ライフサイエンスコーパス: auxin

1 se to auxin requires AUX1 and/or cytoplasmic auxin.

2 ed in cellular responses to the phytohormone auxin.

3 lopmental program coordinated by the hormone auxin.

4 ncreasing nuclear abundance and signaling of auxin.

5 -LIKES (PILS)-dependent nuclear abundance of auxin.

6 root tips were unaffected by the addition of auxin.

7 y resistant to picolinates, but not to other auxins.

8 control during a 48 h time-course where the auxin 1-naphthaleneacetic acid (NAA) was applied to pre-

9 responses to IAA and the membrane-permeable auxin 1-naphthylacetic acid (NAA).

10 formation, 7 days post-treatment (dpt) with auxin, 2,4-D.

11 Auxin, a cardinal plant hormone with morphogen-like prop

12 ravistimulation situation.(1-3) Differential auxin accumulation during the gravitropic response depen

13 nitiation are associated with local peaks of auxin accumulation.

14 nd allowed for rapid circuit activation upon auxin addition.

15 ipt abundance of MdGH3-2 encoding a putative auxin amido conjugate synthase, resulting in a lower fre

16 application of indole-3-acetic acid (IAA) or auxin analogues might effectively protect field crops ag

17 so identified its hyposensitive responses to auxin and abscisic acid treatments and enhanced far-red

18 eral roots were able to respond to exogenous auxin and AtDRO1 gene expression levels in root tips wer

19 StBMI1-1-AS lines revealed downregulation of auxin and brassinosteroid genes, and upregulation of cyt

20 Auxin and brassinosteroids (BR) are crucial growth regul

21 This study examined the effects of elevated auxin and ethylene on the metabolome of Arabidopsis root

22 mone pathways, particularly between those of auxin and ethylene.

23 ient elongation of hypocotyls in response to auxin and for the correct expression of a subset of auxi

24 tiation and outgrowth depends on the hormone auxin and is robust across diverse environments.(3-6) He

25 pathway but prominent suppression of the JA, auxin and light-regulated pathways.

26 jasmonic acid, brassinosteroids, cytokinins, auxin and synthesis of flavonoid, phenylpropanoids and c

27 Auxin and wound-induced turgor pressure changes together

28 Auxins are inhibitors of grape berry ripening and their

29 , during organogenesis, temporal patterns of auxin arise from rhythmic centrifugal waves of high auxi

30 shoot module, a regulatory axis composed of auxin biosynthesis and auxin perception factors controls

31 s an on/off switch to control TAA1-dependent auxin biosynthesis and is required for proper regulation

32 CUC2 regulate the transcription of the local auxin biosynthesis gene YUC4 in a coherent feed-forward

33 lational reporter expression patterns for 14 auxin biosynthesis genes.

34 reduced auxin signaling, and restoration of auxin biosynthesis is sufficient to restore flower outgr

35 suggesting post-translational regulation of auxin biosynthesis may be a general phenomenon.

36 E OF ARABIDOPSIS (TAA1), a key enzyme in the auxin biosynthesis pathway in Arabidopsis thaliana is ph

37 es, both clv2/crn signaling and heat-induced auxin biosynthesis via YUCCA family genes are synergisti

38 the identification of key genes involved in auxin biosynthesis, transport, and signaling.

39 distribution in addition to light-dependent auxin biosynthesis.

40 AFLET SUPPRESSION1 (LLS1) gene, encoding the auxin biosynthetic enzyme YUCCA1 in Medicago truncatula.

41 on, the results suggest that manipulation of auxin biosynthetic pathway genes can be an effective app

42 ing components of the signalling pathways of auxin, brassinosteroids (BRs) and cytokinins.

43 isely controlled temporally and spatially by auxin, brassinosteroids, and light to result in AM initi

44 ed long-term actin cytoskeleton responses to auxin, but plants respond to auxin within minutes.

45 Conversely, ERF115 sensitizes cells to auxin by activating ARF5/MONOPTEROS, an auxin-responsive

46 The quantification of auxin can be compromised by the breakdown of labile auxi

47 Exogenous application of auxin can dramatically enhance the transcription of MUS,

48 tic activity may contribute to regulation of auxin concentration in response to endogenous and/or ext

49 velopmental fate of plant tissues, and local auxin concentration is precisely controlled.

50 onjugate synthase, resulting in a lower free auxin concentration; feeding the transgenic leaves with

51 nscription have largely been elucidated, how auxin controls cell expansion is only now attaining mole

52 o a single, comprehensive explanation of how auxin controls cell expansion, and where more research i

53 The hormone auxin controls many aspects of the plant life cycle by r

54 The phytohormone auxin controls plant growth and development via TIR1-dep

55 e report that in Arabidopsis thaliana roots, auxin controls the spatiotemporal expression of the plas

56 tionally, a dominant mutant (axr2-1) for the auxin coreceptor AUXIN RESPONSIVE2 (AXR2) was strongly s

57 ition in P. patens is underpinned by complex auxin-cytokinin crosstalk that is regulated, at least in

58 for full actin rearrangements in response to auxin, cytoplasmic auxin (i.e. NAA) stimulated a lesser

59 Using an inducible auxin-degron system to rapidly deplete RPB1 (the largest

60 FC recruitment is auxin-dependent and is abolished by treatment with a pol

61 In contrast to auxin-dependent degradation of canonical AUX/IAA protein

62 integration of multiple pathways regulating auxin-dependent flower production.

63 ly, AFB1 has a specialized function in rapid auxin-dependent inhibition of root growth and early phas

64 phot1 may directly regulate ABCB19 to adjust auxin-dependent leaf responses.

65 stalk mechanism integrates BR signaling into auxin-dependent organ growth rates and likely has widesp

66 d promotes lateral root development when the auxin-dependent proteolysis pathway fails.

67 e AID system that incorporates the synthetic auxin derivative 5-Ad-IAA and its high-affinity-binding

68 Auxin determines the developmental fate of plant tissues

69 echanism for the plant hormone auxin whereby auxin directly affects the activity of the atypical auxi

70 owth, organ bending, and subsequent reset of auxin distribution back to the original pre-gravistimula

71 e caused by misregulation of ABCB19-mediated auxin distribution in addition to light-dependent auxin

72 cannot explain the experimentally determined auxin distribution in the root tip.

73 n many auxin-regulated processes, asymmetric auxin distribution, and PIN trafficking.

74 Visualization of auxin distribution, measurement of auxin transport in pr

75 tal auxin fluxes re-capitulates the root-tip auxin distribution.

76 r for auxin signaling and proxy for relative auxin distribution.

77 However, the role of auxin during the late developmental stages and outgrowth

78 It is well established that auxin dynamics depend on the spatial distribution of eff

79 In this review, we focus on rapid auxin effects, their relationship to H(+)-ATPase activat

80 viding a possible mechanism for the observed auxin effects.

81 r transcript abundance of MdPIN1 encoding an auxin efflux carrier but a higher transcript abundance o

82 tivity of polarly localized PIN-FORMED (PIN) auxin-efflux carriers.(1-4) In particular, the timing of

83 nsible for directional transport, namely PIN auxin exporters.

84 patterning through the TRANSPORT INHIBITOR 1/AUXIN F-BOX (TIR1/AFB) transcriptional pathway and can a

85 vascular stem cell death obstructs the polar auxin flux, much alike rocks in a stream, and causes it

86 tes both carrier-mediated and plasmodesmatal auxin fluxes re-capitulates the root-tip auxin distribut

87 ect both carrier-mediated and plasmodesmatal auxin fluxes.

88 nization, providing additional evidence that auxin functions through a transcriptional pathway for tr

89 functions (calcium and MAPK), phytohormones (auxin, gibberellins, abscisic acid, JA and SA), and seco

90 These bioregulators include auxins, gibberellins, cytokinins, abscisic acid, brassin

91 analyzing expression, protein localization, auxin gradient formation, and auxin responsiveness in th

92 root tips, detectable in nuclei, and impacts auxin gradient formation.

93 the L92A/I94A change to AtLAZY1 reversed the auxin gradient normally established across stems by the

94 l roots showed impairment in establishing an auxin gradient upon gravistimulation as visualized with

95 covery opens up new avenues for studying how auxin gradients form across organs and new approaches fo

96 ot apex, but the spatio-temporal dynamics of auxin gradients is unknown.

97 stemergence (POST) wheat-selective synthetic auxin herbicide, using alien substitution (the S genome

98 isregulation of MADS78 and MADS 79 perturbed auxin homeostasis and carbon metabolism, as evident by m

99 This alters auxin homeostasis and transport, consequently leading to

100 munity, growth, and development by affecting auxin homeostasis in planta.

101 rangements in response to auxin, cytoplasmic auxin (i.e. NAA) stimulated a lesser response.

102 of signals via plasmodesmata, is induced by auxin in cells overlying LRP in a progressive manner.

103 directional (polar) transport of the hormone auxin in plants.

104 ement cell-shape acquisition and the role of auxin in this process, we focused on the spirals of youn

105 This localized auxin increase balances wound-induced cell expansion and

106 ed cells in wound perception and detected an auxin increase specific to cells immediately adjacent to

107 usly by two counteracted teams including (1) auxin/indole-3-acetic acid (AUX/IAA)-histone deacetylase

108 lled by two counteracted teams including (1) auxin/indole-3-acetic acid (AUX/IAA)-histone deacetylase

109 All 16 maize auxin/indole-3-acetic acid repressor proteins were degra

110 YABBY5 (CsYAB5), BREVIPEDICELLUS (CsBP), and AUXIN/INDOLEACETIC ACIDS4 (CsAUX4) and promotes their ex

111 Comparison of the auxin-induced changes in gene expression with the patter

112 SISTANT 1 (AUX1) - was considered to precede auxin-induced cytoskeleton reorganization.

113 To address this question, we apply an Auxin-Induced Degron (AID) system to distinguish roles o

114 SPONSIVE2 (AXR2) was strongly suppressed for auxin-induced microtubule array reorganization, providin

115 Auxin-induced root hair initiation and ROS accumulation

116 Auxin-induced TMK1 nanoclustering stabilizes flotillin1-

117 The association of these elements with auxin-induced up-regulation (DR and IR) or down-regulati

118 Here we found that, phytohormone auxin-induced, sterol-dependent nanoclustering of cell s

119 Thus, exogenous auxin induces transverse microtubule patterning through

120 Remarkably, a significant proportion of auxin inducible genes and of targets of the AUXIN RESPON

121 auxin, most likely by regulating a subset of auxin inducible genes related to cell expansion.

122 Through an auxin-inducible approach(10), we degraded the cohesin co

123 Finally, we used CRISPR-Cas9 auxin-inducible degradation to determine that phenotypic

124 Conditional depletion of PICH using the auxin-inducible degron (AID) system resulted in the rete

125 Further assessment using Smc5 cKO and auxin-inducible degron systems demonstrated that absence

126 nd for the correct expression of a subset of auxin-inducible genes In this work, we analyzed the resp

127 Auxin is a crucial plant growth regulator.

128 The hormone auxin is a key signal for plant growth and development t

129 Auxin is a key signal regulating plant growth and develo

130 Auxin is intimately connected to vascular tissue develop

131 In planta, auxin is the first hormone group that was discovered and

132 In addition, we show that auxin itself, in part via TRANS-MEMBRANE KINASE 4 (TMK4)

133 y effect on TMK1 signaling is antagonized by auxin itself, which triggers rapid MAKR2 membrane dissoc

134 asymmetric distribution of the plant hormone auxin, leading to asymmetric growth, organ bending, and

135 et-point angle (GSA) of lateral roots (LRs), auxin levels and auxin transport.

136 Medicago control overall RSA, LR GSA, shoot auxin levels and rootward auxin transport.

137 pling these growth responses and maintaining auxin levels in the root.

138 otoplasts, and direct quantification of free auxin levels suggest these irregularities are caused by

139 dition that causes an increase in endogenous auxin levels.

140 rootward auxin transport and elevated shoot auxin levels.

141 itri Melatonin is a ubiquitously distributed auxin-like metabolite found in both prokaryotes and euka

142 cell organizer that interprets wound-induced auxin maxima.

143 putative cellulose synthases indicated that auxins may preserve cell wall structure.

144 ffinose while simultaneously acting to limit auxin-mediated cell expansion.

145 s a more nuanced role for DRO1 in regulating auxin-mediated changes in lateral branch angle.

146 in regulating PIN polarity, trafficking, and auxin-mediated development.

147 AFB signaling is required and sufficient for auxin-mediated PIN3 re-polarization and shoot gravitropi

148 Auxin metabolism-related and signalling pathway-related

149 terise an alternative symplastic pathway for auxin mobilisation via plasmodesmata, which function as

150 ired for an efficient elongation response to auxin, most likely by regulating a subset of auxin induc

151 The plant hormone auxin must be transported throughout plants in a cell-to

152 es In this work, we analyzed the response to auxin of plants with altered function of the class I TEO

153 The promotive effect of auxin on shoot cell expansion provided the bioassay used

154 feeding the transgenic leaves with exogenous auxin partially restores leaf width.

155 Furthermore, we found that auxin participated in carpel number variation in cucumbe

156 hermoresponsive growth through the phyB-PIF4-auxin pathway.

157 nsive growth, indicating that epidermal PIF4-auxin pathways are essential for the temperature respons

158 armacological interference with ethylene and auxin pathways outlines the hierarchy of responses, plac

159 To investigate the role of plasmodesmata in auxin patterning, we developed a multicellular model of

160 While the mechanisms underlying auxin perception and signaling to regulate transcription

161 tory axis composed of auxin biosynthesis and auxin perception factors controls root responses to high

162 Auxin plays a central role in controlling plant cell gro

163 related genes from duplications and elevated auxin production are associated with aerial root develop

164 henotypic defects characteristic of enhanced auxin production.

165 Before this work, an extracellular auxin receptor - rather than the auxin transporter AUXIN

166 The ZmAFB2/3 b1 auxin receptor was more sensitive to hormone than AtAFB2

167 n of PDLP5 in LRP-overlying cells into known auxin-regulated LRP-overlying cell separation pathways,

168 S3-NCED3 functions as an important avenue in auxin-regulated poplar root growth in response to drough

169 and ala3 T-DNA mutants show defects in many auxin-regulated processes, asymmetric auxin distribution

170 Auxin regulates abundance of the trafficking SNARE SYP13

171 The plant growth hormone auxin regulates development via a family of transcriptio

172 sitional information provided by the hormone auxin regulates rhythmic organ production at the shoot a

173 an be compromised by the breakdown of labile auxin-related compounds during sample preparation.

174 ocalisation, thereby controlling vacuole and auxin-related developmental processes in Arabidopsis emb

175 differentially methylated genes, many within auxin-related gene pathways.

176 idopsis transgenic lines corresponding to 62 auxin-related genes and characterizing the translational

177 Copy number expansion of auxin-related genes from duplications and elevated auxin

178 Forward genetic screens for auxin-related mutants have led to the identification of

179 ads to reduced auxin response and widespread auxin-related phenotypic defects in Arabidopsis (Arabido

180 Expression of the auxin reporter DR5::GUS was also higher in a tcp15 mutan

181 Short-term actin cytoskeleton response to auxin requires AUX1 and/or cytoplasmic auxin.

182 receptor - rather than the auxin transporter AUXIN RESISTANT 1 (AUX1) - was considered to precede aux

183 In Arabidopsis thaliana, AUXIN RESISTANT1 (AXR1) functions as the E1-ligase in th

184 n the cell wall metabolism, lipid transport, auxin response and flavonoid biosynthesis.

185 dataset was enriched with genes involved in auxin response and signaling; and in anatomical structur

186 IFIC PROTEASE14 (TNI/UBP14) leads to reduced auxin response and widespread auxin-related phenotypic d

187 A complete auxin response circuit comprising all maize components,

188 Here, we used the nuclear Auxin Response Circuit recapitulated in yeast (Saccharom

189 growth and development that acts through the AUXIN RESPONSE FACTOR (ARF) transcription factors(2-4).

190 irectly affects the activity of the atypical auxin response factor (ARF), ETTIN towards target genes

191 (AUX/IAA)-histone deacetylase (HDA) and (2) auxin response factor (ARF)-histone acetyltransferase (H

192 (AUX/IAA)-histone deacetylase (HDA) and (2) auxin response factor (ARF)-histone acetyltransferase (H

193 e encoding a microRNA (MIR160B) that targets AUXIN RESPONSE FACTOR (ARF)10 and ARF16 that are involve

194 auxin inducible genes and of targets of the AUXIN RESPONSE FACTOR 6 are regulated by TCP15 and often

195 iption of MUS, which is largely dependent on AUXIN RESPONSE FACTOR 7 (ARF7) and ARF19.

196 on a pair of closely related homologs of the AUXIN RESPONSE FACTOR family, AqARF6 and AqARF8, to expl

197 ng to reduce trans acting small interference Auxin Response Factor production and modulating nodule f

198 Notably, several SAUR genes and the auxin response gene IAA19 also showed reduced expression

199 lts validate the conserved role of predicted auxin response genes in maize as well as provide evidenc

200 ese cells allowed us to demonstrate that the auxin response gradient forms within the cells of the sp

201 formation, suggesting that these fluctuating auxin response gradients are orchestrated via auxin tran

202 pment of mutant cotyledons and root, and the auxin response of mutant seedlings supported the hypothe

203 3b levels likely contributes to the impaired auxin response phenotypes of mta mutant plants.

204 ed the model predictions using the DII-VENUS auxin response reporter, comparing the predicted and obs

205 y identifies a function for TNI/UBP14 in the auxin response through ubiquitin recycling.

206 eins, which normally repress the activity of auxin response transcription factors (ARFs).

207 er reveals strong genome-wide association of auxin response with both inverted (IR) and direct (DR) A

208 In addition to the reduced auxin response, increased levels of DII:VENUS, IAA18:GUS

209 hat brassinosteroid application mimicked the auxin response, showing both early and late microtubule

210 transcription factor involved in the global auxin response, tissue patterning, and organ formation.

211 here laterally peripheral cells have a lower auxin response, which is associated with a lower prolife

212 RNA-Seq and qRT-PCR analyses reveal that auxin-responsive genes and growth-related genes are high

213 Mutants also exhibit misregulation of auxin-responsive genes.

214 based on altered leaf shape, activity of the auxin-responsive reporters DR5::GUS, DR5::nYFP, and IAA2

215 A ARFs(5), are transcriptional activators of auxin-responsive target genes that are essential for reg

216 s to auxin by activating ARF5/MONOPTEROS, an auxin-responsive transcription factor involved in the gl

217 ant mutant (axr2-1) for the auxin coreceptor AUXIN RESPONSIVE2 (AXR2) was strongly suppressed for aux

218 localization, auxin gradient formation, and auxin responsiveness in the atdro1 mutant.

219 ing of collapse of damaged cells and a local auxin signaling activation to coordinate the downstream

220 tion studies suggest that TNI is involved in auxin signaling and acts alongside TIR1, ARF7, and AUX1

221 n as visualized with DII-VENUS, a sensor for auxin signaling and proxy for relative auxin distributio

222 The auxin signaling and root morphogenesis are harmoniously

223 bout the functions of histone acetylation in auxin signaling and root morphogenesis.

224 of histone acetylation in the modulation of auxin signaling as well as in the regulation of root mor

225 stem cell identity and negatively regulates auxin signaling by interacting with ARF10 and ARF16.

226 rentially affect processes downstream of the auxin signaling cascade.

227 em to functionally annotate maize (Zea mays) auxin signaling components, focusing on genes expressed

228 components, including the ZmAFB2/3 b1 maize AUXIN SIGNALING F-BOX (AFB) receptor, was fully function

229 Although auxin signaling function has diverged and expanded, diff

230 that the bHLH TF MdbHLH3 directly modulates auxin signaling in controlling leaf shape in response to

231 el mechanism integrating peptide hormone and auxin signaling in the regulation of flower development

232 us overproliferation when the canonical TIR1 auxin signaling is disrupted.

233 Moreover, the balance in auxin signaling is very critical to contribute to normal

234 In this report, we suggest that the auxin signaling must be controlled harmoniously by two c

235 onate signaling pathway gene, PuMYC2, and an auxin signaling pathway gene, PuAGL12.

236 responses, placing ethylene epistatic to the auxin signaling pathway.

237 nctions of non-canonical AUX/IAA proteins in auxin signaling transduction.

238 itionally, inactivation of epidermal PIF4 or auxin signaling, and overexpression of epidermal phyB su

239 eveloping crn inflorescences display reduced auxin signaling, and restoration of auxin biosynthesis i

240 However, the mechanisms that coordinate auxin signaling, cytoskeletal remodeling and cell expans

241 organ development, meristem development and auxin signaling.

242 gesting a self-regulatory loop whereby local auxin signalling can suppress biosynthesis.

243 levels of class A ARF proteins and modulate auxin signalling output throughout development.

244 rget genes that are essential for regulating auxin signalling throughout the plant lifecycle(2,3).

245 t degradation of canonical AUX/IAA proteins, auxin stabilizes IAA33 protein via MITOGEN-ACTIVATED PRO

246 PHOTOTROPISM2, and alterations in localized auxin streams.

247 this system generally requires high dose of auxin to achieve effective depletion in vertebrate cells

248 Application of auxin to the brassinosteroid synthesis mutant, diminuto1

249 Consistently, the application of auxin to wild-type shoots induced a steeper GSA and auxi

250 tress-associated phytohormones (salicylates, auxins, trans-jasmonic acid, and abscisic acid) and the

251 utation of the acidic residue also abolishes auxin transport activity by ABCB1.

252 The auxin transport activity of ABCB1 was suggested to be re

253 increases both its malate and its background auxin transport activity, suggesting that this motif has

254 ar basis for NPA inhibition of PIN-dependent auxin transport and a logical parsimonious explanation f

255 ing tonoplast proton pumps were defective in auxin transport and distribution.

256 or mutants exhibited an increase in rootward auxin transport and elevated shoot auxin levels.

257 vident by misregulation of genes involved in auxin transport and signaling as well as starch biosynth

258 n of Arabidopsis thaliana, interplay between auxin transport and transcription factors named CUP SHAP

259 1 graft progeny, a phenotype associated with auxin transport based on inhibitor assays.

260 ing as a putative activator of ABCB-mediated auxin transport by cis-trans isomerization of peptidyl-p

261 n phenotypes of PILS5 putative intracellular auxin transport facilitator.

262 All higher plant ABCBs for which auxin transport has been conclusively proven carry this

263 zation of auxin distribution, measurement of auxin transport in protoplasts, and direct quantificatio

264 eptides increased GSA and inhibited rootward auxin transport in wild-type but not in CEP receptor mut

265 t and is abolished by treatment with a polar auxin transport inhibitor.

266 o wild-type shoots induced a steeper GSA and auxin transport inhibitors counteracted the CEP receptor

267 thin protoplasts is dynamic and resistant to auxin transport inhibitors.

268 oboxes, X-ray computed tomography, grafting, auxin transport measurements and hormone quantification

269 uxin response gradients are orchestrated via auxin transport to control lobe formation and determine

270 d is regulated by brassinosteroid signaling, auxin transport, and gibberellin biosynthesis.

271 These genes include, root-specific auxin transport, strigolactone and gibberellin biosynthe

272 pivotal tool in elucidating the unique polar auxin transport-based processes underlying plant growth

273 entations in response to gravity via altered auxin transport.

274 SA) of lateral roots (LRs), auxin levels and auxin transport.

275 is crucial for their function in directional auxin transport.

276 consistent with flavonols modulating ROS and auxin transport.

277 RSA, LR GSA, shoot auxin levels and rootward auxin transport.

278 The major DEGs related to auxin transportation (e.g., PIN-FORMED1) and cell wall d

279 Direct phosphorylation of the auxin transporter ATP-BINDING CASSETTE subfamily B19 (AB

280 tracellular auxin receptor - rather than the auxin transporter AUXIN RESISTANT 1 (AUX1) - was conside

281 which is involved in polar recycling of the auxin transporter PIN-FORMED1.

282 lated transcription factor BZR1/BES1 and the auxin-transporter Dwarf3 were found to be highly correla

283 PIN auxin transporters are important cell polarity markers t

284 We revealed that specific localization of auxin transporters at the different membranes of these y

285 identity marker ATHB8, nor properly polarise auxin transporters to specify new venation paths.

286 Brassinosteroid receptors and auxin transporters, both of which are sustained by CLASP

287 re D/E-P motif that seems to be specific for auxin-transporting ABCBs, which we now refer to as ATAs.

288 rise from rhythmic centrifugal waves of high auxin travelling through the tissue faster than growth.

289 mponent analysis showed that the control and auxin-treated samples were most different at 3 h post-tr

290 Auxin treatment of grape (Vitis vinifera L.) berries del

291 howed reduced expression in the mutant after auxin treatment, while the expression of other SAUR gene

292 mutant than in a wild-type background after auxin treatment.

293 Several SMALL AUXIN UP RNA (SAUR) genes showed decreased expression in

294 anistic framework has emerged, wherein Small Auxin Up RNA (SAUR) proteins regulate protein phosphatas

295 o showed reduced expression of several SMALL AUXIN UP RNA (SAUR)63 subfamily genes, which contain TCP

296 the elongation of hypocotyls in response to auxin was impaired in the mutant.

297 l signalling mechanism for the plant hormone auxin whereby auxin directly affects the activity of the

298 ressor proteins were degraded in response to auxin with rates that depended on both receptor and repr

299 Microtubules respond to exogenous auxin within 5 min, although repatterning of the array d

300 on responses to auxin, but plants respond to auxin within minutes.