1 However,
amoebic appendicitis can sometimes be severe and life th
2 s, only 3 cases were clinically diagnosed as
amoebic appendicitis, including 2 diagnosed at the time
3 AS) staining and PCR to identify undiagnosed
amoebic appendicitis.
4 ng in an underestimation of the incidence of
amoebic appendicitis.
5 a protozoan intestinal parasite that causes
amoebic colitis and amoebic liver abscess.
6 programmed cell death to the pathogenesis of
amoebic colitis and amoebic liver abscess.
7 erythrophagocytosis and the ability to cause
amoebic colitis in human colonic xenografts.
8 One of the hallmarks of
amoebic colitis is the detection of Entamoeba histolytic
9 ne proteinases are a key virulence factor in
amoebic colitis, and provide a novel mechanism for their
10 cysteine proteinases in the pathogenesis of
amoebic colitis, human intestinal xenografts in SCID mic
11 in as an important component of virulence in
amoebic colitis.
12 zoites invade the intestinal mucosa, causing
amoebic colitis.
13 ell eukaryote that is the etiologic agent of
amoebic colitis.
14 results demonstrate for the first time that
amoebic cysteine proteinases are a key virulence factor
15 Amoebic cysteine proteinases have been proposed as impor
16 The critical role of
amoebic cysteine proteinases in human gut inflammation a
17 To test the role of
amoebic cysteine proteinases in the pathogenesis of amoe
18 amage may be explained by our discovery that
amoebic cysteine proteinases possess IL-1B converting en
19 an intestinal protozoan parasite that causes
amoebic dysentery and liver abscess.
20 ntamoeba histolytica, the causative agent of
amoebic dysentery, was determined to have raft-like plas
21 n and might constitute a new drug target for
amoebic dysentery.
22 rtunistic pathogen that causes granulomatous
amoebic encephalitis (GAE), a chronic and often fatal di
23 meningoencephalitis, a chronic granulomatous
amoebic encephalitis (GAE), and a chronic amoebic kerati
24 spp. and B. mandrillaris cause granulomatous
amoebic encephalitis (GAE), which usually presents as a
25 aneous skin lesions as well as granulomatous
amoebic encephalitis, a 'brain-eating' disease.
26 d the almost universally fatal granulomatous
amoebic encephalitis.
27 simple, reliable, and reproducible method of
amoebic enumeration that depends on simply establishing
28 g a most probable number (MPN) technique for
amoebic enumeration.
29 occurred early, because the sequences of the
amoebic enzymes show considerable divergence from those
30 ivergence from those of prokaryotes, and the
amoebic genes encoding these enzymes are in the AT-rich
31 ograms to analyze the promoter regions of 57
amoebic genes that had increased expression specifically
32 Amoebic gill disease (AGD) and complex gill disease (CGD
33 Repurposing antifungal azoles for
amoebic infections has been reported, but their inhibito
34 Amoebic keratitis (AK) is a potentially blinding infecti
35 n corneal tissue model representing clinical
amoebic keratitis infection.
36 Amoebic keratitis is a potentially blinding eye infectio
37 us amoebic encephalitis (GAE), and a chronic
amoebic keratitis.
38 Amoebic liver abscess (ALA) is the most common extrainte
39 range from asymptomatic to invasive disease,
amoebic liver abscess being the most common manifestatio
40 We report a peculiar case of left hepatic
amoebic liver abscess in a patient without a well-known
41 nds inhibit the trophozoite proliferation in
amoebic liver abscess induced in hamster.
42 This case shows that
amoebic liver abscess is possible even in first world co
43 nal parasite that causes amoebic colitis and
amoebic liver abscess.
44 h to the pathogenesis of amoebic colitis and
amoebic liver abscess.
45 Amoebic liver abscesses grow inexorably and, at one time
46 tion, follows a defined trafficking route to
amoebic lysosomal degradation machinery.
47 owleri is known to cause the disease primary
amoebic meningoencephalitis (PAM) and can be found in dr
48 mparatively low levels of infection, primary
amoebic meningoencephalitis (PAM) induced by Naegleria f
49 Primary
amoebic meningoencephalitis (PAM) is a human brain infec
50 ia fowleri is the causative agent of primary
amoebic meningoencephalitis (PAM), which is fatal in >97
51 s the protozoan pathogen that causes primary
amoebic meningoencephalitis (PAM), with the death rate e
52 n uncommon but deadly disease called primary
amoebic meningoencephalitis (PAM).
53 s as a mass, while N. fowleri causes primary
amoebic meningoencephalitis (PAM).
54 eria fowleri is a causative agent of primary
amoebic meningoencephalitis and is highly resistant to c
55 ing further evaluation as a lead for primary
amoebic meningoencephalitis.
56 ausative agent of the rare but fatal primary
amoebic meningoencephalitis.
57 ed in these promoters and specifically bound
amoebic nuclear protein(s).
58 Amoebic pathogenesis entails depletion of the host mucos
59 t both Acanthamoeba trophozoites and soluble
amoebic products induce an early anti-inflammatory monoc
60 , nibble) observed between immune cells, but
amoebic trogocytosis differs because it results in death
61 The discovery of
amoebic trogocytosis in E. histolytica may also shed lig
62 We review this process, termed '
amoebic trogocytosis' (trogo-, 'nibble'), and how this p
63 Amoebic trophozoites activate the transcription factor N
64 ytica infection have begun to illuminate how
amoebic trophozoites cause intestinal disease and liver
65 a to identify the pathogenic behavior of the
amoebic trophozoites.
66 The origins, benefits, and triggers of
amoebic virulence are complex.