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1 wn GABA release and that the results of this autaptic action affect cone voltage-gated Ca2+ channel (
2 meostasis was unperturbed, demonstrated that autaptic activity has significant inhibitory effects on
3                Here we report that GABAergic autaptic activity is present in fast-spiking, but not in
4 ects of synaptic transmission differ between autaptic and dissociated cultures, and the synaptic tran
5 te and gamma-aminobutyric acid (GABA) act as autaptic and heterosynaptic presynaptic inhibitory trans
6 s a result of a larger quantal size, whereas autaptic and heterosynaptic PV-PV synapses differed in t
7 nd mIPSC frequencies did not deviate between autaptic and synaptic connections, the frequency of mEPS
8 receptor ligands exhibited 3.8 +/- 1.9 close autaptic appositions.
9                                              Autaptic conductance prolonged somatic inhibition in pvB
10 in pyramidal neurons by artificial GABAergic autaptic conductances, suggesting that tightly coupled s
11 ing model of neuronal activity, we study how autaptic connections affect activity patterns, and evalu
12 isolation on glial islands formed functional autaptic connections and continued to elaborate new syna
13                Therefore, in the hippocampus autaptic connections contribute to spike AHPs in many in
14              These results suggest a role of autaptic connections in controlling network-wide bursts
15 pairs formed both interneuronal synaptic and autaptic connections indiscriminately.
16 ns are self-innervated by powerful GABAergic autaptic connections reliably activated after each spike
17                                  Adding more autaptic connections to excitatory neurons increased the
18                                 Results show autaptic connections to excitatory neurons with high ave
19 gnificantly affects changes in bursting from autaptic connections.
20 arge numbers of conventional synaptic and/or autaptic contacts that can be easily visualized, making
21 naptic currents were dramatically reduced in autaptic cultures from MALS triple knockout mice due to
22 o different hippocampal neuron preparations: autaptic cultures in which a single isolated cell innerv
23                                       DSE in autaptic cultures is both more robust and elicited with
24 ed that CB1-dependent DSE can be elicited in autaptic cultures of excitatory hippocampal neurons of t
25  that CB1-dependent DSE can be elicited from autaptic cultures of excitatory mouse hippocampal neuron
26 BDNF) on excitatory synaptic transmission in autaptic cultures of hippocampal CA1 neurons.
27                                           In autaptic cultures, the total extent of evoked release, s
28 tatory glutamatergic synapses in hippocampal autaptic cultures.
29 and ABHD6 and determined their expression in autaptic cultures.
30     This change occurred without a change in autaptic current kinetics.
31                                          The autaptic current was abolished by GABA(A) receptor antag
32 epulses reversibly increased fast excitatory autaptic currents (eacs) mediated by alpha-amino-3-hydro
33 ure glia were rendered nonviable, excitatory autaptic currents (EACs) were prolonged in the presence
34 acid (AMPA) receptor component of excitatory autaptic currents (EACs) with an EC50 of 3.8 microM.
35 ak NMDA receptor-mediated (NMDAR) excitatory autaptic currents (EACs) with no effect on the NMDAR EAC
36 aspartate receptors (NMDARs), and inhibitory autaptic currents (iacs) mediated by gamma-aminobutyric
37          Compared to glycine, NMDAR-mediated autaptic currents decayed faster with sarcosine suggesti
38 izing prepulses did not significantly reduce autaptic currents in any neuron studied.
39 d the effect of hyperpolarizing prepulses on autaptic currents in cultured postnatal rat hippocampal
40                An additional requirement for autaptic DSE is filled internal calcium stores.
41 knockdown of DAGLalpha substantially reduces autaptic DSE, shifting the "depolarization-response curv
42 t have a role in determining the duration of autaptic DSE.
43                                              Autaptic endocannabinoid signalling is rich, robust and
44 dopamine neurons evoked a fast glutamatergic autaptic EPSC that showed presynaptic inhibition caused
45 ls of InsP(6) and occluded the inhibition of autaptic EPSCs by exogenous InsP(6).
46  also decreased paired pulse facilitation of autaptic EPSCs evoked by depolarization, indicating that
47            The InsP(6)-induced inhibition of autaptic EPSCs was effectively abolished by coapplicatio
48 ntibody on the InsP(6)-induced inhibition of autaptic EPSCs.
49                                   -Astrocyte autaptic evoked EPSCs, but not IPSCs, displayed an alter
50 used a concentration-dependent inhibition of autaptic excitatory postsynaptic currents (EPSCs) in cul
51 me neuron significantly inhibited the evoked autaptic GABA release.
52 itatory postsynaptic currents (EPSCs) in rat autaptic hippocampal cultures.
53  by systematically varying its expression in autaptic hippocampal glutamatergic neurons from mice of
54                              Here, using rat autaptic hippocampal microcultures, we show that memanti
55 N1/GluN2A and GluN1/GluN2B receptors and rat autaptic hippocampal microisland cultures, we show that
56 subunits modulate the synaptic plasticity of autaptic hippocampal neurons (i.e., Ca(v)beta(2a) induce
57 natomical evidence that MGL regulates DSE in autaptic hippocampal neurons and, taken together with ot
58                                     Cultured autaptic hippocampal neurons exhibit robust DSE.
59                                     Cultured autaptic hippocampal neurons from S426A/S430A mice showe
60  investigated the pharmacology of (+)-CBD in autaptic hippocampal neurons, a well-characterized neuro
61                         Finally, in cultured autaptic hippocampal neurons, CRIP1a overexpression atte
62 idine-1-carboxylate (JZL184)] prolong DSE in autaptic hippocampal neurons, whereas inhibition of ABHD
63 e retrograde inhibition via CB1 receptors in autaptic hippocampal neurons.
64                                  Perisomatic autaptic inhibition is common in both human and mouse pv
65                         To determine whether autaptic inhibition plays a functional role in the adult
66 y postsynaptic current (IPSC) in a PN and an autaptic IPSC.
67 rons and significantly prolongs the decay of autaptic IPSCs and miniature IPSCs in our cultures.
68 D6 is expressed in two distinct locations on autaptic islands, including a prominent localization in
69 eurons of mouse hippocampal brain slices and autaptic microcultures did not, per se, significantly af
70                                           In autaptic mouse neurons cultured on astrocytic microislan
71       Using patch-clamp electrophysiology of autaptic neuronal cultures from Rab3-deficient mouse hip
72 otocol makes it possible to transfect single autaptic neurons as well as mature neurons (15-82 days i
73 estion, we analyzed synaptic transmission in autaptic neurons cultured from RIM1alpha-/- mice.
74  was strongly depressed compared with either autaptic neurons or glutamatergic pairs.
75 ct mEPSC frequencies in either glutamatergic autaptic neurons or in glutamatergic pairs.
76         When expressed in Snap-25-null mouse autaptic neurons, region I mutations reduced the size of
77 ng in vitro assays and rescue experiments in autaptic neurons, we show that interactions within regio
78 otypic" glutamatergic or GABAergic pairs and autaptic neurons.
79 ponsible for a portion of 2-AG production in autaptic neurons.
80 hment rate of the readily releasable pool in autaptic neurons.
81 same drug or one of the others in individual autaptic neurons.
82                                         Peak autaptic NMDA responses were potentiated to 178.9 +/- 22
83         Thus, well-timed inhibition, whether autaptic or synaptic, facilitates precise spike timing a
84  experimentally demonstrate a new electronic autaptic oscillator (EAO) that uses engineered feedback
85 pocampal neurons in culture, the size of the autaptic readily releasable pool before and after stimul
86                                              Autaptic self-inhibition represents an exceptionally lar
87 d a significant impairment in PV interneuron autaptic self-inhibitory transmission, a feature implica
88 The formation of new cholinergic synapses in autaptic single-cell microcultures is inhibited by SPARC
89                                              Autaptic strength was greater than synaptic strength ont
90                           We discovered that autaptic synapses are optimized for maximal transmission
91 ot only interneuronal synapses, but also the autaptic synapses on itself exhibited a trend toward enh
92 ted higher rates of spontaneous release than autaptic synapses.
93                         Overall, single-axon autaptic transmission contributed to approximately 40% o
94                          Indeed, blockade of autaptic transmission degraded temporal precision in mul
95                              The strength of autaptic transmission modulated the coupling of PV-cell
96                                We found that autaptic transmission represents the most powerful inhib
97         These two classes displayed distinct autaptic transmission.
98 ecise self-inhibition mediated by inhibitory autaptic transmission.