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1 e atrioventricular block at the level of the atrioventricular node).
2 t half maximum of 10 mm was delivered to the atrioventricular node.
3 the remaining 7 for pathway proximity to the atrioventricular node.
4 observed in the conduction properties of the atrioventricular node.
5 with antibody-associated disease beyond the atrioventricular node.
6 tract, atrial septum, and in sinoatrial and atrioventricular node.
7 embryos appear to lack the primordium of the atrioventricular node.
8 n, we focused on genetic modification of the atrioventricular node.
9 Bradycardia was induced by ablation of the atrioventricular node.
10 is virtually absent from the sinoatrial and atrioventricular nodes.
11 raphy or the discovery of the sinoatrial and atrioventricular nodes.
12 a position midway between the sinoatrial and atrioventricular nodes.
13 Thirty-minute exposure to MEHP increased the atrioventricular node (147 versus 107 ms) and ventricula
14 dditional animals by applying PF(REV) to the atrioventricular node: 17 low-dose (PF(REV-LOW)) and 10
16 th chronic atrial fibrillation who underwent atrioventricular node ablation and pacemaker implant dem
17 Fourteen piglets 8 weeks of age underwent atrioventricular node ablation and were paced from eithe
18 Patients with cAVB were identified by an atrioventricular node ablation or diagnosis of third-deg
23 gation, we tested the feasibility of in vivo atrioventricular node ablation, in Langendorff-perfused
28 o undergo either pulmonary-vein isolation or atrioventricular-node ablation with biventricular pacing
29 Pulmonary-vein isolation was superior to atrioventricular-node ablation with biventricular pacing
30 dema in another; in the group that underwent atrioventricular-node ablation with biventricular pacing
31 nths (60, vs. 82 in the group that underwent atrioventricular-node ablation with biventricular pacing
32 t pulmonary-vein isolation, and 40 underwent atrioventricular-node ablation with biventricular pacing
33 vagal denervation of the atria and sinus and atrioventricular nodes also denervated the ventricles.
34 ck-down of Prox1 restored the anatomy of the atrioventricular node and His-Purkinje network both of w
35 rug therapy, radio-frequency ablation of the atrioventricular node and implantation of a permanent pa
36 d all patients who underwent ablation of the atrioventricular node and implantation of a permanent pa
38 l of the ventricular rate by ablation of the atrioventricular node and permanent pacing does not adve
39 uired for optimal impulse propagation in the atrioventricular node and stabilizes the level of the co
40 ys (AP) are uncommon connections between the atrioventricular node and the fascicles or ventricles.
43 es such as the right coronary artery and the atrioventricular node, and is the passageway for permane
46 coordinate the spread of excitation from the atrioventricular node (AV node) to ventricular myocardiu
47 lt mice display conduction defects below the atrioventricular node (AVN) as determined by invasive el
51 ng of electrical activity in rabbit isolated atrioventricular node (AVN) preparations that, in the ma
52 y subepicardially via a thin needle into the atrioventricular node (AVN) region of adult rats to crea
54 supply to the sinuatrial node (SAN) and the atrioventricular node (AVN) with 64-section computed tom
55 uction system: the sinoatrial node (SAN) and atrioventricular node (AVN), remains poorly understood.
56 lude injury of the sinoatrial node (SAN) and atrioventricular node (AVN), requiring cardiac rhythm ma
57 of interconnected structures, including the atrioventricular node (AVN), the central connection poin
58 ince the first description of the anatomical atrioventricular nodes (AVNs), a large number of studies
59 mitral) AT that can be overcome by injecting atrioventricular node blockers and signal averaging, res
60 lopment and postnatal function of the murine atrioventricular node by promoting cell-cycle exit of sp
61 pagation of excitation in the sinoatrial and atrioventricular nodes by shortening the space constant
63 ntaining nuclear integrity in heart disease, atrioventricular node cells lacking lamin A exhibit incr
64 tly higher expressed in the right atrium and atrioventricular node compared with left ventricle (P=5.
65 baroreceptor reflex, slowing heart rate and atrioventricular node conduction in response to increase
66 t rate, chronotropic response, and atrial or atrioventricular node conduction velocity may play a rol
72 s of the CCS, including the sinoatrial node, atrioventricular node, His bundle, bundle branches, and
74 ding: Zone I-sinoatrial node region; Zone II-atrioventricular node/His region; and Zone III-bundle br
75 ablation of the right inferior extension of atrioventricular node in 3 cases and by observing a VA i
76 The magnetic catheter was used to ablate the atrioventricular node in 4 animals and to perform linear
77 th atrial fibrillation after ablation of the atrioventricular node is similar to expected survival in
79 s complication of NLS is inflammation of the atrioventricular node leading to congenital heart block
81 This case indicates that although the adult atrioventricular node may be relatively resistant to the
82 via an accessory pathway (n = 4) or via twin atrioventricular nodes (n = 4), ventricular tachycardia
83 n40(-)/connexin45(+) cells is missing in the atrioventricular node of Nkx2-5 heterozygous KO mice.
85 ardia is useful for differentiating atypical atrioventricular node re-entrant tachycardia (AVNRT) fro
90 hmias (bradyarrhythmias, atrial tachycardia, atrioventricular node reentrant tachycardia) are signifi
91 ntricular (AV) reentry (AVRT; n=59), typical atrioventricular node reentry (AVNRT; n=82), atypical AV
92 Current maneuvers for differentiation of atrioventricular node reentry tachycardia (AVNRT) and at
96 atients with double firing properties of the atrioventricular node, separating these into discrete ty
99 atrioventricular bundle during the period of atrioventricular node specification, which results in fe
101 ate electrical conduction through the distal atrioventricular node, where conducting cells densely in
102 n based on this mechanism could occur in the atrioventricular node, where multiple branching is struc
103 iginate from or conduct through the atria or atrioventricular node with abrupt onset, affects 168 to
104 d channel, subtype 4 staining in the compact atrioventricular node with some retention of hyperpolari