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1 y arterial hypertension (PAH) arose owing to anorexigens, acting as indirect serotinergic agonists, c
2                           Interestingly, the anorexigens also caused pulmonary arterial hypertension
3 e; or as a result of the use of fenfluramine anorexigens, amphetamines, or cocaine.
4 th newly diagnosed idiopathic, heritable, or anorexigen-associated PAH (n=80) at baseline and 3 to 4
5 550 patients with idiopathic, heritable, and anorexigen-associated PAH from eight cohorts that had be
6 ients with untreated PAH (8 idiopathic and 2 anorexigen-associated PAH) and 12 control subjects.
7 s of Patient 1 and the six of Patient 2 with anorexigen-associated PH exhibited a monoclonal expansio
8      For the current study, we asked whether anorexigen-associated PH is characterized by monoclonal
9 ls of plexiform lesions of two patients with anorexigen-associated PH.
10 tients (93% idiopathic, 5% heritable, and 2% anorexigen-associated pulmonary arterial hypertension) w
11                         Exclusion of the two anorexigen cases did not alter these results.
12    A melanocortin receptor agonist, a potent anorexigen, decreases AMPK activity in PVH, whereas agou
13 opathic/heritable PAH or PAH associated with anorexigens (HR: 0.99; 95% CI: 0.79-1.25); and 2) PAH as
14 combinant frog leptin (rxLeptin) is a potent anorexigen in frogs, as it is in mammals, but this respo
15 dent patients with idiopathic, heritable, or anorexigen-induced PAH enrolled in the French Pulmonary
16  activated by orexigens and inhibited by the anorexigen leptin.
17                                          The anorexigens may accelerate the growth of pulmonary endot
18                                              Anorexigens such as aminorex fumarate and dexfenfluramin
19                                              Anorexigens that modulate serotonin release and reuptake
20                                       Use of anorexigen therapy is associated with valvular abnormali
21 scular signs and symptoms were similar among anorexigen-treated and untreated subjects.
22 k (RR) of AR were significantly increased in anorexigen-treated patients and were 8.9% in the dexfenf
23 e, 4.0%; and untreated, 8.4%); and following anorexigen treatment were uncommon (dexfenfluramine, 2.3
24 mine group, while the untreated group had no anorexigen use during the previous 5 years.
25 , when adjusted for the median start date of anorexigen use).
26 the scleroderma spectrum of diseases or with anorexigen use.
27 sers, reminiscent of that induced by certain anorexigens used for weight reduction.
28                                              Anorexigens were shown to enhance hypoxic pulmonary vaso