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1 at the cellular level until they entered the axillary lymph node.
2 nel lymph node biopsy, and 10 had a positive axillary lymph node.
3 mapping, and only one patient had a positive axillary lymph node.
4 e morphological characteristic of metastatic axillary lymph node.
5 needle aspiration of a palpable, ipsilateral axillary lymph node.
6 ed between primary breast tumours and paired axillary lymph nodes.
7 y measuring time to peak signal intensity in axillary lymph nodes.
8 The margins are negative, as are all 11 axillary lymph nodes.
9 breast cancer who underwent US assessment of axillary lymph nodes.
10 in the spleen and in inguinal, brachial, and axillary lymph nodes.
11 o residual invasive cancer in the breast and axillary lymph nodes.
12 the regional spread of breast cancer to the axillary lymph nodes.
13 evidence of invasive tumor in the breast and axillary lymph nodes.
14 age breast cancer without involvement of the axillary lymph nodes.
15 idence of invasive disease in the breast and axillary lymph nodes.
16 imary breast cancer and at least 10 involved axillary lymph nodes.
17 of these patients had residual tumor within axillary lymph nodes.
18 ancer patients, independent of the status of axillary lymph nodes.
19 wall and/or the ipsilateral supraclavicular/axillary lymph nodes.
20 t cancer patients with four or more involved axillary lymph nodes.
21 umor cells are unlikely to be found in other axillary lymph nodes.
22 mographic screening (elsewhere) had negative axillary lymph nodes.
23 istologic characteristics of the rest of the axillary lymph nodes.
24 ly 3 cm on exam, and multiple palpable right axillary lymph nodes.
25 multiple conglomerated 1-2 cm level I and II axillary lymph nodes.
26 There was no evidence of enlarged axillary lymph nodes.
27 ologic complete response (pCR) in breast and axillary lymph nodes.
28 absence of invasive cancer in the breast and axillary lymph nodes.
29 even lesions in the neck, five (55%) of nine axillary lymph nodes, 11 (50%) of 22 bone metastases, an
30 < .001) and had a larger number of involved axillary lymph nodes (41% v 26% had > 3 involved nodes;
31 stology) as did other features (27% positive axillary lymph nodes, 63% positive estrogen receptors, a
33 f 20 primary breast tumors and their matched axillary lymph nodes, a high concordance (Fisher's exact
34 er concentration of RPV was delivered to the axillary lymph nodes (AL) (C(max) 2466 ng/g - T(max) 3 d
37 ession in primary breast cancers can predict axillary lymph node (ALN) metastasis, we assessed differ
39 peripheral blood (PB), bone marrow (BM), and axillary lymph node (ALN) tissue of rhesus macaques infe
40 peripheral blood (PB), bone marrow (BM), and axillary lymph node (ALN) tissue of rhesus macaques infe
41 iption polymerase chain reaction (RT-PCR) in axillary lymph nodes (ALN) of breast cancer patients.
42 therapy in patients with 10 or more positive axillary lymph nodes (ALN) to reduce the high loco-regio
44 A was prominently expressed in the liver and axillary lymph nodes, although preliminary data suggest
45 bsence of invasive disease in the breast and axillary lymph nodes, analysed by intention to treat.
46 -10-fold in primary tumor volumes; and in an axillary lymph node and lung metastasis as compared with
47 all survival based on the number of positive axillary lymph nodes and (in N0 patients) pathologic tum
48 perable breast cancer involving four or more axillary lymph nodes and had completed mastectomy or bre
49 enrolled with between one and three positive axillary lymph nodes and Ki-67 of at least 20% as an add
50 icant incidence of metastatic disease to the axillary lymph nodes and may benefit most from selective
51 h biopsy-proved metastatic adenocarcinoma to axillary lymph nodes and occult primary tumor underwent
52 tion is that patients who develop disease in axillary lymph nodes and subsequently undergo ALND have
53 primary breast tumours to that in metastatic axillary lymph nodes and to determine the correlation be
54 ed significantly with the number of positive axillary lymph nodes and with postchemotherapy c-erbB-2
56 nd intraductal disease in the breast and the axillary lymph nodes), and the safety of adding bevacizu
57 tution, prior taxane-based therapy, involved axillary lymph nodes, and centrally determined phenotype
58 T excels in detecting primary breast masses, axillary lymph nodes, and distant metastases; can comple
59 were obtained for the primary breast tumor, axillary lymph nodes, and extraaxillary lymph nodes on e
61 etween radiotracer uptake in spleen, tonsil, axillary lymph nodes, and peripheral blood CD4 T-cell co
62 n of lymph node status by number of involved axillary lymph nodes; and new classifications for metast
63 gn transport of breast epithelial cells into axillary lymph nodes are recently described phenomena.
65 ith combined ART, uptake of [18F]-FDG in the axillary lymph nodes, as measured by TBR, decreased from
66 ne of five Ontario study centers followed by axillary lymph node assessment (ALNA) consisting of sent
67 tients diagnosed with at least four positive axillary lymph nodes at diagnosis who underwent anthracy
69 g NHPs had significant (18)FDG uptake in the axillary lymph nodes at the time of MPXV challenge with
70 mance of breast MRI in diagnosing metastatic axillary lymph nodes based on the pathological result.
71 and high NPV (96.4%) in detecting metastatic axillary lymph nodes, but its specificity was only fair
73 excision of the tumour (1 cm margin) and an axillary lymph-node clearance or sample, all patients re
74 ion took into account the number of involved axillary lymph nodes, clinical tumour stage, oestrogen-r
75 ns of macrophages isolated from the spleens, axillary lymph nodes, colons, jejuna, and livers of heal
77 P < .001), high body mass index ( P = .039), axillary lymph node dissection ( P = .008), and more sev
78 cer diagnosis ( P = .0404), having undergone axillary lymph node dissection ( P = .0464), and receipt
79 relative risk, 0.83 [CI, 0.77 to 0.90]) and axillary lymph node dissection (adjusted relative risk,
80 n with breast cancer, the role of completion axillary lymph node dissection (ALND) after identificati
81 l lymph node biopsy (SLNB) alone, SLNB+RLNR, axillary lymph node dissection (ALND) alone, and ALND+RL
84 staging information with less morbidity than axillary lymph node dissection (ALND) for patients with
85 node biopsy (SLNB) was developed to replace axillary lymph node dissection (ALND) for staging early
88 lymph nodes (SLNs) were negative, SLNB with axillary lymph node dissection (ALND) if SLNB or PET was
89 For breast cancer patients, the role of the axillary lymph node dissection (ALND) in the management
91 l lymph node dissection (SLND) alone without axillary lymph node dissection (ALND) is noninferior to
93 n both sentinel lymph node biopsy (SLNB) and axillary lymph node dissection (ALND) may be because of
94 itivity for macrometastasis (Ma), leading to axillary lymph node dissection (ALND) only when strictly
95 hat consisted of either total mastectomy and axillary lymph node dissection (ALND) or segmental maste
96 inel lymph nodes (SLNs) randomized either to axillary lymph node dissection (ALND) or sentinel lymph
100 ve been proposed to replace the conventional axillary lymph node dissection (ALND) with its concomita
101 e predicted that 20 to 30 cases of SLND with axillary lymph node dissection (ALND) would enable surge
102 trolled trial in which SNB was compared with axillary lymph node dissection (ALND), four limited meta
110 e categorized in 4 groups: SLN-negative with axillary lymph node dissection (ALND; n = 326), SLN-nega
111 ph nodes (SLNs) generally undergo completion axillary lymph node dissection (cALND), either concurren
112 the breast and regional nodes after BCS and axillary lymph node dissection (Group 1) were compared t
113 included body mass index >/= 25 (P = .0236), axillary lymph node dissection (P < .001), regional lymp
115 SLN biopsy (SLNB) alone or SLNB followed by axillary lymph node dissection (SLNB/ALND) between June
117 results (sentinel lymph node biopsy [SNB] or axillary lymph node dissection [ALND]) were compared wit
118 2 patients were randomly assigned to receive axillary lymph node dissection and 2404 to receive axill
119 inel node, 744 had been randomly assigned to axillary lymph node dissection and 681 to axillary radio
121 Breast-conserving surgery combined with axillary lymph node dissection and radiotherapy or maste
122 d the localized lymph node before completion axillary lymph node dissection and used radiography of t
124 er were significantly less likely to receive axillary lymph node dissection as determined by logistic
126 lar to upfront SLNB and reduces the need for axillary lymph node dissection compared with SLNB prior
127 me progressively less extensive, with formal axillary lymph node dissection confined to a dwindling g
130 ence occurred in four of 744 patients in the axillary lymph node dissection group and seven of 681 in
134 oup, 220 (33%) of 672 patients who underwent axillary lymph node dissection had additional positive n
135 ymph nodes (SLNs) are recommended to undergo axillary lymph node dissection if extranodal extension (
136 east cancers while avoiding the morbidity of axillary lymph node dissection if the nodes do not conta
137 gest that it is time to reassess the role of axillary lymph node dissection in patients who undergo c
139 These findings do not support routine use of axillary lymph node dissection in this patient populatio
142 st cancer who have a positive sentinel node, axillary lymph node dissection is the present standard.
144 erated allocation schedule to receive either axillary lymph node dissection or axillary radiotherapy
146 arm was noted significantly more often after axillary lymph node dissection than after axillary radio
147 SLN biopsy followed by completion level I/II axillary lymph node dissection to determine the false-ne
148 ecurrence was 0.43% (95% CI 0.00-0.92) after axillary lymph node dissection versus 1.19% (0.31-2.08)
150 patients undergoing total mastectomy and/or axillary lymph node dissection were randomized to standa
151 the ability to achieve the results of total axillary lymph node dissection without the risks of surg
152 of node-positive patients and of completion axillary lymph node dissection) were analyzed to rule ou
153 bility to obtain prognostic information (ie, axillary lymph node dissection), tends to minimize treat
154 e, approximately 10% of women do not have an axillary lymph node dissection, 11% to 26% do not have t
155 .4% underwent an initial SLNB, 37.2% initial axillary lymph node dissection, and 6.3% no axillary sur
156 eoadjuvant chemotherapy reduces the need for axillary lymph node dissection, and SLNB is an accurate
157 andard primary tumor therapy, to not receive axillary lymph node dissection, and to not receive radia
160 y, followed by breast-conserving surgery and axillary lymph node dissection, which revealed residual
173 ocal recurrence rate and similar survival to axillary lymph node dissection.Preoperative axillary ult
174 y, because a separate incision is needed for axillary lymph-node dissection, and postoperative radiot
177 sentinel-lymph-node involvement, completion axillary-lymph-node dissection can be omitted or replace
178 The previously undisputed gold standard of axillary-lymph-node dissection for staging has now been
179 on the present role and routine practice of axillary-lymph-node dissection in early breast cancer, t
180 h-node biopsy, but also lymph-node sampling, axillary-lymph-node dissection, and observation alone.
183 Despite guidelines recommending against axillary lymph node evaluation in women with DCIS underg
189 (US)-guided fine-needle aspiration (FNA) of axillary lymph nodes for preoperative staging of breast
191 ns of the HEVs are also characterized in the axillary lymph nodes from human breast cancer patients w
195 lymph nodes metastasis and the diagnosis of axillary lymph nodes in patients with breast cancer is i
197 on in both peripheral blood and the draining axillary lymph node, indicating significant BCG vaccine-
198 y breast tumors resulted in viral transit to axillary lymph nodes, infection of lymphatic metastases,
200 rder interactions were considered: number of axillary lymph nodes involved (zero v one to three v fou
201 years or older without prior chemotherapy or axillary lymph node involvement and (2) the hypofraction
203 , age, comorbid disease, prior chemotherapy, axillary lymph node involvement, intensity-modulated rad
206 munohistochemical examination of ipsilateral axillary lymph nodes is a reliable, prognostically valua
207 Reliable pathologic stage classification of axillary lymph nodes is an important determinant of prog
209 d cervical lymph nodes but fewer than 20% of axillary lymph nodes, livers, brown fat samples, kidneys
217 in the breast is correlated with absence of axillary lymph node metastases at final pathology (ypN0)
220 h negative PET scans had such a low risk for axillary lymph node metastases that axillary dissection
223 d with a 1% to 3% reduction in recurrence of axillary lymph node metastases, but is associated with a
224 the 181 patients with lymphatic invasion had axillary lymph node metastases, compared with 19% of the
231 -1 ratio was observed in primary tumors with axillary lymph node metastasis than in node-negative tum
232 y into nude mice, the primary tumor volumes, axillary lymph node metastasis, and lung metastasis were
239 ion of chromosome 14q 31.2 is much higher in axillary lymph node-negative primary breast tumors than
240 d ratio of 0.342 (95% CI, 0.17 to 0.69), and axillary lymph node-negative status yielded a hazard rat
241 uding internal mammary, supraclavicular, and axillary lymph nodes) (nodal-irradiation group) or whole
242 ot sufficiently sensitive to detect positive axillary lymph nodes, nor is it sufficiently specific to
243 LNs removed, no cancer was identified in the axillary lymph nodes of 215 patients, yielding a patholo
244 to those in spleen, IL-10-positive cells in axillary lymph nodes of coinfected animals were predomin
245 capacity to completely clear the breast and axillary lymph nodes of invasive tumor before surgery.
246 to stimulate CD4(+) T cells derived from the axillary lymph nodes of mice vaccinated with irradiated
248 ne mastectomy and had at least four positive axillary lymph nodes or primary tumour stage T3-4 diseas
250 rative activity in a cohort of patients with axillary lymph node-positive breast cancer and compare t
251 identification of a subset of patients with axillary lymph node-positive breast cancer with an impro
260 ll carcinoma and breast cancer metastases to axillary lymph nodes resulted in areas under the curve a
261 otal, 47 paired breast tumour and metastatic axillary lymph node samples were collected in this study
263 or accurate, noninvasive imaging of SLNs for axillary lymph node staging in breast cancer patients.
270 growth factor receptor 2 (HER2) status; and axillary lymph node status with chi(2) or Fisher exact t
271 o age, stage, tumor site, tumor size, grade, axillary lymph node status, extent of surgery, and radio
272 r site, tumor size, grade, ER and PR status, axillary lymph node status, extent of surgery, and RT, t
273 s were analyzed in relation to the patients' axillary lymph node status, menopausal status, disease s
276 ias was not observed in the non-gut-draining axillary lymph nodes, suggesting that the Th17 bias was
277 cancer and at least 10 involved ipsilateral axillary lymph nodes to receive either six cycles of adj
278 tion system (balanced for number of involved axillary lymph nodes, tumour stage, oestrogen receptor s
279 nomas, and metastatic breast carcinomas from axillary lymph nodes using quantitative TaqMan reverse t
282 imination power between benign and malignant axillary lymph nodes was as follows: sensitivity - 60%;
286 ases in primary breast-cancer patients whose axillary lymph nodes were classified, by conventional me
287 for breast cancer confined to the breast and axillary lymph nodes were entered in a prospective study
288 Lymphadenopathy and immune activation in the axillary lymph nodes were evident in IV- and IB-infected
291 ongly with disease-specific survival whether axillary lymph nodes were negative or positive for metas
292 , or IIIA breast cancer involving 10 or more axillary lymph nodes were randomized after surgery and s
293 ng mammography, asymmetrically enlarged left axillary lymph nodes were seen in a healthy 70-year-old
294 st cancer involving four or more ipsilateral axillary lymph nodes were treated with nine cycles of ch
296 s sentinel lymph node biopsy (SLNB), marking axillary lymph node with radioactive iodine seed (MARI),
299 immune response in primary tumors and in the axillary lymph nodes with metastasis (ALN(+)) in breast
300 immune cells in the primary tumor and in the axillary lymph nodes without metastasis (ALN(-)) differe