ライフサイエンスコーパス: avian

1 In 2014, an outbreak of avian A/H10N7 influenza virus occurred among seals along

2 Marek's disease virus (MDV), an avian alphaherpesvirus, causes a deadly lymphoma in chic

3 a membrane, we discovered that the oncogenic avian alpharetrovirus Rous sarcoma virus (RSV) Gag prote

4 nt dorsal intermediate arcopallium (AId), an avian analog of mammalian deep cortical layers with invo

5 biota to initiate intestinal colonization of avian and animal hosts for commensalism and infection of

6 n, warranting continued surveillance in both avian and human populations.

7 ons in H9 haemagglutinin and test binding to avian and human receptor analogues using biolayer interf

8 Pigs appear uniquely susceptible to both avian and human strains of influenza and are often descr

9 Southern Africa is home to diverse avian and mammalian fauna for which almost no informatio

10 xist with other arthropods that reproduce in avian and mammalian nests.

11 ching has responded to top-down selection by avian and mammalian predators using Sahara-Sahel desert

12 al syndrome and the species richness of both avian and mammalian predators; the trends for both preda

13 s-reservoir relationships, consisting of the avian and mammalian reservoir hosts of 415 RNA and DNA v

14 1960s, as well as H2Nx viruses isolated from avian and mammalian species between the 1950s and 2016.

15 nted detail across 354 extant and 37 extinct avian and non-avian dinosaurs.

16 ernative to the use of live vaccines against avian and other emerging coronaviruses.IMPORTANCE Accord

17 influenza virus, but also against all human, avian, and swine serotypes and, therefore, potential pan

18 shows that tail anatomy is not universal in avians, and suggests several possible scenarios regardin

19 "mixing vessels," being susceptible to both avian- and human-origin viruses, which allows the emerge

20 Here, using an avian animal model with complex hearing abilities simila

21 (Melopsittacus undulatus; of either sex), an avian animal model with complex hearing abilities simila

22 Avian ANP32A proteins harbor an additional 33 amino acid

23 Avian ANP32A supports the activity of an avian-origin po

24 munity and in particular, poorly immunogenic avian antigens.

25 The sensory-motor division of the avian arcopallium receives parallel inputs from primary

26 n the regulation of cell death in developing avian B cells.

27 plicated in the morphological development of avian beaks.

28 the chicken egg, including the egg-specific avian beta-defensin 11 (Gga-AvBD11).

29 Out of the 14 avian beta-defensins identified in the Gallus gallus gen

30 phylogenetic diversity of Leucocytozoon (an avian blood parasite) at site and species levels across

31 tes, but large-scale patterns and drivers of avian brain evolution remain elusive.

32 xample of this is seasonal plasticity in the avian brain, where song nuclei exhibit hormonally driven

33 We develop methods to link funding for avian breeding habitat conservation and management at la

34 contributed relatively little to the reduced avian breeding success.

35 iour and geographic distribution of obligate avian brood parasites and their hosts to demonstrate tha

36 Obligate avian brood parasites provide a particularly tractable s

37 Analysis of avian BST-2 genes will shed light on defense mechanisms

38 This indicates that avian BST-2 is involved in host-virus evolutionary arms

39 Characterizing the interaction of avian BST-2 with avian viruses is important in understan

40 odel supports the dietary hypothesis for the avian cecum.

41 olymerase complex activity in both human and avian cell lines than did those from the 2016-17 outbrea

42 erate reliance on multiple infection seen in avian cells but not the added reliance seen in mammalian

43 n-beta and importin-11 have been verified in avian cells, whereas the role of TNPO3 has not been stud

44 ty, even exerting a cytoprotective effect on avian cells.

45 aemorhous mexicanus), to interact with model avian cells.

46 we know very little about the NCL across the avian clade.

47 bringing about large-scale simplification of avian communities.

48 verse (Georgia), whereas in the most diverse avian community (Estonia), hardly any models were attack

49 oexisting species (nine focal species) of an avian community in east-central Illinois, USA.

50 ck risk of moth morphs depended on the local avian community.

51 nes and foster collaborative work within the avian community.

52 reported submillisecond spike timing in the avian cortex can be resolved by superfast syringeal musc

53 been shown to evolve more rapidly in birds, avian cranial morphology is characterised by a striking

54 steroid impact as the main driver of the non-avian dinosaur extinction.

55 fibula and fused caudal vertebrae of the non-avian dinosaur Tyrannosaurus rex.

56 and anisomerism more similar to those of non-avian dinosaurs and crurotarsans than of their own adult

57 In recent decades, intensive research on non-avian dinosaurs has strongly suggested that these animal

58 lian IVD, extant reptile groups and some non-avian dinosaurs independently evolved a synovial ball-an

59 how this variation arose with respect to non-avian dinosaurs is key to understanding how birds achiev

60 he overall skull shape evolved faster in non-avian dinosaurs than in birds across all regions of the

61 organization can discriminate birds from non-avian dinosaurs, and crurotarsans.

62 for an IVD in fossil reptiles, including non-avian dinosaurs, ichthyosaurs, plesiosaurs, and marine c

63 ross 354 extant and 37 extinct avian and non-avian dinosaurs.

64 al, and quadrate-exhibited high rates in non-avian dinosaurs.

65 n in morphological evolution relative to non-avian dinosaurs.

66 in how skull shape evolved in birds and non-avian dinosaurs.

67 xtinction, 66 Ma, included the demise of non-avian dinosaurs.

68 this eggshell type has been inferred for non-avian dinosaurs.

69 thways to herbivory in a large sample of non-avian dinosaurs.

70 iral vectors for protecting against multiple avian diseases.

71 factors, and also provide a global index of avian dispersal ability for use in community ecology, ma

72 106 sites that were originally surveyed for avian diversity in the early 20th century by Joseph Grin

73 ype of a structural family, dubbed herein as avian-double-beta-defensins (Av-DBD).

74 attractive because while it is pathogenic to avians (e.g., chickens), it does not cause significant v

75 body shape that impacts numerous aspects of avian ecology and behaviour - has consistently increased

76 Avian egg production demands resources such as lipids an

77 cts of complex organic extracts derived from avian eggs.

78 Most avian eggshell colours can be produced by a mixture of t

79 eralization of marine invertebrate shells or avian eggshells, respectively.

80 e, only two pigments have been identified in avian eggshells: rusty-brown protoporphyrin IX and blue-

81 The avian embryo permits isolation of the direct effects of

82 he axial and paraxial tissues in the forming avian embryonic body coordinate their rates of elongatio

83 Here, using avian epidermis, we find two major strategies regulate b

84 sed NRG1 secretion and activation of V-ERB-B avian erythroblastic leukemia viral oncogene homolog 3 (

85 The EGFR, a member of the ErbB (avian erythroblastosis oncogene B) family of receptors t

86 Next, we identified ETS1 (v-ets avian erythroblastosis virus E26 oncogene homolog 1) as

87 1,2), is a defining feature of mammalian and avian evolution.

88 no symptoms and 10 healthy subjects with no avian exposure.

89 As avian eyes differ from mammals, we asked whether local m

90 layed a fundamental step in the evolution of avian flight.

91 in the 627 domain of the PB2 subunit, enable avian FluPolA to overcome this restriction and efficient

92 olution is hindered by an exceedingly sparse avian fossil record from the Mesozoic era.

93 Here, we document how avian functional and phylogenetic diversity and structur

94 lish the minimum dimensionality required for avian functional traits to predict subtle variation in t

95 Here, we show that germ-layer patterning in avian gastrulation is ipsilateral despite cells undergoi

96 ne evolution in procellariiform seabirds, an avian group which relies on the sense of olfaction for c

97 made seal H10 hemagglutinin more stable than avian H10 hemagglutinin and similar to human hemagglutin

98 Compared with avian H10 hemagglutinin, seal H10 hemagglutinin showed s

99 indicated that the X-ORFs of equine H3N8 and avian H3N2 influenza viruses encoded 61 amino acids but

100 showed that the PA-X genes of equine H3N8 or avian H3N2 influenza viruses were full-length, with X-OR

101 rains, including H2N1, H5N1, H6N1, H11N9, an avian H3N8, and a human seasonal H3N2 subtype.

102 V strains in comparison to seasonal H3N2 and avian H3N8.

103 However, avian H5N1 influenza virus infections can result in prol

104 ss-inhibited the N1 NAs of highly pathogenic avian H5N1 influenza viruses.

105 e crystal structure of the HA protein of the avian H7N9 influenza virus in complex with a pan-H7, non

106 Namely, we find that avian H9N2 viruses representative of those circulating w

107 spite their importance, genomic resources of avian haemosporidians have proved difficult to obtain, a

108 Here we present a global dataset of avian hand-wing index (HWI), an estimate of wing shape w

109 ture human-type receptor adaptation in novel avian HAs.

110 reened in chicken liver cells by a truncated avian HEV capsid protein (ap237) in which the host prote

111 s or inhibitors, attachment and infection by avian HEV significantly decreased.

112 ntestinal parasite prevalence from 96 and 54 avian host species, respectively, we test the implicatio

113 ection of humans and optimal colonization of avian hosts, senses butyrate likely by indirect means to

114 rt, which affects their fitness to different avian hosts.

115 t also captures the kinetics of seasonal and avian IAV infections, via parameter changes consistent w

116 (i.e., in swine upper respiratory tracts) of avian IAVs affect their spillovers from wild birds to pi

117 wever, only sporadic cases of infection with avian IAVs are reported in domestic swine.

118 of viral replication efficiency exist among avian IAVs but that only a few of these may result in vi

119 One natural barrier for transmission of avian IAVs into humans is the specificity of the recepto

120 olecular mechanisms affecting the ability of avian IAVs to infect swine are still not fully understoo

121 replication efficiency, only a small set of avian IAVs were found to replicate well in epithelial ce

122 ient way for assessment of the risk posed by avian IAVs, such as in evaluating their potentials to be

123 nfects them in the same way as H5N1 and H7N9 avian IAVs.

124 A relational database with avian immune gene evidence from Gene Ontology, Ensembl,

125 n stone or the "seed" for the initial set of avian immune genes is based on the well-studied model or

126 rrently, the database contains 1170 distinct avian immune genes with canonical gene symbols and 612 s

127 y gene property extraction as exemplified by avian immune genes.

128 The avian immune system is characterised by a cascade of com

129 In this article, we present and describe the Avian Immunome DB (AVIMM) for easy gene property extract

130 Avian influenza (AI) affects wild aquatic birds and pose

131 Avian Influenza (AI) is a complex but still poorly under

132 es are considered to be the natural hosts of Avian Influenza (AI), and are presumed to pose one of th

133 The H5N8 highly pathogenic avian influenza (HPAI) clade 2.3.4.4 virus spread to Nor

134 Highly pathogenic avian influenza (HPAI) H5 viruses, of the A/goose/Guangd

135 ly, a human isolate of the highly pathogenic avian influenza (HPAI) H5N1 virus successfully propagate

136 Highly pathogenic avian influenza (HPAI) is a devastating disease of poult

137 Highly pathogenic avian influenza (HPAI) viruses are enzootic in wild bird

138 in poultry.IMPORTANCE H5Nx highly pathogenic avian influenza (HPAI) viruses of the A/goose/Guangdong/

139 Highly pathogenic avian influenza (HPAI) viruses of the H5 A/goose/Guangdo

140 long-distance carriers of highly pathogenic avian influenza (HPAI).

141 man H1N1 and H3N2 viruses and low-pathogenic avian influenza (LPAI) viruses.

142 RNA polymerases of avian influenza A viruses (FluPolA) replicate viral RNA

143 ed this platform using different subtypes of avian influenza A viruses and human samples with respira

144 genesis and tropism.IMPORTANCE Many zoonotic avian influenza A viruses have successfully crossed the

145 inct phenotypes.IMPORTANCE Highly pathogenic avian influenza A(H5N1) viruses have circulated continuo

146 Highly pathogenic avian influenza A(H5N8) viruses first emerged in China i

147 Low-pathogenicity avian influenza A(H9N2) viruses, enzootic in poultry pop

148 infections with clade 2.1 highly pathogenic avian influenza A/H5N1 virus have been reported, associa

149 s chimeric vaccines based on the most common avian influenza H5 and human influenza H1 sequences.

150 method to the analysis of highly pathogenic avian influenza H5N1 clade data in the Mekong region.

151 n a heightened threat for poultry.IMPORTANCE Avian influenza H7N9 viruses have been causing disease o

152 Avian influenza outbreaks have been occurring on smallho

153 We found that an H9N2 avian influenza reassortant virus bearing a human-origin

154 al profiles in individuals who received H5N1 avian influenza vaccine administered with MF59, with alu

155 ement of the human-origin PA gene segment in avian influenza virus (AIV) could overcome barriers to c

156 arly diagnosis of the highly pathogenic H5N1 avian influenza virus (AIV) is significant for preventin

157 ve bird markets (LBMs) are major targets for avian influenza virus (AIV) surveillance programmes.

158 The highly pathogenic avian influenza virus (HPAIV) H5N1 A/goose/Guangdong/199

159 e sustained circulation of highly pathogenic avian influenza virus (HPAIV) H5N1 A/goose/Guangdong/199

160 hown to be associated with highly pathogenic avian influenza virus (HPAIV) H5N1 outbreaks in South-Ea

161 major policies to control highly pathogenic avian influenza virus (HPAIV) infections in chickens.

162 ctural analysis.IMPORTANCE Low-pathogenicity avian influenza virus (LPAIV) subtypes can reassort with

163 t HA head glycosylation of low-pathogenicity avian influenza virus (LPAIV) subtypes.

164 an AS03-adjuvanted versus nonadjuvanted H5N1 avian influenza virus inactivated vaccine.

165 negative regulatory signals during modified avian influenza virus infection.

166 severe and prolonged disease associated with avian influenza virus infections in humans.

167 avian-origin influenza virus polymerases and avian influenza virus replication.

168 ry T cell numbers were decreased in modified avian influenza virus-infected mice.

169 In avian influenza virus-infected patients, the host immune

170 ns, the avian protozoan Eimeria tenella, and avian influenza virus.

171 h the severity of infection with seasonal or avian influenza virus.

172 ls to effectively control a modified form of avian influenza virus.

173 olution to understand virulence evolution in avian influenza viruses (AIV).

174 ces can result in variable susceptibility of avian influenza viruses (AIVs) carrying resistance-assoc

175 H9N2 avian influenza viruses (AIVs) circulate in poultry thro

176 H7N9 avian influenza viruses (AIVs) continue to evolve and re

177 viously found during OS and ZAN selection in avian influenza viruses (AIVs) of the N3 to N9 subtypes

178 Low-pathogenic avian influenza viruses (LPAIVs) are genetically highly

179 H5 and H7 subtypes of low pathogenic avian influenza viruses (LPAIVs) can mutate to highly pa

180 r role in the epidemiology of low-pathogenic avian influenza viruses (LPAIVs), which are occasionally

181 ion and poultry adaptation of H9N2 and other avian influenza viruses and helps us understand the stri

182 d ecology of viruses in this host.IMPORTANCE Avian influenza viruses can jump from wild birds and pou

183 We found that the H5Nx highly pathogenic avian influenza viruses exhibited high virulence in mice

184 enetic clades, while reassortment with other avian influenza viruses has led to the emergence of new

185 Active surveillance of avian influenza viruses in Bangladeshi live poultry mark

186 Avian influenza viruses need several adaptive mutations

187 Avian influenza viruses occasionally infect and adapt to

188 ncern in Bangladesh, where highly pathogenic avian influenza viruses of the A(H5N1) subtype are endem

189 Recurring reports of avian influenza viruses severely affecting humans have s

190 influenza viruses and for highly pathogenic avian influenza viruses that circulate in poultry, but m

191 The inability of avian influenza viruses to effectively bind human-like s

192 lobal concern persists that these or similar avian influenza viruses will evolve into viruses that ca

193 Avian influenza viruses, such as A(H5N1) and A(H7N9), ar

194 hical range seen in these viruses.IMPORTANCE Avian influenza viruses, such as H9N2, cause huge econom

195 PAI viruses and cocirculating low-pathogenic avian influenza viruses.

196 of infectious bronchitis, Newcastle disease, avian influenza, porcine reproductive and respiratory sy

197 Avian leukosis virus subgroup J (ALV-J) is an important

198 Avian-like cellular receptors are the primary target for

199 ceptor-binding avidity toward both human and avian-like receptor analogues, and the A125T+A151T mutat

200 e receptor while maintaining binding for the avian-like receptor.

201 s the evidence that pterosaurs belong to the avian line of archosaurs.

202 de that predates the divergence of different avian lineages, most genes belong to an avian-specific g

203 Avian migrants likely use optimal routes with respect to

204 Assessing climate (change) effects on avian migration phenology has consequently been difficul

205 bution of SV to evolutionary processes in an avian model of incipient speciation.

206 a leading cause of food-poisoning and causes avian necrotic enteritis, posing a significant problem t

207 In avian (non-mammalian) species, however, orexin seemed to

208 including enantiornithine birds and another avian of indeterminate affinities as well as crocodylomo

209 N1]) are all proposed to have been caused by avian or swine influenza viruses that acquired virulence

210 Rarely, an avian- or swine-origin influenza virus adapts to humans

211 We analyzed BST-2 genes in the avian orders Galliformes and Passeriformes and showed th

212 orthologs in multiple avian species from 12 avian orders.

213 Equine-origin H3N8 and avian-origin H3N2 canine influenza viruses (CIVs) preval

214 ids; however, those of equine-origin H3N8 or avian-origin H3N2 CIVs were truncated, suggesting that P

215 PORTANCE Epidemics of equine-origin H3N8 and avian-origin H3N2 influenza viruses in canine population

216 By contrast, poorly adapted avian-origin HAs contain predominately complex-type glyc

217 The avian-origin influenza A virus polymerase is restricted

218 are crucial for their ability to support the avian-origin influenza virus polymerase.

219 32 proteins tested, supports the activity of avian-origin influenza virus polymerases and avian influ

220 Consequently, identification of avian-origin influenza viruses across mammals appears cr

221 However, the avian-origin polymerase is incompatible with human ANP32

222 Avian ANP32A supports the activity of an avian-origin polymerase.

223 nce all human IAV pandemics can be traced to avian origins, there remains ever-present concern over e

224 live observations inside the large, muscular avian oviduct.

225 s region and other higher order areas of the avian pallium.

226 Therefore, we thought to evaluate avian paramyxovirus serotype 3 (APMV-3) strain Netherlan

227 aced with the corresponding ectodomains from avian paramyxovirus serotype 3 (APMV-3).

228 ial step towards the dense representation of avian phylogenetic and molecular diversity, by analysing

229 Our findings reveal new links between avian physiology, ecology, behaviour and life history, w

230 dlife extinctions worldwide, particularly in avian populations considered vulnerable or endangered.

231 t, we scanned for tags regurgitated by a key avian predator (great cormorant Phalacrocorax carbo) at

232 competition and allowing coexistence of four avian predators (snowy owls, glaucous gulls, rough-legge

233 ypothesize higher frequencies are emitted by avian predators and that detecting these auditory cues m

234 nism to partition food resources among these avian predators is spatial segregation, and secondarily

235 Here we show that selection by avian predators on warning colour is predicted by local

236 through eliciting unlearned biases in naive avian predators.

237 e and gram-negative bacterial pathogens, the avian protozoan Eimeria tenella, and avian influenza vir

238 led plumage diversification in this colorful avian radiation.

239 genetic basis of preferences for alternative avian receptors and for human-like receptors, describing

240 duct of accelerated evolution from their non-avian relatives, despite their frequent portrayal as an

241 shuttling of the ARV p17 protein.IMPORTANCE Avian reoviruses (ARVs) cause considerable economic loss

242 (2020) Food availability limits avian reproduction in the city: An experimental study on

243 across the capital versus income spectrum of avian reproduction.

244 l food sources in cities-can explain reduced avian reproductive success in urban areas.

245 its of urban animal populations, for example avian reproductive success is often reduced.

246 ide registration in the European Union (EU), avian reproductive toxicity is characterized after expos

247 nd 53.9% in extant dinosaurs (birds) and non-avian reptiles, respectively.

248 ts antiviral activity against a prototypical avian retrovirus, avian sarcoma and leukosis virus (ASLV

249 st time and demonstrate its activity against avian sarcoma and leukosis virus (ASLV).

250 ity against a prototypical avian retrovirus, avian sarcoma and leukosis virus (ASLV).

251 removal, as we saw a functional response by avian scavengers to increased carrion availability.

252 e to the extensive acquisition of carrion by avian scavengers.

253 nescence in song, offering new insights into avian senescence.

254 Here, we review some of his work on avian sensory specialists and research that he inspired

255 ve and integrative approach to understanding avian sensory systems and provide an example of one syst

256 Extensive prior studies concluded that avian sleep lacked many features characteristic of mamma

257 Late-life declines in avian song, and possibly other sexual traits, may be mor

258 domly distributed among IAVs across multiple avian species and geographic and temporal orders.

259 nd is distributed among IAVs across multiple avian species and geographic and temporal orders.

260 We also identify BST-2 genes in multiple avian species and show that they evolve rapidly in birds

261 ore, we describe BST-2 orthologs in multiple avian species from 12 avian orders.

262 ng from 16 countries and from at least eight avian species were submitted to the presented assays for

263 IAVs infect many avian species wherein they maintain a diverse natural re

264 LDD and SDD may be separate processes in an avian species, and suggests that environmental change ma

265 showing that pair coordination is common in avian species, it remains unclear how environmental and

266 The application of transcriptomics to a wild avian species, naturally exposed to complex chemical mix

267 raphic rates may exist in a diverse array of avian species.

268 H5N2 HPAI virus circulated in different avian species.

269 ormation on incubation and fledging for 3096 avian species.

270 the Wulst in relation to binocularity across avian species.

271 rent avian lineages, most genes belong to an avian-specific gamma-c clade, within which sequences clu

272 hed N-glycans, which is not possible for the avian-specific HA due to geometrical constrains of the b

273 sary to establish the role of the apparently avian-specific neuronal activation in the VMH of zebra f

274 rization event occurred near the base of the avian stem lineage.

275 nalysis of 949 estimates from 204 studies of avian survival and demonstrate that a latitudinal surviv

276 nterparts, but it has not been shown whether avian survival rates covary with latitude worldwide.

277 rs in explaining global patterns of apparent avian survival.

278 ment have seen relatively little research in avian systems, despite ample evidence of their effects i

279 The avian tail embodies a bipartite anatomy, with the proxim

280 ceanic archipelagos worldwide (including 596 avian taxa), and applied a new analysis method to estima

281 The emerging pattern suggests that for some avian taxa, the ontogeny of migratory strategy is a prol

282 ows that these effects on movement extend to avian taxa.

283 erial surface imaging, normally mineralizing avian tendons have been studied with nanometer resolutio

284 dissection, and vessel coagulation) using an avian tissue model (transfer-test).

285 ds (anting), our finding shows that the wild avian tool-use repertoire is wider than previously thoug

286 The avian transition from long to short, distally fused tail

287 al traits for >99% bird species to show that avian trophic diversity is described by a trait space wi

288 from avian viruses exhibit specificity for "avian-type" alpha2-3-linked (NeuAcalpha2-3Gal) receptors

289 ltraviolet (UV) sensitivity to the ancestral avian violet sensitivity, thus improving visual resoluti

290 ill shed light on defense mechanisms against avian viral pathogens.

291 eassortment event between a circulating H2N2 avian virus and the seasonal H1N1 viruses in humans.

292 er distinct from the alpha2-3 specificity of avian virus progenitors.

293 In contrast, HAs from avian viruses exhibit specificity for "avian-type" alpha

294 terizing the interaction of avian BST-2 with avian viruses is important in understanding innate antiv

295 ns, with components of each originating from avian viruses.

296 uggests that BST-2 antagonists exist in some avian viruses.

297 indistinguishable for mammalian and low for avian vision model, which implies effective camouflage.

298 Recent reports have shown that the avian visual dorsal ventricular ridge (DVR) is organized

299 le influenza strains, including pandemic and avian, while largely eliminating the potentially harmful

300 These findings shed new light on avian wing anatomy and the role of unconventional aerody