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1 nscript levels were also induced for gpc4 by anaerobiosis.
2 d by a nutritional downshift or by transient anaerobiosis.
3 rmitted expression of increased tolerance to anaerobiosis.
4 tochondrial NADH/NAD+ ratios associated with anaerobiosis.
5 the FNR-dependent narK operon promoter under anaerobiosis.
6 chondrion reductive evolution on the path to anaerobiosis.
7 heat shock response, DNA recombination, and anaerobiosis.
8 early stages of mitochondrial adaptation to anaerobiosis.
9 hment to surfaces, were downregulated during anaerobiosis.
10 s also required for effective acclimation to anaerobiosis.
11 tes that luminal lactate is a marker mucosal anaerobiosis.
12 itions that was particularly striking during anaerobiosis.
13 mRNA abundance) in the hydEF-1 mutant during anaerobiosis.
14 icroaerobiosis, but not during aerobiosis or anaerobiosis.
15 ignificantly enhanced survival during sudden anaerobiosis.
16 thought to mediate the cellular response to anaerobiosis.
17 that can be induced, among other things, by anaerobiosis.
18 by MvaT and positively controlled by Anr and anaerobiosis.
19 ating persistent state in response to NO and anaerobiosis.
20 issimilatory pathways required for long-term anaerobiosis.
21 l modes of pH regulation were revealed under anaerobiosis.
22 trate- or nitrite-dependent induction during anaerobiosis.
23 by macrophages of M. tuberculosis exposed to anaerobiosis.
24 tion of the ResE phosphatase activity during anaerobiosis.
25 cells, which was completely abolished under anaerobiosis.
26 expression insensitive to H-NS repression in anaerobiosis.
27 romoter, does not appear to be active during anaerobiosis.
28 vity approximately three-fold in response to anaerobiosis.
29 ene is induced nearly 30-fold in response to anaerobiosis.
30 egulated by both Fnr and ArcA in response to anaerobiosis.
31 d the dynamics of the response to short-term anaerobiosis (2 generations) in both catabolite-represse
33 tion defects, suggesting that in addition to anaerobiosis, adaptation to cell envelope stress is a cr
35 using the yeast model of NP-C disease during anaerobiosis, an auxotrophic condition that requires yea
36 thionyl-tRNAs with methionine in response to anaerobiosis and antibiotic exposure via the methionyl-t
37 activated by the Fnr protein in response to anaerobiosis and by the NarQ-NarP two-component regulato
39 bilised in response to aminoglycosides under anaerobiosis and engaged in cell membrane regulation.
40 the FNR transcription factor in response to anaerobiosis and further increased in response to nitrit
41 trate reduction, is increased in response to anaerobiosis and further stimulated by the addition of n
44 ttern of dmsA-lacZ expression in response to anaerobiosis and nitrate addition was identical in both
50 lutionary mechanisms of early transitions to anaerobiosis and shed light on fine-scale adaptations in
51 activates expression of hbaR in response to anaerobiosis and that HbaR, in turn, activates expressio
53 responsive to intracellular pH as well as to anaerobiosis and that residues in the ligand-binding poc
55 ation, (2) production of N2 and N2O requires anaerobiosis, and (3) hybrid N2 is evidence of codenitri
57 e in expression that occurred in response to anaerobiosis, and badR was responsible for a further 5-f
59 activated by the Fnr protein in response to anaerobiosis, and it is further activated and/or repress
60 s of ethylene production such as cold, heat, anaerobiosis, and Li(+) ions enhance or suppress the exp
61 nutrient limitation (particularly iron) and anaerobiosis are major stresses experienced by V. choler
62 row on these resources in vitro, pointing to anaerobiosis as a potential factor limiting growth in th
63 ulation of cupA gene expression and identify anaerobiosis as an inducer of phase-variable cupA gene e
64 r aminoglycoside bactericidal activity under anaerobiosis but its mechanism of action is unclear.
65 od-shaped to filamentous form in response to anaerobiosis by regulating RNase E subcellular distribut
66 rease being independent of the SOS response, anaerobiosis, catabolite repression, and integration hos
67 s elevated in galactose-containing medium in anaerobiosis compared to growth in glucose, and the muta
68 omyces cerevisiae which are expressed during anaerobiosis (DAN1, DAN2, DAN3, DAN4, TIR1, TIR2, TIR3,
69 e cycles, and ecophysiology (cold hardiness, anaerobiosis, desiccation resistance) are discussed.
71 l more pH-regulated proteins; in particular, anaerobiosis enabled induction of several additional cat
72 ensor that deacetylates nuclear eIF5A during anaerobiosis, enabling the cytoplasmic export of eIF5A/T
74 ns are dispensable for UQ biosynthesis under anaerobiosis, even though they were expressed in the abs
75 y carbon starvation, respiratory inhibitors, anaerobiosis, freezing or boiling) lost the ability to g
76 im of this work was to evidence that keeping anaerobiosis has a deep impact on the viability and dive
77 ions to low-oxygen concentrations leading to anaerobiosis have independently arisen in many eukaryoti
78 amely exponential growth, stationary growth, anaerobiosis, heat shock, oxidative stress, nitrogen lim
79 esponse." By approximately 0.2 generation of anaerobiosis in both media, more chronic, heme-dependent
80 deepen our understanding of the evolution of anaerobiosis in ciliates, we predicted the mitochondrial
81 reductase, a major respiratory complex under anaerobiosis in Escherichia coli, is submitted to tight
82 cells in the light depleted O(2) and caused anaerobiosis in the culture, which was necessary and suf
83 olic and regulatory responses that accompany anaerobiosis in wild-type C. reinhardtii cells and a nul
84 tion of sdaA encoding serine deaminase under anaerobiosis; in addition, the glutamate decarboxylase g
85 7H10 Middlebrook medium, both acidic pH and anaerobiosis increased the uptake of the H37Rv strain fo
86 immunofluorescence microscopy, we show that anaerobiosis-induced alginate production by planktonic P
88 that the bacteria experienced conditions of anaerobiosis, iron limitation, and nutrient deprivation
90 h and that adaptation to nutrient stress and anaerobiosis is crucial for H. ducreyi survival in human
91 s examined to determine whether its obligate anaerobiosis is imposed by endogenous reactive oxygen sp
92 pIV-dependent induction of psp expression in anaerobiosis is independent of PspBC, establishing that
93 on of cadC by AphB in response to low pH and anaerobiosis is mirrored in the heterologous organism Es
94 Moreover, the results indicate that mere anaerobiosis is not sufficient to maintain hydrogenase g
100 y, since cells remain sensitive to Mn during anaerobiosis or when hydrogen peroxide biogenesis is sig
101 er oxidation by PSII, (ii) a faster onset of anaerobiosis preserves PSII from irreversible photoinhib
102 E. coli cultures because of reports that in anaerobiosis, pyruvate kinase represents the major route
103 d in opposite conditions: FnrS, expressed in anaerobiosis, represses the expression of iscR while Oxy
104 ars to involve a regulatory cascade in which anaerobiosis, signaled through Anr, stimulates expressio
105 ls to oxidation lies at the root of obligate anaerobiosis, spontaneous mutagenesis, and the use of ox
108 incubation conditions, such as the length of anaerobiosis, the readdition of O2, the presence of acet
110 her, these data provide evidence that during anaerobiosis, the Rpd3 complex acts at the DAN1 promoter
111 n a seconds time scale, of haem a3 following anaerobiosis, there was no indication of accumulation of
112 ox state resembling either reduced oxygen or anaerobiosis, thereby resulting in increased expression
116 d acid (pH 5 to 6) restored acid resistance; anaerobiosis was not required, as it is for acid resista
118 e reductions in activity under conditions of anaerobiosis were found to be primarily the result of re