ライフサイエンスコーパス: argue for

1 Here, I argue for a "person-not-project"-based scheme that would

2 tools and whole-genome sequencing data, and argue for a better integration of population genetics fi

3 Mn-cycling bacteria in the manganese nodules argue for a biologically driven closed manganese cycle i

4 These findings argue for a cautious approach to chronic macrolide use i

5 We use this evidence to argue for a central involvement of root exudates in plan

6 exons into proximity for splicing, our data argue for a central role of introns in mutually exclusiv

7 We argue for a change in the conceptual framework within wh

8 om mid-latitude Andean glaciers, our results argue for a common glacier response to cold conditions i

9 Taken together, these data argue for a complex mechanism of PKCbeta-dependent regul

10 These findings argue for a cortically focal entrainment of ongoing alph

11 We finally argue for a critical approach to the use of animal model

12 Here, we argue for a different approach that focuses less on a bi

13 These data argue for a direct assessment of one-dose efficacy of th

14 We argue for a dual intervention strategy at both the publi

15 We also argue for a focus on the development of individualized t

16 Our results argue for a functional benefit of sparsification that co

17 er dissect the folk concept of "contempt" to argue for a functionally integrated model of attitudes a

18 lutions from a public health perspective and argue for a harmonized approach to clean air and climate

19 Our results argue for a hierarchical stability of secondary structur

20 Our results argue for a largely lipid-dependent process of autophagy

21 of such a functional gradient, other studies argue for a lesser degree of specialization within PFC.

22 I argue for a low value, 1-2 kPa, in frog eggs and perhaps

23 in diabetic peripheral nervous system (PNS) argue for a major role of the ER stress pathways in regu

24 Our data argue for a mechanical control system whereby actin wave

25 to SIL treatment in vivo and in cell culture argue for a mechanism involving NS4B.

26 Hence, we argue for a molecular evolution alongside a clinical and

27 In this Perspective, we argue for a more consistent weight-management approach,

28 417) argue for a more nuanced view of insect decline, and of

29 We argue for a move away from a singular focus on intervent

30 creases in mean wind shear have been used to argue for a negative influence of global warming on seve

31 We argue for a new development paradigm-away from only atte

32 I then argue for a new, integrated framework that differentiate

33 We argue for a new, more precise but more challenging parad

34 These results persuasively argue for a noncell autonomous effect of FAD-linked PS1

35 Based on these results, we argue for a novel mechanistic account for the integratio

36 Our findings argue for a novel model in which human B cells promote s

37 g the results of several ongoing studies, we argue for a pragmatic approach to using low-dose aspirin

38 Our findings therefore strongly argue for a priming mechanism at the photoreceptor ribbo

39 Instead, these data argue for a principal role for transcription factor bind

40 ical tracking of speech at syllable rate and argue for a progressive development of SiN abilities in

41 Consequently, we argue for a research agenda that integrates multiple pla

42 xpand the metabolic potential of Archaea and argue for a revision of the role of Archaea in the carbo

43 And argue for a science of many tongues.

44 We argue for a shift in focus to the evaluation of such tes

45 Alternative models argue for a single boundary distinguishing mood disorder

46 different levels of an action hierarchy and argue for a special status of the distinction between in

47 sodic and declarative memory, whereas others argue for a specific role in the creation of spatial cog

48 We argue for a strengthening of accountability systems acro

49 In addition, we argue for a strong need to explore how disease in dogs r

50 hological descriptions of Crohn disease (CD) argued for a potential defect in lymph transport; howeve

51 Epidemiologists have argued for a single logically consistent definition of i

52 ial selection protocols in biotechnology and argues for a better understanding of mutation supplies i

53 ecific footprint in epigenetic profiles, and argues for a bimodal evolution of gene regulatory progra

54 Such anatomic specificity argues for a framework aimed at understanding vulnerabil

55 This argues for a key role of positional fluctuations in dict

56 ely detected only using 3D EM reconstruction argues for a major role of mitochondrial dynamics in reg

57 ence of nNOS throughout this sensory nucleus argues for a major role of NO in hearing.

58 This argues for a novel humoral immune escape mechanism that

59 This work argues for a novel theory according to which the BG driv

60 Conclusion: This clinical case argues for a possible relationship between SARS-CoV-2 in

61 tprocessing methods in metabolomics and also argues for a proper match between type of metabolomics d

62 ars after vaccination of 9-month-old infants argues for a revision of the single-dose recommendation

63 Alternatively, the searchlight hypothesis argues for a role in focal attentional modulation throug

64 Altogether, this argues for a significant contribution of adrenomedullary

65 In contrast, the cholinergic hypothesis argues for a specific pathology of brain cholinergic pat

66 AD significantly improved protein stability, arguing for a chaperone-like action similar to what has

67 defects similar to the Gln(758) variant(s), arguing for a comparable loss of function.

68 ectivity from frontal to visual cortex, thus arguing for a crucial role of frontoparietal cortex in p

69 to both interrupted and uninterrupted cold, arguing for a digital mechanism integrating long-term te

70 NP-A) and is associated with the centromere, arguing for a direct role of Yta7(ATAD2) in Cse4(CENP-A)

71 ncoupled from the full reactivation program, arguing for a less sharply bimodal definition of latency

72 ants' performance in prosody categorization, arguing for a motor involvement in prosody perception.

73 irect induction of CCL17 by dendritic cells, arguing for a need to address vaccine formulation safety

74 in the proliferation of neural progenitors, arguing for a neuroprotective role of adiponectin.

75 We extend their model by arguing for a perspective that includes not only context

76 ut decreasing the size of the mutated clone, arguing for a predominantly epigenetic effect.

77 maintains its ability to bind mitochondria, arguing for a role for Actr10 in dynactin-mitochondria i

78 RA-RARalpha signaling in Th differentiation, arguing for a role for RA in interleukin 9 (IL-9) relate

79 duced the severity of demyelination in mice, arguing for a role for these cells in white matter damag

80 uced changes in both activity and structure, arguing for a role of DNA-induced structural changes in

81 ship between sleep and memory consolidation, arguing for a significant role of inhibitory neurotransm

82 wellings have been found in tuatara embryos, arguing for a single origin.

83 ic (p)LCDD constitutes an additional feature arguing for a specific entity distinct from the systemic

84 deficient mice show severe skin dysfunction, arguing for a tissue autonomous cause of disease develop

85 However, recent findings argue for additional mechanisms and suggest the decision

86 These data argue for AKT-associated HK2-mediated metabolic reprogra

87 erlying working memory, but recent proposals argue for alternative 'activity-silent' substrates.

88 s of fO2 of Archean magmas are not this low, arguing for alternative explanations for the oxygenation

89 Our data argue for an active role for NO in the remodeling of thy

90 Taken together, our results argue for an active role for Paf1C in promoting H2Bub an

91 We argue for an alternative explanation: that priority itse

92 silencing of the EPHB3 tumor suppressor and argue for an antitumorigenic function of Notch signaling

93 discuss the implications of this dilemma and argue for an approach that will integrate DST into the d

94 Some argue for an early Moon-forming event, approximately 30

95 Our findings might argue for an early surgical intervention in eyes with DM

96 Here, we argue for an evolutionary approach to paranoia across th

97 etect acute changes in STEAP1 expression and argue for an expansion of ongoing efforts to image PCa p

98 imately, in synthesizing this literature, we argue for an HbA(1c) target of <7% for most individuals,

99 ing of pathogens and antimicrobial responses argue for an important role for GH in macrophage priming

100 Here I argue for an increased investment in modern lesion mappi

101 We argue for an integrated T2D knowledgebase, designed for

102 Kline argues for an expanded taxonomy of teaching focusing on

103 ctedly tissue-specific effects in humans and argues for an important role for POLD1 function in adipo

104 A burgeoning literature argues for an important role for the catecholamines dopa

105 enefits of specific interventions in sepsis, argues for an individualized and titrated approach to in

106 cell transfer or immune checkpoint blockade, arguing for an adaptive resistance mechanism.

107 nce to apoptosis but also to steatosis, thus arguing for an early protective effect against alcoholic

108 quipped with a conserved proton-binding site arguing for an electrogenic transport mode.

109 vercome impaired cerebellar cell generation, arguing for an epistatic role of Pals1 in proliferation

110 e control of the levels of free nucleotides, arguing for an essential role of nucleotide metabolism i

111 ry conserved within the green plant lineage, arguing for an evolutionary conserved core machinery of

112 without spatial association with HIF-1alpha, arguing for an importance of hypoxia-independent, i.e.,

113 rved after high-dose steroid treatment, thus arguing for an inflammatory-mediated brain involvement r

114 down-regulating neurons of premotor cortex, arguing for an initiation of selection in the frontal lo

115 ther than tertiary surfactant hydroperoxide, arguing for an orthogonal alkene on water.

116 second semester of life in SGA infants, and argue for better follow-up of these infants after birth,

117 equire rehabilitation after minor stroke and argue for better integration of stroke and dementia serv

118 pocket as VI in all hexon monomers, strongly arguing for binding competition.

119 well-preserved digital pad and skin traces, argues for bipedal trackmakers, here assigned to Batrach

120 These findings argue for both a similar operational 'grammar' and share

121 Altogether, our results argue for both presynaptic and postsynaptic contribution

122 These results also argue for broader discussions of statistical reliability

123 These compiled data argue for CANOMAD to be included in neurologic monoclona

124 We argue for careful patient selection and close longitudin

125 eptor variants not present in mice; however, argue for caution when directly translating these findin

126 This finding further argues for caution when assigning unverified high-valent

127 radigm beyond routine clinical AF management argues for change in the delivery of care to patients wh

128 We argue for close and detailed exploratory analysis of mov

129 We argue for combining data- and hypothesis-driven methods

130 Our findings argue for complement C5 activation during MVA infection

131 point in current staging systems for PTC and argue for considering a revision in how we anticipate pr

132 yond the variance explained by volume alone, arguing for considering multimodal imaging in treatment

133 chieved a level of maturity and success that argues for continued and expanded use of this program.

134 served in human cutaneous melanoma biopsies, arguing for COX activity as a driver of immune suppressi

135 elf-mt peptide bound to P9/HLA-A2 tetramers, arguing for cross-reactivity between T cells with specif

136 th apparently suboptimal drug concentrations argues for development and evaluation of novel therapeut

137 This observation argues for differential modes of action of neutralizing

138 oducing IL-17A(neg)Foxp3(+) T cells strongly argues for direct conversion of Th17 cells into T(reg) c

139 resynaptic Ca2+ chelation as evoked release, arguing for direct triggering of spontaneous release by

140 The results argue for distinct roles for these two regions in learni

141 significant risk of serious adverse events, arguing for earlier intervention.

142 d its numbers can increase 4-fold in 4 days, arguing for early and frequent serum testing.

143 resulted from available screening in men and argue for efforts to identify high-risk individuals who

144 ts and sparse GW data limited the study, and argue for enhancing monitoring efforts.

145 nvolving a cancer-related virus and Netrin-1 argues for evaluating the implication of UNC5 receptor l

146 in the absence of environmental filters, and argue for evolutionarily informed forecasts of invasive

147 We argue for expanding these efforts from resource-rich reg

148 I have tried to distinguish comments that argue for extensions of the theory from those that repre

149 Results argue for frequency-dependent cortical tracking mechanis

150 These observations argue for further investigation of antimicrobial interve

151 enough variation within the two complexes to argue for further resolution into separate species, whic

152 This review argues for further research into hormonal influences on

153 On balance, the evidence still argues for further research to better understand how HDL

154 campus-independent emotional processes, this argues for generic information processing in REM sleep r

155 There are also theories arguing for global precedence, reversed hierarchy, or bi

156 Their findings argue for greater attention to confounding by neuroselec

157 onsequence of single-element management that argues for greater control of nitrogen as well as phosph

158 and instead strongly support native status, arguing for immediate implementation of conservation mea

159 therapeutic activity of anti-IL-1 strategies argues for immunity and inflammation as a metanarrative

160 olic processes for immune cell functions and argue for implementation of IL-15 in adoptive NK-cell ca

161 We conclude by arguing for implementation of guideline-recommended BP m

162 ticularly in intermediate-risk AML patients, arguing for inclusion of mutational testing of these gen

163 more neural information from visual cortex, arguing for increased development of this approach in ba

164 sease, discussing the biological context and arguing for increased investigation into the function of

165 ical distance from visual cortical input and argue for increasingly modified functional microcircuits

166 This "being in two places at once" argues for independent functional regions within the hip

167 sferred to a new spatio-temporal combination arguing for independent neural representations of these

168 by a population of neurons, with some camps arguing, for instance, that mean pairwise correlations o

169 analysis techniques such as space syntax and argue for investigating inter-individual differences in

170 These findings argue for investigation of different mechanistic targets

171 egulators indicates caution is warranted and argues for investment in understanding of their underlyi

172 ms across the tree of life have been used to argue for its antiquity and its optimality.

173 We therefore argue for its implementation across training programs be

174 els of visual word form area (VWFA) function argue for its specific role in decoding written language

175 Application of the method across datasets argues for its competitive predictive ability (AUC of 0.

176 hotics irrespective of treatment-resistance, arguing for its use more generally in schizophrenia.

177 and chronicity of functional motor symptoms argues for larger prospective studies including multiple

178 Several authors argue for maintaining current targets, as opposed to ado

179 e, consolidating review of the literature to argue for major changes in cognitive science.

180 the expansion are hotly contested, with some arguing for matrilineal and matrilocal patterns, whereas

181 Overall our findings argue for more detailed modeling of biological detail ac

182 0-80% and debilitating chemotherapy sequelae argue for more informed treatment selection, which is no

183 These results argue for more intense study of this unique high-elevati

184 decadal Arctic carbon cycle trajectories and argue for more mechanistic models to improve predictive

185 This finding argues for more experimental work aimed at understanding

186 n the germline of older versus younger mice, arguing for mtDNA turnover during oocyte meiotic arrest.

187 r on different homologous chromosomes, which argues for multi-centric origin of respective HCCs.

188 isms of uncoupling the two steps of splicing argue for multiple origins of this pathway.

189 We also argue for new data acquisition and analysis strategies w

190 lenging the generalizability of studies that argue for no causal role for consciousness in decision-m

191 These results argue for non-genetic profiling as a tool for the study

192 Recent evidence argues for nonuniform outcomes after fusion, in which ca

193 i ratios in Archean shales, has been used to argue for ongoing subduction at that time and therefore

194 sts but takes sensory costs into account and argues for optimal control of movement in which feedforw

195 proaches to wildlife management while others argue for physical separation between protected species

196 d safety signal processing in the amygdalae, arguing for predominant generalization of fear and hence

197 P2 antibody prevents long-term potentiation, arguing for previously unknown roles for TIMP2 in normal

198 nal and neuroscientific considerations; some argued for related but distinct viewpoints; a few, thoug

199 This analysis argues for reprioritizing interventions on the basis of

200 e recent history of genetic epidemiology and argue for retaining statistical testing as an important

201 We argue for revisiting methylation enrichment methods that

202 Here, we argue for routine, early and cumulative screening for ge

203 Additionally, we argue for scientists using complex non-linear dynamical

204 Instead, they argue for selective subnetworks of inhibitory and excita

205 patient groups but not in healthy controls, arguing for sensitization of cutaneous mechano- and heat

206 translational protein modifications strongly argue for significant qualitative changes in contractile

207 of the Drosophila AHR-1 homolog, Spineless, argues for similar pathways in mammals.

208 a similar CT load at extragenital locations arguing for similar transmission potential and clinical

209 f socialized spatial cognitive processes and argues for socio-economic analysis in studies investigat

210 ations resembling those in Pbx1 mutant mice, arguing for strong conservation of gene function between

211 Yael Velleman and colleagues argue for stronger integration between the water, sanita

212 TAGIs together with encouraging in vivo data argue for successful translation to the clinic.

213 These data argue for syncytins being a major driving force in the e

214 MDS/AML, often without known family history, arguing for systematic screening.

215 tems from broader defects of the motor unit, arguing for systemic SMN repletion.

216 Together, these data argue for template-assisted folding at more than one poi

217 d performance for difficult discriminations, arguing for temporal integration.

218 o promote discussion around these issues and argue for the adoption of improved standardised PCR corr

219 Here, we argue for the advantages of grounding the psychological

220 Together, these results argue for the apoE deficiency, and not hypercholesterole

221 The results argue for the common neural substrate of RSNs and encour

222 of symptom progression with chronic use, and argue for the continued refinement and development of GM

223 physiological studies, and related fields to argue for the development of a second-person neuroscienc

224 of descending opioid-induced inhibition and argue for the development of sex-specific pain therapeut

225 Our findings argue for the development of therapies aimed at maintain

226 Our experiments argue for the dissociation of spontaneous activity from

227 advances in single-cell transcriptomics and argue for the essential role of epigenomic technologies

228 These results argue for the evaluation of ibrutinib in patients with F

229 These data argue for the evaluation of these agents as treatments f

230 These studies argue for the evolution of a common molecular strategy f

231 These findings strongly argue for the existence of a rat object-processing pathw

232 Furthermore, they argue for the existence of a supra-modal map which keeps

233 Our in vitro data argue for the existence of a tight posttranslational reg

234 These results argue for the existence of an adaptive interdependence b

235 them to those exhibited by chemical bonds to argue for the existence of entropic bonds.

236 that are neurotoxic, and recent observations argue for the existence of soluble helical tetrameric sp

237 We thus argue for the existence of two distinct routes for makin

238 Together, these results argue for the further development of small-molecule inhi

239 evel, backed up by electron microscopy data, argue for the impairment of several forms of endocytosis

240 We discuss gaps in knowledge and argue for the importance of considering bacteria-fungal

241 We outline current gaps in knowledge and argue for the importance of considering infectious and i

242 we view one another's unique approaches and argue for the importance of diversity of perspective in

243 Here we argue for the importance of evaluating environmental sus

244 These findings argue for the importance of inclusion of kidney disease

245 Here we argue for the importance of incorporating economic evalu

246 In this context, we argue for the importance of incorporating techniques in

247 Taken together, our results argue for the importance of resolving linkage in studies

248 rstanding of the cause of schizophrenia, but argue for the importance of the patient's subjective vie

249 Our analyses thus argue for the inclusion (and exclusion) of specific epit

250 Taken together, these results argue for the inclusion of a structural dimension and su

251 In conclusion, we argue for the inclusion of single-dose arms in future tr

252 Baumeister and colleagues argue for the indispensability of groups in human life.

253 mpete the pathogen in warm, estuarine waters argue for the need for a better understanding of this ne

254 lising rheology, calorimetry and microscopy, argue for the occurrence of phase separation phenomena i

255 Thus, we argue for the persistence and physiological relevance of

256 Altogether, these results argue for the potential of 1,25D3-mDCs to restore Ag-spe

257 These findings argue for the prescription of HCV therapy in coinfected

258 Instead, we argue for the presence of a single extensive and variabl

259 continental setting, the isotopic signatures argue for the presence of mantle derived fluids, suggest

260 espondences are pervasive and detailed; they argue for the presence of rods and cones very early in t

261 e for the pericardial tissue compartment and argue for the reevaluation of surgical procedures that r

262 Bentley et al. argue for the social scientific contextualization of "bi

263 encing, while Kyle Rodino and Melissa Miller argue for the use of caution.

264 s was detrimental rather than protective and argue for the use of next-generation TNFR-selective TNF

265 against education of normal cells by TEV and argue for the use of reserpine in adjuvant melanoma ther

266 In particular, we argue for the utility of more standardized data collecti

267 Scholars have long argued for the central role of agency-the size of one's

268 s in the human reference genome, GRCh38, and argues for the construction of a human pan-genome.

269 g-term PFS in patients with IGHV-M after FCR argues for the continued use of chemoimmunotherapy in th

270 Our theory argues for the existence of a novel peripheral mechanism

271 ze local backbone-side-chain contacts, which argues for the inclusion of n-->pi* interactions in the

272 ping that we conducted for knock-out mutants argues for the information content of top-ranked genes.

273 and investigating early disease pathogenesis argues for the need for complementary studies in mice an

274 ions in cesarean section rates in the US and argues for the need for high-quality routine data collec

275 in human and mouse SNpc populations strongly argues for the need of human-focused omics studies.

276 idual patients and health care policies, and argues for the need to (1) lower the prices of cancer dr

277 This perspective argues for the need to investigate somatic variation in

278 nts in an N6-LS-monotreated animal, however, argues for the need to maximize breadth and antiviral ef

279 f an adult hypothalamic neurogenic niche and argues for the potential existence of a migratory path w

280 This argues for the presence of this proofreading in the comm

281 Our study strongly argues for the use of antitumor immunotherapies combined

282 This report argues for the usefulness and practicality of the model

283 Arguing for the computational relevance of phase relatio

284 is less pronounced in freshly spread cells, arguing for the existence of a sorting mechanism acting

285 presence of fewer GR recognition sequences, arguing for the existence of additional mechanisms that

286 iable with only minor morphological defects, arguing for the existence of an alternate lipid II flipp

287 rogen-bonded beta-strands outside the iLBPs, arguing for the generality of the role played by this st

288 cells partially rescued replication, further arguing for the importance of glycosphingolipids in HCV

289 te equivalent charge to GoCs as mf synapses, arguing for the importance of the feedback inhibition.

290 d for ES in the literature is variable, thus arguing for the optimization and standardization of ES t

291 ing-perhaps also in mammalian bones-and thus arguing for the possibility of untapped potential for ce

292 a PGL-dependent fashion following infection, arguing for the potential of PGL-blocking interventions

293 for decisional and postdecisional mechanisms arguing for the supramodality of metacognition.

294 ow efficacy, with recent clinical guidelines arguing for the use of personalized approaches.

295 mical analysis and hierarchical modeling and argue for their integration, demonstrating the value of

296 synonymous variants in human physiology and argue for their routine inclusion in any comprehensive a

297 signals, and their readthrough is regulated, arguing for their importance.

298 partially restored T cell alloproliferation, arguing for their involvement in the immunosuppressive e

299 he biological effect of RTX decreases, which argues for treatment efficacy.

300 found for other G-quadruplex-forming motifs, arguing for widespread applications.