ライフサイエンスコーパス: arterial lesion

1 of their propensity to spontaneously develop arterial lesions.

2 co-localizes with macrophages within murine arterial lesions.

3 that have localized at sites of experimental arterial lesions.

4 raft dysfunction relies on the assessment of arterial lesions.

5 only after histopathologic evaluation of the arterial lesions.

6 anti-inflammatory M2 phenotype in spleen and arterial lesions.

7 ) for evaluating severely calcified coronary arterial lesions.

8 onal properties of resident B cells in human arterial lesions.

9 of 942 patients underwent PI to treat 2,450 arterial lesions (2,365 stenoses or occlusions, 85 aneur

10 Kawasaki disease (kDa) may develop coronary arterial lesions and subsequent coronary events.

11 onstrated reduced KIS gene expression within arterial lesions and these lesions were significantly sm

12 intima is a key event in the development of arterial lesions, apoptosis of VSMCs also plays an impor

13 umber (51%) with previous arterial events or arterial lesions at molecular diagnosis.

14 baseline NIHSS score, lesion volume on DWI, arterial lesion by magnetic resonance angiography, and c

15 hat MMP-9 is critical for the development of arterial lesions by regulating both SMC migration and pr

16 this was paralleled by a marked reduction in arterial lesion C3 deposition despite similar levels of

17 fatal arterial event or any new or worsening arterial lesions detected by systematic head-to-pelvis c

18 chromosome 6 gene, which we call Artles (for arterial lesions), did not affect endothelial cell respo

19 Thus, specific arterial lesions (endarteritis, fibrinoid necrosis, acti

20 The arterial lesions following femoral artery injury in LZ/A

21 Arterial lesions form at sites of low and disturbed bloo

22 Podocan-deficient mice showed increased arterial lesion formation compared with wild-type litter

23 xacerbates injury- or hyperlipidemia-induced arterial lesion formation in mice, possibly by excessive

24 Relative to apoE knockout (KO) mice, arterial lesion formation was significantly decreased in

25 ired endothelial regeneration contributes to arterial lesion formation.

26 trating macrophages, which closely resembles arterial lesions from NF1 patients.

27 Compared with WT mice, arterial lesions grew significantly larger in Tg(p22vsmc

28 iting endothelial regeneration and promoting arterial lesion growth in conditions of endothelial inju

29 In addition, SMC migration and arterial lesion growth were significantly impaired in MM

30 Arterial lesions in cardiovascular diseases are characte

31 and inflammatory burden of acute and chronic arterial lesions in mice.

32 ell subtypes linked to severity of pulmonary arterial lesions in PAH.

33 We have shown that occlusive arterial lesions in patients with heterozygous ACTA2 mis

34 Genes differentially expressed in arterial lesions included 3 products encoded by the mito

35 Reinjury of rat arterial lesions induces an increase in lesion size that

36 percentage of the volume of human restenotic arterial lesions is occupied by extracellular matrix (EC

37 ion into lipid-laden macrophages at sites of arterial lesions, leading to the development of atherosc

38 ix metalloproteinase 9 (MMP-9) is present in arterial lesions of GCA and may be involved in its patho

39 lasty has become particularly attractive for arterial lesions of Takayasu arteritis.

40 that contrast gradient attenuation along an arterial lesion, or transluminal attenuation gradient (T

41 sative relation between oxidative stress and arterial lesion progression remains unclear.

42 The 2-dimensional representation of the arterial lesion provided by angiography is limited in di

43 NE) in the plasma and protein-HNE adducts in arterial lesions than AR(+/+)/apoE(-/-) mice.

44 Chronic rejection in xenografts involves arterial lesions that bear some histological similaritie

45 e characterized by cholesterol-induced focal arterial lesions that impair oxygen delivery to the hear

46 BP allows IFAS to improve early subclinical arterial lesions that tend to progress to plaques and cl

47 A reduction of progression of arterial lesions was observed at all sites.

48 The type and location of coronary arterial lesions were determined by coronary angiography

49 Relevant arterial lesions were frequently detected in the middle

50 All arterial lesions were of atherosclerotic etiology.

51 Their coronary arterial lesions, which were strikingly similar to human

52 ection is characterized by the appearance of arterial lesions with concentric intimal thickening.