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1 for chromatin remodeling possibly leading to anxiolysis.
2 underlying the mechanisms of ethanol-induced anxiolysis.
3 EF2 activation and protein synthesis for its anxiolysis.
4 omotion, and seizure inhibition, but not for anxiolysis.
5 ehavior as well as locomotor stimulation and anxiolysis.
6 ause of negative contrast or novelty-induced anxiolysis.
7 , expensive, and do not consistently provide anxiolysis.
8  role for 5-HT(4) receptors in cognition and anxiolysis.
9 nt differences in the quality of sedation or anxiolysis.
10 eA(AT2R) couples lowered blood pressure with anxiolysis.
11 scillation (30-80 Hz) frequency and promoted anxiolysis.
12  with hypothalamic Crh expression results in anxiolysis.
13 eased in the amygdala during ethanol-induced anxiolysis.
14 s of drug for similar levels of sedation and anxiolysis (14.4 +/- 1.2 mg/8 hrs vs. 1.6 +/- 0.1 mg/8 h
15  of NPS in the basolateral amygdala promotes anxiolysis after chronic ethanol consumption, thereby pr
16 ate that cannabinoid receptors mediate acute anxiolysis and analgesia after running.
17 ist widely used for premedication, sedation, anxiolysis and analgesia.
18 clear complex of the amygdala (BLA) produces anxiolysis and interferes with the generation of conditi
19 al role of treatment expectations in placebo anxiolysis and provide insight into the underlying neura
20                  Unexpectedly, LH-21 induced anxiolysis and reverted obesity-induced anxiety, apparen
21 -2 adrenergic receptor agonist that produces anxiolysis and sleep-like sedation without narcosis or r
22 e mPFC to LC pathway in female mice produced anxiolysis and spatial memory deficits.
23 sant agents, including motor incoordination, anxiolysis, and the elicitation of signs of physical dep
24 nt, the neural mechanisms underlying placebo anxiolysis are poorly understood.
25 cebo treatments on fear and anxiety (placebo anxiolysis) are well known from clinical practice, and t
26 ns seem essential for neuropeptide S-induced anxiolysis, as this effect was blocked by pharmacologica
27 e treatment along with verbal suggestions of anxiolysis can attenuate experimentally induced states o
28                                    sedation; anxiolysis; critical care; midazolam; lorazepam; propofo
29                                 We show that anxiolysis depends on intact cannabinoid receptor 1 (CB1
30 fects of NPY, including vasoconstriction and anxiolysis in animal models.
31   Compound 23 induced significantly stronger anxiolysis in mice than piperine and thus may serve as a
32 anol at concentrations relevant to abuse and anxiolysis, in a cAMP-dependent and ERK-dependent manner
33 cleus of the hypothalamus (PVN), OXT-induced anxiolysis is mediated, at least in part, via activation
34 e contribution of GABAA receptor subtypes to anxiolysis is still controversial.
35                              Ethanol-induced anxiolysis may contribute to alcohol consumption, while
36 h infusion of a specific antagomir, provoked anxiolysis, mimicking the action of ethanol.
37  very anxious patient, possibly secondary to anxiolysis or direct effect on respiratory drive.
38 ced temporal plastic changes in EtOH-induced anxiolysis or withdrawal anxiety, and the presence or ab
39 a-each implemented an independent feature of anxiolysis: reduced risk-avoidance, reduced respiratory
40 cal activities, such as vasoconstriction and anxiolysis, remains to be investigated, adequate tools a
41 gh as a sudden pleasant feeling of euphoria, anxiolysis, sedation, and analgesia.
42 expression of the social familiarity-induced anxiolysis (SFiA) appears dependent on the prefrontal co
43 ytocin (OXT) mediates its actions, including anxiolysis, via its G protein-coupled OXT receptor.
44                        Acute ethanol-induced anxiolysis was measured in adult rats, and amygdaloid ti
45 ress and be most effective in the context of anxiolysis when administered to the dominant nostril.
46                                 Sedation and anxiolysis with lorazepam and midazolam in critically il
47 2-GABAARs) are required for diazepam-induced anxiolysis, with no evidence for an involvement of any o
48 overed to reliably evoke positive affect and anxiolysis without sedation in a patient with epilepsy u
49 g GABAA receptors have been shown to provide anxiolysis without sedation in preclinical models, where
50 e a greater ability to achieve analgesia and anxiolysis without some of the adverse concomitant effec