ライフサイエンスコーパス: asystole

1 (ie, pulseless electrical activity [PEA] or asystole).

2 apy) as well as rhythm (primary or secondary asystole).

3 [95% CI, 0.09-12.26] minutes; P = .047) (vs asystole).

4 le ventricular fibrillation and nonshockable asystole.

5 ebrafish mutant characterized by ventricular asystole.

6 antidepressant overdose was associated with asystole.

7 s presenting in ventricular fibrillation and asystole.

8 se four (16% of total) had potentially fatal asystole.

9 ation at low energies and caused less VT and asystole.

10 balloon catheter during a brief ACh-induced asystole.

11 tion than CPR during postcountershock PEA or asystole.

12 tients with severe episodes and demonstrated asystole.

13 efined as time from WLST to donor mechanical asystole.

14 nduction abnormalities, QTc prolongation and asystole.

15 ced death, with severe bradycardia preceding asystole.

16 /fibrillation or pulseless electric activity/asystole.

17 om respiratory failure, followed by terminal asystole.

18 rioventricular conduction blocks and cardiac asystole.

19 idence and timing of autoresuscitation after asystole.

20 basis of the initial rhythm as shockable or asystole.

21 s until return of spontaneous circulation or asystole.

22 ion period, including the first 2 mins after asystole.

23 rhythm was pulseless electrical activity or asystole.

24 organ function that time from extubation to asystole.

25 ctor of outcome than time from extubation to asystole.

26 ns of (post)ictal cardiac arrhythmias: ictal asystole (103 cases), postictal asystole (13 cases), ict

27 hmias: ictal asystole (103 cases), postictal asystole (13 cases), ictal bradycardia (25 cases), ictal

28 sting was present in 36 patients (86%) (mean asystole, 13.9+/-10.2 seconds).

29 AA included (1) >=3 s electrical pause or asystole; (2) high-grade Mobitz type II atrioventricular

30 40), pulseless electrical activity (20), and asystole (20), in two sets of ten pigs (ten training, te

31 aracterized by bradycardia, tachycardia, and asystole); 22 (9%) patients experienced 38 nonserious ad

32 The first patient had a cardiac asystole 30 min after receiving sofosbuvir and daclatasv

33 = 0.008, OR 1.78) or atrioventricular block/asystole (30% vs. 19%, p < 0.012, OR 1.57) and to requir

34 re pulseless electrical activity (58.7%) and asystole (33.3%).

35 t initial rhythms were nonshockable (83.9%, [asystole = 36.4% and pulseless electrical activity = 47.

36 cumented rhythm at night was more frequently asystole (39.6% [95% CI, 39.0%-40.2%] vs 33.5% [95% CI,

37 rdia 24%, pulseless electrical activity 16%, asystole 48%, other nonshockable 12%; outcomes: Return o

38 571 patients had a first monitored rhythm of asystole (62.6%) and 503 a presumed noncardiac cause of

39 n presentation (41% vs. 30%), women had more asystole (8.8% vs. 7%) and (organized) pulseless electri

40 , pulseless electrical activity (14.6%), and asystole (9.1%).

41 dy tested whether adenosine caused prolonged asystole after transplantation and if it was effective i

42 ular tachycardia/fibrillation in 6 cases and asystole (after approximately 30 minutes of nonresponsiv

43 characterized by 12 minutes of normothermic asystole and a high cardiopulmonary resuscitation rate.

44 Donors with >=50 min between asystole and aortic cross-clamp time in which the heart

45 ed for demographics, cause of death, time of asystole and cold perfusion.

46 postictal apnoea and bradycardia progress to asystole and death.

47 Because of better survival after asystole and PEA, children had better outcomes than adul

48 children vs 11% [2719/24,987] in adults with asystole and PEA; adjusted OR, 2.73; 95% CI, 2.23-3.32).

49 commonly presents with nonshockable rhythms (asystole and pulseless electric activity).

50 Asystole and pulseless electrical activity account for a

51 itals, the initial cardiac arrest rhythm was asystole and pulseless electrical activity in 874 childr

52 ents, 9616 (82.2%) had nonshockable rhythms (asystole and pulseless electrical activity) and 2079 (17

53 iac arrests and separately for nonshockable (asystole and pulseless electrical activity) and shockabl

54 to-definitive care of < or =10 mins (primary asystole) and patients found in ventricular fibrillation

55 bradycardia, ventricular tachycardia, and/or asystole) and the proportion of sK(+) measurements withi

56 spection, 56 patients did not proceed due to asystole, and 134 proceeded to donation.

57 propriate sinus tachycardia and bradycardia, asystole, and atrioventricular blocks) are observed in p

58 m withdrawal of life-sustaining treatment to asystole, and functional warm ischemia time was the time

59 illation, pulseless ventricular tachycardia, asystole, and pulseless electrical activity, as well as

60 Preablation ECVS induced sinus pauses, asystole, and transient atrioventricular block in both g

61 r time, proportionate to the period of donor asystole, and was associated with increasing cellular in

62 F), 45 pulseless electrical activity, and 55 asystole as the initial rhythm.

63 ed incidence of arrhythmias and intermittent asystoles, as well as compromised performance under stre

64 1 and 3 animals were defibrillated into PEA/asystole at 12 minutes.

65 rd of 2 to 5 mins of demonstrated mechanical asystole before declaring death.

66 t a 2- to 5-min observation after mechanical asystole before the declaration of death.

67 Ictal asystole, bradycardia and AV-conduction block were self-

68 urally mediated syncope (NMS) and documented asystole but syncope still recurred in 25% of them at 2

69 rgans based on time from donor extubation to asystole, but data to support this is incomplete.

70 olus intravenously) 5 mins prior to inducing asystole by bupivacaine overdose.

71 rillation, pulseless electrical activity and asystole (by high-dose intravenous pentobarbitone).

72 Death was declared based on cardiac asystole confirmed by auscultation and transthoracic imp

73 Asystole detection at any time during resuscitation is a

74 al and ventricular arrhythmias, heart block, asystole, development of pulmonary congestion, acute mit

75 tricular fibrillation without resuscitation, asystole develops.

76 All patients whose initial rhythm was asystole died before 6 months in both groups.

77 atients with recurrent vasovagal syncope and asystole documented by implantable loop recorder.

78 ng among patients with recurrent syncope and asystole documented by implantable loop recorder.

79 yncope, whereas 94% (15/16) of seizures with asystole duration>6 s led to syncope (P=0.02).

80 No seizure with asystole duration</=6 s led to syncope, whereas 94% (15/

81 Patients who had one or more episodes of asystole during cardiopulmonary resuscitation had the lo

82 Duration of asystole during carotid sinus massage was similar in bot

83 defined as any subsequent rhythm other than asystole during continued prehospital resuscitation.

84 as opposed to pulseless electrical activity/asystole (epilepsy, 26%; no epilepsy, 44%; P=0.002), des

85 observed 76 clinical seizures with 26 ictal asystole episodes, 15 of which led to syncope.

86 ET drugs (n = 25); group 4, postcountershock asystole/ET drugs (n = 18); and group 5, primary or seco

87 ystole/i.v. drugs (n = 39); group 3, primary asystole/ET drugs (n = 25); group 4, postcountershock as

88 During ictal asystole events, 4 patients had left temporal seizure on

89 003 to July 2013 for all patients with ictal asystole events.

90 ccurate predictive tools to estimate time to asystole following the withdrawal of treatment and alter

91 arning model to identify patients with prior asystole from sinus arrest or complete heart block.

92 Pre-hospital asystole from trauma has a universally poor outcome.

93 These latter patients had both asystole >/=3 s (mean 7.6+/-2.2 s) and SBP fall to 63+/-

94 test (bradycardia <40 beats/min for 10 s or asystole >3 s).

95 The incidence of clinically significant asystole (>/=12 seconds after adenosine) was quantified.

96 Ictal asystole>6 s is strongly associated with ictal syncope.

97 Ictal asystole had a mean prevalence of 0.318% (95% CI 0.316%

98 5 minutes or until a significant arrhythmia (asystole, heart block, bradycardia, supraventricular or

99 cale scores, time of extubation, and time to asystole, hypotension, pulseless electrical activity, an

100 lowing groups were defined: group 1, primary asystole/i.v. drugs (n = 39); group 2, postcountershock

101 v. drugs (n = 39); group 2, postcountershock asystole/i.v. drugs (n = 39); group 3, primary asystole/

102 73 +/- 16 years), with presenting rhythms of asystole in 61.5% and ventricular tachycardia or ventric

103 There was one arrhythmic death related to asystole in a single ventricle patient.

104 d by refractory hypotension, bradycardia, or asystole in four patients (two of whom died) and by perf

105 from hemorrhage-induced electrocardiographic asystole in large swine.

106 sepsis; and acute myocardial infarction with asystole in the distribution of the stented vessel).

107 Ictal asystole is a rare, serious, and often treatable cause o

108 f SCD because of pulseless electric activity/asystole is growing, the overwhelming majority of resear

109 thesis that a 2-min observation period after asystole is sufficient for the declaration of death in p

110 ously identify four important abnormalities: asystole, left ventricular dysfunction, right ventricula

111 a complications extraordinarily rare, though asystole may occur as part of the oculocardiac reflex.

112 Asystole might underlie many of these deaths, which woul

113 ation were abnormal ECG alerts, specifically asystole (n = 5), and pulseless electric activity (n = 8

114 of 285 shocks, with 226 shocks that achieved asystole (n=102), organized rhythm (n=120), or monomorph

115 ents (ventricular tachycardia, n = 44; pause/asystole, n = 36; polymorphic ventricular tachycardia/ve

116 going at hospital arrival, arrest rhythm not asystole, no atropine or NaHCO3, fewer epinephrine doses

117 (n = 18); and group 5, primary or secondary asystole/no drug therapy (n = 15).

118 Asystole occurred in 23 (17 died), pulseless electrical

119 ock-induced ventricular tachycardia (VT) and asystole occurred less often after triphasic shocks.

120 ese had documentation of syncope with >/=3 s asystole or >/=6 s asystole without syncope within 12 +/

121 tricular fibrillation is usually followed by asystole or a nonperfusing rhythm.

122 current-induced ventricular fibrillation or asystole or from respiratory arrest secondary to paralys

123 ountershocks (group 1) and patients found in asystole or PEA (primary asystole or PEA) (group 2) were

124 f patients whose first documented rhythm was asystole or PEA (primary asystole or PEA).

125 Patients found in VF who developed asystole or PEA after countershocks (group 1) and patien

126 ng rhythm has a worse prognosis than primary asystole or PEA and may be related to myocardial electri

127 mented pulseless arrest rhythm was typically asystole or PEA in both children and adults.

128 tcome of prehospital VF victims shocked into asystole or PEA with that of patients whose first docume

129 d patients found in asystole or PEA (primary asystole or PEA) (group 2) were included if the reported

130 cumented rhythm was asystole or PEA (primary asystole or PEA).

131 Cardiac arrests in children are typically asystole or PEA.

132 lation, whereas patients with IHCA caused by asystole or pulseless electric activity were stratified

133 ich are associated with better outcomes than asystole or pulseless electrical activity (PEA).

134 r, countershock is most commonly followed by asystole or pulseless electrical activity (PEA).

135 est and decreased survival after in-hospital asystole or pulseless electrical activity arrest.

136 The proportion of cardiac arrests due to asystole or pulseless electrical activity increased over

137 ermia in patients with nonshockable rhythms (asystole or pulseless electrical activity) is debated.

138 ients who experienced nonshockable OHCA (ie, asystole or pulseless electrical activity) were assessed

139 rhythm is most often (81%) nonshockable (ie, asystole or pulseless electrical activity).

140 In patients whose initial rhythm was asystole or pulseless electrical activity, AEDs were ass

141 rdiac arrest, 79.3% had an initial rhythm of asystole or pulseless electrical activity, and 20.7% had

142 l cooling when the initial cardiac rhythm is asystole or pulseless electrical activity, particularly

143 iac arrest with an initial cardiac rhythm of asystole or pulseless electrical activity, pre-hospital

144 tantially worse than those for patients with asystole or pulseless electrical activity.

145 ation from prolonged VF typically results in asystole or pulseless electrical activity.

146 o declined but was offset by more cases with asystole or pulseless electrical activity.

147 patients after pulseless electrical activity/asystole or resistant ventricular fibrillation who were

148 The new valve shows promise for patients in asystole or shock refractory ventricular fibrillation, w

149 which controlled for gender, age, race, and asystole or ventricular fibrillation/ventricular tachyca

150 The prevalence of ventricular fibrillation, asystole, or organized rhythms at 5, 10, or 20 seconds a

151 ng arrhythmia was VF in 48%, PEA in 25%, and asystole/other in the remainder.

152 .98), ventricular fibrillation (p = .14), or asystole (p = .21).

153 During an 8-min asystole period, the animals were randomized to clamp (n

154 nism of hypocalcemia-induced bradycardia and asystole, potentially responsible for the highly increas

155 al occurred with the initiating OHCA rhythm (asystole, pulseless electric activity, or VF/VT).

156 up, whereas prompt epinephrine treatment for asystole/pulseless electric activity was associated with

157 Of 28 842 patients with an IHCA caused by asystole/pulseless electric activity, the rate of 1-year

158 hockable electrocardiographic presentations (asystole/pulseless electrical activity).

159 erapy after resuscitation from 12 minutes of asystole rapidly and reversibly modulated mitochondrial

160 fibrillation who developed postcountershock asystole (secondary asystole) were included.

161 After 8 min of mechanical asystole, the animals were randomly allocated to clamp (

162 rt cold ischemia times (CIT) and constrained asystole times differing by donor age.

163 ized recipients 15, 30, 45, and 90 min after asystole to determine short-term survival patterns, whic

164 The median time from asystole to NRP was 16 min (range 10-23 min).

165 5-minute stand-off period was observed from asystole to skin incision.

166 cy of resuscitation from bupivacaine-induced asystole using lipid emulsion infusion vs. vasopressin,

167 Time from extubation to asystole was 15.9+/-1.9 min and overall warm ischemia ti

168 The prevalence of asystole was 40% (350) in children and 35% (13 024) in a

169 Asystole was achieved with a single dose of bupivacaine

170 However, asystole was an important cause of mortality in sudden c

171 nts with a positive TT even when spontaneous asystole was documented.

172 Mechanical asystole was initially observed in 18 (90%) of 20 cardia

173 ) survived 24 hours after treatment, whereas asystole was observed in 2 patients (0.03%) with 1 resul

174 In 10 patients with ictal asystole, we observed 76 clinical seizures with 26 ictal

175 n times to circulatory arrest and electrical asystole were 8 +/- 1 and 16 +/- 2 minutes, respectively

176 Those presenting with PEA versus VF and asystole were compared with chi(2) tests, ANOVA, and log

177 Times to circulatory arrest and electrical asystole were recorded.

178 veloped postcountershock asystole (secondary asystole) were included.

179 patients (21%) had bradycardia or periods of asystole with subsequent permanent pacemaker insertion.

180 rest whose first documented field rhythm was asystole with time-to-definitive care of < or =10 mins (

181 rolled in the trial (26 shockable rhythm, 10 asystole), with 18 assigned to 34 degrees C and 18 to 32

182 on of syncope with >/=3 s asystole or >/=6 s asystole without syncope within 12 +/- 10 months and met

183 nous bolus of 20 mg/kg bupivacaine to induce asystole (zero time).