ライフサイエンスコーパス: amygdala

1 l telencephalon (i.e., the teleostan pallial amygdala).

2 were observed in the central nucleus of the amygdala.

3 ions depends on the neocortex, striatum, and amygdala.

4 , but not to the cortex, basal forebrain, or amygdala.

5 not prevent hyperactivity in the basolateral amygdala.

6 tes vessels in other brain areas such as the amygdala.

7 to be specific for valence processing in the amygdala.

8 ually culminate in its delayed impact on the amygdala.

9 he interplay between the cmA and superficial amygdala.

10 mation of the glutamatergic circuitry in the amygdala.

11 ntually build up to hyperexcitability in the amygdala.

12 ds, and neuroimmune factors) in the extended amygdala.

13 by the blockade of serotonin reuptake in the amygdala.

14 before dorsolateral prefrontal cortex or the amygdala.

15 enkephalins in the intercalated cells of the amygdala.

16 consistent features of the Eutherian mammal amygdala.

17 d the ventromedial prefrontal cortex and the amygdala.

18 y- and stress-related regions, including the amygdala.

19 h as the striatum, orbitofrontal cortex, and amygdala.

20 er able to engage in top-down control of the amygdala.

21 ll stimuli, but no significant effect on the amygdala.

22 aged individuals is 'Alpha' versus 'Beta' in amygdala.

23 differences in AT, which includes the dorsal amygdala.

24 s, and projection neurons in the basolateral amygdala.

25 triatum, orbitofrontal cortex, and bilateral amygdala.

26 eural ensemble in the central nucleus of the amygdala.

27 ially distributed activation patterns in the amygdala.

28 hens synaptic connectivity days later in the amygdala, a brain area implicated in the affective sympt

29 by chemogenetic manipulation of the central amygdala, a stress-sensitive nucleus that forms a major

30 halin plays a significant role in regulating amygdala activity, but its action is strongly limited by

31 s valence could be easily distinguished from amygdala activity, PL neurons could distinguish both val

32 matic stress disorder (PTSD) show heightened amygdala activity; elevated levels of stress hormones, i

33 e by testing the hypothesis that the primate amygdala acts, in part, like a sensory structure for the

34 n occurring in the developing left and right amygdala after limited bedding exposure, a phenomenon th

35 show that lentiviral delivery of Wnt6 to the amygdala ameliorates locomotor impairment and social beh

36 s in the central-medial boundary zone of the amygdala (Amg(C/M-PAG) neurons) transiently suppressed U

37 density and serotonergic innervation of the amygdala among four macaque species using histological a

38 phosphorylation at Ser845 and Ser831 in the amygdala (AMY), anterior hippocampus (aHIP) and middle g

39 A subjects had decreased positive FC between amygdala and anterior cingulate cortex (ACC), and had in

40 egative functional connectivity (FC) between amygdala and dorsolateral prefrontal cortex (DLPFC), and

41 Seeds in the amygdala and dorsolateral prefrontal cortex were explore

42 n evidence examining the importance of early amygdala and extended amygdala circuitry development to

43 We measured single-neuron activity in the amygdala and found a representation for observational re

44 cal hypothalamus (PeF), periaqueductal gray, amygdala and frontal cortex.

45 NAcc) volume alteration, but not through the amygdala and hippocampal volumes.

46 significantly reduced GAL expression in the amygdala and hypothalamus whilst producing a correspondi

47 ion factor, that is co-expressed with GAL in amygdala and hypothalamus, as being important in the pro

48 Some key brain regions such as the amygdala and insula appear to be primarily involved in t

49 In FND, physical abuse also correlated with amygdala and insula coupling to motor cortices.

50 The change in parahippocampal/amygdala and insula responses during the perception of e

51 To this end, we assessed amygdala and insula treatment-related connectivity chang

52 onnectivity was assessed by subregion in the amygdala and insula, limbic structures key to the disord

53 iction included the cerebellum, hippocampus, amygdala and insular cortex.

54 erefore, we investigated the position of the amygdala and its functional subdivisions within the netw

55 ata indicate greater activation of the right amygdala and midline cerebellar vermis to nonemotional a

56 ivity were associated with activation in the amygdala and posterior temporal regions.

57 LPFC), and had increased negative FC between amygdala and precuneus and superior occipital gyrus (SOG

58 Furthermore, connectivity between the amygdala and prefrontal cortex mediated the relationship

59 AEA, both in the periphery, and also in the amygdala and prefrontal cortex, brain structures critica

60 The FC between left amygdala and right DLPFC had significant correlation wit

61 nt with findings in the ventral striatum and amygdala and show that this monosynaptically connected n

62 (ACC), and had increased positive FC between amygdala and thalamus.

63 am and also reduced connectivity between the amygdala and the anterior cingulate cortex, a network in

64 Functional coupling between the amygdala and the dorsomedial prefrontal cortex (dmPFC) h

65 euronal interactions between the basolateral amygdala and the rostral anterior cingulate gyrus of the

66 ith those in the typically developing (e.g., amygdala) and DBD-only (e.g., dorsal ACC) groups.

67 s (entorhinal cortex), affective processing (amygdala), and motor planning (dorsal premotor cortex) t

68 ion, mediated by the basal ganglia, extended amygdala, and frontal cortex, respectively.

69 We recorded single neurons in the MFC, amygdala, and hippocampus while human subjects switched

70 ria terminalis (BNST), well connected to the amygdala, and hypothalamic structures such as the parave

71 ecreased left frontal inhibition of the left amygdala, and larger decreases were associated with larg

72 analyses were restricted to the hippocampus, amygdala, and parahippocampal cortex, and applied separa

73 the subgenual cingulate cortex, hippocampus, amygdala, and putamen as demonstrating convergent abnorm

74 d decreases in orbitofrontal cortex, insula, amygdala, and temporal cortex.

75 PiB-PET measures in the subcortex (striatum, amygdala, and thalamus), but not in the cortex, were ass

76 ppocampus than in three other brain regions (amygdala, anterior cingulate, and prefrontal cortex).

77 iversity of Pennsylvania cases were cut from amygdala, anterior cingulate, superior/mid-temporal, and

78 tisol during pregnancy is related to newborn amygdala architecture and connectivity in a sexually dim

79 the basolateral nuclear complex (BNC) of the amygdala are critical for the regulation of emotion.

80 al hippocampal CA1 (vCA1) projections to the amygdala are necessary for contextual fear memory.

81 ching signal(s) that drive plasticity in the amygdala are still under debate.

82 in a number of brain regions, including the amygdala, are often sexually dimorphic, and have been re

83 e release onto the intercalated cells of the amygdala as an assay for enkephalin activity, we applied

84 ammals and was detected in post mortem human amygdala, as well as human serum samples.

85 (vCA1) hippocampal projections to the basal amygdala (BA), paired with aversive stimuli, contributes

86 ulation of VTA neurons projects to the basal amygdala (BA).

87 trated significant hippocampal inhibition of amygdala, basal-ganglia, thalamus, orbital frontal corte

88 JNJ-42165279 attenuated activation in the amygdala, bilateral anterior cingulate, and bilateral in

89 of ventral hippocampus (vHipp), basolateral amygdala (BLA) and prefrontal cortex (PFC) inputs reveal

90 the ventral hippocampus (vHipp,) basolateral amygdala (BLA) and prefrontal cortex (PFC) onto identifi

91 rtex neurons that project to the basolateral amygdala (BLA) and reduced CD response compared to subor

92 ctional interactions between the basolateral amygdala (BLA) and the nucleus accumbens core (NAcC).

93 ial prefrontal cortex (mPFC) and basolateral amygdala (BLA) and their reciprocal inhibitory connectiv

94 mption, whereas knockdown in the basolateral amygdala (BLA) decreased alcohol consumption and reduced

95 rtex (GC) and the basolateral nucleus of the amygdala (BLA) however, its synaptic underpinnings are u

96 atory synaptic physiology in the basolateral amygdala (BLA) in male Sprague Dawley rats.

97 The basolateral amygdala (BLA) modulates the consolidation of dorsal hip

98 stored in a genetically distinct basolateral amygdala (BLA) neuronal population that drives reward be

99 The basolateral amygdala (BLA) plays a critical role in fear conditionin

100 The basolateral amygdala (BLA) plays a vital role in associating sensory

101 The basolateral amygdala (BLA) regulates conditioned responses evoked by

102 Whole-cell recordings in basolateral amygdala (BLA) slices from rats revealed higher frequenc

103 e, we show that stress increases basolateral amygdala (BLA) spike firing.

104 ed with anterograde tracing from basolateral amygdala (BLA) to PFC to identify sex-specific innervati

105 F) modulate the responses of the basolateral amygdala (BLA) to stress and are associated with the dev

106 on the dorsal hippocampus (HPC), basolateral amygdala (BLA), and somatosensory cortex (SSCTX).

107 nce requires prelimbic (PL) PFC, basolateral amygdala (BLA), and ventral striatum (VS).

108 sin II inhibition (NMIIi) in the basolateral amygdala (BLA), but not dorsal hippocampus (CA1), select

109 medial prefrontal cortex (mPFC), basolateral amygdala (BLA), dorsomedial striatum (DMS) and olfactory

110 IGNIFICANCE STATEMENT Within the basolateral amygdala (BLA), neuropeptide Y (NPY) is associated with

111 Here, we show that the mouse basolateral amygdala (BLA)-prelimbic prefrontal cortex (plPFC) circu

112 icularly strong candidate is the basolateral amygdala (BLA).

113 repinephrine (NE) release in the basolateral amygdala (BLA).

114 to project downstream to CA1 and basolateral amygdala (BLA).

115 ial prefrontal cortex (mPFC) and basolateral amygdala (BLA).

116 ear, he tells us here, is not wired into the amygdala, but is instead a cognitively assembled underst

117 ntal cortex (vlPFC) and most strongly in the amygdala, but none of the serotonin receptor genes, were

118 f other subcortical structures examined (the amygdala, caudate, globus pallidus, putamen, and thalamu

119 neurons activated by GA in the mouse central amygdala (CeA(GA) neurons).

120 uroadaptations in the central nucleus of the amygdala (CeA) and gene expression changes in the medial

121 ia projections to the central nucleus of the amygdala (CeA) and nucleus accumbens (NAc).

122 ctor (CRF)-expressing neurons in the central amygdala (CeA) antagonize the extinction memory followin

123 opin-releasing hormone gene (Crh) in central amygdala (CeA) are implicated in threat regulation, yet

124 s have implicated the central nucleus of the amygdala (CeA) as an important site for mediating the so

125 ypothalamic area, and central nucleus of the amygdala (CeA) contain the densest concentrations of NTS

126 d nucleus stria terminalis (BNST) or central amygdala (CEA) generates an aversive memory.

127 sin (ChR2) stimulation of central nucleus of amygdala (CeA) in rats with encountering either sucrose,

128 Given the role of the central nucleus of the amygdala (CeA) in the expression of such behaviors [3-5]

129 The central nucleus of the amygdala (CeA) is a key player in alcohol-dependence ass

130 rk using a rat model showed that the central amygdala (CeA) plays an important role in avoidance of a

131 1 d later, whereas silencing the VTA-central amygdala (CeA) projection had no effect.

132 [i.e., neurons that project from the central amygdala (CeA) to the lateral hypothalamus (LH)] mediate

133 GABA signaling in the central nucleus of the amygdala (CeA) underlies key behaviors associated with a

134 the circuit organization within the central amygdala (CeA), a critical regulator of emotional states

135 vely connected to the central nucleus of the amygdala (CEA), and both regions send convergent project

136 BAergic synaptic transmission in the central amygdala (CeA), and circulating cytokine levels were mea

137 in the ventral tegmental area (VTA), central amygdala (CeA), and nucleus accumbens (NAc) shell had no

138 cal recordings in the central nucleus of the amygdala (CeA), we found that rats with high addiction-l

139 eC) and lateral (CeL) nucleus of the central amygdala (CeA).

140 citability in rat lateral nucleus of central amygdala (CeL).

141 s callosum, ventricular system, hippocampus, amygdala, cerebellum and the gyrification index, all rev

142 nalis (BNST), a brain region of the extended amygdala circuit, has been identified as the critical hu

143 functional interactions within the affective amygdala circuit.

144 he importance of early amygdala and extended amygdala circuitry development to the emergence of anxie

145 fferent developmental trajectory than fronto-amygdala circuitry involved in traditional extinction le

146 es vs. neutral faces within the centromedial amygdala (cmA).

147 The basolateral amygdala complex (BLA), extensively connected with both

148 emory and heightened cFos in the basolateral amygdala complex (BLC) with retrieval of the remote (30-

149 This study aims to explore the amygdala connectivity abnormalities in IA.

150 These findings indicate that the amygdala connectivity is altered in IA subjects.

151 oundation for models relating aberrations in amygdala connectivity to psychiatric symptoms in individ

152 onnectivity, (2) induction of negative dlPFC-amygdala connectivity, and (3) local and distributed cha

153 neurons in the VTA that project to the basal amygdala contribute to such a teaching signal for plasti

154 , this work provides a detailed framework of amygdala-cortical interactions that can be used as a fou

155 is refined through the feedback provided by amygdala corticofugal projection (ACPs).

156 eurotensin-expressing neurons in the central amygdala decreases intake of and preference for ethanol

157 The amygdala demonstrated substantial neuronal loss, particu

158 ng how non-conscious processes involving the amygdala detect and respond to danger has contributed to

159 ng how non-conscious processes involving the amygdala detect and respond to danger.

160 The altered FC of amygdala-DLPFC is associated with duration of IA.

161 er, the mechanisms by which stress increases amygdala-dmPFC synaptic strength and generates anxiety-l

162 te depressive symptoms through modulation of amygdala emotional reactivity is unknown.

163 ing that actions of systemic inflammation on amygdala emotional reactivity play a mechanistic role in

164 By contrast, the amygdala encodes overall value accurately.

165 urs are not localized to subdivisions of the amygdala even though the inputs and outputs that carry s

166 The amygdala facilitates odor driven behavioral responses by

167 nd respond to danger has contributed to the 'amygdala fear center' meme, a view he does not endorse.

168 cting anxiety-like behaviors and basolateral amygdala firing.

169 This provides a framework in the amygdala for analyzing how the initial physiological and

170 Structural brain connectivity of the amygdala, fornix, uncinate fasciculus, and cingulum was

171 ed (P = 0.001 and P = 0.04, respectively) in amygdala from patients with ASD (n = 8) compared to non-

172 Larger decreases in amygdala-frontal connectivity and insula-parietal connec

173 Amygdala-frontal connectivity mediated the relationship

174 lizing behavior had a sex interaction in the amygdala-frontal pathway; weaker connectivity (lower fra

175 to characterize developmental transitions in amygdala function underlying age-specific behavioral tra

176 riation in the distribution and magnitude of amygdala functional connectivity with the cerebral corte

177 exhibited more consistent activation of the amygdala, fusiform gyrus, and thalamus than emerging adu

178 umbens, bed nucleus of the stria terminalis, amygdala, habenula, and raphe nucleus), all of which exp

179 how ELS selectively produces effects in one amygdala hemisphere during a critical period of brain de

180 cholinergic innervation patterns within the amygdala, hippocampus, and prefrontal cortex.

181 ly the medial prefrontal cortex, basolateral amygdala, hippocampus, anterior cingulate cortex, and ve

182 ehavior, cognitive, and motor function (e.g. amygdala, hippocampus, cerebellum).

183 ocuses on subcortical structures such as the amygdala; however, less is known about the distributed c

184 hin a network of brain regions including the amygdala, hypothalamus and dorsolateral prefrontal corte

185 es implicated anterior hippocampus (aHC) and amygdala in approach-avoidance decisions under threat, a

186 Bastin et al. fail to include the amygdala in their integrative memory model.

187 nsitivity to threat was confirmed via direct amygdala infusions of a selective serotonin reuptake inh

188 physiological state with hypothalamus-gated amygdala inputs that signal upcoming ingestion of food o

189 rain stimulation was associated with reduced amygdala-insula functional connectivity.

190 ents, including the prefrontal, hippocampus, amygdala, insular, cingulate, cerebellum, caudate, basal

191 The amygdala is a brain area critical for the formation of f

192 The centrolateral amygdala is an important node in the neuronal circuit th

193 The amygdala is an important structure involved in the modul

194 The amygdala is involved in hedonic valuation, but its role

195 sing inhibitory neurons in the centrolateral amygdala is necessary for the inhibition of a conditione

196 sing inhibitory neurons in the centrolateral amygdala is necessary for the long-term storage of condi

197 the insect analog of the mammalian cortical amygdala, is the main target for this olfactory informat

198 lly and in excitatory neurons in the lateral amygdala (LA) impaired long-term memory.

199 lta (PKCdelta)-expressing neurons in central amygdala lateral division (CeL).

200 The prelimbic (PL) area and basolateral amygdala (lateral [LA] and basolateral [BL] nuclei) have

201 left posterior cingulate cortex (PCC), right amygdala, left hippocampus, and right thalamus were sign

202 Amygdala lesions impair performance in reinforcer devalu

203 Our results identify dissociable central amygdala mechanisms of abstinence-dependent expression o

204 double-labeled expression in CeL and central amygdala medial division (CeM).

205 This review uses the example of amygdala-medial prefrontal cortex circuitry development

206 egulation of endogenous opioid modulation of amygdala-mediated emotional and behavioral responses.

207 Furthermore, altered amygdala microstructure was only observed in boys, with

208 ealed three major brain modules: 1) extended amygdala module, 2) midbrain striatal module, and 3) cor

209 ific reduction in mGluR2 function within the amygdala network and facilitates fear, and mGluR2 PAMs c

210 alized coordination in the medial prefrontal-amygdala network underlies social-decision preferences.

211 Here, we aimed to determine whether human amygdala neurons are involved in the computations necess

212 we compared the coding properties of PL and amygdala neurons during a task that requires rats to pro

213 ovides a rare glimpse into the role of human amygdala neurons in social cognition.

214 yses suggested observational value coding in amygdala neurons occurred in a different subset of neuro

215 Additionally, distinct subsets of amygdala neurons represented self-experienced and observ

216 We found a significant proportion of amygdala neurons whose activity correlated with both exp

217 and trial identity as well as or better than amygdala neurons.

218 Collectively, our results suggest that amygdala-nigra interactions represent a previously unapp

219 ting fear and avoidance responses [bilateral amygdala, nucleus accumbens (NAcc), and ventromedial pre

220 vo optogenetic activation of the basolateral amygdala-nucleus accumbens (BLA-NAc) glutamatergic circu

221 in the striatum, anterior cingulate cortex, amygdala, occipitotemporal cortex, and insula (Z > 2.3;

222 zed three functional subdivisions within the amygdala of each individual.

223 documented increased GAS5 expression in the amygdala of individuals with AUD.

224 tensin neurons in the central nucleus of the amygdala of male mice are activated by in vivo ethanol c

225 s to inhibitory neurons in the centrolateral amygdala of mice to block cell-type-specific translation

226 f BLA neurons in vitro in the left and right amygdala of postnatal days 22-28 male and female offspri

227 stability at the expense of lability in the amygdala of rats.

228 We found no evidence for baseline amygdala or ventromedial PFC volume serving as treatment

229 ventromedial prefrontal cortex, basolateral amygdala, or hippocampal CA1 region.

230 ed the same relationship in left hemispheric amygdala (p = 0.010), caudate (p = 0.008), inferior fron

231 pha (Esr1)-expressing cells in the posterior amygdala (PA) as a main source of excitatory inputs to t

232 or frontal gyrus, right angular gyrus, right amygdala/parahippocampal gyrus, and bilaterally in the m

233 ions to the NAcS, but not to the basolateral amygdala, partially reversed suppression of EtOH lever p

234 Although the hippocampal-amygdala pathway has been implicated in the retrieval of

235 tions of neuronal activity in the prefrontal-amygdala pathways critically contribute to social decisi

236 The primate amygdala performs multiple functions that may be related

237 We demonstrate that OT-induced decreases in amygdala perfusion, a key hub of the OT central circuitr

238 The central nucleus of the amygdala plays a significant role in alcohol use and oth

239 e risk for psychopathologies associated with amygdala-prefrontal cortex (PFC) circuits.

240 rder is associated with hyperactivity in the amygdala-prefrontal networks, and normalization of this

241 ELS effects are often sexually dimorphic and amygdala processes exhibit hemispheric asymmetry, we inv

242 The basolateral amygdala projection (BLAp) innervates broad regions of t

243 one-trial learning dependent on basolateral amygdala projection neurons (BLApn).

244 inhibition, displayed elevated hippocampal, amygdala, putamen and thalamus volumes, and evidence of

245 32) and anti-TNF conversely decreasing right amygdala reactivity (across emotional valence) (p = 0.03

246 Heightened amygdala reactivity (particularly to negatively valanced

247 BNC210 reduced amygdala reactivity to fearful faces relative to placebo

248 N-alpha and anti-TNF significantly modulated amygdala reactivity with IFN-alpha acutely enhancing rig

249 Endogenous opioid peptides in the amygdala regulate many of our behaviors and emotional re

250 rate a dissociation between arousal-specific amygdala responding and triggered valence-specific amygd

251 la responding and triggered valence-specific amygdala responding.

252 onth post-psilocybin, negative affective and amygdala response to facial affect stimuli returned to b

253 e mood, increased positive mood, and reduced amygdala response to negative affective stimuli.

254 Further, at greater levels of amygdala response, more securely attached individuals sh

255 ivity with IFN-alpha acutely enhancing right amygdala responses to sad (compared with neutral) faces

256 ted two functional MRI tasks, which measured amygdala responsivity to angry facial expressions and ve

257 specific; no interactions were apparent for amygdala responsivity to neutral faces, or striatal resp

258 The causal relationship between amygdala serotonin levels and an animal's sensitivity to

259 Targeting of amygdala serotonin reuptake with selective serotonin reu

260 s) confirmed the causal relationship between amygdala serotonin transporter and an animal's sensitivi

261 r, these findings provide evidence that high amygdala serotonin transporter expression contributes to

262 Valence-specific patterns of amygdala signaling predicted decisions on food consumpti

263 These results suggest that valence-specific amygdala signals are integrated into the formation of fo

264 SRIs may be mediated by their actions in the amygdala.SIGNIFICANCE STATEMENT Findings here contribute

265 nt and by signaling such events to the basal amygdala.SIGNIFICANCE STATEMENT Powerful mechanisms of f

266 ocations within the primate (Macaca mulatta) amygdala spatially defined and statistically separable r

267 startle electromyography and brainstem- and amygdala-specific functional magnetic resonance imaging

268 In the dorsal ACC, outputs to dPMC and the amygdala strongly overlap in deep layers.

269 rences in the stereotactic locations both of amygdala subdivisions and of cortical functional brain n

270 ng of fMRI signals in the cortex relative to amygdala subdivisions.

271 primarily the cerebral cortex, hippocampus, amygdala, suprachiasmatic nuclei, anterior olfactory nuc

272 We previously identified thalamo-amygdala synapses (T-LA) that project via the interal ca

273 lated intrinsic communication of the striato-amygdala system engaged in reinforcement-based and emoti

274 onstrated that plasticity at thalamo-lateral amygdala (T-LA) synapses is critically involved in the r

275 long-range projections to a number of extra-amygdala targets, but the functions of these projections

276 However, greater hippocampal inhibition of amygdala, thalamus, IFG and dmPFC correlated with hippoc

277 static maladaptation of AEA signaling in the amygdala that drives emotional alterations.

278 ssory olfactory bulb to the posterior medial amygdala-that is necessary for all behavioural responses

279 vity demonstrated abnormal activation in the amygdala, the hippocampal/parahippocampal gyri, the dors

280 The basolateral amygdala therefore facilitates cue-induced control over

281 s allows for mechanistic perturbation of the amygdala to determine its causal contribution to AT.

282 These data suggest that this central amygdala to parabrachial nucleus projection influences t

283 lus, the primary fiber bundle connecting the amygdala to the orbitofrontal cortex and a key component

284 to NAcS, or vmPFC projections to basolateral amygdala, to punished EtOH-SA.

285 The ventral circuit, that includes the amygdala, ventral medial prefrontal cortex, and ventral

286 nts with anxiety disorders exhibited greater amygdala-ventromedial prefrontal cortex (vmPFC) connecti

287 onal autoantibody was associated with larger amygdala volumes (p < 0.05).

288 ICV) and thalamus, putamen, hippocampus, and amygdala volumes and greater lateral ventricle, caudate,

289 egrity on white mater tracts passing through amygdala was also examined.

290 indirect pathway with a relay in the central amygdala was also observed that is similar in its struct

291 indicated that aromatase availability in the amygdala was negatively associated with body mass index

292 ortex (vlPFC) and most strongly in the right amygdala, was associated positively with anxiety-like be

293 tracellular recordings of the mouse cortical amygdala, we identified changes in the electrophysiologi

294 nal and structural connectivity of bilateral amygdala were examined using seed-based connectivity ana

295 ularly in entorhinal cortex, hippocampus and amygdala, were observed between 18F-MK-6240 and global 1

296 luding the orbitofrontal cortex, insula, and amygdala, were persistent and thus may play an important

297 the coupling between the cmA and basolateral amygdala, whereas LZP increased the interplay between th

298 t sensitive to facial expression, as was the amygdala, whereas those on the lower, lateral edge of th

299 cleus accumbens shell (NAcS) and basolateral amygdala, which encode positive and negative valence of

300 pped onto the anatomical organization of the amygdala, while other components reflected integration a