ライフサイエンスコーパス: antigen-specific

1 essor cell-mediated immunosuppression can be antigen specific.

2 antibodies they encode are approximately 70% antigen-specific 1-2 weeks after infection.

3 d transplant rejection, adoptive transfer of antigen-specific 3C-iTregs prevented the induction of ex

4 -specific GC B cells, Tfh cells, and overall antigen-specific Ab after immunization with sheep red bl

5 Overall, we show that antigen-specific activated and germinal centre B cells a

6 ging approach to investigate the response of antigen-specific, activated effector CD8(+) tumor-infilt

7 Here we show that antigen-specific activation of skin Trm cells leads to m

8 This is also known as antigen-specific adaptive tolerance induction for T1D (T

9 ression protocols are not designed to target antigen-specific alloimmunity and are uncapable of preve

10 ma producing memory CD8 T-cells comprised of antigen specific and bystander responses were detected b

11 21(neg) memory B cells, driven by continuous antigen-specific and bystander activation.

12 This MC unresponsiveness is antigen-specific and covers the serum drug concentration

13 1 + T cells in T2D was proved by recovery of antigen-specific and non-specific cytokine production vi

14 These results demonstrate that early antigen-specific and qualitative features of SARS-CoV-2-

15 cellular and molecular evidence of improved, antigen-specific, anti-tumor immune responses which also

16 for the same cohort (cutoff value, 40 mg of antigen-specific antibodies [mgA]/liter).

17 Serological testing to evaluate antigen-specific antibodies in plasma is generally perfo

18 The antigen-specific antibodies induced by TT3-nanoparticles

19 on of germinal centres and the production of antigen-specific antibodies were compromised.

20 We observed a significant increase in antigen-specific antibodies when we applied patches with

21 re assessed using lipopolysaccharide (LPS) O antigen-specific antibodies.

22 lly, and accurately determine the isotype of antigen-specific antibodies.

23 an substantially increase the elicitation of antigen-specific antibodies.

24 e, germinal center (GC) responses to control antigen-specific antibody and B cell memory.

25 er interferometry for the rapid detection of antigen-specific antibody levels in plasma samples, and

26 fferential glycosylation was observed across antigen-specific antibody populations.

27 sion in tissue in conjunction with increased antigen-specific antibody production.

28 ntibody correlate of HIV-1 risk, we examined antigen-specific antibody recruitment of Fcgamma recepto

29 ession of U90 translates into an increase in antigen-specific antibody response in iciHHV-6A/B(+) sub

30 ompared in vitro and their vaccine efficacy (antigen-specific antibody responses and IFN-gamma produc

31 the magnitude and affinity of neutralizing, antigen-specific antibody responses in mice immunized wi

32 route are able to induce serum antigen-specific antibody responses similar to those ind

33 ough the existing flu vaccines elicit strong antigen-specific antibody responses, they fail to provid

34 in a conformational manner for induction of antigen-specific antibody responses.

35 Engineered OMVs elicited high, saturating antigen-specific antibody titers when administered to mi

36 opportunities and challenges of translating antigen-specific approaches for autoimmunity with an emp

37 a-specific cells, suggesting a localized and antigen-specific ASC response.

38 a model for elucidating the roles of non-Dsg antigen-specific AuAbs in the physiological regulation o

39 Antigen-specific B and T lymphocytes were analyzed in an

40 ad in terms of serum antibody production and antigen-specific B and Tfh cell responses.

41 recision cellular immunotherapy approach for antigen-specific B cell depletion.

42 glycolipids which engage iNKT cells support antigen-specific B cell help during inflammasome-mediate

43 Animal models of human antigen-specific B cell receptors (BCRs) generally depen

44 relies on the productive interaction between antigen-specific B cells and T follicular helper (Tfh) c

45 ot spot (ELISpot)-based assay to measure CNS antigen-specific B cells in the blood of MS patients and

46 RARalpha overexpression in antigen-specific B cells promoted differentiation into p

47 inal center (GC) maturation and selection of antigen-specific B cells within secondary lymphoid organ

48 cell differentiation rather than a defect in antigen-specific B-cell tolerance.

49 Serological, antigen-specific B-cell, and interleukin 2-, interferon

50 The ability to isolate antigen-specific B-cells and rapidly solve structures of

51 stence.IMPORTANCE The prevalence and role of antigen-specific Bmem in the CNS during viral encephalom

52 Following secondary infection, antigen-specific BRM cells differentiated in situ, where

53 These results imply that most antigen-specific cCXCR5(+) T cells, including the CD38(-

54 reconstitution of Mycobacterium tuberculosis antigen-specific CD4 T cells in a cohort of HIV-infected

55 alent frequencies and functional profiles of antigen-specific CD4 T cells.

56 cinations at the lowest dose induced durable antigen-specific CD4 T-cell responses with acceptable sa

57 Restoration of the antigen-specific CD4 T-cell subsets mirrored the overall

58 Antigen-specific CD4(+) T cells against Chlamydia are cr

59 pinal cord, decreases integrin expression in antigen-specific CD4(+) T cells, increases the number of

60 TCR re-engagement of antigen-specific CD4(+)CD8alphaalpha(+) IELs by Listeria

61 and stimulate in vivo proliferation of both antigen-specific CD4+ and CD8+ T cells (cross-presentati

62 ft transplantation, depleting Lama5 promoted antigen-specific CD4+ T cell entry into the CR through H

63 rus Ankara (MVA) vector, improved priming of antigen-specific CD4+ T cell help.

64 00 cells/mm3 with >350 cells/mm3 after ART), antigen-specific CD4+ T cell memory to vaccinations/infe

65 n of NKG2D in T cells impairs the ability of antigen-specific CD4+ T cells to promote inflammation in

66 Furthermore, higher frequency of antigen-specific CD4+ T cells was detected in mice immun

67 nalyzed the activation and function of virus antigen-specific CD4+ T cells.

68 ieckmann et al. identified and characterized antigen-specific CD4+ T helper (Th) cells that developed

69 muL; >350/muL after antiretroviral therapy), antigen-specific CD4+ T-cell memory to vaccinations or i

70 We studied antigen-specific CD4+ Tfh cells responding to Plasmodium

71 of CD8+ T-cells in the lungs, percentage of antigen-specific CD4-T-cells in the spleen, and enhanced

72 ion of infection, we identified a persisting antigen-specific CD8 T cell population that was terminal

73 ole of vICA from either virus, in deflecting antigen-specific CD8 T cell-killing of infected cells.

74 Dhx37 knockout enhanced the efficacy of antigen-specific CD8 T cells against triple-negative bre

75 and mice is unusual because the majority of antigen-specific CD8 T cells are not class I restricted

76 They efficiently prime tumor antigen-specific CD8 T cells in vivo, induce CD8 T cell

77 Importantly, all these pDCexos primed naive antigen-specific CD8 T cells only in the presence of bys

78 can provide a context optimal for generating antigen-specific CD8 T cells, as they have natural tropi

79 esponses require the activity of both tumour-antigen-specific CD8(+) and CD4(+) T cells, even in tumo

80 We show that peripheral infections generate antigen-specific CD8(+) memory T cells in the brain that

81 mice, Ythdf1-deficient mice show an elevated antigen-specific CD8(+) T cell antitumour response.

82 kedly vary in their abilities to cross-prime antigen-specific CD8(+) T cells and raise subsequent ant

83 We next evaluated total and antigen-specific CD8(+) T cells in TG.

84 ater proliferation of adoptively transferred antigen-specific CD8(+) T cells in the skin draining lym

85 n-emitting isotopes, can noninvasively image antigen-specific CD8(+) T cells in vivo.

86 eptide-loaded MCF-7 breast cancer cells with antigen-specific CD8(+) T cells in which lytic granules

87 expression and cytotoxic capacity.IMPORTANCE Antigen-specific CD8(+) T cells play a major role in con

88 e identified discrete lineages of intestinal antigen-specific CD8(+) T cells, including a Blimp1(hi)I

89 t cross-presentation and activation of tumor antigen-specific CD8(+) T cells.

90 +) T cells, and a reduction in the number of antigen-specific CD8(+) T cells.

91 l responses, including diminished numbers of antigen-specific CD8(+) T cells.

92 s concurrent with the delayed development of antigen-specific CD8(+) T cells.IMPORTANCE We study the

93 It is thus possible to visualize antigen-specific CD8(+) T-cell populations in vivo, whic

94 The antigen-specific CD8(+) T-cell response was dependent on

95 HC-I expression on tumor cells and promoting antigen-specific CD8(+) T-cell responses to suppress HPV

96 he first time the evolvement of an exhausted antigen-specific CD8(+) TCR repertoire under checkpoint

97 tional DCs (cDCs) in generating potent tumor antigen-specific CD8+ T cell responses.

98 y is specifically induced in vaccine-induced antigen-specific CD8+ T cells in healthy human volunteer

99 Antigen-specific CD8+ T cells respond early during acute

100 on of both surrogate tumor antigen- and oHSV antigen-specific CD8+ T cells within 7 days after oHSV i

101 creased infiltration of both viral and tumor antigen-specific CD8+ T cells, as well as oHSV intratumo

102 lts in a rapid and robust expansion of rare, antigen-specific CD8+ T cells.

103 ing Tregs and increasing the infiltration of antigen-specific CD8+ T lymphocytes in TRAMP-C2-bearing

104 mphocyte and monocyte populations as well as antigen-specific CD8+ T-cell and B-cell responses from p

105 ass I-restricted TCR in PSCs produced naive, antigen-specific CD8alphabeta(+) T cells that lacked end

106 ss surface-displayed nanobodies that mediate antigen-specific cell-cell adhesion to effectively overc

107 erentially induces T(reg) differentiation of antigen-specific cells in these models to impact control

108 d memory-precursor-like TILs contained tumor-antigen-specific cells, exhibited proliferative and effe

109 of mice using these MNAs induces more potent antigen-specific cellular and humoral immune responses t

110 ion (EP) are better able to maintain durable antigen-specific cellular responses in the skin than mic

111 atients by flow cytometry and found a strong antigen specific central memory cell (CMC) response with

112 ed immune impairment; promoted long-lasting, antigen-specific central memory T cell responses; and ac

113 ution of skin vaccinia virus (VV) infection, antigen-specific circulating memory CD8(+) T cells migra

114 Antigen-specific cross-linking of IgE-loaded FceRI on Me

115 , such as cell viability, proliferation, and antigen-specific cytolytic activity.

116 ti-viral and anti-tumor immunity by inducing antigen-specific cytotoxic CD8(+) T-cell responses.

117 ted using this technique were able to induce antigen-specific cytotoxic effector T cell responses tha

118 ived) melanoma cell lines to differentiation antigen-specific cytotoxic T cells and observe strong en

119 DSC accumulation and increased activation of antigen-specific cytotoxic T lymphocytes.

120 First, ovalbumin (OVA) antigen-specific cytotoxic T-cells (CTLs) were incubated

121 CXCR6, CCR1, and CCR5, but CCR expression on antigen-specific effector and memory T cells has not bee

122 facilitate the priming events that generate antigen-specific effector and/or memory CD8(+) T-cell po

123 functional quality and survival of existing antigen-specific effector T cells.

124 ter polyclonal T-cell expansion and improved antigen-specific enrichment of rare T-cell subpopulation

125 eated humanized mice were analyzed using HEV antigen-specific enzyme-linked immunosorbent assay, reve

126 Antigen-specific ex vivo restimulation assays and in viv

127 Optical imaging showed antigen-specific fluorescent signal in PSCA-positive xen

128 cosal environment inhibited the induction of antigen-specific FoxP3 regulatory T cells and the preven

129 ransgenic Nur77-GFP reporter to distinguish "antigen-specific" from "bystander" reactivation, we demo

130 n from CD4(+) T cells reduced frequencies of antigen-specific GC B cells, Tfh cells, and overall anti

131 ment of long-lived plasma cells that secrete antigen-specific, high-affinity antibodies.

132 these sMVA vectors develop robust SARS-CoV-2 antigen-specific humoral and cellular immune responses,

133 vaccines, potentially able to elicit strong antigen-specific humoral and cellular-mediated immune re

134 We show robust antigen-specific humoral and T-cell responses following

135 hting the potential importance of functional antigen-specific humoral immunity to guide patient care

136 All animals exhibited antigen-specific humoral responses already after the pox

137 ENU)-induced germline mutations for aberrant antigen-specific IgE and IgG1 production in response to

138 h indicate control of cancer growth by tumor antigen-specific IgE that recruit and re-educate TAMs to

139 ing immediate (type I) reactions mediated by antigen-specific IgE.

140 sFceRI is produced after antigen-specific IgE/FceRI-mediated activation signals a

141 Plasma KL-6 (units/mL) and pigeon antigen-specific IgG antibody were quantified by enzyme

142 nsitivity and specificity compared to single-antigen-specific IgG determination.

143 -challenge as well as a high magnitude of an antigen-specific IgG response.

144 IgA responses in the GI tract, and enhanced antigen-specific IgG responses in vaginal washes.

145 ic glycan analysis was performed on bulk non-antigen-specific IgG, bulk Fc domain, bulk Fab domain, a

146 cytomegalovirus led to nearly 20-fold higher antigen-specific IgG2b levels relative to controls by we

147 Antigen-specific IgG3 subclass antibodies were quantifie

148 ected both T cell-dependent and -independent antigen-specific IgM-secreting cells into skin.

149 Analysis of antigen-specific immature B cells in early and late onto

150 emerging understanding that, in addition to antigen-specific immune cells, diverse haematopoietic, s

151 tion of this network in evoking a protective antigen-specific immune response in the brain remains un

152 dge is available on the relationship between antigen-specific immune responses and COVID-19 disease s

153 ver, mucosal vaccination often induces serum antigen-specific immune responses of lower magnitude tha

154 xpressing bacteria stimulates systemic tumor-antigen-specific immune responses that reduce the growth

155 T cells generate antigen-specific immune responses to their cognate antig

156 tains the necessary signals to promote tumor antigen-specific immune responses, priming T cells that

157 Although clinical cows expressed antigen-specific immune responses, the profile for subcl

158 h, and antibody production to induce primary antigen-specific immune responses.

159 are necessary for the durable suppression of antigen-specific immune responses.

160 ver, spleen, and brain and induced bacterial antigen-specific immune responses.

161 vels were generally similar and cryptococcal antigen-specific immune stimulation responses did not di

162 els were generally similar, and cryptococcal antigen-specific immune stimulation responses did not di

163 inducing FRalpha-loaded DCs is safe, induces antigen-specific immunity, and is associated with prolon

164 luorescence) imaging enables both presurgery antigen-specific immuno-PET for noninvasive whole-body e

165 ported without cold chain, while maintaining antigen-specific immunogenicity after sublingual vaccina

166 adicate large established melanomas, induced antigen-specific immunological memory and controlled tum

167 cell education, could be exploited to induce antigen-specific immunological tolerance.

168 cognition is also crucial for the pursuit of antigen-specific immunotherapies and implementation of s

169 Antigen-specific immunotherapy (ASI) has been proposed a

170 Thus, antigen-specific immunotherapy holds promise for strengt

171 )-peptide-MHC (pMHC) interaction is the only antigen-specific interaction during T lymphocyte activat

172 erates a highly expanded pool of functional, antigen-specific intestinal Trm, ultimately enhancing pr

173 side cells by involving appropriate pairs of antigen-specific intracellular antibodies.

174 chimeric antigen T cells are more potent in antigen-specific killing of human GBM cells.

175 ment of infection, elicited higher levels of antigen-specific long-lived plasma cells and memory B ce

176 aptive immune system is the proliferation of antigen-specific lymphocytes during an immune reaction t

177 PDAC patient could recognize and kill in an antigen-specific manner a majority of HLA-A*0101 allogen

178 T cells respond to threats in an antigen-specific manner using T cell receptors (TCRs) th

179 an protect heart transplant allografts in an antigen-specific manner.

180 T cells recognize and eliminate tumors in an antigen-specific manner.

181 tibody production by new naive B cells in an antigen-specific manner.

182 Conversely, invasive infection induced antigen-specific memory and protected against reinfectio

183 gainst inactive TcdB involves development of antigen-specific memory B cells and long-lived plasma ce

184 le cell gene expression analysis to evaluate antigen-specific memory B cells in peripheral blood of v

185 ccination under PD-L1 blockade could enhance antigen-specific memory CD4 T-cell responses.

186 CD4(+) T lymphocytes consist of naive, antigen-specific memory, and memory-phenotype (MP) cell

187 tive immunity has been hampered by a lack of antigen-specific models to interrogate the T cell respon

188 to serum IgA, NEI supplementation stimulated antigen-specific mucosal IgA responses in the GI tract,

189 d that the airway prime regimen induced more antigen-specific multifunctional CD4 and CD8 T cells to

190 le to elicit robust and sustained Th1-biased antigen-specific multifunctional CD4(+) T-cell responses

191 in mice would enable the study of endogenous antigen-specific naive T cell responses in disease and i

192 CAR macrophages (CAR-Ms) demonstrated antigen-specific phagocytosis and tumor clearance in vit

193 equency of germinal center (GC) B cells, and antigen-specific plasma cells induced during chronic IL-

194 sion in the liposomes results in an enhanced antigen-specific polyfunctional T cell response.

195 ll-intrinsic type I interferon signaling and antigen-specific priming.

196 T cell memory relies on the generation of antigen-specific progenitors with stem-like properties.

197 To address this issue, we followed the antigen-specific progeny of individual naive CD8+ T cell

198 L-21 did impact the ability of DCs to induce antigen-specific proliferation.

199 erebroventricularly or intravenously mediate antigen-specific protection from tumor rechallenge, both

200 We investigated features of H1N1 influenza antigen-specific pTfh (Ag.pTfh) in virologically control

201 T(EM) cells with innate and help-independent antigen-specific recall capacities.

202 oid cells (ILC) are lymphocytes that lack an antigen-specific receptor and are preferentially localiz

203 SpyCatcher immune receptor allows for rapid antigen-specific receptor assembly, multiantigen targeti

204 preferentially in neonates via induction of antigen-specific regulatory T cells (Tregs).

205 prevented T-cell-mediated diabetes, expanded antigen-specific regulatory T cells and resulted in last

206 GN, confirming the induction of therapeutic antigen-specific regulatory T cells.

207 ly increase both the size and breadth of the antigen-specific response while halving vaccine producti

208 Notably, coordination of SARS-CoV-2 antigen-specific responses was disrupted in individuals

209 on their somatic genetic diversification of antigen-specific responses.

210 iophage with a contractile tail, the multi-O-antigen-specific Salmonella myovirus Det7.

211 us (SLE) arise as a consequence of defective antigen-specific selection or a global enhancement of Ig

212 sublingual vaccines containing EdTx promoted antigen-specific serum IgA responses but also enhanced s

213 s was sufficient to limit infection and that antigen-specific STAT1 R274W CD8(+) T cell responses wer

214 ainage of CNS derived antigens that leads to antigen specific T cell proliferation in the draining ly

215 pathways is an effective way to induce self-antigen specific T cell tolerance to suppress ongoing au

216 cine to generate a large population of tumor antigen specific T cells but found that the presence of

217 asculitis, may enhance crescentic GN through antigen-specific T and B cell activation.

218 PD-1 expression is a hallmark of both early antigen-specific T cell activation and later chronic sti

219 FGL1 inhibits antigen-specific T cell activation, and ablation of FGL1

220 eign circRNAs are potent adjuvants to induce antigen-specific T cell activation, antibody production,

221 To study antigen-specific T cell clonality, we have developed a m

222 T cell-targeted immunotherapies that improve antigen-specific T cell expansion, T regulatory cell dep

223 ancer vaccines hold great promise to produce antigen-specific T cell immunity for personalized therap

224 tetramers to simultaneously detect multiple antigen-specific T cell populations, including from a mo

225 ntation is a common strategy for stimulating antigen-specific T cell populations.

226 ro, as well as on crescentic GN severity and antigen-specific T cell reactivity in the murine model o

227 and functionality, but its influence on the antigen-specific T cell receptor (TCR) repertoire is unk

228 T cells engineered to express antigen-specific T cell receptors (TCRs) are potent ther

229 emporospatially overlaps with the subsequent antigen-specific T cell response.

230 h functional and physical restoration of the antigen-specific T cell response.

231 ida, a STAT1 gain-of-function mutant impedes antigen-specific T cell responses against a common gamma

232 ough augmentation or inhibition of bacterial antigen-specific T cell responses does not alter the eff

233 uces immunoglobulin G1 (IgG1) antibodies and antigen-specific T cell responses in mice.

234 We demonstrate that Siglec-15 suppresses antigen-specific T cell responses in vitro and in vivo.

235 on of patients with epithelial cancers mount antigen-specific T cell responses to "hot spot" p53 muta

236 eased donor chimerism, T cell generation and antigen-specific T cell responses to vaccination.

237 ice and guinea pigs with INO-4800 we measure antigen-specific T cell responses, functional antibodies

238 ors, which demonstrates a high prevalence of antigen-specific T cells against both orthologs.

239 ith induction of neutralizing antibodies and antigen-specific T cells against the SARS-CoV-2 spike pr

240 ansion and tumor infiltration of transferred antigen-specific T cells and demonstrated that the combi

241 er cells could interact with a wide range of antigen-specific T cells and promote anti-cancer respons

242 Antigen-specific T cells are found at low frequencies in

243 lity to induce multifunctional and cytotoxic antigen-specific T cells de novo in samples from healthy

244 D as a general platform to detect endogenous antigen-specific T cells for studying their biology.

245 d as tools to study the impact of particular antigen-specific T cells in immune responses, and may ho

246 a form that enables antigen presentation to antigen-specific T cells in the local lymph nodes.

247 Paradoxically, antigen-specific T cells initially expressing more CD5 (

248 n vitro expansion of the low numbers of self-antigen-specific T cells of sufficient avidity to recogn

249 age malaria relies on a sufficient number of antigen-specific T cells reaching the liver during the t

250 Antigen-specific T cells that recognize endogenously pro

251 To enable detection of antigen-specific T cells with high sensitivity, we devel

252 rained innate mucosal immunity together with antigen-specific T cells, and also indicate that vaccine

253 Despite the importance of antigen-specific T cells, here we show that antigen non-

254 We further show that vaccine-activated antigen-specific T cells, particularly CD8 T cells, in t

255 In antigen-specific T cells, RARalpha strongly promoted the

256 platform, to biochemically label and capture antigen-specific T cells.

257 are a novel population of tissue-restricted antigen-specific T cells.

258 irected toward down-modulation of beta-islet antigen-specific T effector cells.

259 ntigen in atherosclerosis, and activation of antigen-specific T helper 1-type T cells is thought to f

260 ted EAE, which was associated with decreased antigen-specific T helper cell responses.

261 ytokines, serum antibodies, peripheral blood antigen-specific T lymphocytes, and gene expression in n

262 Here we found that antigen-specific T(reg) cells activated by dendritic cel

263 Antigen-specific T(reg) cells formed strong interactions

264 We isolated six nonredundant, antigen-specific T-cell clones, most of which reacting t

265 eptor-2 (VEGFR-2) and PD-1 or PD-L1 enhances antigen-specific T-cell migration, antitumour activity,

266 ucing a sufficient amount of IL-10 to dampen antigen-specific T-cell proliferation and pathogenic cyt

267 des, taken up by dendritic cells and inhibit antigen-specific T-cell proliferation.

268 of genetically engineered T cells expressing antigen-specific T-cell receptors (TCRs), is an appealin

269 nown to be associated with the modulation of antigen-specific T-cell responses.

270 n-situactivation dynamics of quiescent human antigen-specific T-cells interacting with dendritic cell

271 Significantly lower concentrations of antigen-specific T-helper 1 (IL-2, IL-12, interferon-gam

272 nd T-cell signaling domains via bifunctional antigen-specific targeting ligands, universal immune rec

273 These studies suggest that antigen-specific targeting of VAT-localized Treg cells c

274 activity is dispensable for the expansion of antigen-specific Tfh cells during vaccination.

275 on is characterized by the downmodulation of antigen-specific Th1 and Th17 responses and the upregula

276 f GPI-0100, indicating a potential mixed and antigen-specific Th1/Th2 immune response, which is diffe

277 timately, will form the foundation for novel antigen-specific therapeutics.

278 ansgenic lines support negative selection of antigen-specific thymocytes depending on antigen dose.

279 Still, the identification of suitable antigens specific to the tumour that can be targets for

280 Induction of effective antigen specific tolerance depends on concurrent engagem

281 OSD patients indicates that a key element in antigen specific tolerance is activated with this approa

282 a platform for the therapeutic induction of antigen-specific tolerance in autoimmune diseases.

283 himeric and translatable method for inducing antigen-specific tolerance in transplantation.

284 ntigens, and are able to induce systemic and antigen-specific tolerance leading to diabetes preventio

285 Ag GUCY2C to establish a model to study self-antigen-specific tolerance mechanisms.

286 ptotic donor splenocytes effectively induces antigen-specific tolerance to allografts in murine studi

287 crease their antigen presentation and induce antigen-specific tolerance, as indicated by CD4(+) and C

288 hR-activating nanoliposomes (NLPs) to induce antigen-specific tolerance.

289 ansplantation may establish a state of donor antigen-specific tolerance.

290 O peptide suppresses anti-MPO GN by inducing antigen-specific tolerance.

291 ociated with a regulatory network, involving antigen-specific Tr1 cells exhibiting a distinct transcr

292 In these mice, only the ubiquitous antigen-specific TR1 cells suppressed liver autoimmunity

293 Notably, strategies that support an antigen-specific Treg response may limit the immune-infl

294 o persist in the epidermis as efficiently as antigen-specific Trm cells in both contexts.

295 promotes the accumulation and persistence of antigen-specific Trm cells in the epidermal niche.

296 In addition, antigen-specific Trm cells that encountered cognate anti

297 ll clones were recruited into the epidermis, antigen-specific Trm cells were more efficiently retaine

298 in check ensures development of functional, antigen-specific "true" memory phenotype CD8+ T cells th

299 demonstrate that fetal MCs may contribute to antigen-specific vertical transmission of allergic disea

300 er reactions and humoral immune responses by antigen-specific wild-type B cells.