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1 -Feil syndrome, renal agenesis or esophageal atresia.
2  mild pulmonary stenosis to severe pulmonary atresia.
3 bile ducts of mice with experimental biliary atresia.
4 ng sphincter-of-Oddi dysfunction and biliary atresia.
5 ion, showed a limited effect on ovulation or atresia.
6 rapid increase of macrophages and a surge of atresia.
7 ious adverse events in children with biliary atresia.
8 before the surge of macrophage migration and atresia.
9  mimicking the defects associated with canal atresia.
10 coscopic to thoracotomy repair of esophageal atresia.
11 esis and possible therapy for infant biliary atresia.
12 6 years (IQR 0.58-3.0) and 64.3% had biliary atresia.
13 amprey thrives despite developmental biliary atresia.
14 role in pathogenesis in experimental biliary atresia.
15 thout tracheo-esophageal fistula or tracheal atresia.
16  and embryos predominantly develop pulmonary atresia.
17 drives the phenotype of experimental biliary atresia.
18 ion to primary biliary cirrhosis and biliary atresia.
19 mice a partial rupture, resulting in choanal atresia.
20 ith isolated SMCP and/or unexplained choanal atresia.
21 mune response in the pathogenesis of biliary atresia.
22 ion of apoptosis in a mouse model of biliary atresia.
23  have furthered our understanding of biliary atresia.
24  in the pathogenesis of experimental biliary atresia.
25 ant clinical problem in infants with biliary atresia.
26 tion, or to die by programmed cell death, or atresia.
27 ai portoenterostomy for treatment of biliary atresia.
28  mice using an experimental model of biliary atresia.
29 omes, screening, and pathogenesis of biliary atresia.
30 ntributing factor in the etiology of biliary atresia.
31 bility to induce the murine model of biliary atresia.
32 es, and structural proteins at all phases of atresia.
33 jury and obstruction in experimental biliary atresia.
34 ogy of Fallot with complete pulmonary artery atresia.
35 heds light on the underlying causes of canal atresia.
36  of kidney and urinary tract) and esophageal atresia.
37  might be developed for treatment of biliary atresia.
38 ices (EV) in infants with congenital biliary atresia.
39 g thoracoscopy to thoracotomy for esophageal atresia.
40 , resulting in an attenuated form of biliary atresia.
41 e surrounding follicular cells did not begin atresia.
42 the early pathogenesis of congenital biliary atresia.
43 ng laryngeal atresia and the other, tracheal atresia.
44 e balance between follicular development and atresia.
45 s after liver transplantation due to biliary atresia.
46 oses were Ebstein's anomaly (40%), pulmonary atresia (11%), and tetralogy of Fallot (8%).
47 logy of Fallot (15 patients, 25%), tricuspid atresia (12 patients, 20%), Ebstein's anomaly (4 patient
48 omy (5.8% [$21227436]), and small-intestinal atresia (5.1% [$18840546]).
49 dence interval, 45.8 to 64.4); and tricuspid atresia, 74.6% (95% confidence interval, 62.4 to 83.4).
50             Among TOF, 35 (9%) had pulmonary atresia, 98 (26%) had a palliative procedure before repa
51 sus rotavirus (RRV) can also lead to biliary atresia (a neonatal human disease) in mice.
52 est in whether rotavirus could cause biliary atresia, an idiopathic, obliterative infantile disease o
53  congenital malformations comprising choanal atresia, anal abnormalities, post-axial polydactyly, hea
54 e for neonatal disorders such as oesophageal atresia and biliary atresia through clinical trials beca
55 rtility associated with increased follicular atresia and decreased rates of ovulation.
56 f variceal bleeding in children with biliary atresia and high-risk gastroesophageal varices.
57 n causes of cholestatic jaundice are biliary atresia and idiopathic neonatal hepatitis (INH).
58 litis in milder cases with severe intestinal atresia and immunodeficiency in cases with complete loss
59 rs in response to Shh knockdown is pulmonary atresia and is directly related to the abnormal developm
60 ously identified in a patient with tricuspid atresia and large secundum atrial septal defect.
61             A patient with congenital aortic atresia and limited dialysis access options presented to
62             It was associated with pulmonary atresia and moderate or severe aortic regurgitation in u
63           Advances in treatments for biliary atresia and necrotising enterocolitis have been achieved
64 le duct remnant in all patients with biliary atresia and only 1 control.
65 ct remnants, and peripheral blood of biliary atresia and other cholestatic disease controls were char
66 ar and humoral autoimmunity in human biliary atresia and possible interventional strategies therefore
67 formation for assessing prognosis in biliary atresia and primary biliary cirrhosis; and important cli
68  affect infants and children-such as biliary atresia and Reye's syndrome; and (3) redefinition of the
69  on concepts from the mouse model of biliary atresia and rotavirus vaccination programs.
70 wo cases of CHAOS with one showing laryngeal atresia and the other, tracheal atresia.
71            A primary diagnosis of intestinal atresia and the presence of the ileocecal valve may conf
72 hat affect outcomes of patients with biliary atresia and there are no medical therapies that increase
73 virus (RRV)- induced murine model of biliary atresia and whether the T cells are sufficient to result
74 t survival was best in children with biliary atresia and worst in adults with malignancy.
75 (48%) with tetralogy of Fallot and pulmonary atresia, and 4 of 54 (8%) with prosthetic valve endocard
76 ere confirmed (cleft palate, cleft lip, anal atresia, and hypospadias).
77 ver injury and fibrosis in mice with biliary atresia, and increased survival times.
78 ngiomas, pyriform aperture stenosis, choanal atresia, and laryngeal webs, may also have adverse effec
79 rythroblastosis fetalis, septicemia, biliary atresia, and other causes of hyperbilirubinemia.
80                          Male sex, pulmonary atresia, and previous palliations emerged as predictors
81 , familial intrahepatic cholestasis, biliary atresia, and primary sclerosing cholangitis, and clinica
82 sis, primary sclerosing cholangitis, biliary atresia, and progressive familial intrahepatic cholestas
83 apoptotic enterocolitis, multiple intestinal atresia, and recurrent intestinal stenosis.
84 congenital diaphragmatic hernia, oesophageal atresia, and ruptured omphalocele or gastroschisis.
85 ents of autoimmunity exist in murine biliary atresia, and the progressive bile duct injury is due in
86                  The pathogenesis of biliary atresia appears to involve immune-mediated fibro-obliter
87 curring events such as ovulation and ovarian atresia are accompanied with tissue destruction and repa
88 , primary sclerosing cholangitis and biliary atresia are thought to be immune-mediated cholangiopathi
89 ons include traversing occlusions (pulmonary atresia, arterial and venous occlusion, and iatrogenic g
90 etically programmed animal model for biliary atresia, as it loses its bile ducts and gallbladder duri
91 ent hepatoportoenterostomy (HPE) for biliary atresia at 9 U.S. pediatric centers between 1997 and 200
92 ai portoenterostomy as treatment for biliary atresia at the region's largest pediatric hepatology cen
93 omplications of portal hypoplasia in biliary atresia (BA) and acute rejection (AR) are still major co
94 ing Kasai portoenterostomy (KPE) for biliary atresia (BA) and to examine associations between these p
95          Young people (YP) born with biliary atresia (BA) are an emerging population for adult hepato
96 ent in infants with cirrhosis due to biliary atresia (BA) as early as the time of evaluation for live
97                        Children with biliary atresia (BA) have increased maternal cells in their live
98                                      Biliary atresia (BA) is a destructive cholangiopathy of childhoo
99                                      Biliary atresia (BA) is a devastating disease of childhood for w
100                                      Biliary atresia (BA) is a devastating neonatal cholangiopathy th
101                                      Biliary atresia (BA) is a fibroinflammatory obstruction of the e
102                                      Biliary atresia (BA) is a neonatal cholangiopathy of unknown eti
103                                      Biliary atresia (BA) is a neonatal cholestatic liver disease tha
104                                      Biliary atresia (BA) is a neonatal obstructive cholangiopathy th
105                         Extrahepatic biliary atresia (BA) is a pediatric liver disease with no approv
106                                      Biliary atresia (BA) is a progressive fibroinflammatory disorder
107                                      Biliary atresia (BA) is a progressive fibroinflammatory obstruct
108                                      Biliary atresia (BA) is a progressive, inflammatory cholangiopat
109                                      Biliary atresia (BA) is a rare disease in infants, with unknown
110                                      Biliary atresia (BA) is a severe pediatric liver disease resulti
111                                      Biliary atresia (BA) is notable for marked ductular reaction and
112 theory regarding the pathogenesis of biliary atresia (BA) is that bile duct injury is initiated by a
113                                      Biliary atresia (BA) is the most common cause of end-stage liver
114                      The etiology of biliary atresia (BA) is unknown.
115 e, rhesus rotavirus (RRV) can induce biliary atresia (BA), a disease resulting in inflammatory obstru
116                                      Biliary atresia (BA), a progressive fibroinflammatory disorder o
117 th rhesus rotavirus (RRV) results in biliary atresia (BA), and this condition is influenced by the ho
118 iteria to define CCM associated with biliary atresia (BA), or BA-CCM, and correlate presence of BA-CC
119                     In patients with biliary atresia (BA), the extent of intrahepatic biliary fibrosi
120                                      Biliary atresia (BA), the most common cause of end-stage liver d
121 asonography (US) in the diagnosis of biliary atresia (BA), with surgery as the reference standard.
122 o naturally occurring outbreaks of a biliary atresia (BA)-like disease in livestock.
123 er fibrosis, a grave complication of biliary atresia (BA).
124 rotavirus (RRV)-induced experimental biliary atresia (BA).
125 e three main human cholangiopathies (biliary atresia [BA], primary biliary cholangitis [PBC], and pri
126 tional epidermolysis associated with pyloric atresia/beta4 keratinocytes.
127  for children with spina bifida, oesophageal atresia, biliary atresia, diaphragmatic hernia, gastrosc
128 ular septum, single ventricle, and tricuspid atresia born in 1996 to 2003 were identified from the Te
129  populate the livers of infants with biliary atresia, but it is unknown whether neonatal lymphocytes
130                         For example, choanal atresia (CA) is a congenital craniofacial anomaly in whi
131                     In hearts with pulmonary atresia, cardiac neural crest-derived cells, which form
132 s with features of vertebral anomalies, anal atresia, cardiovascular anomalies, trachea-oesophageal f
133 ance to multiple diseases, including biliary atresia, choledochal cysts and gallbladder agenesis.
134 ed immunodeficiency with multiple intestinal atresias (CID-MIA) is a rare hereditary disease characte
135                                      Biliary atresia continues to represent a major challenge with ma
136 d anomalies include ocular coloboma, choanal atresia, cranial nerve defects, distinctive external and
137            Biopsies of patients with biliary atresia demonstrated increased RhoU/Wrch1 and Hey2 expre
138           The reference standard was biliary atresia diagnosed at the region's pediatric hepatology c
139 h spina bifida, oesophageal atresia, biliary atresia, diaphragmatic hernia, gastroschisis, and Down s
140 IM 600992), a condition including esophageal atresia (EA) and tracheoesophageal fistula (TEF), in whi
141 orted in patients after repair of esophageal atresia (EA), but the basis of this association is unkno
142                                   Esophageal atresia (EA/TEF) is a common congenital abnormality pres
143 imary repair (aged <3 months) with pulmonary atresia (early-PA group; n=49), and primary repair betwe
144           Epidermolysis bullosa with pyloric atresia (EB-PA), manifesting with neonatal blistering an
145       During and after developmental biliary atresia, expression of cyp7a1 in intestine increased by
146 sis, including organ and digit duplications, atresia, fistulas, hypertelorism, cleft palate and hamar
147  and haematocervix with cervical and vaginal atresia found on the left side (classification 1.2) with
148                      Among mice with biliary atresia given injections of antioxidants, only NAC reduc
149 operative hemodynamics and overall pulmonary atresia growth were similar, although right pulmonary ar
150      Both groups demonstrated good pulmonary atresia growth.
151 s of ocular coloboma, heart defects, choanal atresia, growth retardation, genital abnormalities, and
152 ere bilateral microphthalmia and oesophageal atresia has a de novo missense mutation, R74P, that alte
153 very of the pathogenic mechanisms of biliary atresia has been limited by the inability to study extra
154 k formation, and that embryos with pulmonary atresia have an outflow tract septum.
155  and neonatal mice with experimental biliary atresia have shown increased expression of proapoptosis
156 , reintervention was associated with virtual atresia (hazard ratio [HR], 0.51; 95% confidence interva
157 dition is characterized by bilateral choanal atresia, hearing loss, cleft lip and/or palate, and othe
158 Relatively common conditions such as biliary atresia, however, remain largely unexplained and the vir
159  mimic of Tetralogy of Fallot with pulmonary atresia; however, subsequent reports describe only a sin
160 uctal stent were less likely to have virtual atresia (HR, 0.36; 95% CI, 0.15-0.85; P=0.02) and more l
161                        Patients with biliary atresia (i.e., obliteration of the biliary tree) suffer
162 neonatal conditions reported were intestinal atresia in 28 (54.9%) studies, abdominal wall defects in
163                           We induced biliary atresia in BALB/c mice by intraperitoneal administration
164  re-examine whether rotavirus causes biliary atresia in children are discussed based on concepts from
165 hest in group A and those <5 kg; and biliary atresia in group C (72.8%).
166 ship between rotavirus infection and biliary atresia in humans.
167  important for biliary infection and biliary atresia in mice.
168 toxin, biliatresone, responsible for biliary atresia in naturally-occurring animal models, that cause
169 erum of mice and children and causes biliary atresia in neonatal mice.
170   Using a model of rotavirus-induced biliary atresia in newborn mice, we found that activated NK cell
171                             Treating biliary atresia in newborns earlier can delay or prevent the nee
172 ing and a significant increase in follicular atresia in the 30 ppb group were observed.
173 ents detected all known infants with biliary atresia in the study population, although the 95% CI aro
174 lts in intestine after developmental biliary atresia, in addition to known mechanisms, such as the re
175 dies in the rotavirus mouse model of biliary atresia indicate that infection of biliary epithelium is
176 onfidence interval, 49.9 to 61.7); pulmonary atresia intact ventricular septum, 55.7% (95% confidence
177 h hypoplastic left heart syndrome, pulmonary atresia intact ventricular septum, single ventricle, and
178 ial intrahepatic cholestasis type 1, biliary atresia, intrahepatic cholestasis of pregnancy, and prim
179                                      Biliary atresia is a devastating disorder of the newborn in whic
180                                      Biliary atresia is a fibro-inflammatory cholangiopathy that obst
181                                      Biliary atresia is a neonatal liver disease with extrahepatic bi
182                                      Biliary atresia is a neonatal obstructive cholangiopathy that pr
183                                      Biliary atresia is a rare disease of infancy, which has changed
184                                      Biliary atresia is an inflammatory fibrosclerosing lesion of the
185                                      Biliary atresia is an inflammatory, fibrosclerosing neonatal cho
186                                      Biliary atresia is associated with oligoclonal expansions of CD4
187 ween ovarian folliculogenesis and follicular atresia is critical for female fertility and is strictly
188 tment typically occurs later because biliary atresia is difficult to detect during its early stages.
189                  The pathogenesis of biliary atresia is not known; one theory is that of a virus-indu
190                         Preantral follicular atresia is observed within the few follicles that develo
191                                      Biliary atresia is the most common cause of end-stage liver dise
192                                      Biliary atresia is the most common cause of end-stage liver dise
193                                      Biliary atresia is the most common cholangiopathy of childhood.
194                                   A follicle atresia kinetic constant (k(A)) was modeled for the two
195 and are associated with extrahepatic biliary atresia, lead to a loss of membrane recognition, but do
196 al and critical care for long-gap esophageal atresia (LGEA) - in comparison to healthy infants - usin
197      Sox17 haploinsufficiency causes biliary atresia-like phenotypes and hepatitis in late organogene
198 iary disease, including extrahepatic biliary atresia, liver disease and transplantation in cystic fib
199                Short stature, auditory canal atresia, mandibular hypoplasia, and skeletal abnormaliti
200 gh much is known about management of biliary atresia, many aspects are poorly understood, including i
201 opment of 46XX females can result in vaginal atresia, masculinization of the urethra, a single urogen
202           Gastroschisis and small-intestinal atresia may be particularly high-yield targets for multi
203                          Multiple intestinal atresia (MIA) is a rare cause of bowel obstruction that
204 ar and cellular basis of multiple intestinal atresia (MIA).
205 astic left heart syndrome (n=346), tricuspid atresia (n=103), tetralogy of Fallot (n=127), pulmonary
206  septal defects (n=22), tricuspid hypoplasia/atresia (n=13), and coronary artery fistulas (n=16).
207 103), tetralogy of Fallot (n=127), pulmonary atresia (n=177), heterotaxy syndrome (n=38), and other (
208  followed by gastroschisis (n=5), intestinal atresia (n=5), and necrotizing enterocolitis (n=4).
209 2], oesophageal atresia [n = 6,303], biliary atresia [n = 3,877], diaphragmatic hernia [n = 6,176], g
210 422], encephalocele [n = 1,562], oesophageal atresia [n = 6,303], biliary atresia [n = 3,877], diaphr
211 and had the underlying diagnoses: intestinal atresia, necrotizing enterocolitis, gastroschisis, and m
212 tions in CHD7 cause Coloboma, Heart Disease, Atresia of Choanae, Retardation of Growth and/or Develop
213 our with learning deficits had hypoplasia or atresia of cingulum.
214  CHARGE (coloboma of the eye, heart defects, atresia of the choanae, retardation of growth and/or dev
215  CHARGE (Coloboma of the eye, Heart defects, Atresia of the choanae, Retardation of growth/developmen
216 yndrome (coloboma of the eye, heart anomaly, atresia of the choanae, retardation, and genital and ear
217 acterized by ocular Coloboma, Heart defects, Atresia of the choanae, Retarded growth and development,
218 acterized by ocular coloboma, heart defects, atresia of the choanae, retarded growth and development,
219 defects (Coloboma of the eye, Heart defects, Atresia of the choanae, severe Retardation of growth and
220 velopment of the mesonephros in males causes atresia of the homolateral ejaculatory duct that results
221 ictly needed, explaining the random culling (atresia) of precursor cells in bilaterians.
222 was increased in patients with porta hepatis atresia (Ohi type II and III vs type I; HR: 2.03, P = 0.
223 nd 123 (39.9%) an operation to treat biliary atresia or choledochal cyst in the preceding year.
224                                              Atresia or hypoplasia of crura and body of fornices was
225 n bile duct (CBD) disorders, such as biliary atresia or ischemic strictures, is restricted by the lac
226 owever, in contrast to patients with biliary atresia or other forms of cholestasis who develop progre
227 stein's anomaly (4 patients, 6.5%), pulmonic atresia or stenosis (7 patients, 11.5%), truncus arterio
228 s with primary biliary cirrhosis and biliary atresia or with Alagille syndrome, two major pediatric c
229 icantly higher occurrence of pulmonary valve atresia (P = 0.001) compared with cases without a detect
230 cases for which 39 (93%) were due to biliary atresia (P<0.001).
231                            In TOF, pulmonary atresia (P=0.003), male sex (P=0.01) and previous pallia
232 vating cytokine IL-33 is elevated in biliary atresia patient serum and in the livers and bile ducts o
233 onfiguration in a paediatric tricuspid valve atresia patient; (3) establish whether the widely used c
234               In the murine model of biliary atresia, perinatal exposure to rhesus rotavirus (RRV) re
235 d compared these phenotypes to the pulmonary atresia phenotype observed following the systemic loss o
236        Senescence may play a role in biliary atresia, primary sclerosing cholangitis, cellular reject
237 tic left heart syndrome and gastrointestinal atresias, probably due to haploinsufficiency for the nei
238 treated with cyclopamine exhibited pulmonary atresia, pulmonary stenosis, and persistent truncus arte
239 ulticenter, double-blind Steroids in Biliary Atresia Randomized Trial (START) was conducted in 140 in
240  immune function, skewed sex ratios, ovarian atresia, reduced egg production, and altered gene expres
241 r, increased apoptosis of GCs and follicular atresia, reduced ovulation rate, and a dramatic decline
242  for anastomotic strictures after esophageal atresia repair is feasible and acceptably safe and provi
243  tonsillectomy and/or adenoidectomy, choanal atresia repair, and/or treatment of gastroesophageal ref
244 as become a routine operation for esophageal atresia repair.
245 n, neonatal hemochromatosis, and the Biliary Atresia Research Consortium have been summarized.
246 ding ocular coloboma, heart defects, choanal atresia, retarded growth and development, genitourinary
247 E syndrome (coloboma, heart defects, choanal atresia, retarded growth, genital anomalies, and ear ano
248 were found for spina bifida, cleft lip, anal atresia, severe congenital heart defects in general, or
249 al injury and obstruction within 7 days, and atresia shortly thereafter.
250 subgroup commonly referred to as the biliary atresia splenic malformation (BASM) syndrome.
251 with cleft palate (aOR = 1.23) and anorectal atresia/stenosis (aOR = 1.40).
252 sia/stenosis (aOR = 2.97) including duodenal atresia/stenosis (aOR = 2.48), and atrial septal defect
253 gmatic hernia (aOR = 2.58), small intestinal atresia/stenosis (aOR = 2.97) including duodenal atresia
254 he prevalence of rectal and large intestinal atresia/stenosis was higher for ART births compared with
255 he aorta, cleft lip, cleft palate, anorectal atresia/stenosis, and limb reduction (upper limit of 95%
256  networks previously unrecognized in biliary atresia, such as the complement components C3ar-1 and C1
257 95-1.01, p = 0.19) and children with biliary atresia surviving with native liver (OR = 0.96, 95% CI 0
258 nique autosomal-recessive lymphedema-choanal atresia syndrome.
259                            Tracheal agenesis/atresia (TA) is a rare but fatal congenital disease in w
260 subjects from families with congenital aural atresia that were heterozygous for TSHZ1 loss-of-functio
261                                      Biliary atresia, the most common cause of childhood cirrhosis, i
262 ; however, a subset of patients with biliary atresia, the most common childhood cholangiopathy, exhib
263 itially designed for patients with tricuspid atresia, this procedure is now offered for a vast array
264 vered that the sea lamprey adapts to biliary atresia through a unique mechanism of de novo synthesis
265 ders such as oesophageal atresia and biliary atresia through clinical trials because of the rarity of
266  we show that androgens attenuate follicular atresia through nuclear and extranuclear signaling pathw
267 d drive the phenotypic expression of biliary atresia, thus constituting a potential therapeutic targe
268  We hypothesized that the T cells in biliary atresia tissue expressed related TCRs, suggesting that t
269 s (7.5% [95% CI, 3.5%-13.8%]) with pulmonary atresia to 497 of 801 (62.0% [58.7%-65.4%]) with coarcta
270 we used a rotavirus-induced model of biliary atresia to investigate the entire biliary transcriptome
271  adults with surgically corrected esophageal atresia/tracheaesophageal fistula (EA/TEF).
272 ntact ventricular septum and 28 with virtual atresia) underwent RV decompression at median 3 (25th-75
273 ollicle reserve is reduced by maturation and atresia until menopause ensues.
274 ll intestine for diseases such as intestinal atresias, volvulus, and necrotizing enterocolitis.
275 plantation or death in children with biliary atresia was determined.
276                                   Intestinal atresia was the most common diagnosis in the deceased gr
277 long with numbers of ovarian macrophages and atresia wave.
278 f cholangiocyte injury in a model of biliary atresia, which is relevant to human BA and may suggest p
279 d chronic biliary disorders, such as biliary atresia, which remains the most common paediatric chroni
280 low-up was predicted by PN dependence and SB atresia, while maximal absolute SB width by birth weight
281 cts including overriding aorta and pulmonary atresia, while none of the sham-operated controls were a
282                   Among infants with biliary atresia who have undergone hepatoportoenterostomy, high-
283 3712]), tracheoesophageal fistula/esophageal atresia (WIQR, $39206; median, $105259 [IQR, $87335-$126
284  identified the 7 known infants with biliary atresia with a sensitivity of 100% (95% CI, 56.1%-100.0%
285 ied included omphalocele, jejunal, and ileal atresia with aberrant mesenteric blood supply, and synda
286 TC7A) for mutations associated with multiple atresia with combined immunodeficiency.
287 aling in the myocardium results in tricuspid atresia with hypoplastic right ventricle associated with
288 ith higher risk of thrombosis were pulmonary atresia with intact ventricular septum (hazard ratio [HR
289 ing catheter valve perforation for pulmonary atresia with intact ventricular septum (PAIVS) 21 years
290 ongitudinal series of fetuses with pulmonary atresia with intact ventricular septum (PAIVS) and/or cr
291      Ninety-nine patients (71 with pulmonary atresia with intact ventricular septum and 28 with virtu
292                      Patients with pulmonary atresia with intact ventricular septum deemed suitable f
293                                    Pulmonary atresia with intact ventricular septum is rarely associa
294 (RV) decompression in infants with pulmonary atresia with intact ventricular septum vary widely.
295 es undergoing RV decompression for pulmonary atresia with intact ventricular septum were included fro
296 -month-old female infant with type I biliary atresia with jaundice (total serum bilirubin, 22.2 mg/dL
297  leads to the common birth defect esophageal atresia with or without trachea-esophageal fistula (EA/T
298 a relatively common birth defect, esophageal atresia with or without tracheoesophageal fistula (EA/TE
299 al diaphragmatic hernia (CDH) and esophageal atresia with tracheoesophageal fistula (EA/TEF) can be r
300 ly repaired tetralogy of Fallot or pulmonary atresia with ventricular septal defect.

 
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