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1  occur (extravasations, adverse, allergic or anaphylactic reactions).
2                     The patient developed an anaphylactic reaction.
3 ent was also observed in an active cutaneous anaphylactic reaction.
4 s) and 95% confidence intervals (CIs) for an anaphylactic reaction.
5 ained arrhythmia, cardiac arrest, and severe anaphylactic reaction.
6 cur with fluorescein angiography, such as an anaphylactic reaction.
7                         Two HCWs experienced anaphylactic reaction.
8 ng signals can dominate to initiate a severe anaphylactic reaction.
9 95.4%) and in all patients with a history of anaphylactic reaction.
10 symptoms ranging from oral pruritus to fatal anaphylactic reaction.
11 7.7% (95% CI, 5.7% to 9.7%) reported a prior anaphylactic reaction.
12 cted to oral challenge, which resulted in an anaphylactic reaction.
13          The TWEAK/Fn14 axis participates in anaphylactic reactions.
14 hereas PGE(2) stabilization protects against anaphylactic reactions.
15 ete Freund's adjuvant has resulted in severe anaphylactic reactions.
16 life of the rMETase apoenzyme and eliminated anaphylactic reactions.
17 diators that cause allergic inflammation and anaphylactic reactions.
18 ths, and no patients experienced allergic or anaphylactic reactions.
19 d minor allergic responses, and 3 (0.7%) had anaphylactic reactions.
20         Drugs are a frequent cause of severe anaphylactic reactions.
21  could also abort ongoing acute IgE-mediated anaphylactic reactions.
22 ot all patients with mastocytosis experience anaphylactic reactions.
23 s organs and severe or even life-threatening anaphylactic reactions.
24 duce macrophage cholesterol efflux in severe anaphylactic reactions.
25 dysfunction and the severity of IgE-mediated anaphylactic reactions.
26 rotective immune responses in the absence of anaphylactic reactions.
27 ere confirmed as allergic, including 11 with anaphylactic reactions.
28 the VE IL-4Ralpha and ABL1 pathway in severe anaphylactic reactions.
29 history of anaphylaxis are at risk of future anaphylactic reactions.
30 posed to contain milk proteins, which caused anaphylactic reactions.
31 portantly, CpG/PN-NP treatment did not cause anaphylactic reactions.
32 can range from mild local symptoms to severe anaphylactic reactions.
33 re were 507 uniphasic and 25 (4.5%) biphasic anaphylactic reactions.
34 patients to prevent further life-threatening anaphylactic reactions.
35 ocument a role for cofactors in about 30% of anaphylactic reactions.
36                  Symptoms range from mild to anaphylactic reactions.
37 , thereby promoting allergic and potentially anaphylactic reactions.
38 ere, including one moderate reaction and two anaphylactic reactions.
39 developed significantly attenuated cutaneous anaphylactic reactions.
40  allergenic food capable of provoking severe anaphylactic reactions.
41 allergenic foods, which can result in severe anaphylactic reactions.
42 eous tissue, are less likely to give rise to anaphylactic reactions.
43  mice represents a new animal model to study anaphylactic reactions.
44  the most common cause of fatal food-induced anaphylactic reactions.
45 lation of SNAP-23 leads to degranulation and anaphylactic reactions.
46 on of IgG1 Abs and to the risk of triggering anaphylactic reactions.
47 4 toxicities included acne-like rash (6.1%), anaphylactic reactions (1.5%), and diarrhea (1.5%).
48 were associated with a 38% increased risk of anaphylactic reaction (40.7 per 100,000 person-years vs.
49 d a non-anaphylactic reaction and 115 had an anaphylactic reaction (88 moderate and 27 severe).
50 hycardia, upper gastrointestinal hemorrhage, anaphylactic reaction, acute kidney injury, and acute my
51 ive patients with a convincing history of an anaphylactic reaction after a hymenoptera sting were tes
52 ort we describe a patient who experienced an anaphylactic reaction after the injection of fluorescein
53                                 Allergic and anaphylactic reactions after the first and second doses
54 uced anaphylaxis (WDEIA) is characterized by anaphylactic reactions after wheat ingestion and physica
55 ble involvement of augmenting factors; after anaphylactic reactions, always ask for possible augmenta
56 gy to peanut is one of the leading causes of anaphylactic reactions among food allergic patients.
57  was conducted with the MeSH of anaphylaxis, anaphylactic reaction, anaphylactic shock, refractory an
58               Thirty-eight experienced a non-anaphylactic reaction and 115 had an anaphylactic reacti
59                The animals were analyzed for anaphylactic reaction and by molecular and immunologic a
60  OcAn, both confirmation of the diagnosis of anaphylactic reaction and identification of the trigger
61 ced a serious infusion reaction (one grade 4 anaphylactic reaction and one grade 3 stridor) during th
62 history of allergy to contrast agents had an anaphylactic reaction and recovered without sequelae.
63 ing patients after complete resolution of an anaphylactic reaction and to dispense with prolonged mon
64                              We suspected an anaphylactic reaction and treated him with intramuscular
65 o beta-lactams are the most common causes of anaphylactic reactions and can be life-threatening.
66 pture, as in case of misdiagnosis, may cause anaphylactic reactions and dissemination.
67 HalphaT(+) patients were more likely to have anaphylactic reactions and less likely to have cutaneous
68  quantify the extravasation underlying human anaphylactic reactions and propose new theragnostic appr
69    Safety monitoring encompassed allergic or anaphylactic reactions and serious adverse events attrib
70                                   Urticaria, anaphylactic reaction, and angioneurotic edema were only
71 ated (four cases of haematoma expansion, one anaphylactic reaction, and one ischaemic stroke) and two
72 p between serum basal tryptase (sBT) levels, anaphylactic reactions, and clonal mast cell diseases wa
73                                 Elicitors of anaphylactic reactions are any sources of protein with a
74            Severe IgE-mediated, food-induced anaphylactic reactions are characterized by pulmonary ve
75 ate that the vast majority of food-triggered anaphylactic reactions are not life-threatening.
76                                              Anaphylactic reactions are not uncommon and are often fa
77                                     Although anaphylactic reactions are potentially life-threatening,
78                                              Anaphylactic reactions associated with the Food and Drug
79     Two patients in the reslizumab group had anaphylactic reactions; both responded to standard treat
80 peptide 1 receptor agonists (GLP-1 RAs) with anaphylactic reactions, but real-world evidence for this
81 city while reducing their potential to cause anaphylactic reactions by essentially eliminating IgE-me
82 ) disease that can lead to potentially fatal anaphylactic reactions caused by excessive MC mediator r
83 tide-induced EAE models led to a rapid-onset anaphylactic reaction characterized by respiratory distr
84 ears [n = 356] without a history of a severe anaphylactic reaction) developing objective symptoms dur
85                                     Systemic anaphylactic reactions did not occur.
86                                           An anaphylactic reaction due to a Hymenoptera sting is a cl
87 he scientific evidence on self-medication of anaphylactic reactions due to Hymenoptera stings, to inf
88                 The underlying mechanisms of anaphylactic reactions due to occupational exposure are
89                                     Biphasic anaphylactic reactions, especially clinically important
90                                              Anaphylactic reaction following contrast agent administr
91                               The effects on anaphylactic reactions following PN challenge and the as
92                            We identified 213 anaphylactic reactions in 192 children (97 male patients
93 teria monocytogenes (HKLM) as an adjuvant on anaphylactic reactions in a mouse model of PN allergy.
94 mpared with milk in the determinism of fatal anaphylactic reactions in children.
95              The prevalence and incidence of anaphylactic reactions in Germany are unknown.
96  roles in initiating and amplifying allergic/anaphylactic reactions in humans.
97 IIA may therefore contribute to allergic and anaphylactic reactions in humans.
98 xtravasation in the severity of IgE-mediated anaphylactic reactions in mice.
99 ast to non-PEGylated rMETase, which elicited anaphylactic reactions in monkeys.
100  antibody production in mice and monkeys and anaphylactic reactions in monkeys.
101  inhibitors have been associated with severe anaphylactic reactions in patients with hymenoptera veno
102 s of pathogenic antibody or life-threatening anaphylactic reactions in protein replacement therapy fo
103 c response to infused VWF concentrates up to anaphylactic reactions in rare cases.
104  early-phase and severely blunted late-phase anaphylactic reactions in response to antigen challenge
105  carrying btk mutations exhibited diminished anaphylactic reactions in response to IgE and antigen.
106                        Putative elicitors of anaphylactic reactions in the context of coronavirus dis
107 ontrast media, the major cause of iatrogenic anaphylactic reactions in the hospital, is explored.
108 s referred to our hospital owing to repeated anaphylactic reactions induced by exercise after meals.
109      Monitoring after complete resolution of anaphylactic reactions is recommended.
110 decrease in body temperature, reflecting the anaphylactic reaction, is substantially enhanced by the
111 with pathological conditions or allergic and anaphylactic reactions, it may contribute beneficially t
112                              She experienced anaphylactic reactions just after the inhalation of Inav
113 veloping anaphylaxis or failure to treat the anaphylactic reaction made up the majority of allegation
114                                              Anaphylactic reactions may occur among 1%-2% of botulinu
115 sis lesions, and clinical characteristics of anaphylactic reaction might be useful for differential d
116 ema (n=69, 20.9%), asthma (n=65, 19.7%), and anaphylactic reaction (n=19, 5.8%).
117 (NARA), total number of reports on suspected anaphylactic reactions, number of reactions where NMBAs
118                                              Anaphylactic reactions occurred in 6 of the 149 treated
119                               Grade 3 severe anaphylactic reactions occurred in seven cases of nine.
120 oducing Treg cells, without causing allergic/anaphylactic reactions or generalized immunosuppression.
121 dren with higher esIgD had decreased odds of anaphylactic reactions (OR =0.48, P = .038).
122  No serious complications such as blindness, anaphylactic reaction, or terminal disease transmission
123     We performed a review of PED records for anaphylactic reactions over 5 years.
124  regimens considerably increase allergic and anaphylactic reactions over avoidance or placebo, despit
125 eaction tryptase level) detected most of the anaphylactic reactions, particularly if baseline levels
126       The odds ratio for developing a severe anaphylactic reaction rather than only mild oral symptom
127 prophylaxis, including one episode of a mild anaphylactic reaction (representing <0.1% of all adminis
128                                         This anaphylactic reaction required a human CD32a transgene b
129 ltuximab (lower respiratory tract infection, anaphylactic reaction, sepsis).
130 s (0.6%) who received pemivibart experienced anaphylactic reactions (serious in 2).
131                    Serum samples from severe anaphylactic reactions show statistically significant lo
132    Moreover, this treatment eliminated fatal anaphylactic reactions that occurred after four to six e
133 phasic - and clinically important biphasic - anaphylactic reactions, the number of transfers to inten
134 ll deficient and protected from IgE-mediated anaphylactic reactions, their dramatically different res
135 e alleviates life-threatening symptoms of an anaphylactic reaction, there are currently no disease-mo
136 on of pork kidney proteins mediating delayed anaphylactic reactions through specific IgE to alpha-Gal
137 tridium cluster XIVa, protected mice from an anaphylactic reaction to a peanut challenge and reduced
138 activation of mast cells is a hallmark of an anaphylactic reaction to allergen.
139 E-dependent effector mechanisms, and a local anaphylactic reaction to an unrelated antigen can enhanc
140                                     Although anaphylactic reactions to blood products are rare, the i
141 nd distress commonly feature in survivors to anaphylactic reactions to drug.
142                                    (iii) The anaphylactic reactions to food derived from non-primate
143 ) is the major cause of fatal and near-fatal anaphylactic reactions to foods.
144 d to treat the few patients who present with anaphylactic reactions to Hymenoptera stings, as well as
145                                              Anaphylactic reactions to immunoglobulin infusions in im
146                      Among the most frequent anaphylactic reactions to insects are those attributed t
147                                              Anaphylactic reactions to neuromuscular blocking agents
148 as to evaluate mortality rate in France from anaphylactic reactions to NMBAs, to identify risk factor
149 f parenteral penicillins, and because severe anaphylactic reactions to oral amoxicillin are rare.
150 tructure and described as major elicitors of anaphylactic reactions to peanut (allergens Ara h 2 and
151 d by both immediate and delayed IgE-mediated anaphylactic reactions to the galactose-alpha-1,3-galact
152  eggs, or occurrence of Sampson Grade 3 to 5 anaphylactic reactions upon egg ingestion.
153 2)=0%, RD 5.7%, moderate-certainty), and non-anaphylactic reactions (vomiting: RR 1.79 [95%CI 1.35-2.
154 with the ingestion of only Citrus unshiu, an anaphylactic reaction was induced by additional acetyl-s
155 nses compared to free allergen in mice while anaphylactic reactions were essentially abolished.
156 ited Kingdom, Canada, and the United States, anaphylactic reactions were reported.
157 associated with a modestly increased risk of anaphylactic reaction when compared with DPP-4 inhibitor
158 lls in multiple tissues and displayed robust anaphylactic reactions when passively sensitized with pa
159  protein injection of immunized mice induced anaphylactic reactions, which were more severe in multip
160 8) generated a modest increase in the HR for anaphylactic reaction, with a wide 95% CI (36.9 per 100,
161                                      Such an anaphylactic reaction would also limit potential therape

 
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