ライフサイエンスコーパス: ankylosis

1 hange in the joint space width, erosions and ankylosis.

2 ve defects, but without clinical evidence of ankylosis.

3 metaplasia, formation of syndesmophytes, and ankylosis.

4 induced apoptosis could play a role in tooth ankylosis.

5 tion, extra-articular fibroplasia, and joint ankylosis.

6 everity index, hindpaw swelling, and hindpaw ankylosis.

7 BMP-2 increase the development of transient ankylosis.

8 saw-tooth and gull-wing lesions and, rarely, ankylosis.

9 urfaces was often associated with a layer of ankylosis (44%, versus 8% of the ePTFE group).

10 cid conditioning promoted significantly more ankylosis (50%) compared with non-acid conditioning (6.3

11 .003), sclerosis (97% vs 81%, P < .001), and ankylosis (92% vs 84%, P = .04).

12 , 97] vs 20% [95% CI: 10, 35], P < .001] and ankylosis (93% [95% CI: 78, 98] vs 70% [95% CI: 47, 87],

13 were stained for identification of new bone, ankylosis and cementum formation.

14 cess of the procedure: complications such as ankylosis and root resorption up to the tooth exfoliatio

15 ion but also are implicated in causing tooth ankylosis and root resorption.

16 d cartilage degradation, bone erosion, joint ankylosis, and deformities in Tnfip6-null animals.

17 ial calcification due to deficiency of CD73, ankylosis, and progeria.

18 infraocclusion, overretained primary teeth, ankylosis, and/or slow orthodontic tooth movement, sugge

19 responsible for the phenotype of progressive ankylosis (ank) in the mouse.

20 Mutation at the mouse progressive ankylosis (ank) locus causes a generalized, progressive

21 syndesmophytes, parasyndesmophytes, and bone ankylosis, but instabilities are rare.

22 tion progressing to a state of near complete ankylosis by day 30.

23 riable micrognathia, temporomandibular joint ankylosis, cleft palate, and a characteristic "question-

24 However, BMPe developed more ankylosis compared with both CONe and CONf while BMPf wa

25 Ankylosis compromised regeneration in sites receiving PG

26 astic prosthesis is a reliable treatment for ankylosis, especially in recurrent cases.

27 ligament, and associated root resorption and ankylosis following surgical implantation of rhBMP-2 in

28 Mutations in the progressive ankylosis gene (Ank/ANKH) cause surprisingly different s

29 nt or recessive mutations in the progressive ankylosis gene ANKH have been linked to familial chondro

30 rs can be moved by orthodontic force without ankylosis, however, at a slower rate compared with those

31 rprisingly, results indicated the absence of ankylosis; however, a marked increase in cementum format

32 he genes involved in determining the rate of ankylosis in AS are likely to be different from those in

33 stic TMJ prostheses for the treatment of TMJ ankylosis in Chinese patients with severe mandibular def

34 Temporomandibular joint (TMJ) ankylosis is a refractory disease that is difficult to p

35 In JIA, early apophyseal bone ankylosis is characteristic, in addition to impaired spi

36 BMP-induced tooth ankylosis is controversial and is poorly understood.

37 l signs or symptoms, and long-segment spinal ankylosis is observed, thoracolumbar CT should be used f

38 The underlying mechanism of tooth ankylosis is unclear.

39 lex with or near the targeted regions of the ankylosis mRNA through UV-induced crosslinking of the an

40 Specific regions of the 3' and 5' UTRs of ankylosis mRNA were targeted by antisense PNAs transport

41 o the conventional method for the release of ankylosis of temporomandibular joint while providing a s

42 chnique using bur and saw for the release of ankylosis of temporomandibular joint.

43 o destruction of the articular cartilage and ankylosis of the joints.

44 in the depiction of erosions, sclerosis, and ankylosis of the SI joints compared with T1-weighted MRI

45 fects the enthesis and may culminate in bony ankylosis of the spine.

46 Ankylosis or root resorption were not observed.

47 Fifteen patients had sacroiliac disease (ankylosis or sclerosis) on computed tomography scans.

48 No evidence of root resorption, ankylosis, or untoward inflammation was seen.

49 chondral new bone formation leading to joint ankylosis over time.

50 ting the crucial role of inflammation in the ankylosis process.

51 meniscal cells, have elevated expression of ankylosis progressive analog (ANKH) and ectonucleotide p

52 Progressive ankylosis protein (ANK) and ectonucleotide pyrophosphata

53 for matrix-gla-protein (MGP), fetuin-A, and ankylosis protein (Ank) as well as alkaline phosphatase

54 fic alkaline phosphatase (TNAP), progressive ankylosis protein (ANK), and ectonucleotide pyrophosphat

55 Ankylosis protein (Ank), which transports intracellular

56 ey PP(i) regulators, genes ALPL, progressive ankylosis protein (ANKH), and ectonucleotide pyrophospha

57 atase/phosphodiesterase 1, CD73, progressive ankylosis protein, and lamin A/C proteins, respectively,

58 asia (CMD), mental retardation, deafness and ankylosis syndrome (MRDA).

59 bsequently found to have a congenital stapes ankylosis syndrome that included hyperopia, a hemicylind

60 n Tomb I displays a conspicuous case of knee ankylosis that is conclusive evidence of lameness.

61 No significant increase in tooth root ankylosis was found among the treatment groups when comp

62 One instance of root resorption and ankylosis was noted with EMD+CAF.

63 No significant new cementum formation or ankylosis was noted.

64 see if such mineral deposition and resulting ankylosis were occurring in the periodontium as well.

65 structural lesions (erosions, sclerosis, and ankylosis) were present on T1-weighted MRI, sCT, and CT

66 stly anchored to the jaw bones by pleurodont ankylosis, where the tooth is held in place on the labia

67 tions observed in these families with stapes ankylosis without symphalangism are predicted to disrupt