ライフサイエンスコーパス: arsenic

1 ESA response was positively associated with arsenic.

2 er of metabolic NADPH) in the absence of any arsenic.

3 ate and tolerate exceptionally high level of arsenic.

4 ged only 10-50 years ago but is still low in arsenic.

5 issolution of iron oxides and the release of arsenic.

6 by increasing access to toxic metals such as arsenic.

7 ptasensors for possible on-site detection of arsenic.

8 calization of Hog1 upon exposure of cells to arsenic.

9 duce the content of the carcinogen inorganic arsenic.

10 erlying the cardiovascular effects caused by arsenic.

11 cal control on the release (or retention) of arsenic.

12 eat shock, endoplasmic reticulum stress, and arsenic.

13 sibility to hazardous trace elements such as arsenic.

14 ditional microbial indicators, chlorine, and arsenic.

15 nd vanadium while positively associated with arsenic.

16 rference was observed in presence of 100 ppb arsenic, 20 ppb copper, 5 ppb cadmium, 10 ppb lead, 10 p

17 e World Health Organization's risk level for arsenic (6.6 ng m(-3)).

18 alysis of the cellular response to trivalent arsenic, a ubiquitous environmental toxin and carcinogen

19 and in vivo studies to provide evidence that arsenic, a widespread environmental toxicant, inhibits e

20 ic levels in cats with or without CIN, renal arsenic accumulation does not appear a primary driver of

21 inorganic arsenic and, potentially, dimethyl arsenic acid are carcinogens widely elevated in rice.

22 lation was dependent on the concentration of arsenic added to the irrigation and the arsenic species.

23 The study quantifies the arsenic adsorption capacity of kaolinite at high tempera

24 II) (hydr)oxide precipitation and decreasing arsenic adsorption onto iron(III) (hydr)oxide.

25 Our global prediction map includes known arsenic-affected areas and previously undocumented areas

26 ensor could detect 0.6 and 0.2 ppb levels of arsenic after 7 and 14 days respectively by exploiting t

27 d Pseudomonas aeruginosa, whose tolerance to arsenic also involves the biosynthesis and transport of

28 Rice accumulates arsenic, an established lung toxicant.

29 rates of 79% for cadmium, 81% for inorganic arsenic and a 66% for DMA.

30 ed to study the seasonal variations of total arsenic and all species known to exist in PM.

31 etical study of halogen bonds to phosphorus, arsenic and antimony in the solid state.

32 Arsenic and cadmium are known cardiovascular toxicants t

33 Our findings of associations for arsenic and cadmium exposures with vascular brain injury

34 We measured arsenic and cadmium in American Indian participants from

35 n Indian elders had measured values of urine arsenic and cadmium several times higher than previous p

36 ibitions of D. mccartyi metabolism caused by arsenic and can inform the design of bioremediation stra

37 d the highest Na and the lowest Se contents; arsenic and Cd levels were found highest in verbena whic

38 accumulation of pro-oxidant elements such as arsenic and depletion of anti-oxidant elements such as z

39 ls detected significant associations between arsenic and higher burden of WMH [grade increase = 0.014

40 fluctuating and transition zones where both arsenic and iron phases are present in oxidized forms, b

41 Arsenic and its inorganic species: As (III), As (V), dim

42 m while positively associated with antimony, arsenic and lead.

43 anganese and arsenic together increased both arsenic and manganese retention.

44 In environments where arsenic and microbes coexist, microbes are the principal

45 evels in some cases constitute >50% of total arsenic and might deserve more attention.

46 Total arsenic and other redox-reactive metal elements were mea

47 A positive correlation between arsenic and phosphorus concentrations in modern groundwa

48 Arsenic and trichloroethene (TCE) are among the most pre

49 bility of trace element contaminants such as arsenic and uranium in irrigated unsaturated soils, acco

50 Cadmium, inorganic arsenic and, potentially, dimethyl arsenic acid are carc

51 atio assessment suggested that metals (i.e., arsenic) and PFOA were the top ranked pollutants that ha

52 ed hyperaccumulator thresholds for chromium, arsenic, and cadmium.

53 axa were associated with levels of antimony, arsenic, and mercury, after adjusting for multiple testi

54 s essential to the carbon, nitrogen, sulfur, arsenic, and selenium biogeochemical cycles.

55 el responses to two heavy metals, copper and arsenic, and their binary mixture across time.

56 cers were used to examine trends in nitrate, arsenic, and uranium concentrations in groundwater benea

57 4 (95% CI: 0.000, 0.028) per 10% increase in arsenic]; and between cadmium and presence of lacunar in

58 heavier p-block elements such as phosphorus, arsenic, antimony, and even bismuth.

59 are enriched in trace metal(loid)s, such as arsenic, antimony, molybdenum, and tungsten.

60 llular functions, others such as cadmium and arsenic are inherently cytotoxic.

61 y metals (mainly mercury, lead, cadmium, and arsenic) are summarized.

62 mical similarity between trivalent inorganic arsenic (arsenite) and antimony (antimonite), we hypothe

63 e that life has adapted to use environmental arsenic as a weapon in the continuing battle for dominan

64 Recent studies often focus on inorganic arsenic as arsenite and arsenate, neglecting the organoa

65 s with real wastewater and concentrations of arsenic as low as 10 ppb.

66 up to 35% of the water+H(2)O(2) extractable arsenic as methylated species, but only dimethylarsinate

67 of the light-sensitive and metastable yellow arsenic (As(4)) is extremely challenging.

68 Inorganic arsenic (As(i)) was the principal form of arsenic in wet

69 lective capture and remediation of trivalent arsenic (As(III)) is a central challenge for water purif

70 Speciated urinary arsenic (As) (inorganic + monomethyl + dimethyl As) was

71 ncrease the concentration of uranium (U) and arsenic (As) above the maximum contaminant levels in wat

72 concentrations of toxic metalloids, such as arsenic (As) and antimony (Sb), on larval amphibians are

73 In bog vegetation and peat, arsenic (As) concentrations and accumulation rates are 1

74 Arsenic (As) exposure adversely affects neurodevelopment

75 was used as an adsorbent for the removal of arsenic (As) from the contaminated water.

76 ur (S) has a contrasting role in the fate of arsenic (As) in peatlands.

77 ce yield is diminished due to the buildup of arsenic (As) in soil from irrigation with high-As ground

78 erials produces wastewater with co-occurring arsenic (As) ions and CeO(2) NPs.

79 Arsenic (As) is a carcinogenic element threatening the h

80 Arsenic (As) is a highly toxic contaminant in the enviro

81 Arsenic (As) is a well-known environmental contaminant.

82 tural organic matter (NOM) can contribute to arsenic (As) mobilization as an electron donor for micro

83 Microbially-mediated methylation of arsenic (As) plays an important role in the As biogeoche

84 the digestion process can alter the forms of arsenic (As) present in food.

85 Effective arsenic (As) removal from groundwater is a pressing need

86 vironmentally relevant mineral substrate for arsenic (As) sequestration in reduced, subsurface enviro

87 als (cadmium (Cd), chromium (Cr), lead (Pb), arsenic (As)) which was found to be < 10%.

88 Chronic exposure to arsenic (As), a human toxicant and carcinogen, remains a

89 s for toxicological effects of cadmium (Cd), arsenic (As), and their binary mixture (Cd/As(mix)) incl

90 chocolate was established and validated for arsenic (As), cadmium (Cd), lead (Pb), and antimony (Sb)

91 cycling, including the toxic trace metalloid arsenic (As).

92 n be used for the decentralized treatment of arsenic(As)-contaminated groundwater.

93 h special focus on resistance to cadmium and arsenic, as well as to biocides and antibiotics.

94 The arsenic-associated CpGs from the meta-analysis were enri

95 xposure resulted in differential declines in arsenic-associated disease.

96 Although arsenic at a high concentration imposes strong selective

97 strated as an effective technology to remove arsenic at an affordable price.

98 ents have been performed by adding inorganic arsenic at concentrations of 10, 100 and 1000 ug L(-1) v

99 icals contained a sulfur atom attached to an arsenic atom.

100 dium), and 12 with known toxicity (antimony, arsenic, barium, beryllium, cadmium, cesium, lead, mercu

101 Together these results suggest arsenic-based metabolisms support organic matter product

102 Speciation reactions influence arsenic behaviour in environmental systems, directly aff

103 Arsenic binding stabilizes the DNA-binding loop-sheet-he

104 s provides substantial support for an active arsenic biogeochemical cycle on the anoxic Archean Earth

105 methods to investigate the evolution of the arsenic biogeochemical cycle.

106 Manganese and arsenic both threaten groundwater quality globally, but

107 Crystal structures of arsenic-bound p53 mutants reveal a cryptic allosteric si

108 equired to adequately assess the atmospheric arsenic burden and subsequent contribution to terrestria

109 Mobilization of uranium and arsenic by land surface activities is suggested by the h

110 alloid levels, including aluminum, antimony, arsenic, cadmium, cobalt, chromium, copper, iron, lead,

111 Arsenic, cadmium, copper, lead, manganese, mercury, sele

112 rate a reducing environment, the presence of arsenic can be problematic because of the potential for

113 PM(2.5) levels of lead, arsenic, chromium, and zinc were significantly enriched

114 rboiled rough rice had an enriched inorganic arsenic compared to nonparboiled milled rice, but parboi

115 of ArsH was linked to a direct action on the arsenic compounds tested, arsH1 and arsH2 genes were exp

116 A survey of highly toxic arsenic compounds, together with some other elements was

117 hate salts, and 3) fluorinated antimony- and arsenic- compounds.

118 Arsenic concentration in maternal drinking water was mea

119 Contrary to other foods, the total arsenic concentration in rice may even be a better healt

120 gregated data points of measured groundwater arsenic concentration.

121 r health hazard indicator than the inorganic arsenic concentration.

122 Larger absolute declines in CWS arsenic concentrations at higher water arsenic quantiles

123 We compared community water system (CWS) arsenic concentrations during 2006-2008 vs. after 2009-2

124 We estimated 3-y average arsenic concentrations for 36,406 CWSs (98%) and 2,740 c

125 The finding explains a steady rise in arsenic concentrations in a pre-Holocene aquifer below s

126 llion people are potentially exposed to high arsenic concentrations in groundwater, the vast majority

127 howed that the average of lead, cadmium, and arsenic concentrations in Indian rice type was significa

128 noi, Beijing and Dhaka sites had annual mean arsenic concentrations that approached or exceeded the W

129 al of arsenic (<10 mug L(-1)) from naturally arsenic-contaminated groundwater sample.

130 r than directly accelerated recharge, caused arsenic contamination of this pre-Holocene aquifer.

131 Arsenic content in grains of red chief genotype was foun

132 The total arsenic content was determined by Hydride Generation-Ato

133 issue and urinary (corrected for creatinine) arsenic content was higher in domestic cats, relative to

134 han dogs, and such foods tend to have higher arsenic content.

135 al a cryptic allosteric site involving three arsenic-coordinating cysteines within the DNA-binding do

136 h available data (N = 687), the median urine arsenic:creatinine ratio was 7.54 mug/g [interquartile r

137 e present evidence of a complete respiratory arsenic cycle, consisting of dissimilatory As(V) reducti

138 w that this downregulation reflects targeted arsenic-dependent degradation of glucose transporters.

139 e spatial and temporal variations in organic arsenic deposition, indicating that local sources of met

140 senate for sorption sites, mobilizing sorbed arsenic derived from past pesticide use or other sources

141 y analytical methods have been developed for arsenic detection in various samples.

142 sors (aptasensors) based-on nanomaterial for arsenic detection, in particular with emphasis on the wo

143 el technology in aptasensors development for arsenic detection, including nucleic acid amplification

144 ineer plant nanobionic sensors for real-time arsenic detection.

145 rochemical pathway for water remediation and arsenic detection.

146 Hog1 promoted arsenic detoxification by stimulating phosphorylation of

147 ducing monomethyl arsenic (MMA) and dimethyl arsenic (DMA).

148 operating times (~hours) to remove dissolved arsenic due to inherent kinetics limitations.

149 , both of which encode proteins that promote arsenic efflux.

150 with 20 day-old rice plants showed lower net arsenic enrichment in IP for plants exposed to monomethy

151 eated a global prediction map of groundwater arsenic exceeding 10 micrograms per liter using a random

152 e research, the molecular mechanism by which arsenic exerts its diabetogenic effects remains unclear.

153 r of differentiation 36 (Cd36), was lower in arsenic-exposed conventional mice but not in AB-treated

154 To assess associations between arsenic exposure and CpG methylation, we used linear reg

155 ts to elucidate potential mechanisms of oral arsenic exposure and diabetes development.

156 he novel and replicable associations between arsenic exposure and DNA methylation at specific CpGs ob

157 We assessed associations between arsenic exposure and genome-wide DNA methylation measure

158 ationwide estimates of public drinking water arsenic exposure are not readily available.

159 These estimates of public drinking water arsenic exposure can enable further surveillance and epi

160 ntional mice exposed to 1 ppm arsenic, while arsenic exposure did not significantly affect the serum

161 Arsenic exposure has been related to numerous adverse ca

162 hat the FAR is facilitating the reduction of arsenic exposure in the US.

163 ghlight a novel molecular mechanism by which arsenic exposure may cause anemia and provide critical i

164 This study assesses the effects of arsenic exposure on the TCE-dechlorinating activities of

165 o characterize potential inequalities in CWS arsenic exposure over time and across sociodemographic s

166 ssing whether differential declines in water arsenic exposure resulted in differential declines in ar

167 Arsenic exposure was related to an increase in LV wall t

168 o determine the ubiquitinated proteome after arsenic exposure, which helped us to identify the ubiqui

169 occurrence data set to estimate public water arsenic exposure.

170 most downregulated proteins in the cell upon arsenic exposure.

171 ad implications for arsenic toxicity and for arsenic-focused chemotherapeutics across human populatio

172 ted mice) were exposed to 0.25 ppm and 1 ppm arsenic for 2 wk.

173 rmula: see text] creatinine-adjusted urinary arsenic [[Formula: see text]].

174 species of arsenic, was the primary form of arsenic found in pet foods.

175 (retention time 19 s) in removing dissolved arsenic from contaminated groundwater in rural Californi

176 s work, we investigate selective sorption of arsenic from simulated groundwaters at pH 8 by a redox-a

177 d-type plants to pre-concentrate and extract arsenic from the belowground environment is exploited to

178 of plants into self-powered autosamplers of arsenic from their environment.

179 Critically, the combination of ATRA and arsenic fully rescues therapeutic response in FLT3-ITD A

180 The inorganic arsenic (iAs) extracted using nanoparticles presented co

181 Chronic exposure to inorganic arsenic (iAs) is a significant public health problem.

182 Inorganic arsenic (iAs) is an environmental toxicant associated wi

183 Inorganic arsenic (iAs/As(2) O(3) (2-) ) is an environmental toxic

184 Arsenic (III) accumulated at higher rates than arsenic (

185 nd could be deployed in the most cadmium and arsenic impacted regions where rice is a staple.

186 glucose transporters are major mediators of arsenic import, providing a potential rationale for this

187 In the context of groundwater arsenic in Bangladesh, we challenge this notion here by

188 Arsenic in dehusked grains was negatively correlated wit

189 aluate levels of mercury, cadmium, lead, and arsenic in dried mushrooms, to determine the effect of c

190 Naturally occurring arsenic in groundwater affects millions of people worldw

191 ence or physical separation of manganese and arsenic in groundwater systems under changing or stratif

192 ple are exposed to toxic levels of dissolved arsenic in groundwater used for drinking.

193 insights into the biogeochemical cycling of arsenic in paddy ecosystems.

194 led stresses of impending climate change and arsenic in paddy soils.

195 Extensive information is available on total arsenic in particulate matter (PM), but little is known

196 of wholegrain rice did not enrich inorganic arsenic in the final milled product.

197 concentration between As(III) and the total arsenic in the sample solution.

198 ied as sorbents for dissolved phosphorus and arsenic in water, respectively.

199 ic arsenic (As(i)) was the principal form of arsenic in wet deposition, with a mean concentration of

200 Arsenic (in As-addition treatments) and Cu in seedling r

201 8 to 2009-2011, mean and 95th percentile CWS arsenic (in micrograms per liter) declined by 10.3% (95%

202 rs hold great promise for rapid detection of arsenic, in particular, nanomaterials-based aptamer sens

203 pore-water arsenite, the more toxic form of arsenic, in the rhizosphere of Californian Oryza sativa

204 Vanadium, nickel, and arsenic increased with time on dialysis while manganese

205 Arsenic-induced liver X receptor/retinoid X receptor (LX

206 xplore whether gut microbiota play a role in arsenic-induced LXR/RXR signaling inhibition and the sub

207 crobiota may be a critical factor regulating arsenic-induced LXR/RXR signaling perturbation, suggesti

208 We found that arsenic interacts with the N- and C-terminal zinc finger

209 Arsenic is a highly toxic heavy-metal pollutant which po

210 Arsenic is a pro-oxidant metallic element.

211 sites, where exposure to lead, cadmium, and arsenic is high.

212 Arsenic is one of these environmental chemicals, with mu

213 h levels of toxic metals such as cadmium and arsenic is only experienced in a handful of special habi

214 Since there was no difference in renal arsenic levels in cats with or without CIN, renal arseni

215 er studies that have shown decreased urinary arsenic levels in the population served by public water

216 E consistently outperformed FeEC in bringing arsenic levels to less than WHO-MCL of 10 mug/L.

217 dults participating in the Health Effects of Arsenic Longitudinal Study (HEALS) who were exposed by d

218 found to be very effective in the removal of arsenic (<10 mug L(-1)) from naturally arsenic-contamina

219 for compliance with the U.S. EPA's 10 mug/L arsenic maximum contaminant level (MCL).

220 s were more likely to continue exceeding the arsenic MCL, raising environmental justice concerns.

221 cides glyphosate, ethephon, and fosamine and arsenic metabolites methylarsonic acid and dimethylarsin

222 Arsenic methylation is dependent on one-carbon metabolis

223 Methylation of iAs by arsenic methyltransferase (AS3MT) controls iAs detoxific

224 C enhanced hepatic S-adenosyl-methionine and arsenic methyltransferase, whereas feeding UL elevated r

225 ansferase, whereas feeding UL elevated renal arsenic methyltransferase.

226 n a partial methylation producing monomethyl arsenic (MMA) and dimethyl arsenic (DMA).

227 Suwannee River DOM (SRDOM) decreased arsenic mobility in the short term (<6 h) via inhibiting

228 opyrite oxidative dissolution, but increased arsenic mobility over a longer experimental time (~7 day

229 ers water chemistry in an aquifer, affecting arsenic mobility.

230 of using alternative sources for generating arsenic moieties and compounds is also discussed.

231 Arsenic occurred most frequently in suboxic to mixed red

232 tion strategy to reduce the toxic effects of arsenic on lipid and cholesterol homeostasis.

233 Inorganic arsenic oxides have been identified as carcinogens in se

234 To combat arsenic pollution and maintain a healthy environment and

235 ic carbon drawn from a clay layer into a low-arsenic pre-Holocene (>12 kyr-old) aquifer promotes the

236 e, In(I)Cl smoothly reacts with a tris(amino)arsenic precursor to yield colloidal InAs quantitatively

237 By combining the global arsenic prediction model with household groundwater-usag

238 need to develop novel analytical methods for arsenic, preferably with the potential for the field-tes

239 tum register using the nanoscale ensemble of arsenic quadrupolar nuclear spins as its hardware.

240 n CWS arsenic concentrations at higher water arsenic quantiles indicate declines are related to MCL i

241 onments and our findings of preadaptation to arsenic raise the intriguing possibility that nematodes

242 to potential prevention and intervention for arsenic-related anemias.

243 try, we investigate the causal mechanisms of arsenic release during MAR via injection in the Orange C

244 In the presence of O(2) arsenic remained oxidized as arsenate under all conditio

245 The effective removing technology for arsenic remains challenging, and one of the reasons is d

246 el to describe the kinetics and mechanism of arsenic removal by the modified DPSC method with this mo

247 and XAS data, we conclusively show that poor arsenic removal in FeEC arises from incomplete As(III) o

248 Arsenic removal was achieved upon aggregation of the As(

249 extremophile sister species [12] reveal that arsenic resistance is a common feature of the genus and

250 Arsenic respiratory genes were expressed in metatranscri

251 Here we tested manganese and arsenic retention under conditions commonly found within

252 increased manganese retention but decreased arsenic retention, while the presence of manganese and a

253 In 2001, the US EPA published the Final Arsenic Rule (FAR) for public drinking water, reducing t

254 nd device integration for the portability of arsenic sensors.

255 e, which was coprecipitated with uranium and arsenic, served as the only iron source in soil.

256 The toxicity of arsenic significantly threatens environmental and human

257 ecrease by 39% compared to yields at today's arsenic soil concentrations.

258 sphorus compound was used as solid-phase for arsenic speciation analysis in seafood samples by ICP-MS

259 ed thioarsenates contribute substantially to arsenic speciation besides the much-better-investigated

260 ast elemental speciation analysis: inorganic arsenic speciation was selected as the first case study

261 exist, microbes are the principal drivers of arsenic speciation, which directly affects bioavailabili

262 s, with trithioarsenate typically dominating arsenic speciation.

263 The accumulation and transformation of arsenic species have been studied in the context of hydr

264 Presented here is a 2 year study quantifying arsenic species in atmospheric deposition collected dail

265 It also was the major thiolated arsenic species in the rhizosphere with concentrations c

266 itution is avoided, and oxidation of reduced arsenic species is minimized.

267 e-reduction of iAs (V) and quantification of arsenic species was accomplished via calibration with on

268 a pre-soaking procedure, while also reducing arsenic species.

269 n of arsenic added to the irrigation and the arsenic species.

270 reduced by PvGSTF1 to arsenite, the form of arsenic stored in the vacuoles of this fern.

271 Arsenic, sucralose, perfluorooctanoic acid (PFOA), atraz

272 access and monitor the internal dynamics of arsenic taken up by the plants via the roots.

273 a can tolerate and hyperaccumulate levels of arsenic that would be toxic to other plants and animals.

274 rate and hyperaccumulate very high levels of arsenic that would kill any other plant or animal outsid

275 osphorus centre into the vacant p-orbital at arsenic; the bonding in 2 has been probed by DFT calcula

276 and require multistep processes to transform arsenic to the less harmful As(V) state.

277 tention, while the presence of manganese and arsenic together increased both arsenic and manganese re

278 n genomic insights into the genetic basis of arsenic tolerance in Andean populations, and utilize thi

279 Amendment of amino acids enhanced arsenic tolerance of D. mccartyi.

280 o skin pigmentation, to hypoxic response, to arsenic tolerance.

281 olved a simple, bacterial-like mechanism for arsenic tolerance.

282 The gut microbiota can influence arsenic toxic effects.

283 rther investigated as potential mediators in arsenic toxicity and as biomarkers of exposure and effec

284 This finding has broad implications for arsenic toxicity and for arsenic-focused chemotherapeuti

285 Arsenic toxicity and mobility in groundwater depend on i

286 Arsenite oxidation decreases arsenic toxicity and mobility in the environment, and th

287 Arsenic trioxide (ATO) was used to investigate the effec

288 identification of small molecules, including arsenic trioxide (ATO), an established agent in treating

289 ing to an increased apoptosis rate following arsenic trioxide treatment.

290 senic (III) accumulated at higher rates than arsenic (V).

291 differences in means and quantiles of water arsenic (via quantile regression) between both 3-y perio

292 A greater accumulation of arsenic was found in roots (0.44-4.10 mg kg(-1)) than in

293 Urine arsenic was higher in domestic cats and dogs with CIN.

294 Arsenic was measured in urine as the sum of inorganic an

295 In some Icelandic hot springs, arsenic was nearly quantitatively thiolated.

296 Inorganic arsenic was present in significant amounts in all sample

297 n organic and relatively harmless species of arsenic, was the primary form of arsenic found in pet fo

298 Sensitivity analyses for urinary arsenic were consistent with rice findings.

299 sential for Hog1-mediated protection against arsenic, were dispensable for the response to osmotic st

300 higher in conventional mice exposed to 1 ppm arsenic, while arsenic exposure did not significantly af