ライフサイエンスコーパス: aneurysm

1 1.93 (95% CI 1.47-2.53) for abdominal aortic aneurysm.

2 14%) developed coronary artery dilatation or aneurysm.

3 ated with the occurrence of abdominal aortic aneurysm.

4 and testing of pathogenic models for aortic aneurysm.

5 oxycycline in blocking MFS-associated aortic aneurysm.

6 erse events, including aortic dissection and aneurysm.

7 duled for second-stage treatment of the LMCA aneurysm.

8 e 3 subarachnoid haemorrhage from a ruptured aneurysm.

9 no ischaemia and successful exclusion of the aneurysm.

10 ing or balloon-assisted coiling for a single aneurysm.

11 ing automated detection and segmentations of aneurysms.

12 y, shock, and development of coronary artery aneurysms.

13 h ruptured and 13.6% (n = 3) with unruptured aneurysms.

14 cm for men, 3.5-4.5 cm for women) infrarenal aneurysms.

15 r subject to pathological conditions such as aneurysms.

16 females and between ruptured and unruptured aneurysms.

17 nce of diastolic dysfunction and no coronary aneurysms.

18 She did not develop coronary aneurysms.

19 likely to undergo repair for isolated aortic aneurysms.

20 velopment and enlargement of thoracic aortic aneurysms.

21 orphological parameters with rupture of PCoA aneurysms.

22 larly with similar effects as unruptured MCA aneurysms.

23 educing the growth of small abdominal aortic aneurysms.

24 oquent areas, and the presence of associated aneurysms.

25 second most common location of intracerebral aneurysms.

26 between ruptured and unruptured intracranial aneurysms.

27 epilepsy, Alzheimer's disease, and cerebral aneurysms.

28 nterest for use in treatment of intracranial aneurysms.

29 tion between disturbed flow and intracranial aneurysms.

30 y on prophylactic operative repair of larger aneurysms.

31 ify individuals at risk for abdominal aortic aneurysms.

32 d in the aneurysmal wall of unruptured brain aneurysms.

33 atification in patients with unruptured PCoA aneurysms.

34 department in endovascular treatment of MCA aneurysms.

35 arterial wall of two unruptured intracranial aneurysms.

36 hy was carried out excluding coronary artery aneurysms.

37 o 978 prophylactic treatments for unruptured aneurysms, 19 procedure-related deaths, and 65 SAH-relat

38 test set consisting of 185 patients with 215 aneurysms (2010-2015).

39 Vascular sequelae included Rasmussen aneurysms (4%), enlarged bronchial arteries (3%), and sy

40 77.78% sensitivity), as were 6 of the 9 flow aneurysms (66.67% sensitivity).

41 , accessory auricular appendages (5), septal aneurysms (8), septal bags (6) and 1 thrombus in the lef

42 tant large iliac diameter (91%) and saccular aneurysm (82%).

43 the most upregulated MMP in abdominal aortic aneurysm (AAA) and, hence, MMP-12-targeted imaging may p

44 study their contribution to abdominal aortic aneurysm (AAA) formation and development, we determined

45 Abdominal aortic aneurysm (AAA) formation is characterized by inflammatio

46 Abdominal aortic aneurysm (AAA) growth after endovascular aneurysm repair

47 Abdominal aortic aneurysm (AAA) is a life-threatening vascular disease wi

48 Abdominal aortic aneurysm (AAA) is a prevalent life-threatening disease,

49 Abdominal aortic aneurysm (AAA) is a severe aortic disease with a high mo

50 Abdominal aortic aneurysm (AAA) is an important cause of cardiovascular m

51 the decision to operate on abdominal aortic aneurysm (AAA) is primarily on the basis of measurement

52 plays an important role in abdominal aortic aneurysm (AAA) pathogenesis, with effects on disease pro

53 Abdominal aortic aneurysm (AAA) remains a fatal disease.

54 enal aortic clamping during abdominal aortic aneurysm (AAA) repair results in ischemia-reperfusion in

55 ective surgical procedures [abdominal aortic aneurysm (AAA) repair, colectomy, coronary artery bypass

56 arteries in open juxtarenal abdominal aortic aneurysm (AAA) repair, the volume effect in these diffic

57 patients requiring elective abdominal aortic aneurysm (AAA) repair.

58 correlated with rupture of abdominal aortic aneurysm (AAA), an age-related vascular disease.

59 ity of periodontitis and of abdominal aortic aneurysm (AAA).

60 mmatory diseases, including abdominal aortic aneurysm (AAA).

61 s the treatment outcomes of abdominal aortic aneurysm (AAA).

62 ficant risk factor of human abdominal aortic aneurysm (AAA).

63 ometric features for future abdominal aortic aneurysms (AAA) growth prediction.

64 ases the risk of developing abdominal aortic aneurysms (AAA).

65 evices for the treatment of abdominal aortic aneurysms (AAAs) are being increasingly used worldwide.

66 Ruptured abdominal aortic aneurysms (AAAs) have mortality estimated at 81%.

67 treatment for asymptomatic abdominal aortic aneurysms (AAAs) with a diameter of at least 55 mm for m

68 Abdominal aortic aneurysms affect more than 3% of US older adults.

69 e- and sex-matched controls (1:10 for aortic aneurysm and 1:100 for aortic dissection) using the Dani

70 ars) had a first-degree relative with aortic aneurysm and 7209 persons (mean age, 39 years) had a fir

71 70 (95% CI, 5.96-7.52) for developing aortic aneurysm and a hazard ratio of 9.24 (95% CI, 5.53-15.44)

72 Increased aortic size, a precursor of aortic aneurysm and a risk factor for dissection, clusters in f

73 Ruptured aortic aneurysm and aortic dissections are potentially preventa

74 Sporadic aortic aneurysm and dissection (AAD), caused by progressive aor

75 rst-degree relatives of patients with aortic aneurysm and dissection had a hazard ratio of 6.70 (95%

76 pecific complications (for example, arterial aneurysm and dissection), integrated physical medicine a

77 d arteries of normal patients and those with aneurysm and dissection.

78 The incidence rates of aortic aneurysm and dissections approach the incidence rates of

79 For both aortic aneurysm and dissections, the absolute event rates appro

80 mice are viable and show protection against aneurysm and injury-induced neointimal hyperplasia, dise

81 sible long-term complications of GCA include aneurysm and stenosis of vessels, even in patients with

82 sk for cerebrovascular complications such as aneurysm and stroke.

83 ere more likely to have a hypogastric artery aneurysm and to undergo hypogastric coiling.

84 most common aortic diseases, namely, aortic aneurysms and acute aortic syndromes.

85 chment shows pleiotropy between intracranial aneurysms and antiepileptic and sex hormone drugs, provi

86 sful medical stabilization of growing aortic aneurysms and aortic root stabilization in Marfan syndro

87 licts young children, causes coronary artery aneurysms and can result in long-term cardiovascular seq

88 ed nations and can result in coronary artery aneurysms and death.

89 contributes to formation of ascending aortic aneurysms and dissections (AADs) induced by smooth muscl

90 (PRKG1, p.Arg177Gln) develop thoracic aortic aneurysms and dissections (TAAD) as young adults.

91 ein kinase 1 (PKG1) leads to thoracic aortic aneurysms and dissections.

92 efects in optic vasculature, which result in aneurysms and eye hemorrhages.

93 the genetic correlation between intracranial aneurysms and other cerebrovascular traits.

94 ifferences in prevalence of abdominal aortic aneurysms and showed more complex iliac anatomy in Asian

95 fected by geometric features of intracranial aneurysms and the surrounding vasculature in a location

96 rst-degree relatives versus 11 to 13 (aortic aneurysm) and 2 to 3 (aortic dissections) per 100 000 pe

97 proteinases during the progression of aortic aneurysm, and provide new insights into the potential th

98 ry edema on thoracic images, coronary artery aneurysms, and extensive right iliac fossa inflammatory

99 nts undergoing FEVAR were older, with larger aneurysms, and more comorbidities.

100 eatures such as aplastic arteries, stenosis, aneurysms, and vessel caliper for endovascular procedure

101 e been implicated in the pathology of aortic aneurysm, aortic dissection, and more recently, vascular

102 gical emergencies (ruptured abdominal aortic aneurysm, aortic dissection, appendicitis, perforated es

103 Aortic aneurysms are a common vascular disease in Western popul

104 Intracranial aneurysms are associated with disturbed velocity pattern

105 Aneurysms are common in the abdominal and thoracic regio

106 in size or morphology of UIAs since growing aneurysms are known to be at high risk for rupture.

107 al arteriovenous fistulas and distal/mycotic aneurysms are often managed with embolization if feasibl

108 ar dysfunction, coronary artery dilation and aneurysms, arrhythmia, and conduction abnormalities.

109 ptured posterior communicating artery (PCoA) aneurysms, as well as patient factors associated with th

110 at frequently presents with ascending aortic aneurysm (AscAA)(4).

111 nfidence intervals (CIs) of abdominal aortic aneurysm associated with physical activity.

112 ociated with enlargement of abdominal aortic aneurysms at 1 year, particularly in aneurysms smaller t

113 Ascending thoracic aortic aneurysm (ATAA) is caused by the progressive weakening a

114 13 and 2016 registered in the Dutch Surgical Aneurysm Audit (DSAA) were included.

115 registered, registered in the Dutch Surgical Aneurysm Audit (DSAA).

116 icipants treated for ruptured and unruptured aneurysms between December 2013 and May 2015.

117 ation devices to reduce the risk of cerebral aneurysm bleeding.

118 that were associated with basilar artery tip aneurysms (BTA) in a location-specific manner.

119 of complications, especially coronary artery aneurysms (CAA).

120 mong those who develop giant coronary artery aneurysms (CAA).

121 ous intravascular methods, treating cerebral aneurysms can be still a therapeutic challenge.

122 Participant demographics, aneurysm characteristics, and endovascular techniques we

123 participant and aneurysm risk factors after aneurysm coiling or balloon-assisted coiling within the

124 Conclusion During aneurysm coiling or balloon-assisted coiling, thromboemb

125 ent neurologic complications of intracranial aneurysm coiling.

126 rgo repair for aortoiliac and isolated iliac aneurysms compared to White and Hispanic patients who we

127 However, aneurysm detection proves to be challenging and time-con

128 Coronary artery aneurysms develop in some untreated children with Kawasa

129 ns between these different factors in aortic aneurysm development and identified a key role for smoot

130 aortic wall, and inflammation, resulting in aneurysm development.

131 PCoA aneurysms diagnosed at older age have morphological feat

132 cal parameters in 409 patients with 432 PCoA aneurysms diagnosed at the Brigham and Women's Hospital

133 The iliac aneurysm diameter was largest in Black and Asian patient

134 plasma biomarkers, systolic blood pressure, aneurysm diameter, and time to aortic rupture and death.

135 ularly deviate from the guidelines regarding aneurysm diameter, with variation between VSUs.

136 arily on the basis of measurement of maximal aneurysm diameter.

137 prolapse, collagen vascular disease, aortic aneurysm, Down syndrome, sleep apnea, depression, hyperl

138 ients with small infrarenal abdominal aortic aneurysms, doxycycline compared with placebo did not sig

139 TAs and a control group of 106 patients with aneurysms elsewhere to control for non-morphological fac

140 el including lumen volume and WSS to predict aneurysm enlargement was superior to maximal diameter al

141 sterolemic diet led to development of aortic aneurysms exhibiting all the features of human disease,

142 Patients with ruptured aneurysms experienced similar adjusted mortality whether

143 rameters that are unlikely to be affected by aneurysm formation could aid in risk stratification for

144 The regional propensity for aneurysm formation in MFS may be related to distinct smo

145 variants in the pathogenesis of intracranial-aneurysm formation is unknown.

146 ) lumen and the wall structure of the aortic aneurysm from CT angiograms (CTA) was compared against a

147 For abdominal aortic aneurysms, genetic associations have been identified, wh

148 ery of novel biomarkers for abdominal aortic aneurysm growth (AAA) prediction.

149 ed with placebo did not significantly reduce aneurysm growth at 2 years.

150 vascular permeability at different stages of aneurysm growth.

151 For aneurysms > 100 mm(3) (mean diameter of ~ 6 mm), a sensi

152 For aneurysms > 30 mm(3) (mean diameter of ~ 4 mm) on the te

153 e that the proposed DLM detects and segments aneurysms > 30 mm(3) in patients with aSAH with high sen

154 ysical activity and risk of abdominal aortic aneurysm has been inconsistent with some studies reporti

155 ion is involved in the development of aortic aneurysms has led to renewed investigations into the rol

156 Traditionally, thoracic aortic aneurysms have been labeled as a degenerative disease, c

157 vity may reduce the risk of abdominal aortic aneurysm, however, further studies are needed to clarify

158 95% CI: 0.62, 0.88), a 28% lower risk of an aneurysm (HR: 0.72; 95% CI: 0.59, 0.88), and a 47% lower

159 scular diseases such as hypertension, aortic aneurysm, hypercholesterolaemia, atherosclerosis, diabet

160 dy period; device; presence of atrial septal aneurysm, hypertension, hyperlipidemia, diabetes, hyperc

161 aorta have a high prevalence of intracranial aneurysms (IA) and suffer subarachnoid hemorrhage (SAH)

162 der that results in aortic root widening and aneurysm if unmanaged.

163 ent for vasospasm secondary to SAH due to an aneurysm, improving CTT and stenosis.

164 t a rare case of a giant treatment-resistant aneurysm in a 65-year-old woman.

165 to treat vasospasm related to SAH due to an aneurysm in four neurointerventional radiology departmen

166 Independent manual segmentations of aneurysms in a 3D voxel-wise manner by two readers (neur

167 at 19 of 24 AAA risk variants associate with aneurysms in at least 1 other vascular territory.

168 el (DLM) to automatically detect and segment aneurysms in patients with aSAH on computed tomography a

169 ed the association of AAA risk variants with aneurysms in the lower extremity, cerebral, and iliac ar

170 4,701 patients with 6,411 intracranial aneurysms, including 1201 prospective patients, who were

171 n with progressive, right-sided intracranial aneurysms, ipsilateral to an impressive cutaneous phenot

172 Intracranial aneurysm is a common life-threatening disease.

173 oid hemorrhage (aSAH), accurate diagnosis of aneurysm is essential for subsequent treatment to preven

174 ow-up in patients with detected intracranial aneurysm is the optimal strategy (cost, $19 839; utility

175 Endovascular treatment of MCA aneurysms is feasible, and our results are convergent wi

176 If annual rupture risk of nongrowing aneurysms is greater than 7.1%, coil placement should be

177 The etiology of aortic aneurysms is poorly understood, but it is associated wit

178 One patient developed a small aortic aneurysm late and has not required treatment.

179 Rupture of an intracranial aneurysm leads to subarachnoid hemorrhage, a severe type

180 downregulated in both human and mouse aortic aneurysm lesions.

181 Aneurysm location (anterior vs. posterior circulation; P

182 ion), 12 were women, 18 had ascending aortic aneurysms (maximal diameter > 4.0 cm), and 10 had bicusp

183 imary outcome was change in abdominal aortic aneurysm maximum transverse diameter measured from CT im

184 Ruptured MCA aneurysms may be treated endovascularly with similar eff

185 stically significant differences in terms of aneurysm morphology between males and females and betwee

186 Data on patients' comorbidities, aneurysm morphology, and treatment course were collected

187 Causes of haemoptysis were: Rasmussen aneurysms (n = 12/24), costocervical trunk pseudoaneurys

188 pseudoaneurysm (n = 1/24), left ventricular aneurysms (n = 3/24), pulmonary arteriovenous malformati

189 ular-repair group, and rupture of a thoracic aneurysm occurred in 1 patient (0.2%) in the open-repair

190 i disease (KD), with coronary dilatation and aneurysm occurring in some.

191 iography showed large (20 x 18 mm) wide neck aneurysm of the right internal carotid artery.

192 Ultrasound (US) examination depicted aneurysm of the right renal artery 6 x 6 cm, with signs

193 woman was diagnosed with three intracranial aneurysms of the right and left middle cerebral artery a

194 Sensitivity analyses with small (<= 3 mm) aneurysms only and with angles excluded, were also perfo

195 e primary treatment modality for most aortic aneurysms, open repair remains an essential treatment mo

196 rates and hazard ratios of developing aortic aneurysm or dissection among first-degree relatives of t

197 first-degree relatives of those with aortic aneurysm or dissection, in comparison with age- and sex-

198 , 1.00-1.37]; P=0.050), and abdominal aortic aneurysm (OR, 2.60 [95% CI, 1.15-5.89]; P=0.022) in the

199 .28; 95% CI: 1.23 to 1.32), abdominal aortic aneurysms (OR: 1.28; 95% CI: 1.20 to 1.37), and stroke (

200 after Endovascular Treatment of Intracranial Aneurysm, or ARETA, cohort.

201 drugs, providing insights into intracranial aneurysm pathophysiology.

202 From 545 eligible patients, 113 aneurysm patients were matched to 113 controls.

203 surements found increased oligomer levels in aneurysm patients with altered aortic wall integrity.

204 fy novel protein markers associated with MFS aneurysm phenotype.

205 This study showed that the presence of aneurysm plays an important role in the remodelling of t

206 ogether, these data suggest that aortic root aneurysm predisposition in this LDS mouse model depends

207 Renal artery aneurysms (RAAs) are a rare vascular pathology with an e

208 the management of descending thoracic aortic aneurysms, recent evidence has suggested that outcomes o

209 of these mAbs, from 3 patients with coronary aneurysms, recognize a specific peptide, which blocks bi

210 The presence of any type of aneurysm related with the AVM was detected in 13 of 15 c

211 s sensitive in detecting intranidal and flow aneurysms related with AVMs.

212 ge; it is also highly sensitive in detecting aneurysms related with AVMs.

213 Major aneurysm-related morphological abnormalities were reveal

214 ntil the end of the study or until any major aneurysm-related morphological abnormality requiring rei

215 urately identified 40 patients where a major aneurysm-related morphological abnormality was present (

216 Current management of aortic aneurysms relies exclusively on prophylactic operative r

217 A robust estimate of the frequency of aortic aneurysms remains to be determined as the majority of su

218 arization (19%-IVSR vs. 16%-VSF), and aortic aneurysm repair (13%-IVSR vs. 13%-VSF) procedures.

219 tic aneurysm (AAA) growth after endovascular aneurysm repair (EVAR) is still unpredictable.

220 surveillance of patients after endovascular aneurysm repair (EVAR), but there is currently no level

221 Endovascular aortic aneurysm repair (EVAR), left ventricular assist device (

222 were most likely to undergo isolated aortic aneurysm repair (W:76%, B:59%, A:68%, H:76%, P < 0.001).

223 rysm repair, or thoracic endovascular aortic aneurysm repair at 20 sites in North America.

224 nts with elective, ruptured, and symptomatic aneurysm repair demonstrated no differences in 30-day mo

225 ysm repair, and thoracic endovascular aortic aneurysm repair devices.

226 and 112 patients (90%) in the placebo group; aneurysm repair in 13 (10%) and 9 (7%), and death in 3 (

227 and Hispanic patients undergoing aortoiliac aneurysm repair in the VQI from 2003 to 2019.

228 ted) of the aorta and also if they underwent aneurysm repair or died.

229 d percutaneous endovascular abdominal aortic aneurysm repair or thoracic endovascular aortic aneurysm

230 llowing carotid, lower extremity, and aortic aneurysm repair procedures.

231 id, 21,428 lower extremity, and 5,800 aortic aneurysm repair procedures.

232 urysm repair or thoracic endovascular aortic aneurysm repair was performed in 53 (20.2%).

233 ary artery bypass grafting, abdominal aortic aneurysm repair, abdominal aortic aneurysm repair, total

234 , percutaneous endovascular abdominal aortic aneurysm repair, and thoracic endovascular aortic aneury

235 , percutaneous endovascular abdominal aortic aneurysm repair, or thoracic endovascular aortic aneurys

236 nal aortic aneurysm repair, abdominal aortic aneurysm repair, total hip arthroplasty, total knee arth

237 aracteristics, and outcomes after aortoiliac aneurysm repair.

238 er abdominal surgery for repair of an aortic aneurysm requiring multiple therapeutic paracenteses.

239 e endovascular repair of an abdominal aortic aneurysm results in lower perioperative mortality than t

240 tions, clinical outcome, and participant and aneurysm risk factors after aneurysm coiling or balloon-

241 actors, play important roles in intracranial aneurysm risk, and drive most of the genetic correlation

242 intensities of activity and abdominal aortic aneurysm risk.

243 Risk of intracranial aneurysm rupture could be affected by geometric features

244 Aneurysm rupture occurred in 7 patients (1.6%) in the en

245 feature has been applied to predict cerebral aneurysm rupture, but not examined as predictor of AAA g

246 needed to inhibit AAA expansion and prevent aneurysm rupture.

247 y and inversely associated with intracranial aneurysm rupture.

248 /20) and was associated with intraprocedural aneurysm rupture.

249 l mechanical properties of murine dissecting aneurysm samples by combining in vitro extension-distens

250 TFEB was measured in human and mouse aortic aneurysm samples.

251 and immunofluorescence on human intracranial aneurysms showed a correlation similar to the mouse vess

252 R, 2.2; 95% CI: 1.1, 4.7; P = .03) and small aneurysm size (OR, 3.0; 95% CI: 1.5, 6.3; P = .003).

253 actors for intraoperative rupture were small aneurysm size and anterior cerebral or communicating art

254 The aneurysm size ranged from 1.9 to 4.7 mm (mean 3.8, SD 0.

255 edict the risk of AAA rupture independent of aneurysm size.

256 aortic aneurysms at 1 year, particularly in aneurysms smaller than 50 mm in diameter.

257 s the risk of development of coronary artery aneurysms, some children have IVIG-resistant Kawasaki di

258 t years for treating wide-necked bifurcation aneurysms, such as balloon-assisted coiling, stent-assis

259 ment is necessary for a comprehensive aortic aneurysm surgery practice.

260 ng on evidence from thoraco-abdominal aortic aneurysm surgery.

261 an isolated, perifoveal, large intraretinal aneurysm surrounded by capillary rarefaction at OCT-A ex

262 Data on thoracic aortic aneurysms (TAA), type B aortic dissections (TBAD), and t

263 vary greatly depending on the location of an aneurysm, the principles remain the same.

264 m both iPSC SMCs and primary MFS aortic root aneurysm tissue confirmed elevated integrin alphaV and r

265 anslating iPSC findings into clinical aortic aneurysm tissue samples highlights the potential for iPS

266 nitiation and progression of thoracic aortic aneurysms to contrast key predisposing risk factors both

267 bral artery (MCA) secondary to SAH due to an aneurysm treated with endovascular angioplasty with a re

268 ent DLMs were trained on 68 patients with 79 aneurysms treated for aSAH (2016-2017) using five-fold-c

269 Forty patients with 41 MCA aneurysms treated in a single centre were included in th

270 Uncertainty in the outcome after aneurysm treatment and quality of life after SAH influen

271 eduction of artifacts following intracranial aneurysm treatment.

272 d risk for having an unruptured intracranial aneurysm (UIA) in women aged between 30 and 60 years and

273 Unruptured intracranial aneurysms (UIAs) are common incidental imaging findings,

274 echnical details of the management of aortic aneurysms vary greatly depending on the location of an a

275 were sorted into stable (<=10-mL increase in aneurysm volume) and progression (>10-mL increase in ane

276 volume) and progression (>10-mL increase in aneurysm volume) groups.

277 A:31%, H:22%, P < 0.001) and isolated iliac aneurysms (W:1.0%, B:3.1%, A:1.5%, H:1.6%, P < 0.001), a

278 Morphological parameters examined included aneurysm wall irregularity, presence of a daughter dome,

279 of albumin-binding-probe enhancement of the aneurysm wall to size of nonenhancing-thrombus-area pred

280 onths later the right middle cerebral artery aneurysm was embolised and the woman was scheduled for s

281 The aneurysm was first diagnosed due to visual disturbances

282 e again admitted with SAH - an enlarged LMCA aneurysm was observed and immediate third-stage embolisa

283 and the genetic architecture of intracranial aneurysms, we performed a cross-ancestry, genome-wide as

284 arotid dissection, and extra or intracranial aneurysm were reviewed retrospectively.

285 86.6% sensitivity); 7 of 9 of the intranidal aneurysms were detected (77.78% sensitivity), as were 6

286 for symptomatic repairs (20%), and ruptured aneurysms were evenly distributed.

287 Thirteen patients/aneurysms were included.

288 Unruptured aneurysms were significantly more frequently treated by

289 than were ruptured aneurysms, while ruptured aneurysms were treated more frequently by coiling alone

290 Cobinamide could be a treatment for aortic aneurysms where oxidative stress contributes to the dise

291 ow angles were associated with ruptured PCoA aneurysms, whereas perpendicular height was inversely as

292 ad to severe malformations, including aortic aneurysms, which are frequently associated with impaired

293 ortant role in the formation of intracranial aneurysms, which is conditioned by the geometry of the s

294 .4% vs. 5.6%, p = 0.0388) than were ruptured aneurysms, while ruptured aneurysms were treated more fr

295 ignificant in the subgroup analysis of small aneurysms (width <= 3 mm) and when angles were excluded.

296 h revealed active bleeding from the ruptured aneurysm with haematoma spreading into the right retrope

297 More advanced aneurysms with higher vascular permeability demonstrated

298 Irregular, multilobed PCoA aneurysms with larger height/width ratios and larger flo

299 Conclusion Screening for intracranial aneurysms with MR angiography in patients with autosomal

300 Coronary-artery aneurysms (z scores >=2.5) were documented in 15 patient