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1 en were diagnosed with ASD, of whom 5689 had autistic disorder.
2 otherwise specified (PDD-NOS), to 0.53, for autistic disorder.
3 ebo-controlled study of NAC in children with autistic disorder.
4 C for treating irritability in children with autistic disorder.
5 isorder than in the parents of children with autistic disorder.
6 rents of children with a diagnosis of DSM-IV autistic disorder.
7 f gastrointestinal symptoms in children with autistic disorder.
8 or self-injurious behavior in children with autistic disorder.
9 o identify susceptibility loci implicated in autistic disorder.
10 nding the development and persistence of the autistic disorder.
11 n of the role of GABRB3 or adjacent genes in autistic disorder.
12 (3) 10 of the 14 met the DSM-IV criteria for autistic disorder.
13 n depletion in drug-free adult patients with autistic disorder.
14 logy of fragile X syndrome (FXS) and related autistic disorders.
15 ave been reported as contributing factors to autistic disorders.
18 ample had been diagnosed with ASDs: 114 with autistic disorder, 56 with Asperger syndrome, and 100 wi
19 s compared with placebo for the treatment of autistic disorder accompanied by severe tantrums, aggres
20 of pervasive developmental disorder (PDD) or autistic disorder (AD) according to International Classi
21 imal 15q have been found in individuals with autistic disorder (AD) and varying degrees of mental ret
23 lastoid cell lines (LCLs) from children with autistic disorder (AD) show mitoplasticity (AD-A), presu
24 ovo mutations (DNMs) from 1628 subjects with autistic disorder (AD), 1873 from 1564 subjects with per
30 -functioning adult male subjects with DSM-IV Autistic Disorder (age 18-45 years; full scale IQ >70; A
31 were compared with scans from 15 adults with autistic disorder and 17 age-matched comparison subjects
32 Overall, 103 of 6959 children (1.5%) with autistic disorder and 180 of 15,830 (1.1%) with mental r
33 -13, revealed linkage disequilibrium between autistic disorder and a marker in the gamma-aminobutyric
36 sociated with a small increase in the RR for autistic disorder and mental retardation compared with I
37 larger in both the parents of children with autistic disorder and the adults with autistic disorder,
38 inkage and association were observed between autistic disorder and the two SNPs, rs2056202 and rs2292
39 8, 1.14) for all ASDs, 1.12 (0.97, 1.30) for autistic disorder, and 1.63 (1.30, 2.04) for ASD-NOS.
41 autism spectrum disorders (childhood autism [autistic disorder], Asperger syndrome, atypical autism,
42 ing the analysis to singletons, the risks of autistic disorder associated with ICSI using surgically
43 ddlers who received a confirmed diagnosis of autistic disorder at approximately 48 months of age and
48 VF treatment overall was not associated with autistic disorder but was associated with a small but st
49 thers took folic acid, 0.10% (64/61,042) had autistic disorder, compared with 0.21% (50/24,134) in th
52 supplements showed no such association with autistic disorder, even though fish oil use was associat
53 atistically significantly increased risks of autistic disorder following ICSI using surgically extrac
56 mated to be 0.50 (95% CI, 0.45-0.56) and the autistic disorder heritability was estimated to 0.54 (95
61 rn in Sweden, the individual risk of ASD and autistic disorder increased with increasing genetic rela
69 dose of secretin, thousands of children with autistic disorders may have received secretin injections
71 lts (age [mean+/-SD], 28.1+/-7.3 years) with autistic disorder (n=17) or pervasive developmental diso
72 with autism spectrum disorder (i.e., DSM-IV autistic disorder or Asperger's disorder) (n = 34) and m
73 were followed up for a clinical diagnosis of autistic disorder or mental retardation until December 3
74 atment approach for medicating children with autistic disorder or other pervasive developmental disor
75 nty-one pediatric subjects with diagnoses of autistic disorder or other pervasive developmental disor
76 Pediatric populations, including those with autistic disorder or other pervasive developmental disor
77 ms with a diagnosis code in any position for autistic disorder or other specified pervasive developme
78 cer was primarily noted for narrowly defined autistic disorder (OR 1.7, 95% CI 1.3-2.1) and ASD with
79 noses of specific autism spectrum disorders (autistic disorder, pervasive developmental disorder-not
80 ental Disorders, Fourth Edition criteria for autistic disorder, pervasive developmental disorder-not
81 n with autistic disorder and the adults with autistic disorder, relative to the comparison subjects.
82 left amygdala was smaller in the adults with autistic disorder, relative to the other two groups.
84 birth to 6 to 14 months; 59% of infants with autistic disorder showed these accelerated growth trajec
85 was significantly larger in the adults with autistic disorder than in the parents of children with a
86 at an abnormal growth rate in toddlers with autistic disorder that was mainly characterized by a qua
87 ay partly explain the failure of people with autistic disorders to inhibit repetitive thoughts and ac
88 ility group, respectively, and the ratios of autistic disorders to other ASD subtypes were 1:2.6 and
89 acebo lead-in phase, drug-free subjects with autistic disorder were administered three different dose
91 t hub function shared across the spectrum of autistic disorders - whether caused by rare highly penet
92 renia and may be beneficial in children with autistic disorder who have serious behavioral disturbanc
93 and diagnostic criteria for the spectrum of autistic disorders will change and become more specific
94 ause of previous reports of individuals with autistic disorder with duplications of the Prader-Willi/
95 were significantly enlarged in toddlers with autistic disorder, with the most severe enlargement occu
96 that parents of children with a diagnosis of autistic disorder would show similar changes in these st