ライフサイエンスコーパス: apneic

1 ariable indicating the presence of increased apneic activity (IAA).

2 If apneic activity does cause acute stress in HF, it should

3 expanded and allow for prolonged periods of apneic airway surgery.

4 entilatory stimuli on the hypocapnia-induced apneic and hypopneic thresholds in sleeping dogs.

5 Lung wet/dry weight was 5.40 +/- 0.93 in apneic animals and 5.00 +/- 0.67 in controls.

6 e experiment fell from 89.6 to 82.8 mm Hg in apneic animals and from 92.2 to 85.5 mm Hg in controls.

7 tor drives to upper airway muscles may cause apneic breathing at birth.

8 This apneic breathing is phenocopied by restricting the mutat

9 In patients with OSAS, the EMGgg for apneic breaths during REM (37 +/- 9%) was lower than dur

10 s significantly larger compared with the pre-apneic control.

11 the post-apneic period compared with the pre-apneic control.

12 digging while feeding near the surface, and apneic dives.

13 roscopy showed gross alveolar fluid in three apneic dogs, and electron microscopy showed interstitial

14 showed interstitial fluid in two additional apneic dogs.

15 tively, to 1.7 to 3.2 Torr at the end of the apneic episode.

16 y characterized by hypoxemia due to frequent apneic episodes and fragmentation of sleep due to the br

17 red cerebrovascular reactivity caused longer apneic episodes and more anxiety, indicating that cerebr

18 The mean duration of apneic episodes did not significantly differ (model poin

19 The mean number of apneic episodes did not significantly differ (model poin

20 of activation during spontaneous obstructive apneic episodes in non-rapid-eye-movement (NREM) sleep:

21 Apneic episodes induced in animals ventilated with 15%,

22 When the apneic episodes were eliminated by application of nasal

23 There is leukocytosis with lymphocytosis and apneic episodes.

24 due to the brief arousals that terminate the apneic episodes.

25 The similar number and duration of apneic events and lack of serious adverse events suggest

26 hospital, our algorithm identified simulated apneic events and successfully injected participants wit

27 = 0.01) and duration (r = 0.63, P = 0.04) of apneic events and the percentage of time breathing was s

28 Apneic events are frightening but largely benign events

29 Repetitive apneic events disrupt the normal physiologic interaction

30 Here, we report apparent life-threatening apneic events in an infant with the homozygous SCN1A(L26

31 l blockers may be considered therapeutic for apneic events in patients with these and other gain-of-f

32 mice, and decreased the frequency of severe apneic events in the patient.

33 that SCN1A(L263V) can cause life-threatening apneic events, which in a mouse model were caused by pro

34 d possibly a higher arousal threshold during apneic events.

35 ear to be linked to the repetitive nocturnal apneic events.

36 The mean amplitude and slope of the post-apneic fEPSP was significantly larger compared with the

37 ves (1 mg lorezapam), and one patient became apneic for a brief interval after receiving additional n

38 ed in the nonprotective group, with the near-apneic group exhibiting the least alterations.

39 Near-apneic group showed significantly less matrix metallopro

40 and mechanical power were lower in the near-apneic group, but no differences were observed in oxygen

41 g histology revealed less injury in the near-apneic group.

42 o relationship between BNP and the number of apneic/hypopneic episodes or the number of arousals.

43 ssociated with the frequency and severity of apneic/hypopneic episodes, intermittent arousals, and hy

44 ssociated with a significant increase in the apneic index in these infants (28.6 +/- 6.4/hour [S] vs.

45 ices showed good agreement with steady-state apneic left ventricular stroke volume values and moderat

46 njector system that measures respiration and apneic motion associated with an opioid overdose event u

47 There was no difference between apneic oxygenation and usual care in incidence of oxygen

48 Apneic oxygenation does not seem to increase lowest arte

49 These findings do not support routine use of apneic oxygenation during endotracheal intubation of cri

50 west arterial oxygen saturation was 92% with apneic oxygenation versus 90% with usual care (95% confi

51 of supplemental oxygen during laryngoscopy (apneic oxygenation) may prevent hypoxemia.

52 high-flow nasal cannula during laryngoscopy (apneic oxygenation) or no supplemental oxygen during lar

53 that hf-TTI stabilized breathing patterns in apneic patients, and was safe and efficacious for prolon

54 p fragmentation are difficult to separate in apneic patients.

55 cm H2O (mean +/- SD) for three breaths in 19 apneic patients.

56 l/kg; respiratory rate, 20 bpm), and 3) near-apneic (PEEP, 10 cm H(2)O; driving pressure, 10 cm H(2)O

57 tilation induced a longer hypocapnia-induced apneic period (51.5 +/- 9.9 versus 11.2 +/- 5.5 seconds,

58 red-pulse facilitation ratio during the post-apneic period compared with the pre-apneic control.

59 diate effect of tracheal stimulation was an "apneic" period at FRC, during which the PCA, a laryngeal

60 After an additional 30 minutes of apneic placental support, lung tissue and bronchoalveola

61 The apneic reaction was associated with a grimace, character

62 al trigeminal complex.(7)(,)(8) Notably, the apneic reflex is not dependent on olfaction as it can be

63 atory regulation, including vagally mediated apneic reflexes.

64 way and the respiratory neurons that mediate apneic reflexes.

65 sults showed that PNE markedly prolonged the apneic response and exacerbated the bradycardic response

66 tent in bronchoalveolar lavage fluid, 2) the apneic response to the right atrial bolus injection of p

67 and 12.1% indicated a passive (i.e., infant apneic) response by the infant, despite excluding period

68 ergence of sensory inputs capable of driving apneic responses and that it may represent a common link

69 However, in our earlier study of apneic responses produced by glutamate stimulation in th

70 Apneic responses were more frequent at earlier postmenst

71 racterize the sites in the pons that produce apneic responses.

72 We found that apneic sites were consistently associated with the inter

73 were no significant differences with gender, apneic spells, jaundice, or phototherapy.

74 ry sensitivity to CO2 between eupnea and the apneic threshold are changeable in the face of variation

75 3-5 mm Hg > eupnea (and 7-10 mm Hg > normal apneic threshold) throughout CMV trials at raised freque

76 e during sleep may lower the value below the apneic threshold, thereby resulting in central apnea.

77 sed a significant (p < 0.05) increase in the apneic threshold, which we attribute to an inhibitory ef

78 ns (relative risk, 0.87; 95% CI, 0.71-1.06), apneic time (mean difference, 10.3 s higher with high-fl

79 severe desaturation, serious complications, apneic time, oxygenation, ICU length of stay, or overall

80 omes included peri-intubation complications, apneic time, PaO2 before and after intubation, PaCO2 aft

81 ess syndrome model supported with ECMO, near-apneic ventilation decreased histologic lung injury and