ライフサイエンスコーパス: atrial pressure

1 ce between the V-wave and the nadir of right atrial pressure).

2 result of a lowering of early diastolic left atrial pressure.

3 on end-diastolic arterial pressure and right atrial pressure.

4 n heart rate, mean aortic pressure, or right atrial pressure.

5 -LVDP) was used as a surrogate for mean left atrial pressure.

6 d elevated minimal LV pressure and mean left atrial pressure.

7 gh increases in cardiac output (.Q) and left atrial pressure.

8 using capacity of carbon monoxide, and right atrial pressure.

9 ion of right ventricular function, and right atrial pressure.

10 +/- 6 mm Hg, p < 0.002) and increased right atrial pressure (10 +/- 6 vs. 8 +/- 4 mm Hg, p < 0.05).

11 1 week post-transplant (higher median right atrial pressure (10 mm Hg [8-13 mm Hg] vs 7 mm Hg [5-11

12 roximately 2-fold greater increases in right atrial pressure (10+/-4 versus 6+/-3 mm Hg; P=0.02), pul

13 ] versus 63 [48-82] mL/m(2), P<0.001), right atrial pressure (12+/-4% versus 6+/-4%, P<0.001), and ri

14 us 5.1+/-1.9 L/min; P=0.01), increased right atrial pressure (12+/-5 versus 4+/-1 mm Hg; P=0.0002), a

15 ), P=0.001) and a higher prevalence of right atrial pressure 15 mm Hg (74% versus 60%, P<0.001) on ec

16 and diuretics administered to decrease right atrial pressure (18+/-2 to 7+/-1 mm Hg), pulmonary wedge

17 0 mg groups, respectively; P<0.05) and right atrial pressure (-2.0+/-0.4, -3.7+/-0.4, and -3.5+/-0.4

18 increased wall stress due to increased left atrial pressures; (2) hemodynamic congestion-induced dec

19 44 mm Hg to 12 +/- 6 mm Hg; p = 0.007), left atrial pressure (29 +/- 11 mm Hg to 20 +/- 8 mm Hg; p =

20 Tolvaptan also reduced right atrial pressure (-4.4 +/- 6.9 mm Hg [p < 0.05], -4.3 +/-

21 as tested prospectively in the estimation of atrial pressure 50 patients.

22 There were significant decreases in right atrial pressure (9+/-1 to 7+/-1 mm Hg, p < 0.01 by ANOVA

23 1.04 (1.03-1.06), P < 0.001], and mean right atrial pressure [adjusted HR 1.07 (1.01-1.13), P = 0.015

24 ular resistance, significantly higher common atrial pressure after Fontan and significantly lower pos

25 tic venous flow dynamics relate best to mean atrial pressure and can be used clinically to estimate m

26 long with higher resting/dynamic PCWP, right atrial pressure and cardiac output (P<0.0001 for all).

27 between adiposity indices with higher right atrial pressure and cardiac output.

28 as no significant relation between mean left atrial pressure and deceleration time.

29 Patients who died had the highest right atrial pressure and heart rate and the lowest mean arter

30 an autoregulatory mechanism to decrease left atrial pressure and improve heart failure (HF) symptoms

31 creased after endotoxin infusion, while left atrial pressure and left ventricular end-diastolic diame

32 Left atrial pressure and left ventricular end-diastolic diame

33 mic hypotension occurred with a fall in left atrial pressure and little change in left ventricular vo

34 levated SCr was associated with higher right atrial pressure and lower cardiac index.

35 yncopal patients presented with higher right atrial pressure and lower cardiac outputs with lower sur

36 , mean pulmonary artery pressure, mean right atrial pressure and mean left atrial pressure) at baseli

37 nges in hemodynamic values (except for right atrial pressure and mean pulmonary artery pressure) were

38 te, mean arterial blood pressure, mean right atrial pressure and peak airway pressure.

39 ases in cardiac output and decreases in left atrial pressure and peripheral resistance but without el

40 elow Veq, we used a servomotor to clamp left atrial pressure and produce nonfilling diastoles, allowi

41 re, pulmonary vascular resistance, and right atrial pressure and provided incremental prognostic valu

42 ormed during exercise, included higher right atrial pressure and pulmonary capillary wedge pressure a

43 mic changes and additionally decreased right atrial pressure and pulmonary vascular resistance.

44 ed for the simultaneous measurement of right atrial pressure and renal sympathetic nerve activity.

45 Ageing, increased left atrial pressure and stiffness, mitral regurgitation, as

46 excursion correlated with WHO-FC, mean right atrial pressure and survival (P<0.05).

47 ure (27% to 39% decrease by 6 h), mean right atrial pressure and systemic vascular resistance, along

48 PV showed positive correlation between intra-atrial pressure and the number of waves emanating from t

49 atrial diastole, resulting in a higher late atrial pressure and thus a normal mean pressure.

50 Atrial pressure and volume loops were prepared from inva

51 right atrial, pulmonary artery and mean left atrial pressures and cardiac output were obtained.

52 Arterial and right atrial pressures and end-tidal CO2 were measured.

53 Left atrial pressures and LV volumes and pressures were measu

54 alculated from Doppler indices reflective of atrial pressures and the diastolic filling volume.

55 sion pressure (diastolic; aortic minus right atrial pressure) and cerebral perfusion pressure (mean a

56 (MAP), pulmonary artery pressure (PAP), left atrial pressure, and cardiac output (CO).

57 aortic pressure, in the left atrium to left atrial pressure, and in all heart chambers to a decrease

58 rized by a decrease in cardiac output, right atrial pressure, and left ventricular (LV) end-diastolic

59 ikely because of marked increase in the left atrial pressure, and preload reduction may unmask the re

60 exercise tolerance, functional class, right atrial pressure, and vasodilator response to adenosine.

61 rdiac output; mean aortic, pulmonary or left atrial pressures; and peak positive and negative first d

62 edural monitoring included vital signs, left atrial pressure, arterial blood pressure, cerebral perfu

63 nsion drugs, oxygen therapy, and lower right atrial pressure, as well as functional class in the grou

64 urements of left ventricular inflow and left atrial pressures, ascending aortic pressure, thermodilut

65 the modified Bernoulli equation, with right atrial pressure assumed to be 10 mm Hg.

66 ass at diagnosis (P < 0.001), and high right atrial pressure at diagnosis (P = 0.002).

67 on fraction is associated with elevated left atrial pressure at rest due to fluid overload or during

68 , and were more likely to have elevated left atrial pressure at rest.

69 re, mean right atrial pressure and mean left atrial pressure) at baseline, during 60 min of atrial fi

70 reload was abruptly reduced by clamping left atrial pressure between 0 and -2 mm Hg in seven open-che

71 y arterial pressure of < 25 mm Hg and a left atrial pressure between 2 and 5 mm Hg.

72 ication developed by cardiac index and right atrial pressure both on echocardiography predicted cardi

73 hypotension or perioperative changes in left atrial pressure, brain natriuretic peptide levels, lacti

74 Shunt implantation reduces left atrial pressures but increases pulmonary blood flow, whi

75 Breathing NO decreased the mean right atrial pressure by 12 +/- 3%, mean pulmonary arterial pr

76 congestion were produced by increasing left atrial pressure by 2 mmHg.

77 balloon in the left atrium to increase left atrial pressure by 5 mmHg.

78 e of at least 25 mm Hg while exceeding right atrial pressure by at least 5 mm Hg.

79 High pulmonary vascular resistance and right atrial pressure by invasive hemodynamic measurements wer

80 scular resistance (by 29%; P=0.03) and right atrial pressure (by 40%; P=0.007), but with only modest

81 measured lung lymph flow after raising left atrial pressure (by inflating a balloon) in sheep that w

82 ion, left ventricular end-diastolic and left atrial pressure can rise to extremely high levels.

83 invasively derived measurements, mean right atrial pressure cardiac index, and mixed venous oxygen s

84 of 6MWD, pulmonary arterial pressure, right atrial pressure, cardiac index and pulmonary vascular re

85 th Organization functional class, mean right atrial pressure, cardiac index, and mixed venous oxygen

86 and mean systemic arterial pressures (MAP), atrial pressures, cardiac output, and arterial blood gas

87 With left atrial pressure clamping, maximal LV pressure decreased

88 domized to the PAC arm (n = 194), only right atrial pressure correlated weakly with baseline SCr (r =

89 aging has been proposed in which early left atrial pressure could be low in the aged heart but rise

90 Right atrial pressure decreased 52% (P=0.012), pulmonary arter

91 Compared with baseline values, right atrial pressure decreased by 34% (17+/-4 versus 11+/-5 m

92 .1 +/- 1.1 to 13.2 +/- 1.3 mm Hg; mean right atrial pressure decreased from 10.9 +/- 1 to 4.8 +/- 1.0

93 whereas diastolic left ventricular and right atrial pressures decreased significantly and proportiona

94 C and correlates with higher swings of right atrial pressure during CC.

95 e data indicate that PAOP overestimates left atrial pressure during endotoxin shock, making it an ina

96 is feasible, seems to be safe, reduces left atrial pressure during exercise, and could be a new stra

97 (PPH), right atrial pressure may exceed left atrial pressure during exercise, resulting in a right-to

98 ogy was defined as inspiratory rise in right atrial pressure during right heart catheterization.

99 rial contraction (a wave); point 2, the left atrial pressure during the start of ventricular systole;

100 ial filling (v wave); point 4, earliest left atrial pressure during ventricular filling; and the line

101 ing right ventricular systolic and mean left atrial pressures during AF.

102 mean difference between the aortic and right atrial pressures during the release phase of chest compr

103 terogeneous clinical syndrome, elevated left atrial pressure-either at rest or with exertion-is a com

104 rtension (PH), with or without elevated left atrial pressure (eLAP), and mortality in candidates for

105 ns between PH, with or without elevated left atrial pressure (eLAP), and mortality in candidates for

106 Left atrial pressure elevation during dextran infusion increa

107 Thus, left atrial pressure elevation increased lymph flow less in d

108 Absence of left atrial pressure elevation was based on combined hemodyna

109 For comparison, we also raised left atrial pressure elevation, plasma oncotic pressures in d

110 We hypothesized that elevation of intra-atrial pressure elicits high-frequency and spatio-tempor

111 complex but characterised by increased left atrial pressure, especially during exertion, which might

112 dge pressure fell from 31 to 18 mm Hg, right atrial pressure from 15 to 8 mm Hg, and SVR from 1,780 t

113 ters also improved with a reduction in right atrial pressure from 22 mm Hg at baseline, to 9 mm Hg an

114 Hg to 11.5+/-3.7 mm Hg; p < .001), and right atrial pressures (from 7.3+/-2.5 mm Hg to 9.8+/-2.5 mm H

115 patients (77%) with a normal predicted left atrial pressure (grade I diastolic dysfunction) had a me

116 24 patients with an elevated predicted left atrial pressure (grade II/III diastolic dysfunction), on

117 %, exercise PCWP >/=25 mm Hg, and PCWP-right atrial pressure gradient >/=5 mm Hg.

118 time, < 180 m/s, which indicated a mean left atrial pressure > or = 20 mm Hg, were both 100%.

119 At intra-atrial pressures >10 cm H2O, the maximum dominant freque

120 capillary wedge pressure (>18 mm Hg), right atrial pressure (>8 mm Hg), and mean pulmonary artery pr

121 ic variables such as cardiac index and right atrial pressure have consistently been associated with s

122 leaflets, which equals left ventricular-left atrial pressure, have been proposed to explain this dyna

123 The classic hemodynamic cutoffs for right atrial pressure (hazard ratio, 1.26 [0.98-1.62]) and mea

124 come; however, cutoffs of 14 mm Hg for right atrial pressure (hazard ratio, 1.59 [1.26-2.00]) and 35

125 on pulmonary-capillary wedge pressure, right atrial pressure, heart rate, or cardiac output.

126 ricle (HR, 10.5; P=0.0429), and higher right atrial pressure (HR, 1.3 per 1 mm Hg; P=0.0016).

127 % CI, 1.76-12.88; P=0.0021), increased right atrial pressure (HR, 1.34; 95% CI, 0.95-1.90; P=0.0992 a

128 Intra-atrial pressure (IAP) was increased in steps of 2 to 3 c

129 ilure, interventions to reduce elevated left atrial pressure improve symptoms and reduce the risk of

130 ers of RV function (RV stroke work and right atrial pressure) improved only in the BPA group.

131 more accurate than P(PAO) in estimating left atrial pressure in cardiac surgical patients.

132 er echocardiographic variables and mean left atrial pressure in group A patients.

133 describe the design of REDUCE Elevated Left Atrial Pressure in Heart Failure (REDUCE LAP-HF I), the

134 ial of a device-based therapy to reduce left atrial pressure in HFpEF.

135 l Inc IASD System II to Reduce Elevated Left Atrial Pressure in Patients with Heart Failure (REDUCE L

136 The REDUCe Elevated Left Atrial Pressure in Patients with Heart Failure (REDUCE L

137 REDUCE LAP-HF I (Reduce Elevated Left Atrial Pressure in Patients With Heart Failure) was a ph

138 , Inc IASD System II to Reduce Elevated Left Atrial Pressure in Patients With Heart Failure), implant

139 heart catheterization confirmed higher right atrial pressure in patients with VC 14 mm than those wit

140 and lower PVR in the PEA group; lower right atrial pressure in the BPA group; and use of pulmonary h

141 trial fibrillation/flutter or from increased atrial pressures in the setting of heart failure with pr

142 A paradoxical inspiratory rise in right atrial pressure (in contrast to the normal fall during i

143 usion pressure (a frequent surrogate of left atrial pressure) in this population.

144 Right atrial pressure increased by 2.5 +/- 1.8 mm Hg (P=0.002)

145 Left atrial pressure increases at exercise with an average up

146 for the first time that an increase in intra-atrial pressure increases the rate and organization of w

147 ignificant interaction between SCr and right atrial pressures (interaction P<0.0001); increased SCr b

148 pulmonary arterial pressure, left and right atrial pressures, intracranial pressure, body temperatur

149 om the passive transmission of elevated left atrial pressure is a notable exception.

150 Evaluation of left atrial pressure is frequently required for mechanically

151 xhibiting directionally opposite firing when atrial pressure is perturbed.

152 the pulmonary artery pulse pressure to right atrial pressure, is a predictor of right ventricular fai

153 The effect of raising the left atrial pressure (LAP) acutely above 25 mmHg (to cause pu

154 of the mitral valve increased the mean left atrial pressure (LAP) by approximately 2.6 and 3.8 mmHg,

155 The mean left atrial pressure (LAP) correlated well with the septal si

156 Increased left atrial pressure (LAP) has been associated with adverse o

157 s have limitations in the prediction of left atrial pressure (LAP) in patients with mitral valve dise

158 ing pressures to direct measurements of left atrial pressure (LAP) via catheterization in 100 patient

159 ally clinically significant increase in left atrial pressure (LAP).

160 Hemodynamic parameters, including mean left atrial pressure (LAP, in mm Hg), mean pulmonary artery p

161 ith higher pulmonary arterial and mean right atrial pressures, lower cardiac index, and impaired exer

162 best predicted death in patients with right atrial pressure <10 mm Hg.

163 c peptide (BNP), and hemodynamics with right atrial pressure <8 mm Hg and cardiac index >2.5 mg/kg/mi

164 ion of right ventricular function with right atrial pressure <8 mm Hg and cardiac index >2.5 to 3.0 l

165 (pulmonary capillary wedge pressure - right atrial pressure), LV myocardial stiffness was nearly 30%

166 artery pressure to stroke volume with right atrial pressure may be most helpful in identifying patie

167 ascular resistance, cardiac index, and right atrial pressure may be used to stratify risk of death.

168 primary pulmonary hypertension (PPH), right atrial pressure may exceed left atrial pressure during e

169 Right atrial pressure (mean/V-wave) decreased significantly, w

170 ators of RV-PV function (i.e., resting right atrial pressure, mean PA pressure, pulmonary vascular re

171 Active treatment significantly lowered right atrial pressure, mean pulmonary artery pressure, and pul

172 ft atrium was directly catheterized for left atrial pressure measurements.

173 tion score 3 versus 2; P<0.001), higher left atrial pressures (median, 14 mm Hg versus 10 mm Hg; P=0.

174 that a mechanical approach to reducing left atrial pressure might be effective in HFPEF.

175 Heart Failure Patients], and LAPTOP-HF [Left Atrial Pressure Monitoring to Optimize Heart Failure The

176 vival on univariate analysis were mean right atrial pressure (mRAP) (p < 0.0001), mPAP (p = 0.034), R

177 a Cox proportional hazards model: mean right atrial pressure (mRAP) more than or equal to 14 mm Hg an

178 renal vascular conductance (RVC), mean right atrial pressure (mRAtP), urine flow, glomerular filtrati

179 falls in arterial pressure (HF: p < 0.001), atrial pressure (Normal: p < 0.001; HF: p < 0.001), and

180 0 mm Hg) PHT, respectively, assuming a right atrial pressure of 5 mm Hg.

181 Increases in left atrial pressure of 5 mmHg increased RAR activity from 3.

182 ressure was increased either by raising left atrial pressure or by aortic constriction.

183 There was no change in right atrial pressure or measures of right ventricular functio

184 approach to AF under conditions of elevated atrial pressure or volume.

185 ing mean arterial pressure, heart rate, left atrial pressure, or left ventricular dP/dt.

186 relaxation, LV end-diastolic pressure, right atrial pressure, or pulmonary pressure in either patient

187 dynamic values, including pulmonary and left atrial pressures, or intrathoracic impedance, which is r

188 rotein C pathway, in rats subjected to acute atrial pressure overload caused by aortic banding.

189 e dP/dt (p < 0.05), an increase in mean left atrial pressure (p < 0.05) and a prolongation of tau, th

190 s of DHEA-S were associated with lower right atrial pressure (P = 0.02) and pulmonary vascular resist

191 e, mean pulmonary artery pressure, and right atrial pressure (P</=0.001, 0.003, 0.017, and 0.031, res

192 0.001) in association with decreases in left atrial pressure (P<0.001), peripheral resistance (P=0.01

193 his study compared a prediction of mean left atrial pressure (P(LA)) ascertained by Doppler echocardi

194 ssociation functional class (P=0.009), right atrial pressure (P=0.037), and stroke volume (P=0.043).

195 ostoperative central venous (P<0.001) common atrial pressure (P=0.042), inotropic score (P<0.001), an

196 tio, 5.3; P<0.001), whereas changes in right atrial pressure (P=0.36) and pulmonary capillary wedge p

197 Elevated left atrial pressure, particularly during exercise, is a key

198 blockade and the absence of change in right atrial pressure, persistent atrial tachycardia caused AR

199 sion pressure is not thought to reflect left atrial pressure (Pla) when alveolar pressure (PA) exceed

200 peak systolic blood pressure (Ps), mean left atrial pressure (PLA), and Doppler-derived IVRT (IVRTDop

201 te wedge pressure (Pcw) from transmural left atrial pressure (Platm) by elevating pleural pressure an

202 essure, coronary sinus pressure (Pcs), right atrial pressure (Pra), and the mean transit time (invers

203 nd EDPR gradients, in conjunction with right atrial pressure, provide Doppler estimates of pulmonary

204 vice that allows shunting to reduce the left atrial pressure provides clinical and hemodynamic benefi

205 In controls subjects, right atrial pressure, pulmonary arterial pressure, and pulmon

206 3 months (p=0.035), with no changes in right atrial pressure, pulmonary arterial pressure, or pulmona

207 LV pressure and volume, left atrial pressure, pulmonary artery pressure and flow, and

208 of whom 69%, 64%, and 79% had elevated right atrial pressure, pulmonary capillary wedge pressure, and

209 Mean pulmonary arterial pressure, mean left atrial pressure, pulmonary vascular resistance, and stat

210 ulmonary artery systolic pressure, and right atrial pressure/pulmonary capillary wedge pressure ratio

211 n (pulmonary artery pulsatility index, right atrial pressure:pulmonary capillary wedge pressure ratio

212 t (=pulmonary capillary wedge pressure-right atrial pressure; r=0.67; P=0.003), suggesting relief of

213 pressure (PCWP) (32 to 14 mm Hg), and right atrial pressure (RA) (19 to 9 mm Hg).

214 tool to identify patients with normal right atrial pressure (RAP <=7 mm Hg) in this population.

215 Diastolic dysfunction was defined as right atrial pressure (RAP) >/=15 mm Hg (right ventricular [RV

216 nal pediatric hemodynamic cutpoints of right atrial pressure (RAP) >12 mm Hg or pulmonary capillary w

217 ographic and invasive measures of mean right atrial pressure (RAP) (r = 0.863; p < 0.0001), systolic

218 Although right atrial pressure (RAP) and pulmonary capillary wedge pres

219 AF was associated with an increase of right atrial pressure (RAP) and right atrial dilatation.

220 Impaired respiratory variation of right atrial pressure (RAP) in severe pulmonary hypertension (

221 undergoing TIPS suggest that elevated right atrial pressure (RAP) may influence survival.

222 dictive value of coronary fistulae and right atrial pressure (RAP) score (comprising the tricuspid va

223 data exists as to the relation of mean right atrial pressure (RAP) to Doppler parameters of right atr

224 Measurements included mean right atrial pressure (RAP), mean pulmonary arterial pressure

225 (IVC dynamics) are used for estimating right atrial pressure (RAP).

226 nvasively determined (RV congestion if right atrial pressure [RAP] 10 mm Hg and LV congestion if pulm

227 dynamic determinants included elevated right atrial pressure, reduced pulmonary artery pulse pressure

228 ry capillary wedge pressure (PCWP) and right atrial pressure responses were compared stratified by ob

229 ue disease, functional class III, mean right atrial pressure, resting systolic blood pressure and hea

230 th these indices: right atrial volume, right atrial pressure, right atrial reservoir strain, right ve

231 y artery pressure to stroke volume and right atrial pressure significantly improved the discriminatio

232 perimental heart failure with elevated right atrial pressure, sodium removal was ~4 times greater tha

233 stolic filling was determined by a simulated atrial pressure source that was either constant or varie

234 terial blood pressure (to 30-35 mm Hg), both atrial pressures, systemic oxygen consumption (by 35%),

235 Cyanotic patients had lower mean right atrial pressures than the control subjects (4+/-3 versus

236 ling wave indexes had the best relation with atrial pressure, the highest being for systolic filling

237 filling have been applied to determine left atrial pressure, their accuracy has been limited by the

238 mm Hg [5-11 mm Hg]; P < 0.001), higher right atrial pressure to pulmonary capillary wedge pressure ra

239 in the EUROMACS score (Hemoglobin and Right Atrial Pressure to Pulmonary Capillary Wedge Pressure ra

240 monary artery pressures and 1 measuring left atrial pressure) to assess the effect on all-cause morta

241 d pulmonary artery systolic pressure or left atrial pressure) variables can assist with diagnosis (H2

242 to 64% reduction in A-loop area of the left atrial pressure-volume relationship, quantifying work pe

243 ventricular end-diastolic diameter, and left atrial pressure vs. left ventricular end-diastolic diame

244 <0.01) per 10-mL/m(2) decrease and for right atrial pressure was 1.05 (95% confidence interval, 1.02-

245 Average initial left atrial pressure was 31 mm Hg.

246 gnificant increase in activity when the left atrial pressure was acutely elevated in both intact and

247 ere common among patients with CS, and right atrial pressure was associated with increased mortality

248 The left atrial pressure was directly related to the E/A ratio (r

249 In the 123 patients, the right atrial pressure was less than 10 mm Hg in 49 patients, t

250 Left atrial pressure was measured with a micromanometer cathe

251 The variation of right atrial pressure was significantly higher in the mechanic

252 Exercise right atrial pressure was the highest in MI+DD followed by MI-

253 pressure (DPP=diastolic blood pressure-right atrial pressure) was associated with more limited hemody

254 ivity (% change in RSNA/mm Hg change in left atrial pressure) was markedly attenuated after PL (pre,

255 l sympathetic nerve activity/mmHg mean right atrial pressure) was measured during isotonic saline vol

256 itation, low cardiac index, and raised right atrial pressure were associated with poor survival for b

257 e renal failure or elevated postbypass right atrial pressure were at increased risk for early mortali

258 fusion pressure, systemic pressure, and left atrial pressure were continuously monitored, electronica

259 stance, stroke volume index (SVI), and right atrial pressure were independently associated with death

260 l pressure, arterial blood pressure and left atrial pressure were measured in paralysed, anaesthetize

261 SVI and right atrial pressure were the hemodynamic variables that were

262 Aortic and right atrial pressures were measured with micromanometers.

263 One mechanism is that when Palv exceeds left atrial pressure, West zone 1 or 2 (non-zone 3) condition

264 re (pulmonary capillary wedge pressure-right atrial pressure), which reflects LV preload independent

265 sure is directly related to an enhanced left atrial pressure, which is common to both heart failure w

266 The dissociation between PAOP and left atrial pressure, while left ventricular and -diastolic d

267 These patients have elevated atrial pressures with exercise as measured with invasive

268 Nitrite reduced right atrial pressures, with no effect on cardiac output or st

269 ary perfusion pressure (CPP) (aortic - right atrial pressure) without epinephrine (A-CPR 21 +/- 8 mm

270 p = 0.577; Q = 14.64, I(2) = 79.51%), right atrial pressure (WMD: 1.01 mmHg, 95%CI: -0.93, 2.96, p =