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1 heart syndrome and an intact or restrictive atrial septum.
2 ling caused inappropriate enlargement of the atrial septum.
3 pulated the atrial free wall rather than the atrial septum.
4 ntial to exert focal pressure upon the intra-atrial septum.
5 h hypoplastic left heart syndrome and intact atrial septum.
6 equency ablated lesions applied to the right atrial septum.
7 over a 6.5-year period, 18 (5.7%) had intact atrial septum.
8 minent on the mesenchymal cap of the primary atrial septum.
9 he spatial relationship of the device to the atrial septum.
10 all of these patients had restriction at the atrial septum.
11 Fifteen patients had an intact atrial septum.
12 re frames designed for anchoring through the atrial septum.
14 he atrial septum, including both the primary atrial septum and dorsal mesenchymal protrusion (DMP).
15 on of the normal physiologic flow across the atrial septum and ductus arteriosus in the human fetus.
16 a compact node of fibers at the base of the atrial septum and forward where it connected with the bu
17 ith: 1) caudocranial activation of the right atrial septum and proximal-to-distal coronary sinus acti
18 mean, 111 +/- 8 ms) present primarily in the atrial septum and right atrium were responsible for main
21 corin mRNA was most abundant in the primary atrial septum and the trabecular ventricular compartment
22 h hypoplastic left heart syndrome and intact atrial septum and to evaluate the relationship of atrial
24 ardial lineages of the distal outflow tract, atrial septum, and in sinoatrial and atrioventricular no
27 acemaker-conduction system, heart valves and atrial septum, and uncover unexpected diversity among ca
28 short cycle length principally involving the atrial septum appear to be critical for maintenance of A
29 malous pulmonary and systemic veins, and the atrial septum are prerequisites for surgical correction
34 hocardiography, Interarterial Shunts, Intact Atrial Septum, Congenital Heart Disease, Unroofing Defec
35 0.5, a previously established model to study atrial septum defects, displayed polydactyly or hypodact
36 0.5, a previously established model to study atrial septum defects, which displayed polydactyly or hy
37 on patterns of known key regulatory genes of atrial septum development, including Nkx2.5, Pitx2, and
38 to other transcription factors implicated in atrial septum development, Odd 1 mRNA expression is rest
39 Odd 1(-/-) mutant mouse embryos fail to form atrial septum, display dilated atria with hypoplastic ve
40 Slowing of electric conduction in the left atrial septum due to antiarrhythmic drugs and/or atrial
41 4 to 396 electrodes) from both atria and the atrial septum during six induced AF episodes in six dogs
42 syndrome and an intact or highly restrictive atrial septum face a neonatal mortality of at least 48%
44 ariants with an intact or highly restrictive atrial septum (HLH-IAS) require immediate postnatal inte
46 gic shunting across the ductus arteriosus or atrial septum in utero (i.e., left to right) can be read
47 d through the dorsal mesocardium to form the atrial septum, including both the primary atrial septum
48 of interatrial communications with an intact atrial septum, including their embryology, pathophysiolo
52 ul ablation of the slow pathway in the right atrial septum near the coronary sinus ostium prevented t
54 ng (age, weight, intact/severely restrictive atrial septum or left ventricle to coronary fistulae), t
56 from one atrium to the other and/or from the atrial septum play an important role in re-formation of
59 trictive interatrial communication or intact atrial septum (RAS) postnatally; however, the ability of
60 S patients with intact or highly restrictive atrial septum remains high despite successful urgent sep
62 normal MV inflow pattern, restrictive/intact atrial septum, retrograde flow in the transverse aortic
63 to additional ablation lines along the left atrial septum, roof, and/or anterior wall where there we
64 pulmonary vein in 46% of patients; the left atrial septum, roof, or anterior wall in all; and the co
67 e and after sequential ablation of the right atrial septum, targeting interatrial conduction zones.
69 esenchyme on the leading edge of the primary atrial septum, the atrioventricular endocardial cushions
70 ); 2) small left atrium with thick, muscular atrial septum (type B, n = 4), and 3) giant left atrium,
71 rt syndrome and intact or highly restrictive atrial septum underwent attempted prenatal intervention.
72 dable, open-cell design stent into the fetal atrial septum via a percutaneous access route through th
73 d by a wave front most often coming from the atrial septum via BB (right atrium, 62%; left atrium, 67
74 GAP43-positive and TH-positive nerves in the atrial septum was 317+/-36 and 155+/-85 per mm(2), respe
79 Anesthetized pigs (n=7) with defects of the atrial septum were catheterized using venous and arteria
80 engendered by the clamp and resection of the atrial septum with possible injury to the internodal con
81 pe B, n = 4), and 3) giant left atrium, thin atrial septum with severe mitral regurgitation (type C,
82 MYH6 homolog eliminates the formation of the atrial septum without overtly affecting atrial chamber f