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1 ting bodies (perithecia) or meiotic progeny (ascospores).
2 carp-forming fungi that physically discharge ascospores.
3 ication of fungicides, which target airborne ascospores.
4  be induced leading to production of meiotic ascospores.
5 a and chlamydospores and in wild habitats by ascospores.
6 and development of barren perithecia without ascospores.
7 ating type and the formation of self-sterile ascospores.
8 y but which were complete devoid of asci and ascospores.
9 le in more mature asci containing developing ascospores.
10  the haploid nuclei are not partitioned into ascospores.
11 l deposited number of spores was highest for Ascospores and Cladosporium.
12  killer haplotypes suppress MSUD even though ascospores are not killed.
13                                              Ascospores are the primary inoculum in Fusarium graminea
14                                              Ascospores are the primary inoculum in Fusarium graminea
15                                              Ascospores are the primary inoculum in the wheat scab fu
16 position of five most prevalent spore genera-Ascospores, Aspergillus, Basidiospores, Cladosporium, an
17 )-anchored protein thought to be involved in ascospore assembly.
18                                  Some mutant ascospores began to bud and underwent additional mitosis
19 nt produced oval, single-celled, binucleated ascospores by selfing.
20 nt for infectious growth and developments of ascospores by the free cell formation process.
21                                 In addition, ascospores carrying null mutations of either gene are in
22 f Spore killer (Sk) x WT (Sk-sensitive), the ascospores containing the Spore killer allele survive, w
23                                              Ascospore delineation does not occur, however, and no se
24 s of temperature, relative humidity (RH) and ascospore density in multiple controlled environment exp
25        Tetrad analyses showed that all small ascospore-derived strains lacked the missing DNA between
26 o direct repeats that was found in all large ascospore-derived strains.
27 rip mutant corrected the defect in ascus and ascospore development in crosses with wild-type.
28  killer targets not only late but also early ascospore development.
29 lement and prevents it from interfering with ascospore development.
30 g that these genes play specialized roles in ascospore development.
31 y shown to control light driven conidial and ascospore development.
32 al pathogen Sclerotinia trifoliorum exhibits ascospore dimorphism and unidirectional mating type swit
33 l in growth and conidiation but defective in ascospore discharge due to the premature breakdown of it
34  editing of its transcripts is important for ascospore discharge in F. graminearum.
35 produced more perithecia and facilitated the ascospore discharge, resembling the phenotype of GzmetE
36 itable allele rescued the defects of amd1 in ascospore discharge.
37  of N. tetrasperma contain four self-fertile ascospores each with nuclei of both mating types (matA a
38              Deletion of FgSRP1 also reduced ascospore ejection and deoxynivalenol (DON) production.
39  Instead of forming four-celled, uninucleate ascospores, Fgama1 mutant produced oval, single-celled,
40 is dispensable for meiosis but important for ascospore formation and discharge.
41 al treatment, but also affected conidiation, ascospore formation and pathogenicity.
42                                              Ascospore formation in yeast is accomplished through a c
43 tant was reduced in growth, conidiation, and ascospore formation.
44 rate, macroconidiation, microconidiation, or ascospore formation.
45                                              Ascospores generated during sexual reproduction are the
46 tead resulted in severe defects in ascus and ascospore genesis.
47 inimum, maximum and optimum temperatures for ascospore germination were 0.0, 29.9 and 21.7 degrees C
48 lar between-slope differences were found for ascospore germination-resistance to acriflavine, with mu
49 spores, it formed eight small, single-celled ascospores in each ascus.
50 evelop on lettuce plants inoculated with dry ascospores in the absence of apparent leaf wetness (requ
51 tical in light of the fact that formation of ascospores in these species requires a long period of ti
52             Instead of producing four-celled ascospores, it formed eight small, single-celled ascospo
53 d a novel screen based on the sensitivity of ascospores lacking dityrosine to treatment with lytic en
54                The Neurospora crassa Asm-1+ (ascospore maturation 1) gene encodes an abundant nucleus
55 , cloning, and molecular analysis of Asm-1+ (Ascospore maturation 1), the Neurospora crassa homologue
56                                    Using the Ascospore maturation-1 (Asm-1) gene, we defined what nee
57 e defective in female sexual development and ascospore maturation.
58 d for formation of female structures and for ascospore maturation.
59 ulated in Fgama1 also resulted in defects in ascospore morphology and budding.
60                                      Progeny ascospores not carrying the killer element fail to matur
61                             The self-fertile ascospores of N. tetrasperma result from first-division
62                                              Ascospores of Neurospora tetrasperma normally contain nu
63  involves microdissecting the four products (ascospores) of a single meiosis and analyzing the config
64 etes produce eight homokaryotic self-sterile ascospores per ascus.
65 otal mutation frequencies over many loci for ascospore pigmentation were 2.3, 3.5 and 4.4% for three
66 ng - self-fertile strains derived from large ascospores produce both self-fertile (large-spores) and
67        The WT RSK protein is dispensable for ascospore production and is not a target of the spore-ki
68 itions, the Delta odeA strain was delayed in ascospore production but produced more ascospores than w
69 d sufficient for perithecial development and ascospore production.
70 cotoxin fumonisin B1 (fum1) among 121 random ascospore progeny from a single cross.
71      Disruption of pairing causes failure of ascospore progeny to mature.
72                               A total of 111 ascospore progeny were analyzed for segregation at 235 l
73 ficantly reduced in virulence and delayed in ascospore release.
74  the most editing sites in Fusarium affected ascospore releasing.
75 ns, the ratio of conidia (asexual spores) to ascospores (sexual spores) is affected by linoleic acid
76 r intermediate (elongated but single-celled) ascospores, suggesting efficient meiotic silencing of un
77 ed in ascospore production but produced more ascospores than wild type over time.
78                       After germination from ascospores, the camk-1 null strains grew slowly, indicat
79 rm or ascus containing intracystic bodies or ascospores, the products of sexual replication.
80 ploids undergo meiosis, but are defective in ascospore wall maturation for they fail to give the fluo
81 ue to the dityrosine-containing layer in the ascospore wall.
82                               Double-deleted ascospores were able to germinate but had a limited capa
83 l cleistothecia carrying a reduced number of ascospores, whereas DeltagprADeltagprB eliminated fruiti
84 ed on its formation of oblate, smooth-walled ascospores within yellow or yellow-green tufts of aerial