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1 al recognition memory and auditory function (auditory brainstem response).
2 accompanying hearing loss (estimated by the auditory brainstem response).
3 ted associations of these disorders with the auditory brainstem response.
4 ncy and threshold in the IC evaluated by the auditory brainstem response.
5 neuropathy, as measured using Wave I of the auditory brainstem response.
6 w greater between-trial phase-locking in the auditory brainstem response.
7 orresponding decline in the amplitude of the auditory brainstem response.
8 at 4 weeks of age as measured by tone burst auditory brainstem responses.
9 have normal auditory thresholds but abnormal auditory brainstem responses.
10 hs of age, after which they display abnormal auditory brainstem responses.
11 anied by permanent changes of latency of the auditory brainstem responses.
12 little changes in the thyroxine level and in auditory brainstem responses.
13 at was confirmed by a complete abrogation of auditory brainstem responses.
14 d intact cochlear amplification but impaired auditory brainstem responses.
15 c emissions and 70-80 dB threshold shifts in auditory brainstem responses.
16 al day 7 (P7) to P96 using voltage-clamp and auditory brainstem responses.
17 distortion product otoacoustic emissions and auditory brainstem responses.
18 ional knock-out (cKO) mice exhibit decreased auditory brainstem responses.
19 with reduced acoustic startle and distorted auditory brainstem responses.
20 h DT at P2, had normal hair cells and normal auditory brainstem responses.
22 nd WT mice in spontaneous eyeblink activity, auditory brainstem response (ABR) amplitudes, and tail-f
23 and functional hearing measures, such as the auditory brainstem response (ABR) and distortion product
25 the CN participates in the generation of the auditory brainstem response (ABR) and receives direct in
26 by reduction in wave I of the suprathreshold auditory brainstem response (ABR) and reduced number of
29 bited normal hearing function as assessed by auditory brainstem response (ABR) measurements, and thei
30 system function is being measured using the auditory brainstem response (ABR) or distortion product
32 ar improvements, including the average click auditory brainstem response (ABR) threshold, the tone-bu
33 shown by a 40-57 dB reduction in the average auditory brainstem response (ABR) thresholds at 0.5-4.0
34 there are no significant differences in the auditory brainstem response (ABR) thresholds between mut
35 ion product otoacoustic emission (DPOAE) and auditory brainstem response (ABR) thresholds during and
38 the results of our previous study that used auditory brainstem response (ABR) thresholds to identify
41 ct on murine hearing when assessed by any of auditory brainstem response (ABR) thresholds, distortion
43 pressure level (SPL) of 55-75 dB had similar auditory brainstem response (ABR) thresholds, wave-1 amp
44 In the current study, a QTL analysis for auditory brainstem response (ABR) thresholds, which indi
45 chlear responses, both in the neural output [auditory brainstem response (ABR) wave 1] and in outer h
46 sensory hair cells, 5) significantly delayed auditory brainstem response (ABR) wave I latencies at lo
51 rst part of this study, we derived the human auditory brainstem response (ABR), a measure of subcorti
53 ed to give close to normal thresholds for an auditory brainstem response (ABR), at least at low to mi
58 either Coch 131-150 or beta-tectorin 71-90, auditory brainstem responses (ABR) showed significant he
60 w2J/dfw2J homozygotes exhibit no discernible auditory brainstem responses (ABR) to sound pressure lev
63 ng testing as a function of sound frequency (auditory brainstem response -- ABR thresholds, and disto
64 ndent auditory threshold shifts (measured by auditory brainstem response, ABR) of up to 73 dB (16 kHz
65 oduct otoacoustic emissions (DPOAEs) and the auditory brainstem responses (ABRs) across the entire ra
66 creased thresholds and reduced amplitudes in auditory brainstem responses (ABRs) and decreased distor
67 ng loss and cochlear pathology, we collected auditory brainstem responses (ABRs) and determined cochl
70 mine this relationship further, we collected auditory brainstem responses (ABRs) from rhesus monkeys
72 dge this gap by using noninvasively recorded auditory brainstem responses (ABRs) to investigate devia
73 rcome some of these limitations by employing auditory brainstem responses (ABRs) to investigate the e
76 P2X4 knock-out (P2X4KO) mice showed improved auditory brainstem responses (ABRs) with smaller latenci
77 C), which is obtained typically by recording auditory brainstem responses (ABRs)-the BIC reflects the
86 ells, in adult mice virtually eliminated the auditory brainstem response and acoustic startle reflex,
87 3, two minke whales provided measures of the auditory brainstem response and data on the frequency ra
88 r 4-8 weeks of ADR treatment, as assessed by auditory brainstem response and distortion-product otoac
89 s seen by morphology and cochlear functions (auditory brainstem response and otoacoustic emissions).
91 d cochlear microphonics, as well as abnormal auditory brainstem responses and cortical auditory-evoke
92 mal cochlear function as indicated by normal auditory brainstem responses and distortion product otoa
94 hlear nerve fibers, and using measurement of auditory brainstem responses and otoacoustic emissions t
95 isplatin-induced ototoxicity, as measured by auditory brainstem responses and scanning electron micro
96 ity with enhanced acoustic startle response, auditory brainstem response, and cochlear microphonics b
97 d noise, a normal sense of balance, a normal auditory brainstem response, and normal transduction cur
98 latency and distortion in the wave I of the auditory brainstem responses, and elevated sensitivity t
99 ion in cochlear neural responses, as seen in auditory brainstem responses, and increased the loss of
100 estly increased interpeak intervals in their auditory brainstem responses, and substantially longer l
101 are approximately 10 dB more sensitive than auditory brainstem responses, and they are very sharply
105 of both strains were selected with matching auditory brainstem response audiograms and gap detection
106 ional measures of auditory function based on auditory brainstem responses, auditory-nerve synapse cou
107 elated changes in mouse hearing by recording auditory brainstem responses before and following exposu
108 e resulted in a temporary threshold shift in auditory brainstem responses but a persistent increase i
109 mouse, the ducky mouse (du), showed elevated auditory brainstem response click and frequency-dependen
110 ence of hearing preservation, as measured by auditory brainstem responses, compared with untreated ea
111 Homozygous mutant mice had no detectable auditory brainstem response, displayed highly disorganiz
112 congenital profound deafness, as assessed by auditory brainstem response, distortion product otoacous
113 on, mitigated NIHL, as evidenced by enhanced auditory brainstem responses, distortion product otoacou
114 ng distortion product otoacoustic emissions, auditory brainstem responses, envelope following respons
115 Height reduction coincides with dampened auditory brainstem responses evoked by low-frequency sti
120 ials) and auditory nerve/brainstem activity (auditory brainstem responses) have made it possible to d
122 fection or treatment and outer hair cell and auditory brainstem responses in children as young as 3 y
125 to function of the SGNs in vivo, we measured auditory brainstem responses in K(Na)1.1/1.2 double knoc
129 ulted in recovery of wave-I amplitude of the auditory brainstem response, indicating effective revers
130 ll transduction and decreased suprathreshold auditory brainstem response input/output gain in WT mice
131 nacin treatment of engrafted animals reduced auditory brainstem response interpeak latency, indicativ
133 distortion product otoacoustic emission and auditory brainstem response measurements in Col11a2 -/-
136 vidence from human temporal bone studies and auditory brainstem response measures suggests that this
139 ry brainstem response thresholds and reduced auditory brainstem response Peak 1 amplitudes showed lim
141 When tested for endocochlear potential and auditory brainstem response, PVM/M-depleted animals show
142 temporary threshold shift (TTS), evident in auditory brainstem response recordings as sound levels r
143 loss at 3 weeks after infection, measured by auditory brainstem response recordings, correlated to th
149 bjects with tinnitus and a normal audiogram, auditory brainstem responses show a significantly reduce
151 less variability when tapping to a beat have auditory brainstem responses that are less variable as w
153 in all patients at baseline, and the average auditory brainstem response threshold in the right (left
154 hat Abcb6 knockout mice exhibit an increased auditory brainstem response threshold, resulting in redu
155 erely hearing-impaired, as shown by elevated auditory brainstem response thresholds and absent endoco
157 a hearing deficit, as indicated by elevated auditory brainstem response thresholds at most frequenci
158 osure, nor were click- or noise-burst-evoked auditory brainstem response thresholds different from co
159 outer HCs is disrupted in Prox1DTA mice and auditory brainstem response thresholds in adults are 40-
160 ed higher hair cell survival rates and lower auditory brainstem response thresholds in injected ears
167 ividual differences observed in behavior and auditory brainstem response timing to cochlear synaptopa
170 develop a mathematical method to measure the auditory brainstem response to running speech, an acoust
172 without developmental dyslexia by measuring auditory brainstem responses to a speech syllable presen
174 tones; and (ii) physiological adaptation of auditory brainstem responses to clicks as a function of
176 processing of complex sounds such as speech (auditory brainstem responses to speech and other complex
177 oor readers have significantly more variable auditory brainstem responses to speech than do good read
178 shold, the middle-ear muscle reflex, and the auditory-brainstem response to clicks in various levels
179 e duration of the wave V-V(n) complex of the auditory brainstem response was studied, as was the effe
180 posure, thresholds had recovered but reduced auditory brainstem response wave 1 amplitudes and audito
184 topathy in humans, namely, modestly abnormal auditory brainstem response Wave I/Wave V ratios in the
185 xonal conduction velocity and caused shorter auditory brainstem response wave VI-I delays, providing
186 on were unaffected in these mutant mice, but auditory brainstem response wave-I amplitude was reduced
187 mal data, we demonstrate that the latency of auditory brainstem response wave-V in noise reflects aud
194 exhibit normal hearing as measured by evoked auditory brainstem responses, which suggests that the ne
195 ermore, 3-week-old double mutant mice lacked auditory brainstem responses, which were present in thei
196 fness characterized by an absent or abnormal auditory brainstem response with preservation of outer h
197 encies and interpeak latencies, evaluated by auditory brainstem response within 48 h after birth, wer