ライフサイエンスコーパス: aorta

1 ing thoracic aorta) and zone III (infrarenal aorta).

2 input function was image-derived (abdominal aorta).

3 n decreases miR-204 in endothelial cells and aorta.

4 erated expansile remodeling of the ascending aorta.

5 demonstrated in an in vivo test in a rabbit aorta.

6 estimation (<12%) in the arch and descending aorta.

7 es occurs at spatially-distinct areas of the aorta.

8 y normalizing the density of iodine with the aorta.

9 with prosthetic replacement of the ascending aorta.

10 ecreased endothelium-dependent relaxation of aorta.

11 hat can involve virtually any portion of the aorta.

12 he graft to the thoracic or thoracoabdominal aorta.

13 ld or no reduction in plaque coverage in the aorta.

14 nd maintaining the molecular identity of the aorta.

15 ecialized endothelial cells of the embryonic aorta.

16 gitation, and uncorrected coarctation of the aorta.

17 ue structure and biology in rabbit abdominal aorta.

18 e of [(99m)Tc]-1 in AAA relative to adjacent aorta.

19 ntrast attenuation for CT angiography of the aorta.

20 terogeneity in EC populations throughout the aorta.

21 o promote their accumulation in the diseased aorta.

22 1-deficient vascular bed, the lateral dorsal aorta.

23 ensive single-cell atlas of all cells in the aorta.

24 ed catheter into either the vena cava or the aorta.

25 cle phase as assessed in wounds, tumors, and aorta.

26 ithin the ascending, thoracic, and abdominal aorta.

27 on that predominantly affects the infrarenal aorta.

28 dynamics in patients with coarctation of the aorta.

29 d), Foxp3 (1.4 folds) and TGF-beta (1.62) in aorta.

30 minantly in the ventral domain of the dorsal aorta.

31 s that predispose to disease of the thoracic aorta.

32 egepant on CGRP-induced relaxations in mouse aorta.

33 ventricle, associated valves, and ascending aorta.

34 d Treg content in mesenteric lymph nodes and aorta.

35 present in both healthy and atherosclerotic aortas.

36 categories that were altered in SR-uPA(+/0) aortas.

37 IE-2 and VEGFR2 in atherosclerotic ApoE(-/-) aortas.

38 ) mice have 4 groups of macrophages in their aortas.

39 e propensity on geometry of patient-specific aortas.

40 e immune cell populations in atherosclerotic aortas.

41 and functional category enrichment in these aortas.

42 ins were reproducibly altered in SR-uPA(+/0) aortas.

43 ealed fewer proliferating macrophages in the aorta (15.6+/-9.79 %) compared with untreated mice (30.2

44 48.2% of individuals (24.4% femorals, 19.3% aorta, 15.8% carotids, and 9.3% iliacs) and plaques in 9

45 I, 77-123]; P<0.001) but not in the thoracic aorta (23% [95% CI, 16-30] versus 28% [95% CI, 19-38]; P

46 creased pulse wave velocity in the ascending aorta (3.4 versus 2.3 m/s for PAH and controls, respecti

47 ssessed by en face Oil Red O staining of the aorta (4.7+/-2.9% versus 1.9+/-1.3%; P<0.01) and cross-s

48 n computed tomography scan (63%), the coated aorta (40%), and the right atrium pseudo-tumoral infiltr

49 It was highest at the ascending aorta (AA) with COVs of 3.6% for intraobserver variabili

50 Flow was measured in the ascending aorta (AAo) and main pulmonary artery (MPA).

51 Flow measurements were made in the ascending aorta (aAo), mid-descending aorta, main pulmonary artery

52 and functional phenotypes for the heart and aorta across 26,893 participants, and explore how these

53 The proximal aorta acts as a coupling device between heart and brain

54 pressing macrophage proliferation within the aorta after a short dosing schedule (7 days) and with a

55 METHODS AND LD were present in EC lining the aorta after the peak in plasma triglycerides initiated b

56 sverse diameter (measured or imputed) of the aorta and also if they underwent aneurysm repair or died

57 eloped more atherosclerotic lesions in whole aorta and aortic root area, with markedly increased SRA

58 scular contrast enhancement in the abdominal aorta and brachiocephalic artery was quantified by measu

59 that aggregates of Medin also develop in the aorta and brain vasculature of wild-type mice in an age-

60 in the abdominal and thoracic regions of the aorta and can cause death due to dissection or rupture.

61 ing molecular-phenotypic data from the mouse aorta and colon, we find that interpretation of decompos

62 e were found to have more severe AMC in both aorta and coronary arteries compared to their littermate

63 e of systemic vascular disease that included aorta and coronary artery atheroma, cardiac hypertensive

64 sociated with calcification of the abdominal aorta and describes a previously unknown role for HDAC9

65 D) is the most common acute condition of the aorta and has a high mortality.

66 nhancer revealed decreased SRF expression in aorta and heart tissues.

67 imation of the circular cross-section of the aorta and identify the implications of this approximatio

68 apoptosis and oxidative stress in Apln (-/y) aorta and in APLN-deficient cultured murine and human ao

69 The donor bladder with abdominal aorta and inferior vena cava was isolated and orthotopic

70 competing vascular trajectories (the dorsal aorta and intercostal arteries).

71 (GCA) causes autoimmune inflammation of the aorta and its large branches, resulting in aortic arch s

72 cluding trauma, but stent-grafts stiffen the aorta and likely increase LV afterload.

73 L-NAME or BH(4) decreased ROS production in aorta and mesenteric arteries supernatant's of both SHR

74 ion, yet despite its prevalence in the human aorta and multiple arterial beds, little is known about

75 ressure loading, IL11 was upregulated in the aorta and neutralizing IL11 antibodies reduced remodelin

76 s that simultaneously occlude the descending aorta and open the aortic arch vessels to atmosphere.

77 have an increased capacity to migrate to the aorta and periaortic lymph nodes compared to Nrp1(-) T c

78 significantly increased calcification in the aorta and peripheral arteries.

79 Phase contrast sequences in the aorta and pulmonary artery showed systemic output of 20

80 d radiofrequency delivery at the root of the aorta and pulmonary artery.

81 rteries (0.15 L/min +/- 0.10) and descending aorta and superior vena cava (0.14 L/min +/- 0.12).

82 +) NK cells were expanded in atherosclerotic aorta and that CD90(+) NK cells produce more IFN-gamma t

83 ula is an abnormal communication between the aorta and the gastrointestinal tract wall.

84 available open chromatin data for the heart, aorta and tibial arteries, and multiple kidney cell type

85 We identified an enrichment of these loci in aorta and tibial artery expression quantitative trait lo

86 26% and 12%, respectively) in the ascending aorta and underestimation (<12%) in the arch and descend

87 reduced the degradation of pyrophosphate in aortas and blood ex vivo, suggesting that these combined

88 hosphate -> phosphate) was increased in both aortas and blood obtained from mice with HGPS.

89 ayed an increased expression of Rap1 both in aortas and circulating lEVs.

90 ver time, eventually resulting in aneurysmal aortas and ectatic esophagi.

91 ted pharmacologic investigations on isolated aortas and generated stable and inducible HEK293 cell li

92 Parallel studies of SR-uPA(+/0) mouse aortas and human plaques identify mechanisms that connec

93 pain-1 (CAPN-1) were significantly higher in aortas and human umbilical vein endothelial cells (HUVEC

94 to a marked reduction in lipid deposition in aortas and isolated macrophages.

95 ajor histological alterations, whereas their aortas and kidneys revealed typical age-related changes

96 tigate caveolae integrity and density in SHR aortas and mesenteric arteries and the role played by ca

97 presence of caveolae-like structures in SHR aortas and mesenteric arteries.

98 linked with PCSK6 mRNA expression in healthy aortas and plaques but also with overall plaque SMA+ cel

99 ne proteins were reduced in both SR-uPA(+/0) aortas and ruptured human plaques.

100 lagens were minimally altered in SR-uPA(+/0) aortas and ruptured human plaques; however, several base

101 Aortas and tail arteries were isolated from young (3-4 m

102 pubis to aortic zone I (descending thoracic aorta) and zone III (infrarenal aorta).

103 Changes in coronary artery, thoracic aorta, and cardiac valve calcium scores and pulse wave v

104 nsient hematopoiesis in the yolk sac, dorsal aorta, and developing heart tube function at their sites

105 pro-inflammatory signature, localized to the aorta, and dynamically interacted with nascent and emerg

106 tomical sites including the yolk sac, dorsal aorta, and heart tube.

107 w that db/db mice overexpress miR-204 in the aorta, and its absence rescues endothelial dysfunction a

108 raft were valve surgery, surgery on thoracic aorta, and other cardiac surgery.

109 trol group in various tissues, including the aorta, and this expression correlated strongly with (99m

110 tegies for major complications involving the aorta, aortic valve annulus, and left ventricle.

111 nly the physiologically loaded states of the aorta are given from in vivo clinical images, inverse ap

112 stent was implanted in the dorsal abdominal aorta as a landmark, followed by the 7 French imaging ca

113 nd impairs mitochondrial function within the aorta, associated with enhanced mitophagy and increased

114 ciency also led to a significant increase in aorta-associated pMos and protected Ldlr(-/-) mice from

115 essed at the ascending (Ao-A) and descending aorta at the pulmonary artery bifurcation (Ao-P) and dia

116 meters and lengths of the ascending thoracic aorta (ATA).

117 m male murine hearts subjected to transverse aorta banding surgery.

118 nformation about blood flow in the ascending aorta, based on maximum values at systole at a single lo

119 rentiating ccRCC from other RCC subtypes are aorta-based corrected AV and aorta-based corrected relat

120 CC subtypes are aorta-based corrected AV and aorta-based corrected relative contrast enhancement valu

121 n atherosclerosis-prone regions of the mouse aorta before lesion formation, and in human atherosclero

122 trast opacification in CT angiography of the aorta between a cohort with fixed trigger delay and a co

123 na cava inferior confluence (CVC), abdominal aorta bifurcation (AB), and iliolumbar ligament were eva

124 ven without equal efficiency in all tissues (aorta, brain and kidney), resulted in rapid lethality ma

125 ted in neointimal formation in the ascending aorta but not in muscular arteries.

126 odulates the levels of MD2 expression in the aorta, but not TLR4, at least in the conditions evaluate

127 This effect was observed ex vivo on isolated aortas, but also in vivo on femoral arteries.

128 cutaneous transcaval access to the abdominal aorta by electrifying a caval guidewire and advancing it

129 ed the effects of antibody deficiency on the aorta by transcriptomics.

130 CAC and thoracic aorta calcification (TAC) were quantified using a convol

131 induced exuberant growth and dilation of the aorta, calcification and ossification of the aortic wall

132 Cardiac and thoracic aorta calcium scores and pulse wave velocity were measur

133 ritability of fluid metrics in the ascending aorta calculated using patient-specific data from cardia

134 The medial layer of the aorta can also be subject to abnormalities (such as Marf

135 The aorta can be affected by a wide spectrum of acute factor

136 nal vascular ultrasound (2DVUS) of abdominal aorta, carotid, iliac, and femoral territories to determ

137 aortic stenosis (SVAS) is a narrowing of the aorta caused by elastin (ELN) haploinsufficiency.

138 lly derived radius along the entire thoracic aorta centerline.

139 hes to the proximal and distal extent of the aorta, cerebral perfusion methods, and cannulation strat

140 on angioplasty for native coarctation of the aorta (CoA) is successful in children and adults but in

141 Coarctation of aorta (COA) results in chronic left ventricular (LV) pre

142 tion develop in patients with coarctation of aorta (COA), we hypothesized that for any SBP, patients

143 ng causes disproportionate stiffening of the aorta compared with the carotid arteries, reducing prote

144 n their spleens, periaortic lymph nodes, and aortas, compared with chow-fed mice.

145 ormation at IEL disruptions in the ascending aorta confirms that the IEL is a critical physical barri

146 evel in the DCM (40-fold) than in transverse aorta constriction (4-fold).

147 ine failing heart of both DCM and transverse aorta constriction mice that was accompanied by a decrea

148 by chronic pressure overload via transverse aorta constriction or chronic neurohormonal stimulation

149 ustly in DCM, and partially after transverse aorta constriction, by stabilizing myocardial NAD(+) lev

150 tion and co-localization with macrophages in aorta cryosections using autoradiography, histology, and

151 saic vessels, wild-type ECs leave the dorsal aorta (DA) and form new vessels while neighboring ECs de

152 ficant role in the positioning of the dorsal aorta (DA), but the reason for hypochordal ossification

153 lium (HE) located in the floor of the dorsal aorta (DA).

154 ain pulmonary artery (PAenh), the descending aorta (DAenh), and whole-lung PBV (WLenh) was calculated

155 lowest at the level of the distal descending aorta (DDA) with median COVs of 1.6% for intraobserver v

156 A PVAT ring separated from the aorta demonstrated more profound stress relaxation than

157 on and coagulation signaling pathways in the aorta, demonstrating that these signaling pathways are c

158 BAV undergoing surgery for aortic stenosis (aorta diameter </=45 mm: BAVnon-dil or >45 mm: BAVdil).

159 Proximal aorta diameters (expressed as Z scores) were modeled in

160 Although Sca-1(+)CD45(+) cells from C57BL/6 aorta did not express CD31, they formed CD31(+) colonies

161 ts with neither AS nor AI, 37% had ascending aorta dilatation (4% severe).

162 n are independent determinants for ascending aorta dilatation in pediatric patients.

163 no AS or AI, there is significant ascending aorta dilatation independent of valve morphology.

164 Ascending aorta dilatation rate was significantly increased in pat

165 nctioning BAV exhibited a very slow proximal aorta dilatation rate.

166 are independently associated with ascending aorta dilatation, suggesting that hemodynamic factors in

167 Complex flow profiles are associated with aorta dilatation, ventricle remodeling, aneurysms, and d

168 c valve dysfunction and progressive proximal aorta dilatation, which can lead to aortic dissection.

169 were independently associated with ascending aorta dilatation.

170 Fifty percent of patients had ascending aorta dilatation.

171 arctation was associated with less ascending aorta dilatation.

172 It is associated with ascending aorta dilatation.

173 rt defects and conditions affecting proximal aorta dimensions.

174 Moreover, in 6 out of 11 tissues (aorta, dorsolateral prefrontal cortex, hippocampus, panc

175 as evident in ApoE(-/-)Irf5(-/-) mice in the aorta, draining lymph nodes, and bone marrow cell cultur

176 blunted accumulation of myeloid cells in the aorta due to the attenuation of Angiotensin II (Ang II)-

177 nal role of mural cells investing the dorsal aorta during early development using the zebrafish.

178 A healthy aorta exerts a powerful cushioning function, which limit

179 nsequently, Poldip2-deficient VSMC and mouse aorta express high levels of contractile proteins and, m

180 sed balloon inflation in the distal thoracic aorta for 60 minutes results in high rates of spinal cor

181 n to screen patients with coarctation of the aorta for IA and suggests screening at ages 10 and 20 or

182 ultrasound molecular imaging of the thoracic aorta for VWF A1-domain and glycoprotein-Ibalpha was per

183 hite fat (superior mesenteric artery) and in aorta from both male and female of another rat strain, t

184 n another set of experiments, we excised the aorta from control and diabetic animals, and measured TL

185 hat are critical in protecting the abdominal aorta from injury.

186 n regulation of vascular tone, compared with aortas from control mice.

187 Aortas from SM22-Cul3Delta9 mice showed increased expres

188 ber breaks, and oxidative stress compared to aortas from wild type littermates.

189 GB1 (rHMGB1) was capable of impairing EDR of aortas from wild-type (WT) mice by an eNOS-dependent mec

190 rysmal disease can affect any segment of the aorta, from the aortic root to the aortic bifurcation.

191 ouse HSC development, but which cells in the aorta-gonad-mesonephros (AGM) microenvironment produce t

192 (HSCs) first arise during development in the aorta-gonad-mesonephros (AGM) region of the embryo from

193 m cells (HSCs) first emerge in the embryonic aorta-gonad-mesonephros (AGM) region.

194 robustly the different stages of EHT in the aorta-gonad-mesonephros (AGM) region.

195 d in a distinct subset of macrophages in the aorta-gonad-mesonephros (AGM).

196 cRNA is pivotal for in vivo HSC emergence in aorta-gonads-mesonephros region.

197 NFL athletes were twice as likely to have an aorta >40 mm after adjusting for the same parameters.

198 he area surrounding the infrarenal abdominal aorta halfway between the right renal artery and aortic

199 Patients with coarctation of the aorta have a high prevalence of intracranial aneurysms (

200 Results showed that aortas have significantly blunted sodium nitroprusside-i

201 the great arteries (n=7), coarctation of the aorta/hypoplastic aortic arch (n=5), tetralogy of Fallot

202 Inserting a cannula in the ascending aorta identifies inadequate occlusion of the descending

203 asurement of blood activity in the ascending aorta (image-based TBR, R (2) = 0.96).

204 ts (annulus too large and extreme horizontal aorta in 2 and 1 unsuccessful cases, respectively).

205 ntiated Treg cells relocated to the inflamed aorta in atherosclerosis-prone low-density lipoprotein r

206 ow optimization, downsizing of the ascending aorta in combination with the valve orientation was the

207 ne marrow and atherosclerotic lesions of the aorta in the CKD-bMPOKO mice was confirmed by immunoblot

208 er, vascular reactivity was tested in rodent aortas in the presence of RBCs pretreated with S-nitroso

209 AC)-to increase wall stress in the ascending aorta-induces severe aortic pathology and mortality from

210 The aorta is the 'greatest artery', through which oxygenated

211 pathological dialogue between the lungs and aorta is undefined.

212 in the ascending aorta (aAo), mid-descending aorta, main pulmonary artery, and superior vena cava.

213 (matrix metalloproteinases) activity in the aorta (mean difference 2554+/-363.9, P=9.95122x10(-6)).

214 The protein composition of healthy aortas (media layer) or with early atheroma (fatty strea

215 y over various image-based patient-dependent aorta models.

216 o and macrocalcifications in atherosclerotic aortas obtained from low-density lipoprotein receptor-de

217 h as an interposition graft in the abdominal aorta of a severe combined immunodeficient Beige mouse m

218 Atherosclerosis was studied in aorta of ApoE(-/-) mice fed western-style diet.

219 ink adducts of HDL apoproteins in plasma and aorta of hyperlipidemic LDLR(-/-) mice, including cross-

220 actility and reduces oxidative stress in the aorta of these animals.

221 on and increased the levels of Parkin in the aortas of aged mice but not young mice.

222 anilic acid (3-HAA) levels in the plasma and aortas of Apoe(-/-) mice, but not in IDO(-/-) mice.

223 In aortas of C57BL/6 mice, Sca-1(+)CD45(+) cells were local

224 Aortas of mice with Myh11-driven IL11 expression were re

225 virus (HIV) infection and colocalize in the aortas of simian immunodeficiency virus-infected nonhuma

226 GB1 mediated EDR impairment was abolished in aortas of TLR4(-/-) mice.

227 We performed shotgun proteomics analyses of aortas of transgenic mice with macrophage-specific overe

228 Methods and Results: We analyzed the aortas of young and aged normolipidemic wild type, disea

229 ed gene expression variations (versus normal aorta) of transforming growth factor-beta1 (TGF-beta1),

230 el of dissection in the suprarenal abdominal aorta, often with a false lumen and intramural thrombus

231 Similarly, in isolated aorta, oleic acid treatment generates LD in EC ex vivo.

232 impacting the VDA, we devised a human dorsal aorta-on-a-chip platform that identified Yes-activated p

233 ifies inadequate occlusion of the descending aorta or any collateral flow and diverts flow away from

234 mulation study to date of coarctation of the aorta (over 70 million compute hours).

235 tricity index (EI), main pulmonary artery-to-aorta (PA/AO) diameter ratio, and pulmonary arterial blo

236 al NRP, complete occlusion of the descending aorta prevents brain perfusion in most cases.

237 to-background ratio (TBR) from the ascending aorta (primary endpoint) (adalimumab: TBR = 0.002, 95% c

238 fts closely approximate the native abdominal aorta properties after just 1 week in vivo.

239 PVAT left attached but not encompassing the aorta provided no benefit in cumulative stress relaxatio

240 PKG activity induces oxidative stress in the aorta, raising concern about the widespread clinical use

241 tative Endovascular Balloon Occlusion of the Aorta (REBOA) has been used clinically to limit torso bl

242 tative endovascular balloon occlusion of the aorta (REBOA) is a novel strategy to obtain earlier temp

243 y managed acute uncomplicated TBAD have late aorta-related complications, such as aneurysmal degenera

244 t management of traumatic IT of the thoracic aorta remains weak.

245 major maternal blood vessels, including the aorta, resulted in a peripheral "vascular storm," with e

246 oradiography and immunohistochemistry of the aorta revealed that (68)Ga-FOL radioactivity co-localize

247 mRNA sequencing of aortas revealed that antibodies are required for the suf

248 Thoracic aorta rings from Sprague Dawley rats were mounted in iso

249 Pushers are novel descending aorta rotary pumps that directly increase renal artery p

250 NG), and superoxide dismutase 3 in ascending aorta samples from 50 tricuspid and 70 patients with BAV

251 Prkg1(R177Q/+) aortas show increased smooth muscle cell apoptosis, elas

252 ield of pyrophosphate in wild-type blood and aortas, showing that eNTPD activity (ATP -> phosphate) w

253 ve size and orientation as well as ascending aorta size reduction was made.

254 Similarly, the concept of pharmaceutical aorta stabilization in Marfan syndrome is supported by a

255 start of treatment in TBR from the ascending aorta (TBR = -0.006, 95% CI = -0.049 to 0.038; P = 0.796

256 antly fewer atherosclerotic plaques in their aortas than the control mice due to reduced cholesterol

257 baseline multi-layered model for the healthy aorta that delineates medial elastic lamellae and intra-

258 ied as primary when they develop on a native aorta that has not undergone an intervention and as seco

259 ter discrepant measurements of the ascending aorta that impede, complicate, and impair appropriate cl

260 h cytokines regulate inflammation within the aorta, their contribution to AAA via distant alterations

261 genic, present in the yolk sac, placenta and aorta, through an endothelial-to-haematopoietic transiti

262 or-matched healthy and atherosclerotic human aorta tissue (n = 15) and human carotid plaque samples (

263 mpliant, elastic aortic arch that allows the aorta to accommodate blood ejected by the heart and main

264 st was positioned in the descending thoracic aorta to estimate the PET-derived radioactivity concentr

265 ats by exposing the infrarenal, intraluminal aorta to PPE (porcine pancreatic elastase) under pressur

266 visualize its transport in situ through the aorta to the renal parenchyma and its subsequent filtrat

267 models showed altered reactivity of isolated aortas to phenylephrine and acetylcholine, as well as ma

268 sis (tetralogy of Fallot, coarctation of the aorta, transposition of the great vessels, hypoplastic l

269 aortic root) and neural crest (NC, ascending aorta/transverse arch) SMC lineages to model MFS aortic

270 ncreased hemodynamic forces on the ascending aorta, typically due to poorly controlled hypertension,

271 0 000 cells from the caudal arteries (dorsal aorta, umbilical, vitelline) of 9.5 days post coitus (dp

272 wth-promoting transcription factors and that aortas undergo large-scale metabolic reprograming in the

273 structure and organization in a Marfan mouse aorta using ex vivo small chamber myography.

274 genic endothelium (HE) in the ventral dorsal aorta (VDA), support lifelong hematopoiesis.

275 - and IsoLG-lysyl content in atherosclerotic aortas versus 4-HOBA.

276 he arterial blood pressure (BP) waves in the aorta was developed and experimentally validated.

277 maging with the CIRPI system, the descending aorta was flushed with contrast agent, and OCT images we

278 The abdominal aorta was imaged in vivo in both atherosclerotic rabbits

279 Enzymatic dissociation of 4 whole mouse aortas was followed by single-cell sequencing of >10 000

280 eukocyte infiltrate in atherosclerotic mouse aortas was recently analyzed in 9 single-cell RNA sequen

281 g for IA in patients with coarctation of the aorta, we developed and calibrated a Markov model to mat

282 aortic valve and dilated proximal ascending aorta, we sought to assess (1) factors associated with i

283 dioactivity concentrations in the descending aorta were compared with blood samples counted on a cali

284 border with tumor, nontumoral pancreas, and aorta were recorded on pancreatic parenchymal phase (PPP

285 ere immediately sacrificed after imaging and aortas were excised for mass spectrometry imaging: Matri

286 ates the recruitment of CD4 T cells into the aorta where they can be pathogenic.

287 these patients to dilatation of the thoracic aorta, which is generally silent but can precipitate aor

288 els of the inflammatory cytokine IL-6 in the aorta, which participates in a positive feedback loop wi

289 se in atherosclerotic lesions throughout the aorta, which were significantly attenuated with MCC950 (

290 n of PAH, we hypothesized that the ascending aorta will present signs of apparent stiffness in childr

291 018), CT angiography of the thoracoabdominal aorta with bolus tracking was performed in two different

292 ts, which individually look at the ascending aorta with different perspectives and dimensional defini

293 s of dorsoventral polarized signaling in the aorta with gene expression profiling of sorted cell popu

294 d four-dimensional (4D) flow of the thoracic aorta with navigator gating was performed as a reference

295 solved protein changes taking place in human aortas with early atherosclerosis to find new potential

296 cular relaxation, we cultured mouse thoracic aortas with the FoxO inhibitor and conducted ex vivo two

297 degrees of local dilatations of the thoracic aorta, with enlargement typically exacerbated in cases w

298 and activity of matrix metallopeptidase 2 in aortas without affecting metabolic parameters.

299 decreases atherosclerosis by 60% in en face aortas, without changing plasma cholesterol.

300 The mean ascending aorta Z score progression rate for BAV patient with a no