ライフサイエンスコーパス: asphyxia

1 in water (19 min) at 37 degrees C (perinatal asphyxia).

2 probe was placed into the peritoneum before asphyxia.

3 tion from anaerobic metabolism during severe asphyxia.

4 o i.v. NaCN (20 microg) and transient (10 s) asphyxia.

5 o 10 degrees C during 30 min after perinatal asphyxia.

6 racteristics induced by repeated episodes of asphyxia.

7 seizures-signs commonly attributed to birth asphyxia.

8 beta 1-null mice die at birth from asphyxia.

9 g-valve-mask (BVM) ventilation for perinatal asphyxia.

10 arbia and hypoxia, which ultimately leads to asphyxia.

11 europrotective effect of cooling after birth asphyxia.

12 a and autoheparinization in drowning-related asphyxia.

13 frequently considered to be caused by birth asphyxia.

14 to 45-minute normocapnic hypoxia followed by asphyxia.

15 e most common resuscitation method for birth asphyxia.

16 None of the patients had birth asphyxia.

17 ve related these reactions to signs of birth asphyxia.

18 , 5% CO(2)), and (d) combined resistance and asphyxia.

19 reporting of prematurity (0.2 % median) and asphyxia (0.3 % median) both significantly increased.

20 eeding (Esbilac; 200 cal.kg(-1).day(-1)) and asphyxia (100% N(2) for 50 seconds followed by cold expo

21 ined as neonatal infections; 331 [30%]), and asphyxia (151 [14%]).

22 rm birth (28%), severe infections (26%), and asphyxia (23%).

23 isease; 2) an etiology of arrest drowning or asphyxia; 3) higher pH, and 4) bilateral reactive pupils

24 The leading causes of neonatal death were asphyxia (35% [291 of 834]), prematurity (30% [247 of 83

25 Asphyxia (37% [246 of 663]) and infection (28% [50 of 17

26 inical data, 476 (39.9%) died from perinatal asphyxia, 562 (47.0%) died from neonatal sepsis, and 156

27 lion, UR 0.717 million-1.216 million), birth asphyxia (9%, 0.814 million, 0.563 million-0.997 million

28 hyxia; however, recent studies indicate that asphyxia accounts for less than 10% of cerebral palsy ca

29 These results demonstrate that asphyxia activates a brainstorm, which accelerates prema

30 One neonatal death occurred from perinatal asphyxia after shoulder dystocia in the standard care gr

31 girl who died of anoxic encephalopathy from asphyxia after the accidental ingestion of fresh hemlock

32 Asphyxia alone did increase blood pressure (+7.0 +/- 1.1

33 improved treatment prospects for babies with asphyxia and altered understanding of the theory of neur

34 ephalography in rats undergoing experimental asphyxia and analyzed cortical release of core neurotran

35 Rats were subjected to 10-min asphyxia and cardiopulmonary resuscitation (CPR).

36 th cow's milk-based formula and subjected to asphyxia and cold stress to develop NEC.

37 The patient had asphyxia and cyanosis confirmed by medical staff when hi

38 enrolled in the High-dose Erythropoietin for Asphyxia and Encephalopathy (HEAL) study.

39 enrolled in the High-dose Erythropoietin for Asphyxia and Encephalopathy (HEAL) trial or who were eli

40 analysis of the High-Dose Erythropoietin for Asphyxia and Encephalopathy (HEAL) trial, which enrolled

41 enrolled in the High-Dose Erythropoietin for Asphyxia and Encephalopathy (HEAL) Trial, which tested t

42 neonates in the High-dose Erythropoietin for Asphyxia and Encephalopathy trial, which recruited parti

43 the treatment of term newborns who sustained asphyxia and exhibit acidosis and/or encephalopathy (wea

44 lecting a stronger association between birth asphyxia and FA in the case group than the control group

45 acental abruption, have an important role in asphyxia and fetal death.

46 aharan Africa and South Asia, with perinatal asphyxia and neonatal sepsis being the leading causes of

47 brain, BDNF may be a potential treatment for asphyxia and other forms of acute injury in the perinata

48 ntial of brain cooling for epilepsy, stroke, asphyxia and other neurological diseases.

49 C, with higher values in patients with birth asphyxia and similar effect sizes as observed for FA.

50 icular fibrillation cardiac arrest and birth asphyxia and tissue plasminogen activator for ischemic s

51 Associations between birth asphyxia and WM regions of interest diffusion metrics, i

52 s, preterm birth and low birth weight, birth asphyxia, and intracranial hemorrhage of the newborn sig

53 rural environment, preterm birth, perinatal asphyxia, and multiple births were associated with an in

54 uses of deaths in 2008 were pneumonia, birth asphyxia, and preterm birth complications, each accounti

55 sepsis, necrotizing enterocolitis, perinatal asphyxia, and the immune thrombocytopenias), aid the pra

56 ia (FiCO2 = 7.5%), hypoxia (FiO2 = 12%), and asphyxia (apnea for 1 min).

57 iencies in brain oxygenation, known as birth asphyxia, are associated with WM of patients with severe

58 Deaths due to asphyxia as well as following acute poisoning with sever

59 In a model of asphyxia-associated pediatric cardiac arrest, hemodynami

60 Heart Association care in a porcine model of asphyxia-associated VF cardiac arrest.

61 Perinatal asphyxia at term remains a significant cause of infant d

62 The combination of resistance and asphyxia both reduced gain and displaced the curve to hi

63 y of brain injury that occurs not just after asphyxia, but also when cerebral perfusion is impaired d

64 Rats offsprings were exposed to 19 min of asphyxia by immersing the uterus horns in water at 37 de

65 Hyperoxia following asphyxia CA is more damaging to the brain and lungs but

66 Upon systemic IR injury initiated by asphyxia CA, hyperoxia-induced injury exacerbated inflam

67 Rats were resuscitated from 10 min of asphyxia CA.

68 Birth asphyxia can cause moderate to severe brain injury.

69 ults suggest that inspiratory resistance and asphyxia cause changes in the baroreceptor reflex which

70 rol group) or exposed to 7.5 min of in utero asphyxia, causing acidosis and hypoxia.

71 During reventilation after asphyxia, CBF increased more in cocaine than in control

72 of ischemic stroke in cerebral palsy; birth asphyxia, congenital malformations, placental pathology,

73 Birth asphyxia constitutes a major global public health burden

74 Birth asphyxia constitutes a major global public health burden

75 ty percent of patients with drowning-induced asphyxia developed overt disseminated intravascular coag

76 ia for 72 hours in infants who had perinatal asphyxia did not significantly reduce the combined rate

77 mework to quantify the welfare impact of air asphyxia during fish slaughter, using rainbow trout as a

78 y the onset of severe hypoxemia and eventual asphyxia during snow burial.

79 3) complications of delivery (uterine atony, asphyxia, emergency Cesarean section).

80 Twenty-two swine underwent 7 minutes of asphyxia followed by ventricular fibrillation and random

81 After 7 minutes of asphyxia, followed by VF, 20 female 3-month-old swine ra

82 ldren younger than 5 years, apart from birth asphyxia, for which a level-2 intervention is available.

83 associated with accidents, cardiac events or asphyxia, generally had normal pH.

84 The receptor density (Bmax) in the untreated asphyxia group was decreased compared to control animals

85 In contrast, Bmax in the allopurinol treated asphyxia group was similar to control (1.06+/-0.37); and

86 without seizures), and evidence of perinatal asphyxia (group 1); and those without other evidence of

87 For cases with perinatal asphyxia, guideline adherence ranged from 12.2% (n = 77)

88 atal morbidity arising from birth hypoxia or asphyxia has not changed significantly in recent years d

89 cerebral palsy are often attributed to birth asphyxia; however, recent studies indicate that asphyxia

90 effects in the newborn, including perinatal asphyxia, hypoxia, and hypercapnia.

91 with meningitis, and 25 (9%) with perinatal asphyxia/hypoxia.

92 n conditions (intrauterine hypoxia and birth asphyxia [ICC, 0.27], other perinatal conditions [ICC, 0

93 show that moderate hypothermia within 6 h of asphyxia improves survival without cerebral palsy or oth

94 aluated using farmed rainbow trout killed by asphyxia in air or percussion.

95 Following severe perinatal asphyxia in both humans and rats, thalamic neurons displ

96 ctivity was decreased from control following asphyxia in both the untreated and treated animals (47.7

97 The role of intrapartum asphyxia in neonatal encephalopathy and seizures in term

98 help determine the prognosis after suspected asphyxia in term infants, including obstetric informatio

99 Hypercapnia, hypoxia, and asphyxia increased contralateral phrenic burst amplitude

100 With severe asphyxia induced by complete cord occlusion for 10 min,

101 ere was no effect of allopregnanolone on the asphyxia induced impairment of the input/output (I/O) cu

102 Asphyxia-induced arousal likely requires the combined ac

103 IHCA, toddler-aged piglets were subjected to asphyxia-induced CA, followed by ventricular fibrillatio

104 Rats were then subjected to 20 min asphyxia-induced cardiac arrest followed by 30 min cardi

105 2 hrs postasphyxia from animals subjected to asphyxia-induced cardiac arrest for 7 or 9 mins (n = 8/g

106 preserving mitochondrial integrity following asphyxia-induced IHCA.

107 ase in long term potentiation at P5, and the asphyxia-induced increase in IP(3)R1 expression in CA1 p

108 ngle dose of this steroid could reduce birth asphyxia-induced losses in hippocampal function at 5 day

109 ur objective was to test the hypothesis that asphyxia induces bleeding by hyperfibrinolytic dissemina

110 nsient global ischemia, we found that ~8 min asphyxia induces considerable injury of CA1 neurons 4 h

111 howed a significant diagnostic group x birth asphyxia interaction (F(1, 843) = 11.46; P = .001), refl

112 layed a significant diagnostic group x birth asphyxia interaction (F(1, 843) = 9.28; P = .002) in the

113 chypnea of the newborn, infective pneumonia, asphyxia, intracerebral hemorrhage, seizure, cardiomyopa

114 Perinatal asphyxia, intraventricular hemorrhage and stroke are com

115 hemic brain injury in survivors of perinatal asphyxia is a frequently encountered clinical problem fo

116 Moderate cooling after birth asphyxia is associated with substantial reductions in de

117 In south Asia, fetal asphyxia is the major cause of stillbirth.

118 Asphyxia is the most common cause of death after avalanc

119 Perinatal asphyxia is the most important cause of acute neurologic

120 mortality and morbidity among neonates with asphyxia is unknown.

121 -six rabbits were submitted to 13 minutes of asphyxia, leading to cardiac arrest.

122 sympatho-respiratory activity induced by CNS asphyxia-like stimuli, suggesting they bestow a life-or-

123 athing disorders with repetitive episodes of asphyxia may adversely affect heart function.

124 ies in the PLIC of adult patients with birth asphyxia may suggest a greater susceptibility to hypoxia

125 Birth asphyxia measures were identified in 15% to 16% of parti

126 After rats were resuscitated from the 10-min asphyxia, mechanical ventilation was restarted at an FIO

127 This applied model of voluntary human asphyxia might have broader implications for the managem

128 In a porcine asphyxia model, we characterized the hemodynamic, volume

129 udden infant death syndrome (n = 544 [44%]), asphyxia (n = 74 [6.0%]), septicemia (n = 61 [4.9%]), an

130 five of neonatal morbidity, comprising birth asphyxia (n=3), septicaemia (n=1), and neonatal convulsi

131 All cases of drowning-induced asphyxia (n=49) were compared with other patients with c

132 Events such as perinatal asphyxia, near drowning, respiratory arrest, and near su

133 t work is to analyze the effect of perinatal asphyxia on different subpopulations of GABAergic neuron

134 derlying causes of stillbirth were perinatal asphyxia or hypoxia (130 [72%] of 180 stillbirths) and c

135 e most common underlying cause was perinatal asphyxia or hypoxia (17 [29%]) and the most common immed

136 187 [42%] of 449 neonatal deaths), perinatal asphyxia or hypoxia (98 [22%]), and neonatal sepsis (50

137 the underlying cause of death was perinatal asphyxia or hypoxia in 60 (53%) and birth defects in 24

138 ith perinatal brain injury (especially birth asphyxia or hypoxia ischemia [HI]) is unclear.

139 Perinatal asphyxia or hypoxia, infections, and birth defects accou

140 With either preterm birth asphyxia or induced acute cerebral hypoxia-ischemia, min

141 records of neonates who died from perinatal asphyxia or neonatal sepsis determined by postmortem dia

142 timal among neonates who died from perinatal asphyxia or neonatal sepsis.

143 t the time of death and had either perinatal asphyxia or neonatal sepsis.

144 ed before causation is ascribed to perinatal asphyxia or other aetiologies.

145 newborn (OR = 1.10; 95% CI, 1.02-1.19), and asphyxia (OR = 1.34; 95% CI, 1.03-1.75).

146 ased to the 60% level, and he had occasional asphyxia over 10 seconds with no thoracic motion after a

147 The perinatal asphyxia (PA) group showed a significant decrease in cal

148 The consequences of perinatal asphyxia (PA) include alterations which may manifest as

149 cantly correlated with the features of birth asphyxia, particularly a history of seizures.

150 irth-weight, congenital anomalies, perinatal asphyxia, postsurgical, and sepsis categories.

151 Co-existing infection/inflammation and birth asphyxia potentiate the risk of developing neonatal ence

152 he first two groups, cardiac arrest followed asphyxia produced by neuromuscular blockade with and wit

153 With no evidence of trauma or asphyxia, profound accidental hypothermia with cardiac a

154 ned a clinically relevant model of perinatal asphyxia providing intrapartum hypoxia in rats.

155 Only 4.4% of neonates (28) with perinatal asphyxia received all recommended treatments.

156 esults suggest that the hypoxic component of asphyxia reduces baroreceptor-vascular resistance reflex

157 alformations (aRR, 1.61; 95% CI, 1.43-1.81), asphyxia-related complications (aRR, 1.75; 95% CI, 1.26-

158 death, congenital anomalies (APC = -7.87%), asphyxia-related conditions (APC = -9.43), immaturity-re

159 ed with increased risks of preterm delivery, asphyxia-related neonatal complications, and congenital

160 at full term and was partly mediated through asphyxia-related neonatal complications.

161 y in full-term children was mediated through asphyxia-related neonatal morbidity.

162 ss index (BMI) in early pregnancy and severe asphyxia-related outcomes in infants delivered at term (

163 Risks of severe asphyxia-related outcomes in term infants increase with

164 Sham-operated piglets (n=8) underwent no asphyxia-reoxygenation.

165 s myocardial injury in newborn piglets after asphyxia-reoxygenation.

166 Preterm birth and term birth asphyxia result in brain injury from inadequate oxygen d

167 Moderate hypothermia after perinatal asphyxia resulted in improved neurocognitive outcomes in

168 5, 1.6), apnea (RR = 5.8, 99% CI: 5.1, 6.5), asphyxia (RR = 8.5, 99% CI: 5.7, 11.3), respiratory dist

169 (term=39 days) and 1h before inducing birth asphyxia, spiny mice dams were injected subcutaneously (

170 Asphyxia stimulates a robust and sustained increase of f

171 d were subjected to formula feeding and cold asphyxia stress or were delivered naturally and were mot

172 ed but increases in formula feeding and cold asphyxia stress, correlating with induced inducible NO s

173 tus more susceptible to the acute hypoxia or asphyxia that can accompany relatively uncomplicated lab

174 real time physiological biomarkers for birth asphyxia that constitutes a major global public health b

175 r of the anterior commissure produced by the asphyxia that was prevented by hypothermic treatment.

176 After resuscitation of an infant with birth asphyxia, the emphasis has been on supportive therapy; h

177 In neonates with asphyxia, the LMA was safe in the hands of midwives but

178 dicolegal significance attached to perinatal asphyxia, the neuropathological basis of this condition

179 of environmental factors, particularly birth asphyxia, the specific cause of cerebral palsy remains u

180 bjected to 7-min (n = 14) and 9-min (n = 14) asphyxia times.

181 be caused by special circumstances, such as asphyxia, trauma, pulmonary embolism, accidental hypothe

182 ed sham rats (all surgical procedures except asphyxia) treated with induced hyperthermia at 24 hrs (n

183 xia-ischaemia in a piglet model of perinatal asphyxia using clinically relevant magnetic resonance sp

184 oxia-ischemia in a piglet model of perinatal asphyxia using magnetic resonance spectroscopy (MRS) bio

185 Birth asphyxia was defined based on measures from standardized

186 No neonatal asphyxia was observed in newborn babies.

187 ceptor responses to hypoxia, hypercapnia and asphyxia were examined in a superfused in vitro rat caro

188 A convenience sample with neonatal asphyxia were monitored for twenty hours in the first da

189 m clinicians accurately identified perinatal asphyxia were more likely to receive BVM ventilation tha

190 anglia and thalami of infants with perinatal asphyxia were predictive of worse clinical outcomes.

191 s to alter CBF regulation to hypercapnia and asphyxia, which may put the drug exposed newborn at risk

192 Asphyxia, which occurs during obstructive sleep apnoeic

193 bilical cord produced moderate but sustained asphyxia, which resolved after the end of the compressio

194 In fact, asphyxia, which was the most stressful, induced a higher

195 sts the hypothesis that repeated episodes of asphyxia will lead to alterations in the characteristics

196 (LV) function after cardiac arrest caused by asphyxia with that of cardiac arrest induced by dysrhyth

197 ally buried by an avalanche typically die of asphyxia within 35 minutes, often making timely rescue i

198 o were cooled for HIE secondary to perinatal asphyxia without CP (cases), and controls matched for ag

199 rmia induced at 24 hrs vs. rats subjected to asphyxia without induced hyperthermia (33 +/- 13 vs. 67

200 thology damage scores than rats subjected to asphyxia without induced hyperthermia (9.3 +/- 1.5 vs. 6

201 fter the initiation of hypothermia for birth asphyxia would result in further improvement.