega1 subtype of the gamma-aminobutyric acid A receptor, zolpidem tartrate presents

10 Antagonism of gamma-aminobutyric acid A receptors (GABA(A)Rs) by bilobalide

11 Here, we report on the effects of beta-aminobutyric acid, a priming agent with an exceptionally

12 o neurotransmitter receptor genes, the gamma-Aminobutyric acid-A receptor delta and gamma-aminobutyri

13 protein-like 2), glycine receptor, and gamma-aminobutyric acid-A receptor.

14 -isoxazolepropionic acid, glycine, and gamma-aminobutyric acid-A receptors), were prevalent in patien

15 terminants containing substitutions of alpha-aminobutyric acid (AABA), a cysteine analogue containing

16 gamma-Aminobutyric acid aminotransferase (GABA-AT) is a pyrido

17 in barrier and inhibit the activity of gamma-aminobutyric acid aminotransferase (GABA-AT), the enzyme

18 ivator that regulates transcription of gamma-aminobutyric acid aminotransferase (GABA-AT; GabT) upon

19 GAD65 synthesizes gamma-aminobutyric acid, an important autocrine and paracrine

20 e stress, and is associated with GABA (gamma-aminobutyric acid, an inhibitory neurotransmitter) signa

21 particular, bicyclic gamma-amino acids-gamma-aminobutyric acid analogues-were synthesized.

22 furosine (2-FM-lysine), 2-furoylmethyl-gamma-aminobutyric acid and 2-FM-arginine were detected.

23 some neurotransmitter systems, such as gamma-aminobutyric acid and glutamate, mainly in the cortex.

24 ethod to monitor glutamate, glutamine, gamma-aminobutyric acid and lactate in the brains of unaffecte

25 sidues p2C and p5M to the conservative alpha-aminobutyric acid and norleucine, respectively, signific

26 agonist bicuculline, the agonist GABA (gamma-aminobutyric acid), and the classical benzodiazepines al

27 ll as regulating release of glutamate, gamma-aminobutyric acid, and acetylcholine from presynaptic ax

28 d by l-glutamate (l-Glu), l-aspartate, gamma-aminobutyric acid, and acetylcholine, with l-Glu eliciti

29 ed by the neurotransmitters glutamine, gamma-aminobutyric acid, and dopamine.

30 eractions between the endocannabinoid, gamma-aminobutyric acid, and glutamate systems and their indiv

31 nesis including synthesis of glutamate/gamma-aminobutyric acid as a potential transcriptional target

32 the neurons also release glutamate and gamma-aminobutyric acid as cotransmitters, with striking regio

33 ropic glutamate receptors (mGluRs) and gamma-aminobutyric acid B (GABA(B)) receptors (GABA(B)Rs) most

34 SNs) triggered astrocyte signaling via gamma-aminobutyric acid B (GABA(B)) receptors.

35 Aminobutyric acid-A receptor delta and gamma-aminobutyric acid B receptor subunit 1; their differenti

36 beta-aminobutyric acid (BABA) is an endogenous stress metabol

37 n genes responsive to the priming agent beta-aminobutyric acid (BABA) revealed IMPAIRED OOMYCETE SUSC

38 donic acid (AA), jasmonic acid (JA) and beta-aminobutyric acid (BABA).

39 luding two pathways, phenylacetate and gamma-aminobutyric acid catabolism, which were found to be imp

40 BrdU(+) /GABA(+) (gamma-aminobutyric acid) cells were also found but no new Cr(+

41 the specific impairment of perisomatic gamma-aminobutyric acid circuits are hallmarks of the schizoph

42 d lower limb was associated with lower gamma-aminobutyric acid concentration in the sensorimotor cort

43 Additionally, the reduced gamma-aminobutyric acid concentration may contribute to the ne

44 r adults to a likely decrease in GABA (Gamma Aminobutyric Acid) concentration in visual cortex, an as

45 ls were used to compare differences in gamma-aminobutyric acid concentrations between patients and co

46 injection to the NAcSh decreased local gamma-aminobutyric acid concentrations.

47 Fast glutamate and gamma-aminobutyric acid cotransmission convey discrete pattern

48 cally relevant concentrations, whereas gamma-aminobutyric acid does not.

49 the distribution of neurotransmitters-gamma-aminobutyric acid, dopamine and serotonin-with high spec

50 real-time-corrected three-dimensional gamma-aminobutyric acid-edited magnetic resonance (MR) spectro

51 As the inhibitory gamma-aminobutyric acid-ergic (GABAergic) transmission has a p

52 iety of neurotransmitters, such as the gamma-aminobutyric acid-ergic system, the study of prefrontal

53 part by direction-selective release of gamma-aminobutyric acid from starburst amacrine cells onto dir

54 Type A gamma-aminobutyric acid (GABA(A)) receptors are pentameric lig

55 The effects of gamma-aminobutyric acid (GABA) A receptor activation on physio

56 mine whether antecedent stimulation of gamma-aminobutyric acid (GABA) A receptors with the benzodiaze

57 Gamma-Aminobutyric acid (GABA) accumulates in plants following

58 gamma-Aminobutyric acid (GABA) administration has been shown t

59 The differentiation of gamma-aminobutyric acid (GABA) and dopamine neurons, but not g

60 Gamma-aminobutyric acid (GABA) and glutamate are the most abun

61 aining dopamine (DA), serotonin (5HT), gamma-aminobutyric acid (GABA) and glutamate neurons.

62 PVN -> NAc neurons and quantified both gamma-aminobutyric acid (GABA) and glutamate release and phosp

63 amino acid neurotransmitter systems of gamma-aminobutyric acid (GABA) and glutamate, respectively, pl

64 Levels of gamma-aminobutyric acid (GABA) and glutamic acid decarboxylase

65 e inhibitory transmitters include both gamma-aminobutyric acid (GABA) and glycine (GLY).

66 health-promoting metabolites, such as gamma-aminobutyric acid (GABA) and phenolic compounds.

67 Glutamate (GLU) and gamma-aminobutyric acid (GABA) are the major excitatory (E) an

68 elease the inhibitory neurotransmitter gamma-aminobutyric acid (GABA) at non-image-forming brain targ

69 pertensive activity due to their large gamma-aminobutyric acid (GABA) content (6.8-10.6 mg/g) and ang

70 ble structures, including amido-diols, gamma-aminobutyric acid (GABA) derivatives, and heterocycles.

71 f the cockroach Periplaneta americana, gamma-aminobutyric acid (GABA) has been identified as the pote

72 Altered neurotransmission of gamma-aminobutyric acid (GABA) has been implicated in the path

73 to map histamine (HA), FMRF-amide, and gamma-aminobutyric acid (GABA) immunoreactivity in the central

74 Abnormally elevated levels of gamma-aminobutyric acid (GABA) in the medial prefrontal cortex

75 ilk and regular-fat cheese enriched in gamma-aminobutyric acid (GABA) influences daytime ambulatory b

76 Despite its importance for gamma-aminobutyric acid (GABA) inhibition and involvement in n

77 o reductions of parvalbumin containing gamma-aminobutyric acid (GABA) interneurons and volumes of the

78 rtate receptor (NMDAR) hypofunction on gamma-aminobutyric acid (GABA) interneurons disinhibiting pyra

79 Gamma-aminobutyric acid (GABA) is a widely conserved signaling

80 he role of VGLUT3 in neurons releasing gamma-aminobutyric acid (GABA) is not settled.

81 gamma-Aminobutyric acid (GABA) is one of the major inhibitory

82 gamma-Aminobutyric acid (GABA) is the major inhibitory neurotr

83 gamma-Aminobutyric acid (GABA) is the major inhibitory transmi

84 gamma-Aminobutyric acid (GABA) is the most abundant inhibitory

85 gamma-Aminobutyric acid (GABA) is the primary inhibitory neuro

86 In contrast, KATPHI islets had low gamma-aminobutyric acid (GABA) levels and lacked (13)C incorpo

87 Low-pressure plasma also increased gamma-aminobutyric acid (GABA) levels from approximately 19 to

88 in excitatory glutamate and inhibitory gamma-aminobutyric acid (GABA) levels, including in brain regi

89 bitory), schizophrenia (excitatory and gamma-aminobutyric acid (GABA) mediated) and bipolar disorder

90 he density of cartridges detectable by gamma-aminobutyric acid (GABA) membrane transporter 1 immunore

91 enzyme GAD67, a critical actor of the gamma-aminobutyric acid (GABA) metabolism as it catalyses the

92 Early-born gamma-aminobutyric acid (GABA) neurons (EBGNs) are major compo

93 stimulation of mouse zona incerta (ZI) gamma-aminobutyric acid (GABA) neurons or their axonal project

94 Stimulation of dorsal raphe gamma-aminobutyric acid (GABA) neurons promoted movement in ne

95 show that optogenetic inhibition of LH gamma-aminobutyric acid (GABA) neurons restricted to cue prese

96 s expression in VTA 5-HT2CR expressing gamma-aminobutyric acid (GABA) neurons, but not 5-HT2CR expres

97 Aberrant glutamate and gamma-aminobutyric acid (GABA) neurotransmission contribute to

98 The gamma aminobutyric acid (GABA) neurotransmission system has be

99 ACE) resulted in an enhancement of the gamma-aminobutyric acid (GABA) neurotransmitter system in the

100 either the amino acid (AA) transmitter gamma-aminobutyric acid (GABA) or glutamate.

101 ns, scaffold and adaptor proteins, and gamma-aminobutyric acid (GABA) or glycine receptors.

102 s did not confirm colocalizations with gamma-aminobutyric acid (GABA) or the circadian coupling pepti

103 Given the heterogeneity within the gamma-aminobutyric acid (GABA) receptor and transporter famili

104 neurites are also immunopositive for a gamma-aminobutyric acid (GABA) receptor subunit (GABAA Ralpha1

105 Nonselectively reducing intrastriatal gamma-aminobutyric acid (GABA) receptor-A inhibition synchroni

106 ngly, severe antagonists of ionotropic gamma-aminobutyric acid (GABA) receptors.

107 rinking, and basal and alcohol-induced gamma-aminobutyric acid (GABA) release in the central nucleus

108 Our results demonstrate that the gamma-aminobutyric acid (GABA) release is lower and opioid eff

109 We found that local gamma-aminobutyric acid (GABA) release on dendrites of mouse c

110 es revealed a significant reduction in gamma-aminobutyric acid (GABA) release probability without alt

111 f the metabotropic GABA(B) receptor by gamma-aminobutyric acid (GABA) results in prolonged inhibition

112 nitrate stimulated PGC1alpha-mediated gamma-aminobutyric acid (GABA) secretion from muscle.

113 ncreased mitochondrial activity causes gamma aminobutyric acid (GABA) sequestration in the mitochondr

114 Gamma-aminobutyric acid (GABA) serves diverse biological funct

115 , Gao and colleagues identify that the gamma-aminobutyric acid (GABA) shunt is upregulated with the o

116 xendin-4, a GLP-1 receptor agonist, on gamma-aminobutyric acid (GABA) signaling in hippocampal CA3 py

117 Altered gamma-aminobutyric acid (GABA) signaling in the prefrontal cor

118 inflammation, synaptic plasticity and gamma-aminobutyric acid (GABA) signaling, and liver inflammati

119 target, NKCC1, initiates the switch in gamma-aminobutyric acid (GABA) signaling, limits early spontan

120 ndmark is the excitatory to inhibitory gamma-aminobutyric acid (GABA) switch caused by reciprocal cha

121 ociated with reduced concentrations of gamma-aminobutyric acid (GABA) that are normalized by antidepr

122 onally stained with antibodies against gamma-aminobutyric acid (GABA) to identify GABAergic interneur

123 A) exhibited a significant increase in gamma-aminobutyric acid (GABA) transmission compared with rats

124 te signaling (P-value=7.22 x 10(-15)), gamma-aminobutyric acid (GABA) transport (P-value=1.36 x 10(-1

125 We saw that stimulation of vesicular gamma-aminobutyric acid (GABA) transporter (VGAT)-expressing B

126 encoded by SLC6A1, is one of the major gamma-aminobutyric acid (GABA) transporters in the brain and i

127 evels of glutamate+glutamine (Glx) and gamma-aminobutyric acid (GABA) were measured before, during, a

128 eic acids, as well as higher amount of gamma-aminobutyric acid (GABA) were observed in biodynamic gra

129 f endogenous alpha5-subunit-containing gamma-aminobutyric acid (GABA)(A) receptors (alpha5-GABARs).

130 excitatory (glutamate) and inhibitory (gamma aminobutyric acid (GABA)) neurotransmitter circuits in a

131 Gamma aminobutyric acid (GABA), a neurotransmitter of the cent

132 tant ion channels, gated by glutamate, gamma-aminobutyric acid (GABA), and acetylcholine, also have a

133 ibitory neurotransmitter of the brain, gamma-aminobutyric acid (GABA), can be released through the re

134 the relative quantity of glutamate to gamma-aminobutyric acid (GABA), DA, and glutamate neurons with

135 zyme synthesizing the neurotransmitter gamma-aminobutyric acid (GABA), define how artemisinins also i

136 rentially methylated probes implicated gamma-aminobutyric acid (GABA), dopamine and serotonin neurotr

137 nthesis of neurotransmitters including gamma-aminobutyric acid (GABA), dopamine, and serotonin.

138 r various neurotransmitters-glutamate, gamma-aminobutyric acid (GABA), dopamine, serotonin, acetylcho

139 dicted to form anion channels gated by gamma-aminobutyric acid (GABA), glutamate, histamine, or chang

140 ajor sleep-promoting neurotransmitter, gamma-aminobutyric acid (GABA), in the GABA shunt generates ca

141 ay of the inhibitory neurotransmitter, gamma-aminobutyric acid (GABA), in this putative imbalance: GA

142 effects of postharvest treatments with gamma-aminobutyric acid (GABA), methyl jasmonate (MeJA) or met

143 Low levels of gamma-aminobutyric acid (GABA), one of two major neurotransmit

144 rs for the inhibitory neurotransmitter gamma-aminobutyric acid (GABA), particularly within the fronta

145 ecently, it has been demonstrated that gamma-aminobutyric acid (GABA), the chief inhibitory neurotran

146 gamma-Aminobutyric acid (GABA), the major inhibitory neurotran

147 A considerable increase in gamma-aminobutyric acid (GABA), together with some other amino

148 acting neurotransmitters glutamate and gamma-aminobutyric acid (GABA), two major neurotransmitters in

149 a potential lipophilic bioisostere of gamma-aminobutyric acid (GABA), various late-stage diversifica

150 The inhibitory neurotransmitter, gamma-aminobutyric acid (GABA), was then actively delivered th

151 nal fluid (CSF)-induced enhancement of gamma-aminobutyric acid (GABA)-A receptor activity was found i

152 spartate (NMDA) glutamate receptor and gamma-aminobutyric acid (GABA)-A receptor during progression o

153 ith a positive allosteric modulator of gamma-aminobutyric acid (GABA)-A receptors in cerebrospinal fl

154 unique role of GABARAPs, in particular gamma-aminobutyric acid (GABA)-A-receptor-associated protein-l

155 n is likely due to a reduced number of gamma-aminobutyric acid (GABA)-ergic boutons, which may result

156 eractions between parvalbumin-positive gamma-aminobutyric acid (GABA)-ergic interneurons and pyramida

157 uses stress-sensitive, PVH-projecting, gamma-aminobutyric acid (GABA)-ergic neurons as representing a

158 Dysfunction related to gamma-aminobutyric acid (GABA)-ergic neurotransmission in the

159 hanges the number of dopamine (DA)- or gamma aminobutyric acid (GABA)-expressing neurons, with corres

160 This study describes the gamma-aminobutyric acid (GABA)-immunoreactive (GABA-ir) neuron

161 Calretinin- and gamma-aminobutyric acid (GABA)-immunoreactive (IR) cells were

162 spartate (NMDA) excitation balanced by gamma-Aminobutyric acid (GABA)-mediated inhibition and leads t

163 , salt reabsorption in the kidney, and gamma-aminobutyric acid (GABA)-mediated modulation in neurons.

164 but not the parvalbumin subset of SNr gamma-aminobutyric acid (GABA)-releasing (GABAergic) neurons w

165 Here, we found that gamma-aminobutyric acid (GABA)-releasing neurons of the mouse

166 At the circuit level, these gamma-aminobutyric acid (GABA)-releasing projections target hi

167 lease the inhibitory neurotransmitter, gamma-aminobutyric acid (GABA).

168 ion of the inhibitory neurotransmitter gamma-aminobutyric acid (GABA).

169 tions in regulation of the HPA axis by gamma-aminobutyric acid (GABA).

170 (93%) on dendrites that do not contain gamma aminobutyric acid (GABA).

171 ue to significant, tonic inhibition by gamma-aminobutyric acid (GABA).

172 e-limiting enzyme in the production of gamma-aminobutyric acid (GABA).

173 h slightly different sensitivities, on gamma-aminobutyric acid (GABA)A , alpha-amino-3-hydroxy-5-meth

174 to involve functional deficits in both gamma-aminobutyric acid (GABA)ergic and glutamatergic synaptic

175 change factor selectively localized to gamma-aminobutyric acid (GABA)ergic and glycinergic postsynaps

176 ergent findings indicate that cortical gamma-aminobutyric acid (GABA)ergic circuitry is altered in sc

177 inergic systems and is extended to the gamma-aminobutyric acid (GABA)ergic elements involved with the

178 inergic systems and is extended to the gamma-aminobutyric acid (GABA)ergic elements of these nuclei.

179 Finally, we revealed a role for the gamma-aminobutyric acid (GABA)ergic functions of LTSIs in lear

180 s glutamatergic excitatory neurons and gamma-aminobutyric acid (GABA)ergic inhibitory interneurons.

181 uperior colliculus receives inhibitory gamma-aminobutyric acid (GABA)ergic input from the basal gangl

182 gamma-Aminobutyric acid (GABA)ergic inputs are strategically p

183 egmental nucleus (RMTg), which contain gamma-aminobutyric acid (GABA)ergic neurons that mediate rewar

184 types (cholinergic, glutamatergic, and gamma-aminobutyric acid (GABA)ergic neurons) across its differ

185 ognitive alterations and damage to the gamma-aminobutyric acid (GABA)ergic system in the hippocampus

186 he retina, the ratio of glycinergic to gamma-aminobutyric acid (GABA)ergic to amacrine cells remained

187 Abnormalities in prefrontal gamma aminobutyric acid (GABA)ergic transmission, particularly

188 tic connectivity and function of a few gamma-aminobutyric acid (GABA)ergic wide-field amacrine cells

189 ent solely in neurons, specifically in gamma-aminobutyric acid (GABA)ergic, glutamatergic, and catech

190 d mPFC glutamate + glutamine (Glx) and gamma-aminobutyric acid (GABA+) concentrations in 64 women wit

191 The gamma-aminobutyric acid (GABAA) receptor gamma2 subunit gene,

192 iated with mutations in the inhibitory gamma-aminobutyric acid (GABAA) receptor gamma2 subunit gene,

193 ne, a positive allosteric modulator of gamma-aminobutyric acid (GABAA) receptors, for the treatment o

194 synaptic inhibition mediated by type A gamma-aminobutyric acid (GABAA) receptors, which are Cl(-)-per

195 Ionotropic receptors of gamma-aminobutyric acid (GABAAR) regulate neuronal inhibition

196 ting to the risk of each first IA, and gamma-aminobutyric acid (GABAs) associated with the first auto

197 Val > Lys, Tyr, Pro > hydroxyproline > alpha-aminobutyric acid > Gln, Thr, Ser > Glu, Ala, Gly, Asn,

198 altered brain levels of glutamate and gamma-aminobutyric acid have been identified in both animal an

199 uropil organization and the pattern of gamma-aminobutyric acid immunostaining of the medulla and LOX

200 tostatin (SST), a marker of inhibitory gamma-aminobutyric acid interneurons that target pyramidal cel

201 Gamma-aminobutyric acid is the principle inhibitory neurotrans

202 differences in prefrontal or striatal gamma-aminobutyric acid level.

203 s of this study were to investigate if gamma-aminobutyric acid levels (i) are abnormal in patients wi

204 Specifically for each unit decrease in gamma-aminobutyric acid levels (in mM), there was a predicted

205 ate + glutamine, N-acetylaspartate, or gamma-aminobutyric acid levels in the left DLPFC.

206 This study suggests that reduced gamma-aminobutyric acid levels reflect pathological abnormalit

207 Abnormal glutamate and GABA (gamma-aminobutyric acid) levels have been found in the early p

208 rmalities (e.g., serotonin, glutamate, gamma-aminobutyric acid) likely interact to facilitate illness

209 nterneurons (FSIs), a strong source of gamma-aminobutyric acid-mediated synaptic inhibition in the NA

210 impulsive actions, most likely through gamma-aminobutyric acid-mediated synaptic inhibition of medium

211 athways related to ethanol production, gamma-aminobutyric acid metabolism, and endotoxin biosynthesis

212 The gamma-aminobutyric acid modulator propofol induces neuronal ce

213 Mice lacking GIRK2 in gamma-aminobutyric acid neurons (GAD-Cre:Girk2(flox/flox) mice

214 nd a specific population of inhibitory gamma-aminobutyric acid neurons (i.e., parvalbumin-containing

215 endent signaling in dorsal hippocampal gamma-aminobutyric acid neurons but no evident behavioral phen

216 It is principally composed of gamma-aminobutyric acid neurons.

217 ting and inhibiting neighbouring GABA (gamma-aminobutyric acid) neurons in the ventral tegmental area

218 he pre- and postsynaptic components of gamma-aminobutyric acid neurotransmission and in the density o

219 y supports the idea that modulation of gamma-aminobutyric acid neurotransmission may be an important

220 is in part mediated by specific GABA (gamma-aminobutyric acid)-producing neurons in the bed nucleus

221 e, we asked whether mesoaccumbal GABA (gamma-aminobutyric acid) projections enhance adaptive respondi

222 ion causes changes in the rodent brain gamma-aminobutyric acid receptor (GABAAR) subunit composition

223 echanisms controlling the metabotropic gamma-aminobutyric acid receptor (GABAB) cell surface stabilit

224 he type of excitability: a depolarized gamma-Aminobutyric acid receptor (GABAR) reversal potential or

225 d-aspartate receptor in 4 patients and gamma-aminobutyric acid receptor A in 1 patient of 111 patient

226 e (AgRP) neurons because inhibition of gamma-aminobutyric acid receptor in the ARC did not prevent th

227 behavioral results suggest that local gamma-aminobutyric acid receptor signaling mediates the hypoph

228 in's effects on energy balance through gamma-aminobutyric acid receptor signaling.

229 duced lysosomal degradation of RhoB in Gamma-aminobutyric acid receptor-associated protein (GABARAP)+

230 ociated protein 1 light chain 3 (LC3), gamma-aminobutyric acid receptor-associated protein (GABARAP)]

231 ell-surface antibodies, mainly against gamma-aminobutyric acid receptors (53% vs 11%; P < .001).

232 Metabotropic gamma-aminobutyric acid receptors (GABA(B)) are involved in th

233 become appreciated that activation of gamma-aminobutyric acid receptors (GABA-Rs) on ss-cells can pr

234 The accumulation of gamma-aminobutyric acid receptors (GABAARs) at the appropriate

235 ncludes the metabotropic glutamate and gamma-aminobutyric acid receptors.

236 synaptic inhibition upon activation of gamma-aminobutyric acid receptors.

237 B1R); 3) binds to CB1R, which inhibits gamma-aminobutyric acid release from the cholecystokinin-conta

238 se brain, prevented ethanol-stimulated gamma-aminobutyric acid release in the central amygdala, and r

239 Elevated gamma-aminobutyric acid release under chronic stress is accomp

240 donoylglycerol-dependent inhibition of gamma-aminobutyric acid release without altering postsynaptic

241 t chronic stress increases presynaptic gamma-aminobutyric acid release.

242 ell types include hypocretin and GABA (gamma-aminobutyric-acid)-releasing neurons of the lateral hypo

243 esult from the synchronous activity of gamma-aminobutyric acid-releasing cells.

244 including the migration of GABAergic (gamma-aminobutyric-acid-releasing) neurons from ventral to dor

245 GABAergic (gamma-aminobutyric-acid-releasing) oviposition inhibitory neur

246 Here we show that a GABAergic (gamma-aminobutyric-acid-releasing) pathway originating from th

247 Moreover, up-regulation of the gamma-aminobutyric acid shunt and alanine metabolism explained

248 nate-semialdehyde dehydrogenase in the gamma-aminobutyric acid shunt pathway and an aconitase family

249 mentation, alanine metabolism, and the gamma-aminobutyric acid shunt, while [(13)C]glutamate and [(15

250 be temporarily reverted by inhibiting gamma-aminobutyric acid signaling but not by a Ras-ERK blockad

251 links impulsive behavior in rodents to gamma-aminobutyric acid signaling in the NAc.

252 This results in delayed gamma-aminobutyric acid switch and higher susceptibility to se

253 process, the excitatory-to-inhibitory gamma-aminobutyric acid switch; defects in this switch have be

254 Immunohistochemistry for the gamma aminobutyric acid synthesizing enzyme glutamic acid deca

255 urrent techniques for monitoring GABA (gamma-aminobutyric acid), the primary inhibitory neurotransmit

256 strong antagonism toward receptors of gamma-aminobutyric acid, the major inhibitory transmitter.

257 The binding of GABA (gamma-aminobutyric acid) to extrasynaptic GABA(A) receptors ge

258 reated with melatonin exhibited higher gamma-aminobutyric acid transaminase (GABA-T) enzyme activity

259 es the CG7433 protein, a mitochondrial gamma-aminobutyric acid transaminase (GABAT), and reduces GABA

260 sion or pharmacological enhancement of gamma-aminobutyric acid transmission.

261 bitory synaptic currents and vesicular gamma-aminobutyric acid transporter (vGAT) staining intensity

262 isorder genes, SLC6A1, which encodes a gamma-aminobutyric acid transporter, was associated with misse

263 nts in the gene GABRB2, coding for the gamma-aminobutyric acid type A (GABA(A) ) receptor subunit bet

264 Gabazine, a gamma-aminobutyric acid type A (GABA(A)) receptor antagonist,

265 predict peak current responses of the gamma-aminobutyric acid type A (GABA(A)) receptor.

266 a3, and gamma2 subunits comprising the gamma-aminobutyric acid type A (GABA(A)) receptor.

267 ugs act through positive modulation of gamma-aminobutyric acid type A (GABA(A)) receptors to dampen n

268 ial firing by local application of the gamma-aminobutyric acid type A (GABA-A) agonist muscimol incre

269 entified four de novo mutations in the gamma-aminobutyric acid type A (GABAA ) receptor beta3 subunit

270 Proteostasis maintenance of gamma-aminobutyric acid type A (GABAA) receptors dictates thei

271 isplayed micromolar affinity for brain gamma-aminobutyric acid type A (GABAA) receptors.

272 The extrasynaptic alpha-5 gamma-aminobutyric acid type A receptor (alpha5-GABAAR) regula

273 steroids are efficacious modulators of gamma-aminobutyric acid type A receptor (GABA(A)) receptor fun

274 highly selective for the slow type of gamma-aminobutyric acid type A receptor (GABA(A)R) could have

275 The gamma-aminobutyric acid type A receptor (GABAA-R) is a major i

276 deficient neurons show reduced surface gamma-aminobutyric acid type A receptor (GABAAR) levels and im

277 that inhibit alpha5 subunit-containing gamma-aminobutyric acid type A receptor function improve cogni

278 sporter expression and impaired spinal gamma-aminobutyric acid type A receptor function, indicative o

279 wever, DCS-LTD persists despite either gamma-aminobutyric acid type A receptor or N-methyl-D-aspartat

280 uding GABRB3, GABRA5, GABRG3, encoding gamma-aminobutyric acid type A receptor subunits (beta3, alpha

281 Alpha-5 gamma-aminobutyric acid type A receptors (alpha5-GABAARs) are

282 Subunit-selective modulation of gamma-aminobutyric acid type A receptors (GABA(A)R) is conside

283 The function and pharmacology of gamma-aminobutyric acid type A receptors (GABA(A)Rs) are of gr

284 eroids (NS) are the main modulators of gamma-aminobutyric acid type A receptors (GABA(A)Rs), which ar

285 c acetylcholine receptors (nAChRs) and gamma-aminobutyric acid type A receptors (GABAARs) are members

286 t act as convulsants and inhibitors of gamma-aminobutyric acid type A receptors (GABAARs) rather than

287 Extrasynaptic gamma-aminobutyric acid type A receptors (GABAARs),which contr

288 suggest that alpha5 subunit-containing gamma-aminobutyric acid type A receptors are novel targets for

289 he scaffolding protein Gephyrin and of gamma-aminobutyric acid type A receptors at inhibitory neurona

290 nhibition of alpha5 subunit-containing gamma-aminobutyric acid type A receptors attenuated cognitive

291 nhibition of alpha5 subunit-containing gamma-aminobutyric acid type A receptors attenuated deficits i

292 t inhibiting alpha5 subunit-containing gamma-aminobutyric acid type A receptors would improve cogniti

293 elective for alpha5 subunit-containing gamma-aminobutyric acid type A receptors, 30 minutes before un

294 function of alpha5 subunit-containing gamma-aminobutyric acid type A receptors.

295 ic animals, supporting the notion that gamma-aminobutyric acid type A signaling can also initiate ict

296 ing synaptic inhibition through GABAA (gamma aminobutyric acid type A) and glycine receptors depends

297 ned neuronal inhibition is mediated by gamma-aminobutyric acid type B (GABAB) receptors, which are he

298 ol, like rapid antidepressants, alters gamma-aminobutyric acid type B receptor (GABABR) expression an

299 und the sushi 1 domain specific to the gamma-aminobutyric acid type B receptor subunit 1a (GABA(B)R1a

300 The GABA(B) (gamma-aminobutyric acid type B) receptor is one of the princip