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1 GLP-1R) expression in alpha-cells using both antibody-dependent and antibody-independent strategies.
6 activity, anti-CMV IgG, and NK cell-mediated antibody-dependent cell cytotoxicity were present in ale
7 expression and degranulation, and natural or antibody-dependent cell cytotoxicity, in comparison with
10 as prominent effector cells and induction of antibody-dependent cell phagocytosis as one of the prima
11 the immunologic synapse and potently induce antibody-dependent cell-mediated antiviral responses: (i
13 d with their parental forms, potent in vitro antibody-dependent cell-mediated cytotoxicity (0.1-0.3 m
14 ental insights into the relationship between antibody-dependent cell-mediated cytotoxicity (ADCC) and
15 histochemistry, for affinity by BIACORE, for antibody-dependent cell-mediated cytotoxicity (ADCC) and
17 Results from serum transfer experiments and antibody-dependent cell-mediated cytotoxicity (ADCC) ass
18 to ipilimumab by multicolor flow cytometry, antibody-dependent cell-mediated cytotoxicity (ADCC) ass
20 es that enhance tumor cell susceptibility to antibody-dependent cell-mediated cytotoxicity (ADCC) by
21 but their potential for cancer treatment via antibody-dependent cell-mediated cytotoxicity (ADCC) has
24 te Fc gama receptors (FcgammaRs) and mediate antibody-dependent cell-mediated cytotoxicity (ADCC) may
25 ogenic species of Ebola viruses and elicited antibody-dependent cell-mediated cytotoxicity (ADCC) res
26 the susceptibility of HIV-infected cells to antibody-dependent cell-mediated cytotoxicity (ADCC), an
27 n, endowed with increased ability to mediate antibody-dependent cell-mediated cytotoxicity (ADCC), du
28 effector functions of antibodies, including antibody-dependent cell-mediated cytotoxicity (ADCC), in
29 asure the killing of virus-infected cells by antibody-dependent cell-mediated cytotoxicity (ADCC), we
30 iller (NK) cells to kill tumor cells through antibody-dependent cell-mediated cytotoxicity (ADCC).
31 enhanced virus replication and resistance to antibody-dependent cell-mediated cytotoxicity (ADCC).
32 ic antibodies, arising in part from enhanced antibody-dependent cell-mediated cytotoxicity (ADCC).
33 ured by glycans that either promote or block antibody-dependent cell-mediated cytotoxicity (ADCC).
34 ability to eliminate virus-infected cells by antibody-dependent cell-mediated cytotoxicity (ADCC).
35 unctions of the innate immune system such as antibody-dependent cell-mediated cytotoxicity (ADCC).
36 their surface are preferentially targeted by antibody-dependent cell-mediated cytotoxicity (ADCC).
37 eutralization, binding to infected cells, or antibody-dependent cell-mediated cytotoxicity (ADCC).
38 Substantial effector functional activities (antibody-dependent cell-mediated cytotoxicity and antibo
39 (hYP7 and hYP9.1b) in the IgG format induced antibody-dependent cell-mediated cytotoxicity and comple
41 anti-neuraminidase antibodies weakly induced antibody-dependent cell-mediated cytotoxicity and enhanc
42 binding, neuraminidase inhibition, in vitro antibody-dependent cell-mediated cytotoxicity and in viv
45 feron gamma production, resulting in greater antibody-dependent cell-mediated cytotoxicity compared w
46 In addition, SEFL variants demonstrated no antibody-dependent cell-mediated cytotoxicity in vitro a
47 odies displayed cellular phagocytosis and/or antibody-dependent cell-mediated cytotoxicity in vitro O
48 outbreak variants of Ebola virus and mediate antibody-dependent cell-mediated cytotoxicity in vitro.
49 ying specificities regulate the magnitude of antibody-dependent cell-mediated cytotoxicity induction.
50 ing antibodies, neutralising antibodies, and antibody-dependent cell-mediated cytotoxicity measured 2
51 eptor affinity maturation and defects in the antibody-dependent cell-mediated cytotoxicity pathway.
53 ations in the MICA and MICB genes related to antibody-dependent cell-mediated cytotoxicity were ident
54 chanisms (complement-dependent cytotoxicity, antibody-dependent cell-mediated cytotoxicity, and antib
55 TF:FVIIa-dependent intracellular signaling, antibody-dependent cell-mediated cytotoxicity, and rapid
56 e activation of natural killer (NK) cells by antibody-dependent cell-mediated cytotoxicity, both RIFI
57 d optimized structure for the enhancement of antibody-dependent cell-mediated cytotoxicity, complemen
58 receptor alpha that depletes eosinophils by antibody-dependent cell-mediated cytotoxicity, for patie
59 ween milk or plasma neutralization activity, antibody-dependent cell-mediated cytotoxicity, or HIV-1
60 ticular the IgG1 and IgG3 subclass mediating antibody-dependent cell-mediated cytotoxicity, seem to p
61 and engagement of myeloid effector cells for antibody-dependent cell-mediated cytotoxicity, were simi
62 N9) vaccines elicited robust, cross-reactive antibody-dependent cell-mediated cytotoxicity-mediating
67 ent cellular cytotoxicity (ADCC), and modest antibody-dependent cell-mediated virus inhibition (ADCVI
68 ody-dependent cell-mediated cytotoxicity and antibody-dependent cell-mediated virus inhibition) were
69 l blood mononuclear cell assay, and moderate antibody-dependent, cell-mediated cytotoxicity activity
70 and fixation as well as Fc-gamma-dependent, antibody-dependent, cell-mediated cytotoxity in both mur
72 ecificity) and effector function activities (antibody dependent cellular phagocytosis, cellular cytot
73 the five neutralizing MAbs exhibited strong antibody-dependent cellular cytotoxicity (ADCC) activity
74 (Abs) to the V1V2 region of gp120 with high antibody-dependent cellular cytotoxicity (ADCC) activity
75 and that this protection may correlate with antibody-dependent cellular cytotoxicity (ADCC) activity
76 lobulin G1 antibody variant with compromised antibody-dependent cellular cytotoxicity (ADCC) activity
77 ust cross-clade binding and neutralizing and antibody-dependent cellular cytotoxicity (ADCC) activity
78 is V3 specificity correlated with measurable antibody-dependent cellular cytotoxicity (ADCC) activity
79 ngly, the presence of antibodies with potent antibody-dependent cellular cytotoxicity (ADCC) activity
80 bodies) show reduced FcgammaR engagement and antibody-dependent cellular cytotoxicity (ADCC) activity
81 n both complement-dependent cytotoxicity and antibody-dependent cellular cytotoxicity (ADCC) activity
83 ility to opsonize viral particles, to direct antibody-dependent cellular cytotoxicity (ADCC) against
84 b) directs natural killer (NK) cell-mediated antibody-dependent cellular cytotoxicity (ADCC) against
86 iminating latent HIV infection, specifically antibody-dependent cellular cytotoxicity (ADCC) and anti
87 d recycling, Fc effector activities, such as antibody-dependent cellular cytotoxicity (ADCC) and phag
88 These Treg suppressed cetuximab-mediated antibody-dependent cellular cytotoxicity (ADCC) and thei
90 s immunized by only the i.m. route had lower antibody-dependent cellular cytotoxicity (ADCC) antibody
91 s by competition with ACE2 but could involve antibody-dependent cellular cytotoxicity (ADCC) as IgG1
93 Natural killer (NK) immune cells mediate antibody-dependent cellular cytotoxicity (ADCC) by aggre
94 ycan fucosylation have been shown to improve antibody-dependent cellular cytotoxicity (ADCC) by allow
95 ible (CDi) neutralizing epitopes targeted by antibody-dependent cellular cytotoxicity (ADCC) effector
96 oss-reactive antibodies capable of mediating antibody-dependent cellular cytotoxicity (ADCC) effector
97 onses at an early time point correlated with antibody-dependent cellular cytotoxicity (ADCC) function
98 virus type 1 (HIV-1) Env and able to mediate antibody-dependent cellular cytotoxicity (ADCC) have bee
99 ng evidence supports a role for HIV-specific antibody-dependent cellular cytotoxicity (ADCC) in contr
100 we examined type-specific and cross-reactive antibody-dependent cellular cytotoxicity (ADCC) in HIV-1
101 to increase FcgammaRIII binding and improve antibody-dependent cellular cytotoxicity (ADCC) in vitro
106 Nonneutralizing antibodies (Abs) involved in antibody-dependent cellular cytotoxicity (ADCC) may prov
107 Treg depletion was mediated through the antibody-dependent cellular cytotoxicity (ADCC) mechanis
108 but not natural killer (NK) cells to induce antibody-dependent cellular cytotoxicity (ADCC) of alpha
110 levels of HIV-1-neutralizing antibodies and antibody-dependent cellular cytotoxicity (ADCC) response
111 itopes) as targets of potentially protective antibody-dependent cellular cytotoxicity (ADCC) response
113 cellular phagocytosis of HIV-1 virions, and antibody-dependent cellular cytotoxicity (ADCC) response
115 e HIV-1 reservoir.IMPORTANCE Mobilization of antibody-dependent cellular cytotoxicity (ADCC) to elimi
116 protein expressed by infected cells mobilize antibody-dependent cellular cytotoxicity (ADCC) to elimi
118 ating the factors that modulate HIV-specific antibody-dependent cellular cytotoxicity (ADCC) will hel
119 d more stem-specific antibodies, with higher antibody-dependent cellular cytotoxicity (ADCC), and bet
120 e breadth of HIV-1 gp120 and V1V2 responses, antibody-dependent cellular cytotoxicity (ADCC), and low
122 Very high IgG binding titers, substantial antibody-dependent cellular cytotoxicity (ADCC), and mod
123 functions, including tier 1 neutralization, antibody-dependent cellular cytotoxicity (ADCC), infecte
124 antibodies (MAbs) frequently mediate potent antibody-dependent cellular cytotoxicity (ADCC), making
125 cts on virus infectivity, antibodies mediate antibody-dependent cellular cytotoxicity (ADCC), the kil
126 ese epitopes and sensitize infected cells to antibody-dependent cellular cytotoxicity (ADCC), we trea
127 e, core fucosylation significantly decreases antibody-dependent cellular cytotoxicity (ADCC), whereas
129 ase of cell reinfection, and protection from antibody-dependent cellular cytotoxicity (ADCC), which i
130 Cross-reactive influenza virus-specific antibody-dependent cellular cytotoxicity (ADCC)-activati
131 ed that human natural killer (NK) cells, via antibody-dependent cellular cytotoxicity (ADCC)-like mec
132 elope glycoproteins is the primary target of antibody-dependent cellular cytotoxicity (ADCC)-mediatin
133 tion with either lineage induces HA-specific antibody-dependent cellular cytotoxicity (ADCC)-mediatin
151 sponses (characterized by increased in vitro antibody-dependent cellular cytotoxicity [ADCC] activity
152 infected cells by natural killer (NK) cells (antibody-dependent cellular cytotoxicity [ADCC]) or comp
153 g to FcgammaRIIIA and thereby decreasing the antibody-dependent cellular cytotoxicity activities.
155 An afucosylated Fc form (Pr20M) directed antibody-dependent cellular cytotoxicity against PRAME+H
156 fic CD16(pos) gammadelta T cells can perform antibody-dependent cellular cytotoxicity against stromal
157 ti-IL1RAP antibody capable of both achieving antibody-dependent cellular cytotoxicity and blocking of
158 pecific monoclonal antibodies display robust antibody-dependent cellular cytotoxicity and CD4-depende
160 tes killing of infected cells by Fc-mediated antibody-dependent cellular cytotoxicity and complement-
161 oinflammatory direct cell killing to promote antibody-dependent cellular cytotoxicity and complement-
162 f a CD20-targeting antibody had no impact on antibody-dependent cellular cytotoxicity and did not cha
164 odification in the Fc domain that eliminates antibody-dependent cellular cytotoxicity at clinically r
165 ivation of the classical complement pathway, antibody-dependent cellular cytotoxicity by innate immun
166 he asymmetrically engineered Fc variants for antibody-dependent cellular cytotoxicity enhancement cou
167 Our study highlights the potential role that antibody-dependent cellular cytotoxicity might play in a
168 mulation, by the use of antibodies to induce antibody-dependent cellular cytotoxicity or to block iKI
169 an HIV-1 Env vaccine increased C1C2-specific antibody-dependent cellular cytotoxicity potency and bre
170 NSCC sensitivity in a manner associated with antibody-dependent cellular cytotoxicity rather than EGF
171 levels of IgA antibodies, and high levels of antibody-dependent cellular cytotoxicity responses and H
173 In each study, protection correlated with antibody-dependent cellular cytotoxicity specific for CD
175 r not only induced gp140-specific IgG, ADCC (antibody-dependent cellular cytotoxicity) and some neutr
176 ed in N. benthamiana are capable of inducing antibody-dependent cellular cytotoxicity, an activity no
177 including complement-dependent cytotoxicity, antibody-dependent cellular cytotoxicity, antibody-depen
178 ts from a panel of exploratory immunoassays (antibody-dependent cellular cytotoxicity, CD4+ T-cell cy
180 2M activates natural killer cells to enhance antibody-dependent cellular cytotoxicity, mediates compl
181 d functional activity (virus neutralization, antibody-dependent cellular cytotoxicity, phagocytosis,
182 f natural killer cells, the key mediators of antibody-dependent cellular cytotoxicity, to human AMR i
183 IgG are potent effectors of complement- and antibody-dependent cellular cytotoxicity, which are crit
184 biting complement-dependent cytotoxicity and antibody-dependent cellular cytotoxicity, which suggests
185 ) arm that was derived from broadly binding, antibody-dependent cellular cytotoxicity-mediating antib
193 bs, readily induced by vaccines, can trigger antibody-dependent cellular effector functions, through
195 ed functional activities, including ADCC and antibody-dependent cellular phagocytosis (ADCP) activiti
197 ve antitumor antibody responses by enhancing antibody-dependent cellular phagocytosis (ADCP) in xenog
198 ecruitment of Fcgamma receptors (FcgammaRs), antibody-dependent cellular phagocytosis (ADCP), and the
199 xenografts by enhancing macrophage-mediated antibody-dependent cellular phagocytosis (ADCP), but syn
201 h a SIRPalpha-CD45 RIPR molecule potentiates antibody-dependent cellular phagocytosis beyond that of
202 wo FCGR2B SNPs influenced whether anti-gp140 antibody-dependent cellular phagocytosis correlated sign
203 ody levels and immunodominant specificities, antibody-dependent cellular phagocytosis of HIV-1 virion
204 6M0-mcMMAF recruits macrophages and mediates antibody-dependent cellular phagocytosis of MM cells.
205 dy-dependent cell-mediated cytotoxicity, and antibody-dependent cellular phagocytosis) and direct apo
206 ores, neutralizing antibody (nAb) responses, antibody-dependent cellular phagocytosis, CD4+ polyfunct
208 y, antibody-dependent cellular cytotoxicity, antibody-dependent cellular phagocytosis, programmed cel
209 to avidly capture HIV virions and to mediate antibody-dependent cellular phagocytosis, suggesting a r
211 D16, the Fc receptor (FcgammaIII) to mediate antibody-dependent cellular toxicity (ADCC), for a precl
212 their surface are preferentially targeted by antibody-dependent cellular-mediated cytotoxicity (ADCC)
213 I antibodies of various isotypes in ADCC and antibody-dependent cellular-phagocytosis (ADCP) assays.
215 ng capacity) and has increased resistance to antibody-dependent complement-mediated killing compared
216 ndings indicate that increased resistance to antibody-dependent complement-mediated killing secondary
217 ors of complement activation (RCA) to resist antibody-dependent complement-mediated lysis (ADCML).
219 ation activity limited to tier 1 viruses and antibody-dependent cytotoxicity responses (ADCC) after D
224 v antigen engagement which lead to effective antibody-dependent effector function directed to the non
225 s provide additional evidence that effective antibody-dependent effector function in the cluster A re
226 Might COVID-19 vaccines sensitize humans to antibody-dependent enhanced (ADE) breakthrough infection
228 ion showed significant capacity for in vitro antibody dependent enhancement of Dengue-1, 2, 3 and 4 s
229 Crosslinking of Fc with FcgammaRIIB mediates antibody-dependent enhancement (ADE) activity by MW05.
230 evere dengue disease focus on the process of antibody-dependent enhancement (ADE) as a primary risk f
237 protection against DENV disease and prevents antibody-dependent enhancement (ADE) of disease in mice.
239 rns for cross-reactive responses that induce antibody-dependent enhancement (ADE) of heterologous fla
241 cination and antiviral strategies.IMPORTANCE Antibody-dependent enhancement (ADE) of viral entry has
243 mmune serum, it has been shown in vitro that antibody-dependent enhancement (ADE) of ZIKV infection c
244 ble of both cross-neutralization, as well as antibody-dependent enhancement (ADE) of ZIKV infection.
245 cross-reaction to ZIKV and was able to drive antibody-dependent enhancement (ADE) of ZIKV infection.
248 e virus (DENV) infections is associated with antibody-dependent enhancement (ADE), and it was recentl
254 Progression to DHF/DSS is attributed to antibody-dependent enhancement (ADE); however, because o
255 tions in flavivirus immune vaccinees such as Antibody-Dependent Enhancement (ADE, a phenomenon involv
256 ovide statistical support for the process of antibody-dependent enhancement (but not original antigen
259 ines identified cellular immunopathology and antibody-dependent enhancement as potential safety issue
263 ZIKV co-circulate, understanding the role of antibody-dependent enhancement in the context of pregnan
270 ucleoside-modified mRNA vaccines do not show antibody-dependent enhancement of infection in vitro.
271 mRNA vaccines, including one that minimizes antibody-dependent enhancement of infection, elicited hi
276 st closely paralleled clinical outcomes, IgE antibody-dependent functional assays remained inhibited
277 lactose epitope bound by IgM that results in antibody-dependent killing via the classical pathway of
278 ly induced lymphoma Gal-1 expression ablated antibody-dependent lymphoma phagocytosis in vitro and ly
280 tor IV expression on macrophages, leading to antibody-dependent macrophage-mediated depletion of regu
284 ovide epidemiological evidence that multiple antibody-dependent mechanisms contribute to protective i
286 n a prespecified immune correlates analysis, antibody-dependent monocyte phagocytosis and antibody bi
288 of FcgammaR function dramatically inhibited antibody-dependent murine ITP and successfully circumven
290 ted functional antibody responses, including antibody-dependent neutrophil/monocyte phagocytosis, com
291 pact of NK cell differentiation phenotype on antibody-dependent NK cell activation, with highly diffe
294 to the production of class-switched anti-MOG antibodies, dependent on the presence of hemagglutinin-s
295 ed binding antibody against scaffolded V1V2, antibody-dependent phagocytic activity against VLP-coate
296 y-dependent cellular cytotoxicity (ADCC) and antibody-dependent phagocytosis (ADP), are unclear.
298 PPD-specific isotype/subclass, PPD-specific antibody-dependent phagocytosis, cellular cytotoxicity,