ライフサイエンスコーパス: amnesic

1 hbourhood, to which he moved after he became amnesic.

2 CA1 of brain histamine-depleted rats (hence, amnesic) 10 min before the retention test restored IA me

3 reciprocal manner during the anxiolytic and amnesic action of the benzodiazepine agonist.

4 nerative diseases, included 6 3 with typical amnesic Alzheimer disease and 3 with atypical variants (

5 ude of [18F]AV-1451 binding (3 patients with amnesic Alzheimer disease, r = -0.82; P < .001; r = -0.7

6 We addressed this issue by asking MTL amnesics and controls to generate exemplars of three typ

7 egy; (ii) following the organization trials, amnesics and non-amnesics retained information to a comp

8 These data suggest that at least some severe amnesics are able to acquire new semantic knowledge, pro

9 emory function, from healthy young adults to amnesic cases.

10 ptide is thought to contribute to the subtle amnesic changes in Alzheimer's disease (AD) by causing s

11 ry during the 13-year period since he became amnesic, despite having no measurable anterograde episod

12 The labels generated and used by the amnesics during interactions with their partners became

13 social recognition test in mice revealed an amnesic effect at 5 mg/kg intraperitoneally.

14 The present experiment investigated the amnesic effect of lidocaine and ECS in 25 rats trained o

15 previous learning protected animals from the amnesic effect of NMDA antagonists on new learning (of a

16 cessary to render memories vulnerable to the amnesic effect of PKMz-antisense.

17 aversive memories renders them liable to the amnesic effect of reconsolidation inhibitors.

18 Furthermore, the amnesic effect persists for at least 24 h, cannot be att

19 These two distinct amnesic effects are abolished if protein degradation by

20 at potential for clinical practice, the fear-amnesic effects are typically demonstrated through Pavlo

21 tic effects during the post-shock period and amnesic effects during a retention test 24 h later.

22 The amnesic effects of atr/scop in the MSDB have been presum

23 campus, is a critical locus in mediating the amnesic effects of atr/scop.

24 cilitative effects that outweighed potential amnesic effects of cortical beta amyloid.

25 experiment 2, we determined the anterograde amnesic effects of large lesions of the hippocampus that

26 methamphetamine, and an insensitivity to the amnesic effects of the cholinergic receptor antagonist,

27 nd therefore support the hypothesis that the amnesic effects of this lesion are caused primarily by t

28 The results show that the amnesic effects of TS+AM+FX cannot be generally attribut

29 ures for faster onset of action, predictable amnesic effects, and relatively rapid recovery.

30 esis that STEP may be required for ethanol's amnesic effects.

31 otoxic, antidepressive, anxiolytic, and anti-amnesic effects.

32 over a 30-min delay; (iii) two subgroups of amnesics emerged, those subjects impaired in acquisition

33 In addition to the amnesic episodes, many patients describe significant int

34 e patients with otherwise typical FTD can be amnesic from presentation, or even present solely with a

35 Studies of fluency in MTL amnesics have yielded mixed results but were not designe

36 E.P. became profoundly amnesic in 1992 after viral encephalitis, which damaged

37 ive temporal lobe damage, all of whom became amnesic in a known year, were given tests of anterograde

38 trieval despite the fact that these mice are amnesic in long-term memory tests when natural recall cu

39 Patients with typical AD and non-amnesic initial presentation had a significantly higher

40 scertain since damage to this tract in human amnesics is invariably accompanied by atrophy to surroun

41 We report the case of R.S., a 49-year-old amnesic man, who we found to have acquired information a

42 ficits in multiple cognitive domains and 31% amnesic MCI.

43 Amnesic participants made more errors than nonamnesic pa

44 us in spatial learning and memory, we tested amnesic participants with hippocampal damage in a virtua

45 s hypothesis by examining the performance of amnesic participants with hippocampal lesions (one femal

46 Amnesic participants' latencies were longer and distance

47 The amnesic participants' learning resulted in their arrivin

48 perceptual memory (priming) in a profoundly amnesic patient (E.P.), despite at-chance recognition me

49 We tested a profoundly amnesic patient (E.P.), who has virtually complete bilat

50 Amnesic patient D.A. demonstrated intact TP, and perform

51 The amnesic patient E.P. has demonstrated normal levels of r

52 r semantic (fact) learning in the profoundly amnesic patient E.P., who has extensive damage limited p

53 Although neuropsychological studies of the amnesic patient H.

54 trasted the findings for E.P. with the noted amnesic patient H.M, whose surgical lesion is strikingly

55 ted to the hippocampal formation, a severely amnesic patient with extensive medial temporal lobe dama

56 he ability of bilateral medial temporal lobe amnesic patients (MT; n=8) and normal participants (NC;

57 Amnesic patients (n = 8), who have severely impaired dec

58 Amnesic patients acquired delay conditioning at a normal

59 Amnesic patients also produced significantly more nonada

60 easures that vary with word repetition in 12 amnesic patients and 12 control subjects: (i) a late pos

61 Seven bitemporal amnesic patients and 7 controls were randomly presented

62 Amnesic patients and control participants generated exem

63 this issue, resting fMRI was performed with amnesic patients and control participants.

64 Amnesic patients and controls listened to verbal descrip

65 Amnesic patients and controls showed comparable total le

66 Amnesic patients and nondemented patients with Parkinson

67 Eight medial temporal lobe amnesic patients and their demographically matched contr

68 Amnesic patients are impaired on recall tests [1, 2], an

69 reported perceptual deficits in the group of amnesic patients at the heart of this debate.

70 ning of the transverse patterning problem by amnesic patients derives from their general impairment i

71 Controls performed well, but the amnesic patients did not acquire categorical knowledge.

72 Amnesic patients exhibited normal learning of the task b

73 Two amnesic patients failed to recover fear responses follow

74 emonstrations of intact category learning by amnesic patients for dot patterns, artificial grammars,

75 Specifically, it was widely held that amnesic patients have impaired explicit memory performan

76 Amnesic patients K.C. and R.F.R. were tested to provide

77 ruction observed in some hippocampal-damaged amnesic patients may be supported by residual function i

78 Amnesic patients often show improved performance when st

79 Experiment 1 examined the performance of 6 amnesic patients on 11 to 25 different recognition memor

80 On the tests of semantic knowledge, the amnesic patients performed markedly better than the pati

81 mained stable across brief breaks in typing, amnesic patients showed evidence of impaired access to m

82 relations in paired-associate learning, the amnesic patients showed remarkably robust collaborative

83 ntrols retained these gains over the breaks; amnesic patients slowed down and compensated for these l

84 of conflicting results from studies in human amnesic patients suggests that extraneous damage to extr

85 The amnesic patients tended to be impaired on the same items

86 stem, providing preliminary support from MTL amnesic patients that the default network can be fractio

87 ion is a processing mechanism that may allow amnesic patients to compensate for relational memory def

88 The amnesic patients underwent a sadness induction procedure

89 The nondeclarative memory available to amnesic patients was relatively inflexible and available

90 In the present studies, category learning in amnesic patients was tested with stimuli that both exhib

91 Amnesic patients were able to distinguish between 2 tone

92 Amnesic patients were as competent as healthy volunteers

93 o discourage the use of episodic memory, the amnesic patients were impaired and E.P. performed at cha

94 The amnesic patients were impaired at solving both the trans

95 Amnesic patients were not able to reverse the previously

96 Numerous results in the amnesic patients were predicted in advance by the single

97 In contrast, amnesic patients were unimpaired on the category decisio

98 investigated object perception in two human amnesic patients who were chosen on the basis of their l

99 nd familiarity judgement tasks compared with amnesic patients with Alzheimer's disease and healthy co

100 ings are described here for three additional amnesic patients with bilateral damage limited to the hi

101 Three amnesic patients with damage limited to the hippocampal

102 Amnesic patients with damage to the hippocampal formatio

103 Here, we addressed this topic in amnesic patients with damage to the hippocampus (HC grou

104 Most amnesic patients with damage to the medial temporal lobe

105 To resolve this discrepancy, amnesic patients with either selective hippocampal damag

106 Three amnesic patients with extensive bilateral medial tempora

107 It is unknown whether amnesic patients with hippocampal damage would exhibit s

108 ddressed in 5 experiments with E.P., 3 other amnesic patients with hippocampal damage, and 8 healthy

109 ces, we compared the performance of severely amnesic patients with large medial temporal lobe lesions

110 me intervals has sometimes been described in amnesic patients with medial temporal lobe damage.

111 Amnesic patients with medial temporal system damage show

112 Specifically, amnesic patients with MTL damage were impaired at probab

113 Amnesic patients with MTL lesions that converged on the

114 hy participants, but occurred only rarely in amnesic patients with severe damage to the hippocampus.

115 Human amnesic patients with well characterized MTL damage and

116 It was found that amnesic patients' CRs occurred significantly earlier tha

117 matomotor network connectivity was intact in amnesic patients, indicating that bilateral MTL lesions

118 with the findings from patient RB and other amnesic patients, make three important points about memo

119 ruous than for incongruous words, as did the amnesic patients, who performed less well overall.

120 ative memory that are beyond the capacity of amnesic patients.

121 nimals are consistent with the findings from amnesic patients.

122 nd medial prefrontal cortex were observed in amnesic patients.

123 dementia performed markedly better than the amnesic patients.

124 d an aspect of memory that was intact in the amnesic patients.

125 a 'Schema for the Examination of Aphasic and Amnesic Persons'.

126 45 [IQR 0.035-0.056]) than did those with an amnesic presentation (0.041 [0.031-0.057]; p=0.001).

127 We observed two distinct amnesic profiles in bvFTD patients that could reflect tw

128 Studies have investigated the nootropic and amnesic properties of both systems.

129 ciceptive effects of THC and its detrimental amnesic properties.

130 in individuals with Alzheimer disease and in amnesic rats have demonstrated that DBS targeted to the

131 BLA of brain histamine-depleted rats, hence amnesic, restored long-term memory; however, the time fr

132 ng the organization trials, amnesics and non-amnesics retained information to a comparable extent ove

133 This amnesic shadow follows a dose-response function, becomes

134 ting occurs on these affective memories, the amnesic shadow itself is induced by conscious suppressio

135 Casting an amnesic shadow over harmful, reactivated memories thereb

136 ion through retrieval suppression induces an amnesic shadow that impairs the encoding and stabilizati

137 Combining the amnesic shadow with subliminal reactivation may offer a

138 y subliminally reactivating them within this amnesic shadow.

139 P), diarrheic shellfish poisoning (DSP), and amnesic shellfish poisoning (ASP) toxins in seafood is a

140 o detect the primary marine biotoxin groups: amnesic shellfish poisoning toxins, paralytic shellfish

141 for the neurologic condition referred to as amnesic shellfish poisoning.

142 rent evidence suggests that after an initial amnesic stage in Alzheimer's disease, attention is the f

143 Ten chronic and stable amnesic subjects (nine with bilateral medial temporal lo

144 paired declarative memory for the tasks, the amnesic subjects demonstrated acquisition and retention

145 dings of impaired item memory in hippocampal amnesics suggest a more nuanced role for the hippocampus

146 presentation: a typical pattern with initial amnesic syndrome (n = 4 cases), progressive visual dysfu

147 ropine and scopolamine (atr/scop) produce an amnesic syndrome in humans, subhuman primates, and roden

148 eptal muscarinic agonists also alleviate the amnesic syndrome produced by systemic administration of

149 rief survey of the clinical varieties of the amnesic syndrome, transient and persistent, selected the

150 sly accessible memories, producing a classic amnesic syndrome.

151 particular, mechanisms underlying the human amnesic syndrome.

152 ral lobes can result in profound anterograde amnesic syndromes.

153 when active, the internal state provided by amnesic treatments is represented and integrated within

154 major findings were: (i) immediate recall in amnesics was improved by providing an organizational str

155 Amnesic windows might also occur in healthy people due t