ライフサイエンスコーパス: atopic march

1 ergic disease, is a phenomenon known as the "atopic march".

2 nt of asthma in a disease course called the 'atopic march'.

3 ay account for one mechanism underlying the 'atopic march'.

4 opic dermatitis (AD), a phenomenon known as 'atopic march'.

5 maintenance of allergic inflammation and the atopic march.

6 hma development in a phenomenon known as the atopic march.

7 orbidity, ADAM33 may also be involved in the atopic march.

8 ntervention studies to potentially alter the atopic march.

9 nantly eczema loci increase the risk for the atopic march.

10 on and allergic sensitization initiating the atopic march.

11 tigation into environmental modifiers of the atopic march.

12 tical to establishing a causal nature of the atopic march.

13 econdary prevention strategies to arrest the atopic march.

14 as therapeutic interventions to inhibit the atopic march.

15 s) follow trajectory profiles resembling the atopic march.

16 oietin (TSLP) is thought to drive AD and the atopic march.

17 , may play a critical role in triggering the atopic march.

18 conjunctivitis, a process referred to as the atopic march.

19 AD with the goal to stop or even reverse the atopic march.

20 gnized as being a treatable component of the atopic march.

21 ew/worsened event was considered one step of atopic march.

22 titis, which commonly precedes asthma in the atopic march.

23 c diseases as well as the advancement of the atopic march.

24 ren, as well as for diseases involved in the atopic march.

25 ng that IL-22 plays an important role in the atopic march.

26 esent possible ways to prevent or modify the atopic march.

27 basophils and the underlying drivers of the atopic march.

28 as well as for diseases associated with the atopic march.

29 by eight latent classes: no disease (51.3%), atopic march (3.1%), persistent eczema and wheeze (2.7%)

30 As a key component of the atopic march, AD demonstrates the interactive nature of

31 ut also as a potential means of stopping the atopic march and further progression of this systemic al

32 n this review, we provide an overview of the atopic march and immune mechanism underlying the sensiti

33 data reveal critical roles for IL-33 in the "atopic march" and will offer a new therapeutic target in

34 barrier and immune function genes across the atopic march, and FA in particular.

35 s and how these therapies might mitigate the atopic march by facilitating tolerance.

36 features with these diseases as part of the "atopic march." Controversy exists as to whether immune a

37 other allergic diseases, a link termed 'the atopic march', debate continues as to whether this repre

38 The atopic march describes the progression from atopic derma

39 The atopic march describes this temporal relationship in the

40 bling studies to understand the aetiology of atopic march diseases.

41 s on plausible biological mechanisms for the atopic march focus on defective skin barrier function an

42 The frequently observed atopic march from early AD to asthma, allergic rhinitis,

43 demonstrate a role for TSLP and IL-25 in the atopic march from skin sensitization to food allergic re

44 Although the concept of the atopic march has been refined and strengthened by many c

45 The atopic march has been studied mostly in White population

46 The term "atopic march" has been used to imply a natural progressi

47 The atopic march hypothesis proposes that eczema precedes th

48 Assessment of the atopic march in association with asthma or wheeze reveal

49 We sought to define the atopic march in Black and White children and explore mec

50 pulmonary function with age, reflecting the atopic march in patients with AD.

51 a, there is yet no definitive proof that the atopic march is the primary causal factor in childhood a

52 As AE is a first step in the atopic march, its prevention and appropriate treatment a

53 worsened asthma-like allergy in a model of 'atopic march.' Mice deprived of dietary AhR ligands disp

54 ssion was associated with increased risk for atopic march pathways.

55 manifestations of these symptoms followed an atopic march pattern.

56 s between birth and 11 years: "No disease," "Atopic march," "Persistent eczema and wheeze," "Persiste

57 y framework, with no evidence for sequential atopic march progression.

58 A popular hypothesis known as the atopic march proposes a set of sequential allergy and re

59 The atopic march recognizes the increased occurrence of asth

60 The atopic march refers to the coexpression and progression

61 Atopic march refers to the sequential development of all

62 AD and asthma, the mechanisms underlying the atopic march remain poorly understood.

63 In this context, the atopic march remains a promising area of investigation.

64 While the underlying cause of the atopic march remains unknown, recent evidence suggests t

65 llergic rhinitis, in a process known as the "atopic march." Signaling between epithelial cells and in

66 Mouse models of atopic march suggest that systemic, skin-derived thymic

67 , our findings provide a new paradigm for an atopic march that is inclusive of Black children.

68 triggers and is often the first step in the atopic march that results in asthma and allergic rhiniti

69 xamine the allergic phenotypes that form the atopic march, to determine whether such analyses of data

70 understanding of the role of microbes in the atopic march towards asthma, and in causing exacerbation

71 The atopic march was described more than 20 years ago on the

72 rsening of allergic conditions suggestive of atopic march was observed in a pooled adult/adolescent A

73 also spreads beyond the skin to initiate the atopic march, which includes food allergy, asthma, and a

74 evelopment of allergic conditions across the atopic march, while unique risk loci provide opportuniti

75 s new insights into our understanding of the atopic march, with innate immunity initiating dermatitis