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1 e double outlet right ventricle and a common atrioventricular valve.
2 r valves have generally been better than the atrioventricular valves.
3 the development of myxomatous changes of the atrioventricular valves.
4 direct formation of the semilunar valve and atrioventricular valves.
7 and superior), and the atrial aspect of the atrioventricular valves also express high levels of Tbx5
8 rings (AVRs) surround the atrial orifices of atrioventricular valves and are hotbed of ectopic activi
11 had a single mitral valve, 130 had a common atrioventricular valve, and 97 had a single tricuspid va
14 ive small GTPases, RhoA and Rac1, coordinate atrioventricular valve (AV) differentiation and morphoge
16 s applied to derive principal strains at the atrioventricular valve (AVV) and apical short-axis level
18 how mutations disrupt the interplay between atrioventricular valve (AVV) morphogenesis and function
20 monary bypass time, operation prior to 1991, atrioventricular valve (AVV) replacement at the time of
21 a low incidence of semilunar valve defects, atrioventricular valve defects and double outlet right v
23 specific aspects of trials in patients with atrioventricular valve disease are reviewed in this arti
24 y and safety for transcatheter treatments of atrioventricular valve disease in patients with heart fa
25 of patients, the dynamic nature of secondary atrioventricular valve disease severity, the role of hea
30 sed by Doppler determination of the systemic atrioventricular valve flow velocity at the conclusion o
32 (-/-) double null embryos exhibited abnormal atrioventricular valve formation, a phenotype never seen
34 ived cells to the individual leaflets of the atrioventricular valves has also important pragmatic con
35 ediating intracellular kinase activation for atrioventricular valve morphogenesis using well defined
36 equirement for FOG-1 in the outlet tract and atrioventricular valves of the heart that depend on expr
37 ice, and thrombi were detected at either the atrioventricular valves or within the atria of 2 of 13 j
38 tion of EPDCs to the various leaflets of the atrioventricular valves provides a new paradigm in valve
39 quent impact on transplant-free survival and atrioventricular valve regurgitation (AVVR) as well as t
41 pulmonary artery pressure >15 mm Hg (n=16), atrioventricular valve regurgitation (n=5), and decrease
42 ic dysfunction (P < .01), moderate or severe atrioventricular valve regurgitation (P < .01), higher F
43 en in those patients with moderate or severe atrioventricular valve regurgitation (p = 0.07) and in t
44 ailure late in pregnancy because of systemic atrioventricular valve regurgitation and required valve
46 ventricular dysfunction, moderate or greater atrioventricular valve regurgitation on pre-catheterizat
47 ded complete heart block (n=2) and increased atrioventricular valve regurgitation requiring surgical
48 stenosis and regurgitation; to semilunar and atrioventricular valve regurgitation, and to major risks
49 irculatory failure, ventricular dysfunction, atrioventricular valve regurgitation, arrhythmia, protei
50 rtension, pulmonary regurgitation, pulmonary atrioventricular valve regurgitation, pulmonary and syst
54 with left ventricular outflow tract surgery, atrioventricular valve replacement, or ventricular L-Loo
55 no procedure; (2) ventricular L-looping; (3) atrioventricular valve replacement; (4) and absence of p
56 regarding the long-term outcomes of systemic atrioventricular valve (SAVV) intervention (morphologic
58 tion fraction (SVEF) at the time of systemic atrioventricular valve (SAVV) replacement as a predictor
61 ular, single tricuspid, single mitral, and 2 atrioventricular valves was 56% (95% confidence interval
63 E/end diastolic volume) and flow through the atrioventricular valve were computed and compared betwee
64 eliminates expression in the atrium and the atrioventricular valve while expression is retained in t