ライフサイエンスコーパス: attachment level

1 ding on probing, probing depth, and clinical attachment level.

2 ing depth, bleeding on probing, and clinical attachment level.

3 XCL8 were positively related to the clinical attachment level.

4 probing, visible plaque, probing depth, and attachment level.

5 keratinized tissue width (KTW), and clinical attachment level.

6 inical parameters (P < 0.05) except clinical attachment level.

7 for repeat measurements of probing depth and attachment level.

8 H loss were noted at the 5.1-5.4 mm clinical attachment level.

9 cells, but NanI-producing strains had higher attachment levels.

10 PD), and vertical (VAL) and horizontal (HAL) attachment levels.

11 lative vertical (RVAL) and horizontal (RHAL) attachment levels.

12 gatively correlated, with MARGI-reported RNA attachment levels.

13 nths: 1) probing depth; 2) relative clinical attachment level; 3) GR; 4) thickness of KT (TKT); and 5

14 depth; (2) probing depth (PD); (3) clinical attachment level; (4) width of keratinized tissue (wKT);

15 his discovery GWAS of interproximal clinical attachment level-a measure of lifetime periodontal tissu

16 1 and 2 had significantly different clinical attachment level and gingival recession changes by the e

17 gy (AAP) criteria using measures of clinical attachment level and probing depth (PD).

18 e secondary outcomes were change in clinical attachment level and proportion of sites with bleeding o

19 cate a possible correlation between clinical attachment level and salivary IgG (P = 0.07; r(2) = 0.08

20 use (versus none) was associated with higher attachment levels and more teeth only among participants

21 GI], bleeding on probing [BOP], and clinical attachment level) and photographs from 53 participants (

22 e outcome (clinically determined periodontal attachment level) and predictors (self-reported dental s

23 ions included: 1) probing depth, 2) clinical attachment level, and 3) oral radiographs for alveolar c

24 Plaque index, probing depth, clinical attachment level, and bleeding on probing were evaluated

25 ncluding periodontal probing depth, clinical attachment level, and bleeding on probing, as well as cr

26 al examination using probing depth, clinical attachment level, and bleeding on probing.

27 refore plaque index, probing depth, clinical attachment level, and bleeding on probing.

28 arameters related to probing depth, clinical attachment level, and bone loss around teeth increased t

29 included changes in probing depth, clinical attachment level, and defect fill as revealed by reentry

30 ed by measurement of probing depth, clinical attachment level, and extent and severity of disease.

31 by criteria based on probing depth, clinical attachment level, and extent and severity of periodontal

32 Probing depth, clinical attachment level, and gingival and plaque indices in eac

33 , glycated hemoglobin, sampled-site clinical attachment level, and gingival index (P <0.05).

34 Recession height, probing depth, clinical attachment level, and keratinized tissue width and thick

35 8-OHdG, MDA, probing depth, clinical attachment level, and percentage of sites bleeding on pr

36 ngival index, plaque control record, probing attachment level, and probing pocket depth were assessed

37 on the percentage of root coverage, probing attachment level, and the amount of keratinized tissue i

38 s were measured, and probing depth, clinical attachment levels, and bleeding on probing were used to

39 in the percentage of root coverage, probing attachment levels, and increased keratinized tissue.

40 rrelated with probing pocket depth, clinical attachment levels, and RANKL concentrations in GCF.

41 Preoperative probing depths, attachment levels, and transoperative bone measurements

42 nges in probing depths and relative clinical attachment levels appeared to reflect changes in the und

43 laque index (PI), pocket depth, and clinical attachment level at days 14, 28, 42 (treatment), and 60

44 ue index, gingival index, probing depth, and attachment level at six sites per tooth.

45 Probing depth (PD), attachment level, bleeding on probing (BOP), and interpr

46 arameters, including probing depth, clinical attachment level, bleeding on probing, and gingival and

47 nation included probing depth (PD), clinical attachment level, bleeding on probing, and plaque index.

48 inical examinations including probing depth, attachment level, bleeding on probing, and root caries r

49 obtained first, with probing depth, clinical attachment level, bleeding on probing, plaque index scor

50 er, there were no significant differences in attachment levels, bleeding upon probing, or extent of s

51 vely related with probing depth and clinical attachment level; blood glucose was related only to blee

52 ism and periodontal lesions (i.e., decreased attachment level, bone loss, tooth mobility/migration, a

53 ith probing depth (PD) >/= 5 mm and clinical attachment level (CAL) >/= 3 mm were randomly divided in

54 with probing depth (PD) >/=5 mm and clinical attachment level (CAL) >/=2 mm at the same site.

55 defined as percentage of cases with clinical attachment level (CAL) >/=2.7 mm and linear bone growth

56 0.05) of mean PD (1.4 +/- 0.7 mm), clinical attachment level (CAL) (1.3 +/- 0.8 mm), and BOP (33.4%

57 und between viral load and moderate clinical attachment level (CAL) (rho = 0.638, P <0.05), CD4+ nadi

58 mary outcome variable was change in clinical attachment level (CAL) after 12 months.

59 ) of at least 5 mm and 2 mm loss of clinical attachment level (CAL) after initial non-surgical therap

60 ed change in probing depth (PD) and clinical attachment level (CAL) after the therapy.

61 this study is to report changes in clinical attachment level (CAL) and bone fill of periodontal IBDs

62 s to assess the association between clinical attachment level (CAL) and bone mineral density (BMD) at

63 Probing pocket depth (PPD), clinical attachment level (CAL) and gingival recession (GR) were

64 changes in clinical factors such as clinical attachment level (CAL) and gingival recession (GR).

65 cluding Probing pocket depth (PPD), Clinical attachment level (CAL) and Horizontal probing depth (HPD

66 alivary cytokines was found between clinical attachment level (CAL) and IL-21 (P = 0.02).

67 rimary efficacy parameters included clinical attachment level (CAL) and probing depth (PD).

68 val index (GI), probing depth (PD), clinical attachment level (CAL) and recession height (GRH), reces

69 ater mean gain in relative vertical clinical attachment level (CAL) and relative horizontal CAL were

70 dontal destruction was assessed via clinical attachment level (CAL) and the number of missing teeth.

71 related with probing depth (PD) and clinical attachment level (CAL) as well as with GCF levels of TNF

72 CF was inversely associated with clinical attachment level (CAL) at baseline before therapy in all

73 en serum and salivary PCT values to clinical attachment level (CAL) at P < 0.001 and rho = 0.78 and 0

74 periodontal probing depth (PD) and clinical attachment level (CAL) at six sites per tooth.

75 of variables on the 1-year post-op clinical attachment level (CAL) changes and the tooth survival we

76 Root coverage (RC) and clinical attachment level (CAL) did not differ significantly betw

77 Pocket depth (PD) and clinical attachment level (CAL) for six sites/tooth were ascertai

78 combination of clinically relevant clinical attachment level (CAL) gain (>=3 mm) and pocket closure

79 cket Probing Depth reduction (PPD), Clinical Attachment Level (CAL) gain and radiographic bone gain w

80 , probing depth (PD) reduction, and clinical attachment level (CAL) gain in both statin and placebo/n

81 obing depth (PD) reduction and mean clinical attachment level (CAL) gain was greater in the ALN group

82 n terms of probing depth reduction, clinical attachment level (CAL) gain, and bone level (clinical an

83 st improvements of recession depth, clinical attachment level (CAL) gain, and keratinized tissue (KT)

84 t postoperative probing depth (PD), clinical attachment level (CAL) gain, or radiographic defect reso

85 cally significant PD reductions and clinical attachment level (CAL) gains > or =2 mm compared to 56%

86 The clinical attachment level (CAL) measurements were stable througho

87 bing (BOP), probing depth (PD), and clinical attachment level (CAL) of all teeth were examined.

88 d probing depth (PD) with a gain in clinical attachment level (CAL) to treat advanced periodontal dis

89 on probing, probing depth (PD), and clinical attachment level (CAL) were carried out in four sites pe

90 ding on probing, probing depth, and clinical attachment level (CAL) were determined.

91 on probing, probing depth (PD), and clinical attachment level (CAL) were measured at baseline and 3 a

92 bing (BOP), probing depth (PD), and clinical attachment level (CAL) were performed.

93 Probing depth (PD) and clinical attachment level (CAL) were recorded at baseline and aft

94 rs including probing depth (PD) and clinical attachment level (CAL) were recorded.

95 bleeding index, probing depth, and clinical attachment level (CAL) were significantly higher in CAD+

96 severity of disease measured as the clinical attachment level (CAL) when healthy and diseased groups

97 dex (GI), 2) probing depth (PD), 3) clinical attachment level (CAL), 4) radiologic defect depth, and

98 acterized using probing depth (PD), clinical attachment level (CAL), alveolar crest height (ACH), and

99 and 12 months, probing depth (PD), clinical attachment level (CAL), and bleeding on probing (BOP) we

100 improvements in probing depth (PD), clinical attachment level (CAL), and bleeding on probing (BOP) we

101 val index (GI), probing depth (PD), clinical attachment level (CAL), and bleeding on probing (BOP), w

102 ental calculus, probing depth (PD), clinical attachment level (CAL), and bleeding on probing (BOP).

103 ng index (GBI), probing depth (PD), clinical attachment level (CAL), and bleeding on probing (BOP).

104 d examiners measured probing depth, clinical attachment level (CAL), and bleeding on probing on all t

105 Probing depth (PD), clinical attachment level (CAL), and bone PD were recorded.

106 Clinical parameters, including PD, clinical attachment level (CAL), and BOP, and GCF IL-1beta levels

107 ), probing depth (PD), relative GR, clinical attachment level (CAL), and cervical lesion height cover

108 ments, including pocket depth (PD), clinical attachment level (CAL), and gingival index (GI) were per

109 bleeding index, probing depth (PD), clinical attachment level (CAL), and gingival marginal level, inc

110 val index (GI), probing depth (PD), clinical attachment level (CAL), and HbA1c level, of all particip

111 g index (mSBI), probing depth (PD), clinical attachment level (CAL), and IBD depth, were recorded at

112 Probing depth, clinical attachment level (CAL), and keratinized gingival width (

113 were plaque index, gingival index, clinical attachment level (CAL), and PD.

114 evaluated were probing depth (PD), clinical attachment level (CAL), and percentage of sites with PD

115 Measurements of probing depth (PD), clinical attachment level (CAL), and radiographic BDA were done a

116 ents, including probing depth (PD), clinical attachment level (CAL), and radiographs, were used to cl

117 duced probing depth (PD), increased clinical attachment level (CAL), and reduced bleeding on probing

118 gingival bleeding on probing (BOP), clinical attachment level (CAL), and surfaces with plaque were re

119 rmined plaque score, probing depth, clinical attachment level (CAL), and the number of remaining teet

120 REC, probing depth, clinical attachment level (CAL), and width of keratinized tissue

121 ation-including probing depth (PD), clinical attachment level (CAL), bleeding on probing (BOP) and mi

122 mean changes in probing depth (PD), clinical attachment level (CAL), bleeding on probing (BOP), and g

123 eters including probing depth (PD), clinical attachment level (CAL), bleeding on probing (BOP), and p

124 asurements were probing depth (PD), clinical attachment level (CAL), bleeding on probing (BOP), gingi

125 Probing depth (PD), clinical attachment level (CAL), bleeding on probing (BOP), gingi

126 Probing depth (PD), clinical attachment level (CAL), bleeding on probing (BOP), gingi

127 Full-mouth probing depth (PD), clinical attachment level (CAL), bleeding on probing (BOP), gingi

128 analyzed, including probing depth, clinical attachment level (CAL), bleeding on probing, and percent

129 Probing depth (PD), clinical attachment level (CAL), bleeding on probing, and plaque

130 At baseline, probing depth (PD), clinical attachment level (CAL), bleeding on probing, and plaque

131 d tissue (KTw), probing depth (PD), clinical attachment level (CAL), clinician rating of color and te

132 gingival index, probing depth (PD), clinical attachment level (CAL), defect base level (DBL), and cre

133 intrabony defects was the change in clinical attachment level (CAL), for furcations the change in hor

134 nd post-therapy probing depth (PD), clinical attachment level (CAL), gingival recession (GR), and rad

135 cal parameters (probing depth (PD), clinical attachment level (CAL), gingival recession (GR), gingiva

136 PD, clinical attachment level (CAL), gingival recession, plaque index

137 l parameters, with the exception of clinical attachment level (CAL), had significantly (P <0.05) impr

138 ere recession depth, probing depth, clinical attachment level (CAL), height of keratinized tissue (wK

139 ters, including probing depth (PD), clinical attachment level (CAL), IBD depth, and percentage defect

140 ameters such as probing depth (PD), clinical attachment level (CAL), IBD depth, and percentage defect

141 meters, such as probing depth (PD), clinical attachment level (CAL), intrabony defect depth, and perc

142 ical recession (VR), probing depth, clinical attachment level (CAL), keratinized tissue width (KTW),

143 que index (PI), probing depth (PD), clinical attachment level (CAL), modified gingival index (GI), an

144 ing index (GI), probing depth (PD), clinical attachment level (CAL), number of teeth, CD4+ T lymphocy

145 ements, probing pocket depth (PPD), clinical attachment level (CAL), plaque index (PI), gingival inde

146 ters, including probing depth (PD), clinical attachment level (CAL), plaque index, and gingival index

147 ed by measuring probing depth (PD), clinical attachment level (CAL), plaque, bleeding on probing, vis

148 Clinical attachment level (CAL), probing depth (PD), and bleeding

149 The clinical attachment level (CAL), probing depth (PD), and gingival

150 using alveolar crest height (ACH), clinical attachment level (CAL), probing depth (PD), and percenta

151 cal gingival recession depth (VRD), clinical attachment level (CAL), probing depth (PD), and width of

152 ts meta-analyses were conducted for clinical attachment level (CAL), probing depth (PD), bleeding on

153 res of alveolar crest height (ACH), clinical attachment level (CAL), probing depth (PD), gingival ble

154 width of keratinized gingiva (KW), clinical attachment level (CAL), probing depth (PD), gingival ind

155 iological parameters were assessed: clinical attachment level (CAL), probing depth (PD), gingival rec

156 d 6 months were probing depth (PD), clinical attachment level (CAL), recession height (RH), width of

157 Gain in clinical attachment level (CAL), reduction in probing depth (PD),

158 (GTR) support substantial gains in clinical attachment level (CAL), reductions in probing depth (PD)

159 on measures of probing depth (PD), clinical attachment level (CAL), the radiographic pattern and ext

160 Recession, probing depth (PD), clinical attachment level (CAL), treatment time, and PROs were as

161 ng on probing (BOP), probing depth, clinical attachment level (CAL), waist circumference (WC), hsCRP,

162 dex (mSBI), probing depth (PD), and clinical attachment level (CAL), were recorded at baseline (befor

163 aque index, probing depth (PD), and clinical attachment level (CAL), were recorded at baseline.

164 bing (BOP), probing depth (PD), and clinical attachment level (CAL), were recorded before the treatme

165 x, plaque index, probing depth, and clinical attachment level (CAL), were recorded.

166 bing (BOP), probing depth (PD), and clinical attachment level (CAL), which were recorded at baseline

167 BOP); 3) probing depth (PD); and 4) clinical attachment level (CAL).

168 (BS); 3) probing depth (PD); and 4) clinical attachment level (CAL).

169 n in probing depth (PD) and gain in clinical attachment level (CAL).

170 dex (mSBI), probing depth (PD), and clinical attachment level (CAL).

171 que score, bleeding on probing, and clinical attachment level (CAL).

172 women, low BMD was associated with clinical attachment level (CAL).

173 The primary outcome variable was clinical attachment level (CAL).

174 bing (BOP), probing depth (PD), and clinical attachment level (CAL).

175 , mean probing depth (PD), and mean clinical attachment level (CAL).

176 bing depth, gingival recession, and clinical attachment level (CAL).

177 (GI); 4) probing depth (PD); and 5) clinical attachment level (CAL).

178 dex (mSBI), probing depth (PD), and clinical attachment level (CAL).

179 (FMBS), gingival recession, PD, and clinical attachment level (CAL).

180 margin position (GMP), and relative clinical attachment level (CAL).

181 zed tissue, probing depth (PD), and clinical attachment level (CAL).

182 index (GI), probing depth (PD), and clinical attachment level (CAL).

183 depth (PD), gingival recession, and clinical attachment level (CAL).

184 nd inflammatory outcomes, primarily clinical attachment level (CAL).

185 index (PI), probing depth (PD), and clinical attachment level (CAL).

186 dex (mSBI), probing depth (PD), and clinical attachment level (CAL).

187 e primary outcome was the change in clinical attachment level (CAL).

188 th including probing depth (PD) and clinical attachment level (CAL).

189 fferences in probing depth (PD) and clinical attachment level (CAL).

190 obing depth (PD): 9.03 +/- 1.62 mm; clinical attachment level (CAL): 11.16 +/- 1.81 mm) were treated

191 f the 12-month follow-up period: 1) clinical attachment level (CAL); 2) presence or absence of mobili

192 periodontal probing depth (PD); 2) clinical attachment level (CAL); 3) gingival recession; and 4) pe

193 t (NSPT): 1) probing depth (PD); 2) clinical attachment level (CAL); 3) plaque index (PI); 4) gingiva

194 x (mSBI); 3) probing depth (PD); 4) clinical attachment level (CAL); and 5) IBD depth.

195 measures of probing depth (PD) and clinical attachment levels (CAL) with 95% confidence intervals (C

196 d upon probing pocket depths (PPD), clinical attachment levels (CAL), and whole-mouth gingival bleedi

197 (probing depths [PDs] and vertical clinical attachment level [CAL-V]) and standardized radiographs w

198 index, probing depth [PD], vertical clinical attachment level [CAL-V]) were available for patients at

199 aque index, probing depth [PD], and clinical attachment level [CAL]) were recorded at baseline and 2

200 aque index, probing depth [PD], and clinical attachment level [CAL]) were recorded at baseline, 2 mon

201 aque index, probing depth [PD], and clinical attachment level [CAL]) were recorded at baseline, 3 mon

202 index [GI], probing depth [PD], and clinical attachment level [CAL]).

203 erapy clinical (probing depth [PD], clinical attachment level [CAL], and gingival recession [GR]) and

204 que index [PI], probing depth [PD], clinical attachment level [CAL], and percentage of sites with ble

205 inical (probing pocket depth [PPD], clinical attachment level [CAL], gingival recession [GR]) and rad

206 l measurements (probing depth [PD], clinical attachment level [CAL], recession, mobility, plaque inde

207 Clinical attachment levels (CALs; primary outcome), probing depth

208 points examined included changes in clinical attachment level, changes in bone level/fill, and probin

209 mple size >500, half-mouth minimum, clinical attachment level) containing prevalence data on destruct

210 ive trait of CP (mean interproximal clinical attachment level determined by full-mouth periodontal ex

211 Probing depth (PD), clinical attachment level, dichotomous presence or absence of sup

212 bsolute change in probing depth and clinical attachment level from baseline to 1-year follow-up.

213 uating plaque index, probing depth, clinical attachment level, furcation involvement, bleeding on pro

214 p had statistically significant open probing attachment level gain (95% confidence level, 3.18 to 4.3

215 Outcomes were clinical attachment level gain (CALg) and probing depth reduction

216 measure was absolute change in open probing attachment level gain and percentage defect resolution f

217 , gingival index, plaque index, and clinical attachment level gain at 90 days, demonstrating effectiv

218 eduction in deep probing depths and clinical attachment level gain in three of four specimens, in add

219 pared to 13.8% by SRP alone), and a clinical attachment level gain of 1.16 mm (compared to 0.80 mm by

220 actor-BB (rhPDGF-BB) led to greater clinical attachment level gain of approximately 1 mm compared to

221 greater probing depth reduction and clinical attachment level gain than the control group after 3 and

222 was 2.97 mm (95% CI: 2.38-3.56 mm), clinical attachment level gain was 1.65 mm (95% CI: 1.17-2.13 mm)

223 Probing depth reduction and clinical attachment level gain were obtained in three-fourths of

224 ical (RVCAL) and horizontal (RHCAL) clinical attachment level gain were shown to be greater in the AL

225 luded probing depth (PD) reduction, clinical attachment level gain, bleeding on probing (BOP) reducti

226 arameters: probing depth reduction, clinical attachment level gain, bleeding on probing reduction, an

227 us was determined by probing depth, clinical attachment level, gingival bleeding index, and the prese

228 exams that included probing depth, clinical attachment level, gingival bleeding, and radiographic al

229 parameters including probing depth, clinical attachment level, gingival index and plaque index were r

230 tal parameters (probing depth [PD], clinical attachment level, gingival index, bleeding on probing, a

231 ecession width, probing depth (PD), clinical attachment level, gingival index, plaque index, patient

232 imary outcome, bleeding on probing, clinical attachment level, gingival recession, interleukin-1beta,

233 s, and 6 months were probing depth, clinical attachment level, GR height, width of keratinized gingiv

234 mm, bleeding on probing [BOP], and clinical attachment level >/= 2 mm) and one healthy site (PD </=

235 rproximal probing depth >/=6 mm and clinical attachment level >/=4 mm were randomized into two groups

236 exhibiting probing depth >/=4 mm or clinical attachment level >/=4 mm.

237 site with probing depth >=5 mm and clinical attachment level >=2 mm at the same site.

238 leeding on probing (PD/BOP) or with clinical attachment level >=4 mm (CAL).

239 rs of periodontal probing depth and clinical attachment level, have been proven in multiple clinical

240 ent in the following: 1) horizontal clinical attachment level (HCAL); 2) vertical clinical attachment

241 xamined including: 1) interproximal clinical attachment level (iCAL), 2) interproximal probing depth

242 ve correlations with probing depth, clinical attachment level, IL-1beta, and IL-6.

243 subjects were stratified based on gender and attachment level into two groups.

244 bing depths (mean: 3.18 mm; SD: 0.59 mm) and attachment levels (mean: 3.93 mm; SD: 0.19) at 6 years w

245 s that are heavily influenced by probing and attachment level measurements alone.

246 s a reference for probing depth and relative attachment level measurements prior to surgery.

247 g index, probing depths, recession, clinical attachment level, mobility, furcation involvement, numbe

248 (2.42 mm vs. 1.32 mm) and a gain in clinical attachment level of 22% versus 7% (1.58 mm vs. 0.42 mm)

249 rease, but no significant effects on probing attachment level or percentage of root surface coverage.

250 efect coverage, keratinized tissue, clinical attachment level, or clinical healing for treatment of r

251 ontal probing depth (P <0.05), mean clinical attachment level (P <0.05), and sites with bleeding on p

252 ness, keratinized tissue width, and clinical attachment level (P <0.05).

253 l groups both for PD (P = 0.03) and clinical attachment level (P = 0.11).

254 rs recorded included probing depth, clinical attachment level, plaque index, and gingival index.

255 Probing depth, clinical attachment level, plaque index, and papilla bleeding ind

256 significant improvements in clinical status (attachment level, probing depth, plaque, gingivitis, and

257 to teeth with an initial reduced periodontal attachment level, provided adequate periodontal treatmen

258 ding index, probing depth (PD), and relative attachment level (RAL) were recorded at baseline and 3,

259 g index (mSBI), probing depth (PD), relative attachment level (RAL), and gingival marginal level (GML

260 Relative clinical attachment level (RCAL) and probing depth (PD) measures

261 PPD and relative clinical attachment level (rCAL) improved for all groups during f

262 uch as probing depth (PD), relative clinical attachment level (rCAL), and gingival margin level (GML)

263 3) probing depth (PD); 4) relative clinical attachment level (rCAL); and 5) gingival marginal level

264 ngival index (GI), PD, and relative clinical attachment levels (rCALs) was done at baseline and at 4

265 ter surgery, data on probing depth, clinical attachment level, recession depth and width, amount of k

266 ional periodontal measures, such as clinical attachment level, recession, and bleeding on probing.

267 Relative clinical attachment level (relative CAL) and probing depth (PD) m

268 e GRD type based on the midbuccal/midlingual attachment level respective to the interproximal bone le

269 achment level (RVAL) and relative horizontal attachment level (RHAL), and intrabony defect depth were

270 index, probing depth (PD), relative vertical attachment level (RVAL) and relative horizontal attachme

271 ve vertical and relative horizontal clinical attachment level [rvCAL and rhCAL], intrabony defect dep

272 6 months 3.68 +/- 0.86, P < 0.001), clinical attachment level (test BL 6.93 +/- 1.5, 6 months 5.3 +/-

273 (WKT), probing depth (PD), vertical clinical attachment level (VAL), visual plaque score (VPS), and b

274 n specific combinations of probing depth and attachment level values.

275 tical probing depth (VPD), vertical clinical attachment level (VCAL), gingival recession, and horizon

276 ttachment level (HCAL); 2) vertical clinical attachment level (VCAL); 3) probing depth (PD); and 4) l

277 Gain in clinical attachment level was 2.8 +/- 0.6 mm in OF and 2.3 +/- 0.

278 bing (BOP), probing depth (PD), and clinical attachment level, was performed at all PMT visits during

279 on probing, probing depth (PD), and clinical attachment level were carried out in four sites per toot

280 amount of keratinized gingiva, and clinical attachment level were evaluated.

281 ng, suppuration, probing depth, and clinical attachment level were measured at all teeth present.

282 Probing depth and clinical attachment level were measured at baseline and 1 year.

283 ameters including probing depth and clinical attachment level were measured, and a gingival tissue sa

284 ingival indices, probing depth, and clinical attachment level were measured.

285 probing depth, keratinized tissue (KT), and attachment level were recorded at baseline and 8 months

286 ing depth, bleeding on probing, and clinical attachment level were recorded during the second trimest

287 robing depth (PD), recession depth (RD), and attachment level were recorded.

288 ion, keratinized tissue, probing depths, and attachment levels were made initially, at 3 months, and

289 bleeding scores, probing depths and clinical attachment levels were observed for both test and contro

290 Probing depth and clinical attachment levels were obtained.

291 ing on probing, probing depths, and clinical attachment levels were performed at baseline, after 6 we

292 Gingival and periodontal pocket depth and attachment levels were recorded.

293 bleeding index, probing depth, and clinical attachment level) were recorded and samples of serum, sa

294 gingival index, probing depth, and clinical attachment level) were recorded.

295 x, plaque index, probing depth, and clinical attachment level, were recorded before GCF collection.

296 x, plaque index, probing depth, and clinical attachment level, were recorded.

297 ctions in probing depths and improvements in attachment levels while producing no detectable recessio

298 ded probing depth, recession depth, clinical attachment level, width of keratinized tissue, mobility,

299 aphs, and changes in probing depth, clinical attachment level, width of keratinized tissue, percussio

300 r correlations for pocket depth and clinical attachment level with bacterial amounts were observed fo