ライフサイエンスコーパス: arterial pressure

1 brovascular counter-regulation of changes in arterial pressure.

2 dant increases of resting metabolic rate and arterial pressure.

3 th radiotelemetry devices for recording mean arterial pressure.

4 ressed partly by increases in heart rate and arterial pressure.

5 this exposure to a 5 mm Hg reduction in mean arterial pressure.

6 adjusted for age, body mass index, and mean arterial pressure.

7 voke a pressor reflex known to increase mean arterial pressure.

8 Mean pulmonary arterial pressure.

9 vides prognostic utility beyond that of mean arterial pressure.

10 c BP, diastolic BP, pulse pressure, and mean arterial pressure.

11 rdiovascular outcomes, independently of mean arterial pressure.

12 d a norepinephrine infusion to maintain mean arterial pressure.

13 brain hypoperfusion during acute increase in arterial pressure.

14 n II, and resulted in better preservation of arterial pressure.

15 Neither candesartan nor CAP affected arterial pressure.

16 n 21% O(2) ), to selectively lower pulmonary arterial pressure.

17 67 dynes; p < 0.001) with no effects on mean arterial pressure.

18 echocardiographic cardiac index and the mean arterial pressure.

19 ricular stroke volume without affecting mean arterial pressure.

20 affected by baseline serum lactate and mean arterial pressure.

21 ean arterial pressure was above optimal mean arterial pressure.

22 d this correlated with post-brain death mean arterial pressures.

23 st circumference (-1.1 to -1.9 cm), and mean arterial pressure (0.0 to -1.1 mm Hg) at 6 months and Fr

24 8.7, -5.1]; p(group) < 0.0001), similar mean arterial pressure (-1.1 mm Hg [95% confidence limit, -2.

25 0+/-4 versus 6+/-3 mm Hg; P=0.02), pulmonary arterial pressure (22+/-8 versus 11+/-4 mm Hg; P=0.0001)

26 ation, terlipressin decreased mean pulmonary arterial pressure (-6.5 +/- 1.8 mm Hg; p = 0.005) and te

27 7 +/- 451 mL; p = 0.005), and mean pulmonary arterial pressure (-7 +/- 1 mm Hg; p < 0.001) and increa

28 7 mm Hg; 95% CI, -25 to -8; p < 0.001), mean arterial pressure (-7 mm Hg; 95% CI, -12 to -1; p = 0.02

29 : 0.4, 1.7; p = 0.001), 0.8-mmHg higher mean arterial pressure (95% CI: 0.2, 1.4; p = 0.01), and no s

30 nd COMACARE (Carbon Dioxide, Oxygen and Mean Arterial Pressure After Cardiac Arrest and Resuscitation

31 In contrast, an elevated mean arterial pressure along with increased central and perip

32 performed a comprehensive time-weighted mean arterial pressure analysis (time-weighted-average-mean a

33 resulted in a 37 mmHg reduction in systolic arterial pressure and 19% inhibition of angiotensin conv

34 sin II caused a significant increase in mean arterial pressure and a rapid reduction in catecholamine

35 Antagonism of P2X3 receptors also reduces arterial pressure and basal sympathetic activity and nor

36 assess the relationship between optimal mean arterial pressure and brain tissue oxygenation.

37 elation was observed between changes in mean arterial pressure and cardiac index (r = 0.035, p = 0.79

38 assessed for each time-weighted-average-mean arterial pressure and cumulative-time-below mean arteria

39 ressure analysis (time-weighted-average-mean arterial pressure and cumulative-time-below various mean

40 The lower arterial pressure and enhanced natriuresis during high s

41 s, we demonstrate that ELA and 3 both reduce arterial pressure and exert positive inotropic effects o

42 Approximately 200 had portal venous and arterial pressure and flow measurements before and after

43 timate Pglom in patients from combined renal arterial pressure and flow measurements.

44 ucted a model on the basis of proximal renal arterial pressure and flow velocity measurements that pr

45 urons also resulted in a significant fall in arterial pressure and heart rate that was similar in mag

46 and the hypoxia-induced CR (O(2) -CR), mean arterial pressure and heart rate were significantly grea

47 sociation between time-weighted-average-mean arterial pressure and ICU-mortality for each threshold r

48 in RN-NSC-grafted rats reduced resting mean arterial pressure and increased heart rate in all but 2

49 variability in the relationship between mean arterial pressure and indices of brain oxygenation.

50 An inverse relationship between mean arterial pressure and mortality was identified (p = 0.00

51 MRI, in combination with measurement of peak arterial pressure and MRI-derived timing of valvular eve

52 Low mean arterial pressure and need for high doses of vasopressor

53 howed higher diuresis and natriuresis, lower arterial pressure and plasma NT-proBNP.

54 Mean arterial pressure and PP were continuously recorded and

55 terial baroreflex gain, and provoked smaller arterial pressure and R-R interval fluctuations, which w

56 re no differences between groups in the mean arterial pressure and R-R interval responses to non-burs

57 rum tryptase levels and directly with median arterial pressure and recurrent flushing episodes.

58 ncreased risk for incident CVD, whereas mean arterial pressure and relative wave reflection (correlat

59 Baseline mean arterial pressure and renal blood flow were similar in b

60 o-obliteration leading to elevated pulmonary arterial pressure and resistance, right ventricular dysf

61 tion, the steady-state relationships between arterial pressure and sodium excretion, a correlation th

62 ru), a setting that increases both pulmonary arterial pressure and sympathetic outflow.

63 adratic) association between the lowest mean arterial pressure and the primary outcome of myocardial

64 minute; P<0.01) despite a reduction in mean arterial pressure and was inversely related to pulse pre

65 strongly correlated with pulse and systolic arterial pressures and with total arterial stiffness, re

66 activity index and identify the optimal mean arterial pressure, and 3) assess the relationship betwee

67 D with forward pressure wave amplitude, mean arterial pressure, and global reflection coefficient der

68 tonically suppresses splanchnic SNA (SSNA), arterial pressure, and heart rate via projections to the

69 lla (RVLM) lower sympathetic nerve activity, arterial pressure, and heart rate, or when administered

70 percentage of total blood volume (TBV), mean arterial pressure, and heart rate, which were recorded a

71 iomarkers, such as WBC, oxygen content, mean arterial pressure, and heart rate, yielded estimation ac

72 ldosterone improved 5-day survival, invasive arterial pressure, and in vivo and ex vivo arterial resp

73 e, uses shock index as a substitute for mean arterial pressure, and incorporates serum lactate as a b

74 ependently decreased PP, did not modify mean arterial pressure, and increased SVR.

75 pressure, cerebral perfusion pressure, mean arterial pressure, and jugular venous bulb oxygen satura

76 cipient age, body mass index, mean pulmonary arterial pressure, and pretransplant diagnosis, higher E

77 s subjects, right atrial pressure, pulmonary arterial pressure, and pulmonary capillary wedge pressur

78 ood pressure, diastolic blood pressure, mean arterial pressure, and pulse pressure from the Internati

79 erformed for systolic BP, diastolic BP, mean arterial pressure, and pulse pressure.

80 s to describe changes in cardiac index, mean arterial pressure, and their relationship to other indic

81 iovascular parameters, including heart rate, arterial pressures, and body temperature.

82 As the patients' actual mean arterial pressure approached optimal mean arterial press

83 Systolic and pulse arterial pressures, as well as indices of vascular funct

84 ts during vasoactive drug-induced changes in arterial pressure assessed at the internal carotid and v

85 Every 10 mmHg drop from baseline in mean arterial pressure associated with a 3% increase in ische

86 There was no difference in mean arterial pressure at 1, 24, or 48 hours between groups.

87 e blood volume, subsequent titration of mean arterial pressure at 35 mm Hg), anesthetized and instrum

88 Mean arterial pressure at 6 hours was 72.2 mm Hg in the renin

89 guidelines, which recommend maintaining mean arterial pressure at 85 to 90mm Hg for a week after spin

90 nd point was a response with respect to mean arterial pressure at hour 3 after the start of infusion,

91 on, norepinephrine titrated to maintain mean arterial pressure at preshock values, mechanical ventila

92 imates and invasive measurement of pulmonary arterial pressure at rest and peak exercise were simulta

93 ic pressure was calculated as 0.9 x systolic arterial pressure at the carotid, femoral, and radial ar

94 oups in the median cumulative time with mean arterial pressure below 60 mm Hg (7 vs 7 minutes; differ

95 erial pressure: for 1 mm Hg decrease in mean arterial pressure below 75, 70, 65, 60, and 55 mm Hg, th

96 systolic BP (beta=-4.11; P=2.8x10(-4)), mean arterial pressure (beta=-3.50; P=8.9x10(-6)), and reduce

97 , consisting of age, oxygen saturation, mean arterial pressure, blood urea nitrogen, C-Reactive prote

98 is not attributable to factors such as mean arterial pressure, blood viscosity and pH.

99 Pulmonary ventilation and pulmonary arterial pressure both rise progressively during the fir

100 an arterial pressure approached optimal mean arterial pressure, brain tissue oxygenation increased (p

101 MCS devices increased forward blood flow and arterial pressure but other effects varied among devices

102 Advancing age and lower mean arterial pressure, but not the presence of a transthyret

103 L-NAME increased mean arterial pressure by approximately 17 mm Hg in both grou

104 Time-weighted-average-mean arterial pressure captures both the severity and duratio

105 ut no effect on macrocirculatory parameters (arterial pressure, cardiac index, heart rate, and pulse

106 PP, mean arterial pressure, cardiac output, SVR, and ascites volu

107 ood pressure, diastolic blood pressure, mean arterial pressure, carotid intima-media thickness and bo

108 s: preflight, late mission and landing day.) Arterial pressure changed systematically from preflight

109 Although mean arterial pressure, characteristic aortic impedance, and

110 uencies and probabilities increased, even as arterial pressure climbed to new levels); or altered pul

111 NOS3 and GUCY1A3 expression and reduced mean arterial pressure, combined them into a genetic score, a

112 he 25th percentile (78 mm Hg) of lowest mean arterial pressure compared with at the median of 87 mm H

113 than the intact group (2-month fall in mean arterial pressure: control-intact, -10 +/- 1 mm Hg; cont

114 cardiographic measures of systolic pulmonary arterial pressure correlated reasonably well with invasi

115 Mean arterial pressure decreased similarly during endotoxemia

116 degree of hemodynamic instability (mean [SD] arterial pressure decreases of 25 [1] and 41 [11] mm Hg,

117 nd 170 mm Hg of steady-state changes in mean arterial pressure, defined as static CA.

118 ve cyclooxygenase-2 inhibitor, abolished the arterial pressure difference between the knockout and co

119 Work rate and mean arterial pressure during exercise were similar in contro

120 regarding its accuracy to estimate pulmonary arterial pressure during exercise.

121 l mice, Pkd1 knockout mice exhibited reduced arterial pressure during high salt intake; this associat

122 y two and fivefold, respectively, normalized arterial pressure during LVR, and lowered plasma lactate

123 We additionally collected mean arterial pressure, end-tidal CO2, and temperature.

124 C) activation and reflexively increases mean arterial pressure; endomorphin release is also increased

125 These results indicate that arterial pressure equilibrates within the endothelium an

126 isodes of hypotension were common, with mean arterial pressure falling by a median of 22 mmHg (interq

127 Arterial pressure fell and then rose in space, and drift

128 In response to hemorrhage, mean arterial pressure fell in all groups, with the fall bein

129 CR, or HS/CR+MC-2 (HS = 40% of baseline mean arterial pressure for 60 minutes; CR = return of shed bl

130 also nominated an appropriate range for mean arterial pressure for each patient during surgery.

131 Time-weighted-average-mean arterial pressure: for 1 mm Hg decrease in mean arterial

132 litude, and 1 ms pulse width, restoring mean arterial pressure from 0 to 37 mmHg.

133 rom 351 +/- 55 to 182 +/- 67 mL/min and mean arterial pressure from 96.7 +/- 18.2 to 41.5 +/- 4.6 mm

134 o extracorporeal membrane oxygenation, lower arterial pressure, fungal pneumonia, and advancing age.

135 5-year-old female uni-x and sham sheep, mean arterial pressure, glomerular filtration rate, and renal

136 ding PTA versus renal stent placement, intra-arterial pressure gradient greater than 10%, diastolic B

137 tropic Score greater than 50 to reach a mean arterial pressure greater than 65 mm Hg despite adequate

138 uded the following physiologic targets: mean arterial pressure greater than 70 mm Hg, cerebral perfus

139 than or equal to 20 mm Hg, decrease in mean arterial pressure greater than or equal to 10 mm Hg, dec

140 s the need for vasopressors to maintain mean arterial pressure greater than or equal to 65 mm Hg and

141 sistent with POPH (defined as mean pulmonary arterial pressure >25 mm Hg and pulmonary vascular resis

142 responders were defined as CI >=10% and mean arterial pressure >=10%, respectively.

143 d treatment with vasopressors targeting mean arterial pressure (>/=65 mm Hg) and blood transfusion (f

144 ood flow in response to transient changes in arterial pressure has been used to assess dynamic CA.

145 s associated with incident CVD, whereas mean arterial pressure (hazard ratio, 1.10; 95% confidence in

146 objective physical variables (including mean arterial pressure, heart rate, respiratory rate, and oxy

147 PDE9-I also produced progressive falls in arterial pressure (HF: p < 0.001), atrial pressure (Norm

148 tion (HR: 23.2; P = 0.01), and baseline mean arterial pressure (HR: 0.92; P = 0.01) were found to be

149 An increased mean arterial pressure in addition to resolution of tachycard

150 atory modulation of sympathetic activity and arterial pressure in both normotensive and CIH hypertens

151 tensive and CIH hypertensive rats, but basal arterial pressure in CIH rats remained higher compared t

152 Iron deficiency augments hypoxic pulmonary arterial pressure in healthy individuals and exacerbates

153 ale: Exogenous angiotensin II increases mean arterial pressure in patients with catecholamine-resista

154 fect lactic acid-mediated reflex increase in arterial pressure in patients with PAD.

155 r agonist), caused a graded increase in mean arterial pressure in rats with sinoaortic denervation an

156 asal blood pressure and acute change in mean arterial pressure in response to angiotensin II (Ang II)

157 erence in the primary outcome of 6-hour mean arterial pressure in septic shock patients receiving vas

158 ained by transcranial Doppler sonography and arterial pressure in the radial artery was obtained by t

159 r 1 (BLT1) receptor with CP-105,696, reduced arterial pressure in the SHR compared to the normotensiv

160 tive arterial elastance despite similar mean arterial pressures in control subjects.

161 At baseline HR, diastolic and mean arterial pressures in IST and POTS were higher versus co

162 After infusion of angiotensin II, mean arterial pressure increased by 61.6 mmHg in MD-NOS1KO mi

163 Mean arterial pressure increased significantly in MD-NOS1KO m

164 investigated whether reductions in pulmonary arterial pressure influenced sympathetic outflow and bar

165 Mean arterial pressure initially decreased further under bolu

166 The Intraoperative Norepinephrine to Control Arterial Pressure (INPRESS) study was a multicenter, ran

167 d perfusion within proximity of optimal mean arterial pressure is associated with increased brain tis

168 Mean arterial pressure is critically important in patients wi

169 nd carotid sinus baroreceptors when systemic arterial pressure is lowered.

170 A significant increase in mean arterial pressure is observed in early adulthood in both

171 In humans, pulmonary arterial pressure is positively related to basal muscle

172 pital location, era, systolic pressure, mean arterial pressure, lactate, bundle compliance, amount of

173 tcomes included 24-hour survival rates, mean arterial pressure, lactate, hemoglobin, and estimated in

174 Exclusion criteria for both groups were mean arterial pressure less than 60 mm Hg, contraindications

175 nificant associations only remained for mean arterial pressure less than 65 mm Hg (odds ratio, 1.07;

176 tality included: a single occurrence of mean arterial pressure less than 65 mm Hg (p = 0.0051) or sys

177 ial pressure less than 80 mm Hg and any mean arterial pressure less than 70 mm Hg.

178 itivity analyses based on every hour of mean arterial pressure less than 80 mm Hg and any mean arteri

179 In two models of arterial pressure loading, IL11 was upregulated in the a

180 sure reactivity index can yield optimal mean arterial pressure, lower and upper limit of autoregulati

181 ty index-based determination of optimal mean arterial pressure, lower and upper limit of autoregulati

182 The mean optimal mean arterial pressure, lower and upper of autoregulation wer

183 oach, we demonstrate that reducing pulmonary arterial pressure lowers basal MSNA in healthy humans.

184 (systolic blood pressure </=90 mm Hg or mean arterial pressure </=65 mm Hg) presenting to the emergen

185 ndomized to strict or usual BP control (mean arterial pressure </=92 mmHg or 102-107 mmHg, respective

186 dynamic deterioration with an intrinsic mean arterial pressure <60 mm Hg during a sustained episode.

187 albumin <2.5 g/dL, heart rate >90 bpm, mean arterial pressure <60 mmHg, white blood cell count >/=15

188 usion or intervention, hypotension (systolic arterial pressure <=90 mm Hg), and pneumonia.

189 or titration suggest a universal target-mean arterial pressure (MAP) >65 mm Hg.

190 es to identify the relationship between mean arterial pressure (MAP) and cerebral blood vessels' diam

191 or reflex (EPR) is defined by a rise in mean arterial pressure (MAP) and heart rate (HR) in response

192 -wide gene-smoking interaction study of mean arterial pressure (MAP) and pulse pressure (PP) in 129 9

193 Hypotension was defined as a mean arterial pressure (MAP) below 65 mm Hg for at least 1 mi

194 to observe the systolic, diastolic, and mean arterial pressure (MAP) correlations.

195 The measured mean arterial pressure (MAP) exhibited measurable deviation f

196 is study aimed to determine the optimal mean arterial pressure (MAP) in patients with AMI and shock a

197 vels of estradiol-17beta (E2) increases mean arterial pressure (MAP) in young female Sprague-Dawley (

198 nt of CA during steady-state changes in mean arterial pressure (MAP) induced by intravenous infusion

199 Mean arterial pressure (MAP) was defined as diastolic BP plus

200 BP), diastolic blood pressure (DBP) and mean arterial pressure (MAP) were significantly (P < 0.05) re

201 Renal blood flow (RBF), mean arterial pressure (MAP), and heart rate (HR) were contin

202 IOP, mean arterial pressure (MAP), and HR increased rapidly and si

203 Mean arterial pressure (MAP), heart rate (HR), BT, motor acti

204 ow systolic blood pressure (SBP) and/or mean arterial pressure (MAP).

205 ood pressure, diastolic blood pressure, mean arterial pressure (MAP)], brachial artery blood flow ( Q

206 dural increase of median SBP (+11%) and mean arterial pressure (MAP, +10%, both p < 0.001), and a uni

207 4-hours after ICU admission (median: 25 mean arterial pressure measurements per-patient).

208 ension (PH) is diagnosed by a mean pulmonary arterial pressure (mPAP) value of at least 25 mm Hg duri

209 that showed correlation with mean pulmonary arterial pressure (mPAP) were used to create a regressio

210 class (WHO FC); and change in mean pulmonary arterial pressure (mPAP), pulmonary vascular resistance

211 ression of Kv1.5 channels), we measured mean arterial pressure, myocardial blood flow, myocardial tis

212 erial load parameters did not change in mean arterial pressure-nonresponders.

213 oventricular CMT-3 attenuated increased mean arterial pressure, normalized sympathetic activity, and

214 o, 2.71; 95% CI, 1.67-4.39; p < 0.001), mean arterial pressure (odds ratio, 0.979; 95% CI, 0.963-0.99

215 agic shock by blood withdrawn until the mean arterial pressure of 30 mm Hg and maintained at this pre

216 c events rose rapidly below an absolute mean arterial pressure of 60 mmHg.

217 a vasopressor requirement to maintain a mean arterial pressure of 65 mm Hg or greater and serum lacta

218 n daily dose of vasopressor to insure a mean arterial pressure of 65-75 mm Hg.

219 in for more than 24 hours to maintain a mean arterial pressure of 70 mm Hg or greater.

220 These data suggest that maintaining a mean arterial pressure of greater than 65 mm Hg may be a reas

221 ension was defined as a decrease in the mean arterial pressure of greater than or equal to 15% compar

222 by a progressive elevation in mean pulmonary arterial pressure, often leading to right ventricular fa

223 o assess the association between lowest mean arterial pressure on each intensive care day, considered

224 ltivariate linear regression models for mean arterial pressure or SVRI in patients with severe malari

225 changes in right atrial pressure, pulmonary arterial pressure, or pulmonary resistance.

226 atinine, bilirubin or albumin, baseline mean arterial pressure, or study design, size or time period.

227 on and magnitude of clinically observed mean arterial pressure outside optimal mean arterial blood pr

228 1) and a two-fold increase in mean pulmonary arterial pressure (p < 0.0001) compared with baseline.

229 Pulmonary embolism increased mean pulmonary arterial pressure (p < 0.0001), pulmonary vascular resis

230 s (p < 0.05), cardiac index (p < 0.05), mean arterial pressure (p < 0.05), PaO2/FIO2 (p < 0.05), and

231 as low recruiters experienced lower systolic arterial pressure (P = 0.008).Conclusions: A single-brea

232 cording of hemodynamic parameters [pulsatile arterial pressure (PAP) and heart rate (HR)].

233 Abnormal pulmonary arterial pressure (PAP) responses to exercise have been

234 ounts of hypotension (defined by lowest mean arterial pressures per day) were strongly associated wit

235 ated that independent of differences in mean arterial pressure, pH and blood viscosity, race accounts

236 bserved that despite a threefold increase in arterial pressure power <0.03 Hz with oscillatory LBP, t

237 e 0.03-0.10 Hz, both cerebral blood flow and arterial pressure power more than doubled.

238 use, blood pressure medication use, and mean arterial pressure, PP quartile was still associated with

239 In vehicle-treated sheep, mean arterial pressure progressively declined from 25 to 32 h

240 an+/-SD, 8.3+/-2.8 per subject) of pulmonary arterial pressure, pulmonary arterial wedge pressure and

241 ure were not correlated with changes in mean arterial pressure (r = 0.0002; p = 0.85).

242 y correlated with decreases in the diastolic arterial pressure (r = 0.92) and to a lesser extent with

243 udy was to examine the effect of RDN on mean arterial pressure, renal function, and the reflex respon

244 n Earth with electrocardiogram, non-invasive arterial pressure, respiratory carbon dioxide concentrat

245 ctrocardiogram, finger photoplethysmographic arterial pressure, respiratory carbon dioxide levels, ti

246 Cardiac index responders and mean arterial pressure-responders were defined as CI >=10% an

247 The mean arterial pressure response to fluid bolus in cardiac ICU

248 eat fluid bolus based solely on lack of mean arterial pressure response to the initial fluid, since t

249 Cardiac index-responsiveness and mean arterial pressure-responsiveness rates were 33% and 56%,

250 etween cardiac index-responsiveness and mean arterial pressure-responsiveness.

251 herapy in the improvement of 6MWD, pulmonary arterial pressure, right atrial pressure, cardiac index

252 Mean arterial pressure SD score increased with HD only.

253 Factors associated with higher cIMT and mean arterial pressure SD-scores were HD group, higher ultraf

254 inephrine required to maintain a target mean arterial pressure; secondary outcomes included hemodynam

255 Wireless transmission of detected arterial pressure signals to a smart phone demonstrates

256 atment prevented the further decline in mean arterial pressure, substantially reduced heart rate and

257 CFH is associated with mean arterial pressure, SVRI, and peripheral perfusion in pat

258 Ventilation, arterial pressure [systolic blood pressure, diastolic bl

259 in maximum negative dP/dt (dP/dt(Min)), mean arterial pressure, systolic pressure, diastolic pressure

260 ungal infection, vasopressor use, and a mean arterial pressure target of 80-85 mm Hg.

261 als are needed to determine the optimal mean arterial pressure-targets in this patient population.

262 the lactic acid-mediated reflex increase in arterial pressure that is MOR stimulation-independent an

263 After the rise in mean arterial pressure, there was a significant increase in c

264 rial pressure and cumulative-time-below mean arterial pressure threshold (55, 60, 65, 70, and 75 mm H

265 ity and duration of hypotension below a mean arterial pressure threshold and cumulative-time-below is

266 e-below is the total time spent below a mean arterial pressure threshold.

267 ssure and cumulative-time-below various mean arterial pressure-thresholds) during the first 24-hours

268 ctrocardiogram, finger photoplethysmographic arterial pressure, tidal carbon dioxide concentrations a

269 ctrocardiogram, finger photoplethysmographic arterial pressure, tidal volume, respiratory carbon diox

270 HS rats (removal of blood to reduce arterial pressure to 30 +/- 2 mm Hg, 90 minutes, followe

271 ters, grafting RN-NSCs restored resting mean arterial pressure to normal levels and remarkably allevi

272 dex was then calculated as the ratio of mean arterial pressure to regional cerebral blood flow.

273 ents with grade A TR signals, mean pulmonary arterial pressure-to-workload ratio at a threshold of 1.

274 reduction of right ventricular and pulmonary arterial pressures, toward normal levels of right-side p

275 roteinuria were repeat transplantation, mean arterial pressure, transplant glomerulopathy, microcircu

276 ontributes significantly to the elevation of arterial pressure under these conditions.

277 increased noradrenaline dose to elevate mean arterial pressure up to 85-90 mm Hg before collecting a

278 Arterial pressure, vagal tone and muscle sympathetic out

279 QRS-gated DPD demonstrated higher pulmonary arterial pressures versus isolated postcapillary pulmona

280 nce (FVC; Doppler ultrasound, brachial intra-arterial pressure via catheter) to local intra-arterial

281 Basal mean arterial pressure was 15 mm Hg higher and glomerular fil

282 Mean arterial pressure was 3.5 mm Hg (4%) higher (median 84.5

283 minophen-induced hypotension, the nadir mean arterial pressure was 64 mm Hg (95% CI, 54-74).

284 ionship did not persist when the actual mean arterial pressure was above optimal mean arterial pressu

285 Arterial pressure was monitored via an arterial catheter

286 LT1) receptors were blocked with CP-105,696, arterial pressure was reduced in the SHR compared to the

287 Mean arterial pressure was reduced to 30 mm Hg for 90 minutes

288 Mean arterial pressure was slightly higher in SHR but within

289 mathematical model for long-term control of arterial pressure was the model of Guyton and Coleman; r

290 aneous vascular conductance (CVC = flux/mean arterial pressure) was expressed as a change from baseli

291 g pump controller parameters and noninvasive arterial pressure waveforms, central aortic pressure, ou

292 Changes in mean arterial pressure were closely correlated with decreases

293 Leg blood flow and mean arterial pressure were determined, whereas leg vascular

294 PVR and mean pulmonary arterial pressure were not significant predictors of pos

295 tmax, systolic, diastolic, and pulse femoral arterial pressure were obtained from the pressure wavefo

296 olume, cardiac output and reductions in mean arterial pressure were similar between age groups and co

297 ar otolith system and a decrease in the mean arterial pressure when a person stands up.

298 ficant increases (P < 0.05; n = 7-8) in mean arterial pressure, which were generally accompanied by s

299 Increasing mean arterial pressure with noradrenaline in septic shock pat

300 one of the other indices including pulmonary arterial pressure (WMD: -0.97 mmHg, 95%CI: -4.39, 2.44,