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1 y inside the ectoderm, and generation of the archenteron.
2 ension movements during the formation of the archenteron.
3 to examine the mechanical properties of the archenteron.
4 both ectodermal lineages and portions of the archenteron.
5 efore this territory invaginates to form the archenteron.
6 y of animal blastomeres to contribute to the archenteron.
7 s also regulated to that new position in the archenteron.
8 derm and fusing at the dorsal midline of the archenteron.
9 promote expression in the vegetal plate and archenteron.
10 o locate expression in the vegetal plate and archenteron.
11 oisson effect (necking) that occur in actual archenterons.
12 ere also common, including reduced or absent archenterons.
13 t primordium is similar to elongation of the archenteron and also of the entire embryonic axis (both
14 e, at the gastrula stage in the whole of the archenteron and in postgastrular stages only in the midg
17 endodermal target genes are expressed in the archenteron and might be terminal differentiation enzyme
18 (4) that interactions between the tip of the archenteron and the presumptive oral ectoderm are not re
19 region and the posterior of the invaginating archenteron, and finally to the midgut and hindgut of th
21 eposited in the basal lamina surrounding the archenteron as well as in other areas of the blastocoel
24 ge embryos SpHmx is expressed throughout the archenteron, but particularly strongly in delaminating s
25 e layered epithelium, and whether or not the archenteron cavity actually gives rise to the gut lumen.
29 ondary invagination involves the addition of archenteron cells and an increase in volume of the arche
30 during secondary invagination, the number of archenteron cells increased by at least 38% (over 50% wh
32 RhoA induces precocious invagination of the archenteron, complete with the actin rearrangements and
33 eneration gives rise to larvae containing no archenteron derivatives at all, endoderm only, or both e
34 alone each regenerate the full complement of archenteron derivatives; thus, they are uninformative as
36 mall micromere descendents at the tip of the archenteron during gastrulation and are then enriched in
37 econdary mesenchyme cells and the elongating archenteron during gastrulation; Cadherin (G form) has a
39 earrangement can account for key features of archenteron elongation and provides a useful starting po
40 chanical model, we show that key features of archenteron elongation can be accounted for by passive c
41 lation of secondary mesenchyme cells late in archenteron elongation does not result in extensive elas
42 cells to the base of the archenteron late in archenteron elongation leads to excessive cell rearrange
47 teron cells and an increase in volume of the archenteron epithelium, we conclude that secondary invag
50 s an extensive rearrangement of cells of the archenteron giving rise to secondary mesenchyme at the a
52 ort-range communication between cells of the archenteron in order to reorganize the tissues and posit
54 bilaterally symmetric, and flank the ectopic archenteron, in some cases resulting in mirror-image, sy
55 , but they accompany the invagination of the archenteron initially, in much the same way vertebrate m
59 egetal plate and primary invagination of the archenteron, involves only the Endo16-expressing cells o
64 ing the addition of cells to the base of the archenteron late in archenteron elongation leads to exce
65 be into a long thin tube; secondly, that the archenteron lining does not become the definitive gut lu
69 re, as an initial step towards examining how archenteron precursors are specified, a clonal analysis
70 eric embryo approach, we show that implanted archenteron precursors differentiate autonomously to pro
71 s demonstrate that mesoderm induction in the archenteron requires contact with ectoderm, and loss-of-
74 al effect of disrupting morphogenesis of the archenteron, revealing a previously unsuspected function
76 nsion of deep ectoderm just underlain by the archenteron roof is twice that of not-yet-underlain deep
77 nic mesenchyme, the CyIIa gene, expressed in archenteron, skeletogenic and secondary mesenchyme, and
78 After removal of any or all parts of the archenteron, the remaining veg 1 and /or veg 2 tissue re
80 n giving rise to secondary mesenchyme at the archenteron tip followed by the foregut, midgut and hind
81 rived from the host, the ectopic presumptive archenteron tissue can act to 'organize' ectopic axial s
82 ition, the ectopically implanted presumptive archenteron tissue induces ectopic skeletal patterning s
88 ve capacity of this structure, pieces of the archenteron were removed or transplanted at different st