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1 ssels within the dermis, resembling a tufted angioma.
2 ation therapy is the development of sporadic angiomas.
3 erning, with no evidence of malformations or angiomas.
4 ents had multiple classic cerebral cavernous angiomas.
5 ite kindred with familial cerebral cavernous angioma and confirm the mapping to 7q11-22, including th
6 It lacks the "hemosiderin rim" of cavernous angioma and demonstrates increased susceptibility only o
9 Kaposiform hemangioendothelioma and tufted angioma are less common than IH but more often associate
15 n and spinal-cord hemangioblastomas, retinal angiomas, clear-cell renal carcinoma, neuroendocrine tum
19 lso used the technique to distinguish cherry angiomas from normal human skin by comparing the molecul
21 responsible for familial cerebral cavernous angioma in a large Hispanic kindred was mapped to human
22 aims to describe the behavior of a cavernous angioma in its natural history, documenting: a) its de n
27 phVEGF165-treated sites showed macroscopic angioma-like structures at the injection site while cont
29 ith those of infantile hemangioma and tufted angioma of children, but features of the clinical presen
30 epatic metastasis, one patient had a hepatic angioma, one patient had an extraadrenal pheochromocytom
31 sociated with upper eyelid or leptomeningeal angiomas, seizures, prior hemispherectomy, or pulsed-dye
32 er vascular tumors include congenital tufted angiomas (TAs), kaposiform hemangioendotheliomas (KHEs),
34 rient the etiological diagnosis of cavernous angioma versus other alternatives: de novo appearance, f