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1 , primarily manifested by significantly less atelectasis.
2 tem bronchus was needed to relieve left lung atelectasis.
3 ry function to those with DPT and no rounded atelectasis.
4 and 20 cm H2O to induce different levels of atelectasis.
5 ailable tool to differentiate pneumonia from atelectasis.
6 ctioning during extubation may contribute to atelectasis.
7 ecision tree to differentiate pneumonia from atelectasis.
8 , pleural effusion, pulmonary opacities, and atelectasis.
9 tilation, and 32 with clinically significant atelectasis.
10 n inflammation, consolidation, flooding, and atelectasis.
11 e sickness to airway obstruction, apnea, and atelectasis.
12 ral ventilation is present it prevents lobar atelectasis.
13 oss of sighs, which was associated with lung atelectasis.
14 tilation in order to induce lobar absorption atelectasis.
15 pressure in the recruitment of diaphragmatic atelectasis.
16 ors, particularly alveolar derecruitment and atelectasis.
17 nical ventilation often develop pneumonia or atelectasis.
18 ty-related changes consistent with dependent atelectasis.
19 ociated lung injury is cyclic recruitment of atelectasis.
20 verage precision scores of 0.25 +/- 0.03 for atelectasis, 0.34 +/- 0.03 for consolidation, 0.33 +/- 0
21 eving macro AUC scores of 0.72 +/- 0.004 for atelectasis, 0.75 +/- 0.007 for consolidation, 0.76 +/-
22 for lungs and pleura labels were as follows: atelectasis, 0.77 (95% CI: 0.74, 0.81); nodule, 0.65 (95
23 15 of 40), pulmonary edema (40%, 16 of 40), atelectasis (10%, 4 of 40), adult respiratory distress s
24 s included hypertrophic scar formation (35), atelectasis (12), pleural effusion (13), recurrent stern
25 two, by slight thickening or minimal linear atelectasis; 16, by thicker linear or subsegmental atele
26 ontinuation of narcotics), 3 (0.3%) cases of atelectasis, 2 (0.2%) corneal abrasions, 1 (0.1%) subacu
29 r in group 1 compared with group 2 patients: atelectasis, 44% vs. 73% (p < .001); postextubation stri
32 nt positive end-expiratory pressure to avoid atelectasis, a tidal volume that is limited to less than
33 lmonary infiltrates, aspiration pneumonitis, atelectasis, acute respiratory distress syndrome, pleura
34 ning in interstitial edema, and deflation in atelectasis all result in similar reductions in dark-fie
35 ventilation) or injury from ventilation with atelectasis and alveolar flooding at end-expiration (ope
37 barrier functions are compromised by purely atelectasis and dysregulated by additional systemic infl
39 e correlates with characteristic patterns of atelectasis and pleural fluid collection on conventional
41 ictive pulmonary disease, ineffective cough, atelectasis and pneumonia, and chronic respiratory insuf
44 tion, C/EBPgamma-deficient newborns die from atelectasis and respiratory failure, which can be mitiga
46 e was placed on a lung area with significant atelectasis and the following parameters measured: (1) t
51 tusion, acute respiratory distress syndrome, atelectasis) and the nonspecific radiographic and clinic
52 tusion, acute respiratory distress syndrome, atelectasis) and the nonspecific radiographic and clinic
53 ators that are focused around the regions of atelectasis, and 2) ventilate in a patient-dependent man
54 four conditions: pneumonia, fluid overload, atelectasis, and acute respiratory distress syndrome.
55 tal lethality, lung abnormalities resembling atelectasis, and dwarfism characterized by aberrant cart
57 duce the FIO2, reduce the risk of absorption atelectasis, and maintain the same alveolar PO2, by incr
58 h monolayer collapse pressures, help prevent atelectasis, and minimize the flow of lung surfactant ou
61 (stroke), pulmonary complication (pneumonia, atelectasis, and prolonged ventilation), and renal compl
63 rmal lungs (baseline), unilateral whole-lung atelectasis, and unilateral and bilateral lung injuries
64 , hyper-attenuated lung, and multi-segmental atelectasis as a diagnostic "triad of anthracosis" was 1
65 EEP are useful not only to treat hypoxia and atelectasis as the consequence of one-lung ventilation,
66 injured pig lung, in the absence of cyclical atelectasis (as determined with dynamic computed tomogra
68 ecame more homogeneous, and 3) the amount of atelectasis at end-expiration decreased from 24% to 16%
69 ree consecutive patients had left lower lobe atelectasis believed to be related to endotracheal tube
70 sing the left atrium and causing medium lobe atelectasis; bilateral pleural effusion was also present
71 normalities (pleuroparenchymal bands, linear atelectasis, bronchiectasis and/or bronchiolectasis) wer
72 onsolidation, pleuroparenchymal band, linear atelectasis, bronchiectasis and/or bronchiolectasis, ret
73 riable shunt fraction, such as with cyclical atelectasis, but it is generally presumed to remain cons
74 days, six of seven animals developed patchy atelectasis; by day 11, two of seven animals demonstrate
75 lations suggests that the static behavior of atelectasis cannot be accurately extrapolated to predict
76 y14 dataset, thoracic abnormalities included atelectasis, cardiomegaly, effusion, infiltration, mass,
77 n, aspiration pneumonitis, pleural effusion, atelectasis, cardiopulmonary edema, and pneumothorax.
78 be-specific agreement was for the finding of atelectasis/consolidation for both pulmonary ultrasound
79 MP alone), total inflammatory changes (i.e., atelectasis/consolidation plus total MP plus ground-glas
81 bilateral pleural effusions and multi-lobar atelectasis/consolidation, which were significantly high
82 ng findings, including pleural effusions and atelectasis, correlated with cytokine release syndrome g
83 issure correlated with a curvilinear band of atelectasis coursing inferomedially and obliquely from i
85 trauma (high-stretch), but not atelectrauma (atelectasis), directly activates monocytes within the pu
86 levated pleural pressure (Ppl) and worsening atelectasis during mechanical ventilation in patients wi
87 vated pleural pressure (P(PL)) and worsening atelectasis during mechanical ventilation in patients wi
89 wo of 24 [8.3%; 95% CI: 0, 20]; P = .04) and atelectasis (eight of 14 [57%; 95% CI: 30, 84] vs six of
90 ch as those due to cancer, pleural effusion, atelectasis, emphysema, infiltrates, ground-glass opacit
91 sure (3 cm H2O) and sustained inflation; and atelectasis group received the same tidal volume as cont
96 pared with sham, LI animals had irreversible atelectasis, higher lung infection rates (P<0.0001) and
97 tress syndrome in preterm infants, including atelectasis, hyaline membranes, and the lack of pulmonar
98 preceded by a recruitment maneuver reverses atelectasis, improves lung mechanics, distribution of ve
99 ring cyclic recruitment and derecruitment of atelectasis in acute respiratory failure and might harm
102 itive end-expiratory pressure for recruiting atelectasis in dependent and diaphragmatic regions.
105 airway pressure is required to recruit lung atelectasis in patients with ARDS and class III obesity
106 airway pressure is required to recruit lung atelectasis in patients with ARDS and class III obesity
107 ges in 21 (33%), perigraft leak in 13 (21%), atelectasis in six (10%), mural thrombus within the sten
108 7, the chest radiographs showed patchy lobar atelectasis in six animals, which cleared by day 11 in a
110 s and linear opacities (fundamentally linear atelectasis) in the first chest X-ray with pathologic fi
112 ventilation in the control group resulted in atelectasis, increased concentrations of bronchoalveolar
119 Overdistension of aerated lung occurs during atelectasis is detectable using clinically relevant magn
122 w, deep ventilation was associated with less atelectasis, less alveolar formation, and more elastin w
123 mal values, and that in lobar pneumonia with atelectasis, lung HA would be further decreased because
129 oxygenation, retractions, capillary refill, atelectasis or pneumonia on chest radiograph, and pleura
130 defined as pneumonia, clinically significant atelectasis, or prolonged mechanical ventilation (> 24 h
134 eep vein thrombosis, respiratory depression, atelectasis, pneumonia, ileus, and postoperative nausea
135 r complications were anastomotic leak (13%), atelectasis/pneumonia (2%), intrathoracic hemorrhage, re
136 infrequent: wound infection/dehiscence, 3%, atelectasis/pneumonia, 2%, intrathoracic hemorrhage, rec
137 scans, four lung compartments-not inflated (atelectasis), poorly inflated, normally inflated, and hy
138 However, differential diagnosis of rounded atelectasis poses a challenge to pulmonary specialists a
139 piratory time constants, and 3) reduce tidal atelectasis, preventing hyperinflation.Methods: Three po
141 ng all offspring within 24 h of birth due to atelectasis-producing pulmonary hypoxia, which recapitul
142 ive days, including pneumonia, bronchospasm, atelectasis, pulmonary congestion, respiratory failure,
144 ations include pleural effusion, hemothorax, atelectasis, pulmonary edema, diaphragmatic dysfunction,
145 eural sparing was also evaluated in cases of atelectasis, pulmonary laceration, and a control group o
148 ent of ARDS, pneumonia, pneumothorax, severe atelectasis, severe hypoxemia, or need for rescue therap
149 rio theorizes that the initiating problem is atelectasis that develops as a result of a surfactant ab
151 ccurrence of alveolar hypoxia and absorption atelectasis, thus optimizing the residual lung function.
153 tasis; 16, by thicker linear or subsegmental atelectasis; two, by contiguous tumor infiltration; one,
154 I, 1.7-2.2; p < 10), including pneumothorax, atelectasis, ventilator-associated pneumonia, hypoglycem
155 130 proteins were differentially abundant in atelectasis versus aerated lung, mostly (n = 126) with l
156 lation between severity of URI and degree of atelectasis was analysed by multiple linear regression.
157 ored using a questionnaire and the degree of atelectasis was defined by sonographic findings showing
159 An animal model of obesity with reversible atelectasis was developed by placing fluid filled bags o
160 , hyper-attenuated lung, and multi-segmental atelectasis was identified as a reliable set of imaging
161 antly different for pneumonia, but dependent atelectasis was more common in patients with early ARDS
163 chiectasis, cicatricial emphysema, and lobar atelectasis were similar in the two patient groups (p >
164 atively combinations of fibrosis, edema, and atelectasis, were present in all premature infants.
165 lowering distal airway pressure may lead to atelectasis, whereas raising distal airway pressure may
166 physiologic changes like edema formation and atelectasis, which are commonly seen in bacterial pneumo
167 General anaesthesia per se also induces atelectasis, which may worsen with URIs and yield detrim
168 similar across groups, with the exception of atelectasis, which was less common in the restrictive ox
170 F-beta signaling were negatively enriched in atelectasis with decreased adhesive glycoprotein THBS1 r