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1 mpered by the genetic intractability of this anucleate cell.
2 ization in the regulatory repertoire of this anucleate cell.
3  mechanisms that control endocytosis in this anucleate cell.
4 tivation leading to increased frequencies of anucleate cells.
5 owth were impaired in viability and produced anucleate cells.
6 tions nor, obviously, transcription by these anucleate cells.
7 he temperature-sensitivity and production of anucleate cells.
8  to a 100-fold increase in the production of anucleate cells.
9 spoIIIE, led to an increase in production of anucleate cells.
10 utants are temperature-sensitive and produce anucleate cells.
11 segregation and divide frequently to produce anucleate cells.
12              Consequently, polynucleated and anucleated cells accumulated in these cultures.
13                          The chains included anucleate cells and cells with aberrantly dense or diffu
14                               Conversely, in anucleate cells, asters alone can support furrow inducti
15 ed on their surface antigens and the smaller anucleated cells based on their size.
16 hey are sometimes called cells, small cells, anucleated cells, cell fragments, or megakaryocyte fragm
17 ture by cellular force and tested platelets (anucleate cells), CHO-K1 cells (nucleated cells), and ot
18  segregation defects, including formation of anucleate cells, compact nucleoids confined to one half
19 y closer to the cell extremities, whereas in anucleated cells (deletion mutants for mukB), the Tsr cl
20 g aberrant nuclear division, as well as many anucleate cells, demonstrating that the TRF4/5 function
21               This increase in production of anucleate cells depended on recA, indicating that there
22      There is also an enhanced population of anucleated cells, each containing a single kinetoplast k
23              Moreover, MksBEF can complement anucleate cell formation in SMC-deficient cells.
24 s, nor did it suppress the high frequency of anucleate cell formation.
25 l chromosome segregation, and an increase in anucleated cell formation.
26                          Blood platelets are anucleate cell fragments that play a critically importan
27                            Platelets (small, anucleate cell fragments) derive from large precursor ce
28               Blood platelets are functional anucleate cells generated by specialized fragmentation o
29 ther examination of FtsZ ring positioning in anucleate cells generated by the parC and mukB mutants:
30 mutation dramatically enhanced production of anucleate cells in an smc null mutant.
31 in the production of approximately 0.9 to 3% anucleate cells in prfA cultures grown at 30 or 37 degre
32 epithelial cells histologically that contain anucleated cells in the center surrounded by basophilic
33 e ion channels are functional in these tiny, anucleate cells is difficult to assess by direct electro
34 duces proinflammatory cytokine production in anucleate cells lacking NF-kappaB is unknown.
35 ogressively lose organelles and convert into anucleate cells or corneocytes.
36  they suppressed temperature sensitivity and anucleate cell production of cells containing null or po
37 ricted to low temperature with production of anucleate cells, reflecting chromosome segregation defec
38           This, along with its expression in anucleate cells, suggests that Bcl-3 has previously unre
39                                Platelets are anucleated cells that circulate in the bloodstream.
40                            Although they are anucleated cells that lack a complex secretory apparatus
41 also confirm that this is a feature of other anucleate cells through transcriptome sequencing of matu
42 s in protein oxidation have been reported in anucleate cells, where no transcription occurs.
43                    Human blood platelets are anucleate cells whose response to extracellular stimuli
44 cytokinesis to produce one nucleated and one anucleate cell (zoid).
45                  A significant population of anucleate cells (zoids), apparently derived from kinetop