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1 therapy with corticosteroids and polyclonal antilymphocyte globulin.
2 patients who received alemtuzumab or rabbit antilymphocyte globulin.
3 on; CsA pigs (n= 14) received CsA (5 mg/kg), antilymphocyte globulin (10 mg/kg for 10 days), predniso
4 ding to the use of induction therapy: rabbit antilymphocyte globulin (5-year rate 6.8%, 4/59), alemtu
8 uction, old antibody era, 1987 to 1993, when antilymphocyte globulin and muromonab-CD3 were the major
9 antithymocyte globulin (rATG; 1999) replaced antilymphocyte globulin and muromonab-CD3, with maintena
10 onsisted of induction therapy with Minnesota antilymphocyte globulin/antithymocyte globulin/OKT3 in m
12 after treatment with corticosteroids equine antilymphocyte globulin (ATGAM), or the mouse anti-human
13 es in patients who received either Minnesota antilymphocyte globulin for 5 days or more or OKT3 for 7
14 ey transplants, graft failure and the use of antilymphocyte globulin for rejection are associated wit
16 mycophenolate mofetil, azathioprine, OKT-3, antilymphocyte globulin), only the use of antilymphocyte
17 us or cytomegalovirus status, induction with antilymphocyte globulin or antithymocyte globulin (ATG),
18 ents received 5-7 days of induction therapy (antilymphocyte globulin or antithymocyte globulin), wher
19 course of antithymocyte globulin, Minnesota antilymphocyte globulin, or OKT3 before developing PTLD.
20 d irradiation (TLI), rabbit antithymocyte or antilymphocyte globulin (RATG or RALG), and a single don
21 3, antilymphocyte globulin), only the use of antilymphocyte globulin when used to treat rejection was