ライフサイエンスコーパス: aortic injury

1 lumbar vein injuries, 1 renal artery, and 1 aortic injury).

2 verely injured blunt trauma patients without aortic injury.

3 model in rats on the basis of CaCl2-mediated aortic injury.

4 management have expanded for solid organ and aortic injury.

5 no evidence of PH near the diaphragm and no aortic injury.

6 horacic aorta to evaluate potential thoracic aortic injury.

7 ng a high-cholesterol diet after endothelial aortic injury.

8 ed in patients suspected of having traumatic aortic injury.

9 years) were evaluated for possible traumatic aortic injury.

10 graphy directly without CT, 10 had traumatic aortic injury.

11 dictive value for the detection of traumatic aortic injury.

12 d mediastinal hematoma or direct findings of aortic injury.

13 th CT scans that were positive for traumatic aortic injury.

14 heir diagnostic algorithm for acute thoracic aortic injury.

15 ssion of iNOS in response to balloon-induced aortic injury.

16 of protease activity in a rat model of focal aortic injury.

17 insensitive but relatively specific sign for aortic injury after blunt trauma.

18 thors present a series of four patients with aortic injury after invasive coronary angiography and in

19 ockout mice were resistant to CaCl2-mediated aortic injury and did not develop elastin degeneration a

20 oups of patients who are suspected of having aortic injury and have indeterminate angiograms may bene

21 with moderate to high suspicion of traumatic aortic injury and in all patients with CT scans that wer

22 For aortic injury and mediastinal hemorrhage, respectively,

23 naling in a well-defined model of pathologic aortic injury and observed Angiotensin II (Ang II) incre

24 There was no significant difference in major aortic injury and permanent pacemaker implantation rates

25 The combined incidence of aortic injury and stent-related complications was 14%.

26 d PH near the level of the diaphragm without aortic injury, and 72 had no evidence of PH near the dia

27 the thorax (HCTT) for the diagnosis of blunt aortic injury (BAI).

28 iography to indicate suspicion for traumatic aortic injury before angiography resulted in savings of

29 for 1.5% of thoracic trauma, blunt thoracic aortic injury (BTAI) is a rare disease with a high morta

30 hemorrhage alone is sensitive for traumatic aortic injury, but the finding of aortic injury is more

31 Endovascular repair of blunt traumatic aortic injury can be performed with a low morbidity and

32 ve cases of endograft treatment of traumatic aortic injury from December 2004 to November 2008 were r

33 Likewise, the therapy for acute aortic injuries has changed substantially.

34 The imaging evaluation of acute aortic injuries has undergone radical change over the pa

35 Endovascular repair of thoracic aortic injury has been shown in multiple studies to have

36 s at angiography were positive for traumatic aortic injury in 19 (90%).

37 CT signs of traumatic aortic injury in 21 patients included contour abnormalit

38 racic CT was performed to evaluate traumatic aortic injury in 677 patients with positive or equivocal

39 s at angiography were negative for traumatic aortic injury in 77 (97%).

40 facilitating the diagnosis of acute thoracic aortic injury in the patient with blunt chest trauma has

41 Endovascular repair of traumatic aortic injury is becoming routine.

42 traumatic aortic injury, but the finding of aortic injury is more specific.

43 verely injured blunt trauma patients without aortic injury, IT was not associated with additional in-

44 Moreover, endovascular imaging of the focal aortic injury model enabled successful measurement of en

45 ; 95% CI: 1.81, 7.64; P = .0001), as well as aortic injuries (n = 4 vs n = 0; P = .0015; OR, unavaila

46 = 197), pulmonary vein stenosis (n = 2), or aortic injury (n = 2) were routinely reviewed in transve

47 d be performed only in the presence of local aortic injury or aneurysm.

48 advances, timely diagnosis of acute thoracic aortic injury remains a challenge.

49 Aortic injury represents a rare but potentially fatal co

50 both PH near the level of the diaphragm and aortic injury; six had aortic injuries without PH, five

51 Aortic injury status was determined by reviewing angiogr

52 e B aortic dissections (TBAD), and traumatic aortic injuries (TAI) treated with TEVAR from 2012 to 20

53 been used for serial examination of minimal aortic injuries that are treated conservatively.

54 iology and pathophysiology of acute thoracic aortic injury, the current status of the individual imag

55 ositive likelihood ratio for the presence of aortic injury was 10.8, and the negative likelihood rati

56 near the level of the diaphragm as a sign of aortic injury was 70%; specificity, 94%; positive predic

57 age, respectively, specificity for traumatic aortic injury was 99% and 87% and sensitivity was 90% an

58 m there was very high suspicion of traumatic aortic injury were examined with aortography only.

59 scans positive for mediastinal hemorrhage or aortic injury were retrospectively reviewed and interpre

60 us of imaging and therapy of acute traumatic aortic injuries will be reviewed.

61 of the diaphragm and aortic injury; six had aortic injuries without PH, five had PH near the level o