ライフサイエンスコーパス: axon terminal

1 st of an axon terminating on another axon or axon terminal.

2 ey induce cytoskeletal reorganization at the axon terminal.

3 evidence that GABAARs are localized at MNTB axon terminals.

4 ealed eukaryotic ribosomes in CB1-expressing axon terminals.

5 ise control of neurotransmitter release from axon terminals.

6 ive vesicles were also observed in axons and axon terminals.

7 ric acid, and acetylcholine from presynaptic axon terminals.

8 nal transport, must be actively localized to axon terminals.

9 tic nicotinic receptors (nAChRs) on dopamine axon terminals.

10 varying numbers of varicosities representing axon terminals.

11 to the hippocampus in order to illuminate RE axon terminals.

12 present on both GABAergic and glutamatergic axon terminals.

13 fferent transmitter released from peripheral axon terminals.

14 endritic spines, and clusters of vesicles in axon terminals.

15 ximately 10% of EAAT2 molecules are found in axon terminals.

16 uctural alterations in these cells and their axon terminals.

17 pharmacology at the level of individual cone axon terminals.

18 optical excitation or inhibition of amygdala axon terminals.

19 tive and closely juxtaposed to prefrontal D1 axon terminals.

20 bits synaptic transmission at CB1-expressing axon terminals.

21 rimary role of the 5-HT1B receptors in these axon terminals.

22 B)Rs was also present at lower levels on PVI axon terminals.

23 in inhibiting the growth of R7 photoreceptor axon terminals.

24 elease of synaptic vesicles from presynaptic axon terminals.

25 e also expressed both in PC dendrites and BC axon terminals.

26 -level tonic calcium influx into presynaptic axon terminals.

27 None was found in axon terminals.

28 % of the postsynaptic targets of cholinergic axon terminals.

29 tic tips and somas and was weak or absent in axon terminals.

30 ic (inhibitory-type) synapses from unlabeled axon terminals.

31 on firing patterns, are poorly understood at axon terminals.

32 ate neuropeptides from secretory granules in axon terminals.

33 rather than direct activation of interneuron axon terminals.

34 o areas where alpha-MSH1-13 is released from axon terminals.

35 5%-10% is expressed in neurons, primarily in axon terminals.

36 rites of nonpyramidal neurons, as well as in axon terminals.

37 tic cannabinoid CB1 receptors on juxtaposing axon terminals.

38 destabilization and secondary retraction of axon terminals.

39 predominantly localized to the cytoplasm of axon terminals.

40 astrocytes but also in neurons, primarily in axon terminals.

41 on disease, characterized by degeneration of axon terminals.

42 occurred throughout dendrites, axons, and in axon terminals.

43 which preserve in vivo-like connectivity of axon terminals.

44 support fast, highly processive runs toward axon terminals.

45 by GABAC receptors, probably on bipolar cell axon terminals.

46 ion of myelin and VGLUT1-positive excitatory axon terminals.

47 nfection of projection neurons through their axon terminals, (3) targeted infection of genetically sp

48 tration of d-aspartate (an EAAT2 substrate), axon terminals accumulate d-aspartate as quickly as astr

49 ity in hundreds of cell bodies or long-range axon terminals, across all six layers in visual cortex o

50 nsport and function has no effect on sensory axon terminal activity but does negatively impact motor

51 at retrograde transport of mitochondria from axon terminals allows replacement of the axon terminal p

52 t of phospholipids cause degeneration of the axon terminal and loss of synaptic vesicles.

53 Imaging studies identified the BC axon terminal and PC dendrites as loci of AC-dependent K

54 al activity directs fine-scale topography of axon terminals and a new system to study this process at

55 in Nav subtype expression between CH and SR axon terminals and between CH and SR dendrites and spine

56 elease coincided with the degeneration of DA axon terminals and decreased expression of DA neuron-enr

57 onstrates that both kinases are contained in axon terminals and dendritic spines adjacent to the syna

58 1 cannabinoid receptors (CB1) are located at axon terminals and effectively control synaptic communic

59 in neurons, highlighted by concentrations in axon terminals and euchromatin-rich nuclear domains.

60 synaptic transmission, Na(+) accumulates in axon terminals and facilitates glutamate uptake into syn

61 light stimulus-evoked Ca(2+) signals in cone axon terminals and horizontal cell dendrites as well as

62 to area 9), m2 receptors predominated in ACC axon terminals and in more than half of the targeted den

63 onal dystrophy, including abnormally dilated axon terminals and increased amounts of synapses contain

64 d at the somatodendritic compartment, or the axon terminals and it can be transported anterogradely,

65 leads to accumulation of aged organelles in axon terminals and loss of cell body mitochondria.

66 es delta opioid receptors that reside on FSI axon terminals and negatively couple to adenylyl cyclase

67 The density of mitochondria in axon terminals and perisynaptic astrocytes was increased

68 synaptic inhibitory synapses on bipolar cell axon terminals and possibly amacrine cell dendrites.

69 taneously recorded from presynaptic pallidal axon terminals and postsynaptic thalamocortical neurons

70 e-strand RNA viruses, infect neurons through axon terminals and spread trans-synaptically in a retrog

71 The lack of degeneration of bare motor axon terminals and the morphological remodeling that occ

72 r maintaining stable contacts between basket axon terminals and the Purkinje AIS during pinceau organ

73 uences the branching pattern of regenerating axon terminals and the redistribution of acetylcholine r

74 release of neurohormones at the interface of axon terminals and vasculature.

75 synthesis decreases LE/Lys abundance at the axon terminal, and shortens axon length in a CPT1C-depen

76 l features of chemically distinct classes of axon terminals, and a major autonomic source of axons la

77 motor neurons, lack of mitochondria in motor axon terminals, and immature neuromuscular junctions.

78 NDCBE is in most glutamatergic axon terminals, and is also present in the terminals of

79 ne, cytoplasmic, and plasmalemmal domains of axon terminals, and subjacent to the postsynaptic densit

80 to be localized at the soma membrane, axon, axon terminals, and the nodes of Ranvier of induced plur

81 66% were somatodendritic profiles, 22% were axon terminals, and the remaining 12% were associated wi

82 1 [labeling all ON and OFF bipolar cell (BC) axon terminals] and G-protein gamma13 (labeling all ON B

83 inct neuronal morphologies with a variety of axon terminal arborizations subserving their functions.

84 how that not all mitochondria that leave the axon terminal are degraded; rather, they persist over se

85 of anterograde mitochondrial movement toward axon terminals are beginning to be understood, the frequ

86 Physiologic studies have shown that large axon terminals are found in pathways issued from motor s

87 In primary auditory cortex the bulk of MGV axon terminals are in layer IV/lower layer III with mino

88 Comparable experiments in central axon terminals are prohibitively difficult but the bioph

89 These findings show that axon terminals are unstable without M(2) and that muscle

90 nstrate that responses in dendrites, but not axon terminals, are end inhibited by flanking gratings b

91 r acute 5-HT(4)R activation in glutamatergic axon terminals arising from the medial prefrontal cortex

92 Finally, ordered geometric axon terminal arrangements that are not required for neu

93 he presynaptic plasma membrane of a neuron's axon terminals as a precondition for fusing with the mem

94 evidence that S1P can be produced at mature axon terminals as well as at immature growth cones in re

95 anogaster, for GABAergic synaptic markers on axon terminals as well as dendrites.

96 matergic corticostriatal and thalamostriatal axon terminals at dendritic spines of medium spiny neuro

97 Activation of thalamocortical axon terminals at different frequencies disrupted cortic

98 d with fusion probability at active zones of axon terminals at frog neuromuscular junctions.

99 ules composing active zone material (AZM) in axon terminals at mouse neuromuscular junctions.

100 d network coupled by gap junctions while the axon terminals (ATs) form a third independent network in

101 Both of these nuclei send columnar axon terminals back to the same tectal position receivin

102 while HCN1 channels were concentrated in the axon terminal boutons.

103 odistributing with anterogradely labeled Ipc axon-terminal boutons, further supporting a glutamatergi

104 naling molecules have been identified in the axon terminal, but their specific role in axon guidance

105 in forming synaptic specializations at their axon terminals, but in excess of the usual number.

106 subtypes on dendrites, dendritic spines, and axon terminals, but the proportion of pre- and post-syna

107 of intrabouton Ca(2+) influx into GABAergic axon terminals by CB1, leading to the effective suppress

108 Correcting the SV content in dopamine neuron axon terminals by impairing anterograde SV trafficking m

109 However, orderly positioning of axon terminals by positional cues and synaptic activity

110 otal gray matter (by 10%) and in parvalbumin axon terminals (by 49%).

111 eactivity was preferentially localized to CH axon terminals compared to SR, and in SR dendrites and s

112 d with dendrites, M5R was less often seen in axon terminals, comprising only 10.8% (n = 102) of the t

113 ry cortex and found that practically all ChC axon terminals contact axon initial segments, with an av

114 g: [1] lateral efferent olivocochlear (LEOC) axon terminals contain endogenous dynorphin neuromodulat

115 s associated with the formation of retracted axon terminals containing multivesicular bodies and seco

116 The mesoaccumbens axon terminals containing VGluT2 vesicles make asymmetri

117 tial to travel from the neuronal soma to the axon terminal, defining the temporal manner in which inf

118 An acute and highly reproducible motor axon terminal degeneration followed by complete regenera

119 pTrkB-ir was in axons, axon terminals, dendrites, and dendritic spines of neuro

120 roBDNF) stabilizes or eliminates presynaptic axon terminals, depending on its proteolytic conversion

121 le fibers, postsynaptic differentiation, and axon terminal development.

122 trastructurally defined synapses per GPe-STN axon terminal doubled with no alteration in terminal/syn

123 adder and developed varicosities along their axon terminal endings.

124 ial-like processes to a circuit in which RGC axon terminals establish synapses with dendritic shafts

125 imulation of ChR2-expressing thalamocortical axons/terminals evoked robust synaptic responses in cort

126 inct clusters of acetylcholine receptors and axon terminals exhibited numerous terminal varicosities.

127 In the IPL, DB1 bipolar cell axon terminals expressed the glycine receptor, GlyRalpha

128 icroscopy, we resolved the ultrastructure of axon terminals fated for laminar stabilization versus th

129 ae were implanted above the indirect pathway axon terminal field in the dlVP, or the direct pathway t

130 tent of collateralization of D1-MSN to these axon terminal fields we used retrograde labeling in tran

131 g of dendrites, dendritic spines, axons, and axon terminal fields within a few hours to a few days af

132 nts revealed reduced Ca(2+) influx into MNTB axon terminals following activation of presynaptic GABA(

133 y, our results indicate that horizontal cell axon terminals form two independent sets of homotypic ga

134 target nucleus (LA or BL), ~90% of cortical axon terminals formed asymmetric synapses with dendritic

135 In both cases, most (76-86%) PHAL(+) axon terminals formed asymmetric synapses, typically wit

136 Virtually all PHAL-positive CMT and PVT axon terminals formed asymmetric synapses.

137 und that double-labeled PHA-L (+)/VGluT2 (+) axon terminals formed synaptic contacts on dendrites of

138 form inhibitory-type synapses, the unlabeled axon terminals forming asymmetric synapses are the likel

139 mate transporter 1-positive terminals and of axon terminals forming asymmetric synapses in the dorsol

140 s with CB1r-ir typically received input from axon terminals forming asymmetric-type synapses.

141 s in the LC, and also revealed CB1r-positive axon terminals forming synaptic contact with MOR-contain

142 at mCherry-VGluT2 varicosities correspond to axon terminals, forming asymmetric synapses on neighbori

143 ervations of anterogradely labeled (PHAL(+)) axon terminals found at perirhinal sites adjacent to or

144 neurons establish local synapses, we tagged axon terminals from resident VTA neurons by intra-VTA in

145 C1 level, there was extensive withdrawal of axon terminals from thalamus and cortex, detectable a de

146 ncode object motion retinotopically, but the axon terminals fuse into a glomerular structure in the c

147 Although CMT and PVT axon terminals generally contacted dendritic spines, a s

148 n pathway was shown previously to inhibit R7 axon terminal growth.

149 ation of the cholinergic neurons or their V1 axon terminals improved performance of a visual discrimi

150 s/cm(2)/s) light-evoked calcium spikes in Mb axon terminals in an NEM-sensitive manner, but light res

151 elrhodopsin (ChR2) in CST cell bodies and in axon terminals in cervical spinal cord.

152 sing long-range posteromedial (POm) thalamic axon terminals in cortex and induced CaMPARI conversion

153 neurons in dorsal root ganglia (DRG) and on axon terminals in lamina II (the substantia gelatinosa)

154 maged the regeneration of trigeminal sensory axon terminals in live zebrafish larvae following laser

155 loss of motor neurons and atrophy of distal axon terminals in muscle, resulting in loss of motor fun

156 c activation of VTA glutamate cell bodies or axon terminals in NAc was sufficient to support reinforc

157 es to the actin cytoskeleton, is enriched at axon terminals in neurons, and activates the axon growth

158 By imaging OSN axon terminals in olfactory bulb glomeruli as well as OS

159 so evident in the innervation pattern of OSN axon terminals in olfactory bulbs.

160 been implicated in compensatory sprouting of axon terminals in paralyzed or denervated muscles.

161 tion of SC-projecting neurons in V1 or their axon terminals in SC sufficiently elicits the behavior,

162 urons are known to enlarge their presynaptic axon terminals in size and strength, thereby compensatin

163 ffect was strongest when puffed to STN-->SNr axon terminals in SNr, indicating a primary role of the

164 dition, illumination of Mrgprd-ChR2-Venus(+) axon terminals in spinal cord slices evoked EPSCs in hal

165 ceive excitatory inputs from ON bipolar cell axon terminals in sublamina-b of the inner plexiform lay

166 mus, we next recorded directly from pallidal axon terminals in thalamic nucleus DLM, and found that a

167 k, we also found that directly activating M2 axon terminals in the auditory cortex suppresses spontan

168 ing this period, the density of mPFC-derived axon terminals in the BA also decrease significantly, an

169 ly, silencing VTA dopamine neurons, or their axon terminals in the BA during the footshock, reduced t

170 ibution and morphology of CCK+ INs and their axon terminals in the BNC of the monkey was investigated

171 CCK+ axon terminals in the BNC were found both in the neuropi

172 ruit both GABA and GABA-A receptors to their axon terminals in the EB, and optogenetic stimulation co

173 tes, cells of the dermis, and on nociceptive axon terminals in the epidermis.

174 B1Rs are located on Schaffer collateral (Sc) axon terminals in the hippocampus, where they inhibit gl

175 mical analysis revealed the absence of GLP-1 axon terminals in the HPFv, suggesting volume transmissi

176 ealed abundant rod bipolar cells (RBCs) with axon terminals in the innermost sublamina of the inner p

177 e release from more than 13,000 bipolar cell axon terminals in the intact retina, we show that bipola

178 C dendrites in the outer plexiform layer and axon terminals in the IPL, as well as a putative amacrin

179 Cs) were recorded extracellularly from their axon terminals in the medial sublamina of tectal retino-

180 found mainly extra-synaptically in axons and axon terminals in the NAc and are enriched in glutamater

181 unt of GluD1-positive unmyelinated axons and axon terminals in the primate striatum.

182 operties and synaptic targets of CMT and PVT axon terminals in the rat BL.

183 extracellular GABA concentration around RHT axon terminals in the SCN.

184 ulder responsive sites in CN densely labeled axon terminals in the shoulder representation in VPL, bu

185 tant characteristic of dopamine release from axon terminals in the striatum is that it is rapidly mod

186 Conversely, inhibition of PVN OXT axon terminals in the VTA decreased social interactions.

187 nd that 20-Hz optogenetic stimulation of MFC axon terminals increased curvature of time-response hist

188 over, optogenetic activation of serotonergic axon terminals increased excitability of fusiform cells.

189 receives S1 inputs, and activation of the S1 axon terminals increases the response to noxious stimuli

190 tyramine, which displaces noradrenaline from axon terminals) induced vasoconstriction.

191 i, and strong optogenetic stimulation of the axon terminals induces distinct escape behaviours and au

192 to the periphery and relocate photoreceptor axon terminals into the center.

193 asion occurs prenatally, organization of PrV axon terminals into whisker-specific rows and patches ta

194 Although degeneration of the axon terminal is dependent on neural activity, activatio

195 the outward transport of dynein from soma to axon terminal is driven by direct interactions with the

196 tials, and the rate of depolarization of the axon terminal is instead governed by the rate of rise of

197 ed from the infected neuron cell body to the axon terminal is poorly understood.

198 Cabp5 immunolabeling of type 3 bipolar cell axon terminals is reduced in Vsx1-null mice.

199 ological contexts, but whether they do so in axon terminals is unknown.

200 that in rhesus monkeys (Macaca mulatta) most axon terminals labeled from tracers injected into ACC ar

201 etina, with significantly less dendritic and axon terminal labeling in TRPM1 knockout compared to wil

202 These data suggest that GLT-1 expressed in axon terminals may be important in maintaining energy me

203 Competitive interactions between axon terminals may determine the number of synapses each

204 GABA spillover excitation between MNTB axon terminals may entrain neighboring MNTB neurons, whi

205 Excitatory GABA spillover between inhibitory axon terminals may have important implications for the d

206 R immunoreactivity (M1R-ir) was also seen in axon terminals, most of which formed asymmetrical synaps

207 M2R-ir was also seen in axon terminals, most of which formed asymmetrical synaps

208 Traditional fluorescent dyes label axon terminals near an injection site, but unfortunately

209 es and late endosomes occurs continuously at axon terminals, non-degradative roles of autophagy at bo

210 The autoreceptor population located on the axon terminals of 5-HT neurons is a difficult population

211 hy on tissue sections from fixed and stained axon terminals of active and resting frog neuromuscular

212 The axon terminals of cb5b bipolar cells costratify with the

213 Nav 1.6 immunoreactivity were found between axon terminals of CH and SR or between dendrites and spi

214 g melanopsin cells also received inputs from axon terminals of dopaminergic amacrine cells.

215 at the orexin neurons are heavily apposed by axon terminals of glutamatergic and GABAergic neurons of

216 Axon terminals of glutamatergic retinal rod bipolar cell

217 In the rod pathway of the mammalian retina, axon terminals of glutamatergic rod bipolar cells are pr

218 Direct recordings from the large axon terminals of goldfish retinal bipolar cells (BCs) h

219 puncta were predominantly localized on large axon terminals of horizontal cells.

220 ransmitter GABA by directly depolarizing the axon terminals of inhibitory interneurons, thus bypassin

221 affected a number of synaptic boutons at the axon terminals of larval neuromuscular junction.

222 modulates autophagy of synaptic vesicles in axon terminals of motoneurons via its function as a guan

223 We show that calcium signals in the axon terminals of olfactory receptor neurons (ORNs) do n

224 The presynaptic axon terminals of one neuron release neurotransmitters t

225 used to quantify GAD67 protein levels in the axon terminals of parvalbumin-containing GABA neurons, w

226 ranslation of the protein, especially in the axon terminals of parvalbumin-containing neurons, suppor

227 We found that axon terminals of perisomatically projecting GABAergic i

228 , a synaptic lamina that is comprised of the axon terminals of photoreceptors and the dendrites of ho

229 s to defects in inner and outer segments and axon terminals of photoreceptors.

230 PTEN protein is enriched in cell bodies and axon terminals of purified motor neurons.

231 LRRTM4 is enriched at GABAergic synapses on axon terminals of rod bipolar cells (RBCs).

232 to NL was mapped, and the morphology of the axon terminals of SON neurons in NL was examined in chic

233 motoneurons, axons in the sciatic nerve, and axon terminals of the neuromuscular junctions.

234 targeted dendrites adjacent to glutamatergic axon terminals, often near dendritic bifurcations.

235 e lipase, which is located in the inhibitory axon terminal, or by alpha-beta-hydrolase domain 6, whic

236 ts with C1 cell lesions, the mCherry-labeled axon terminals originating from the transfected noncatec

237 ynapses from inhibitory- and excitatory-type axon terminals, over 88% of which were unlabeled and oth

238 ns have reached the medulla; ttk69 mutant R7 axon terminal overgrowth begins shortly after this time

239 rom axon terminals allows replacement of the axon terminal population within a day.

240 Early loss of synaptic vesicles in axon terminals preceding motor deficits, accumulation of

241 The distance between cell bodies and axon terminals predicts that glutamine-glutamate cycle i

242 nologies, is highly specific for determining axon terminal projections within particular neuronal pop

243 and Orco protein is trafficked to developing axon terminals, raising the possibility that ORs play a

244 The HBC(R/MC)s with axon terminals ramifying between 0% and 30% of the inner

245 uts from rods and M-cones, the HBC(MC)s with axon terminals ramifying between 10% and 50% of the IPL

246 marily from M-cones, and the HBC(M/SC)s with axon terminals ramifying between 25% and 50% of IPL rece

247 ity preceded by the occurrence of dystrophic axon terminals, reduced axonal transport, organelle-fill

248 to Bar, and optogenetic stimulation of these axon terminals reliably provokes voiding.

249 The function of GLT-1 in axon terminals remains unknown.

250 ux into medial nucleus of the trapezoid body axon terminals, resulting in reduced synaptic strength.

251 EM) reconstruction of rat CA3 pyramidal cell axon terminals revealed approximately 1.7-1.9 times high

252 Ca(2+) responses in the ePN axon terminals show no detectable suppression by iPNs, a

253 Based on their axon terminal stratification, these bipolar cells could

254 ophy, reduces intrinsic excitability, alters axon terminal structure, and impairs striatal dopamine r

255 e neuropeptide distribution from the soma to axon terminals, suggesting that the generation of glucos

256 2 contained mainly Lewy neurites in presumed axon terminals, suggesting the involvement of the EC -->

257 Basket cells, whose axon terminals surround principal cell somata and proxim

258 morphology and stratification level of their axon terminal system in the inner plexiform layer and in

259 rtical slices revealed the morphology of the axon terminal system of individual bipolar cells.

260 lysis revealed that GABAergic CB1-containing axon terminals targeted dendrites adjacent to glutamater

261 types of immunoreactivities: basally located axon terminals that are colocalized with myoinhibitory p

262 2) travels retrogradely to nearby inhibitory axon terminals that express the primary type 1 cannabino

263 New AChR clustering is also induced by axon terminals that follow SC processes extended during

264 ith immunogold labeling demonstrated labeled axon terminals that formed symmetric synapses on dendrit

265 2) in axon segments that are continuous with axon terminals that lack VMAT2, but contain vesicular gl

266 2), we found densely innervated CTB-positive axon terminals that were mainly located in the lateral a

267 CB1 immunoreactivity was detected in axon terminals that were typically large, complex, and c

268 anscript into protein, and protein levels in axon terminals, the key site of GABA production and func

269 abeled projection neurons from (outputs) and axon terminals to (inputs) the ACC of adult rhesus monke

270 nner, leading to tiled arrangements of their axon terminals to allow optimal allocation of serotonin

271 neurotransmitter GABA can spill over between axon terminals to cause excitation of nearby synapses to

272 osis signal is retrogradely transported from axon terminals to cell bodies to induce cell death.

273 ced ambiguities in the correct assignment of axon terminals to identified motor units imaged at lower

274 id signaling stimulates protein synthesis in axon terminals to induce long-term depression of hippoca

275 n-dependent transport of organelles from the axon terminals to the cell bodies is essential to the su

276 sites of input from DB6 diffuse bipolar cell axon terminals to the inner stratifying type of melanops

277 ry, neuroinvasive herpesviruses traffic from axon terminals to the nuclei of neurons resident in peri

278 of GABAAR-mediated depolarizations from MNTB axon terminals to the soma, some hundreds of microns awa

279 e number of synaptic connections per GPe-STN axon terminal, to substantial strengthening of the GPe-S

280 ns and damaged mitochondria, particularly at axon terminals, ultimately might overwhelm the capacity

281 t localize ionotropic GABA receptors on cone axon terminals using electron microscopy, we suggest tha

282 by inhibiting Ca(2+) entry into presynaptic axon terminals via N-type (Cav2.2) Ca(2+) channels.

283 onal innervation by CCK-positive basket cell axon terminals was confirmed by reduced frequency of inh

284 with this, membrane localization of NHE1 at axon terminals was greatly reduced in Chp1-deficient Pur

285 to the cytoplasm, whereas CB1r-ir located in axon terminals was more commonly localized on the plasma

286 ABA transporter and bassoon coimmunoreactive axon terminals was unchanged; and (5) the number of ultr

287 Using viral vectors taken up at axon terminals, we expressed chemogenetic actuators sele

288 e FG injection, and (2) GLP-1-immunoreactive axon terminals were observed adjacent to the ventricular

289 Vesicles transport Nfasc186 to the soma and axon terminal where they fuse with the neuronal plasma m

290 EAATs reside on rod bipolar cell axon terminals where GABA and glycine receptors also med

291 lso expressed in cerebellar basket cell (BC) axon terminals, where its blockade increases BC inhibiti

292 ein synthesis by cytoplasmic RNP granules in axon terminals, where RNP granules regulate local RNA me

293 that accumbens DBS antidromically stimulates axon terminals, which ultimately activates GABAergic int

294 l bodies and dendrites, as well as unlabeled axon terminals, which, in turn, form inhibitory-like syn

295 Our approach was to label RGC axon terminals with an indicator of activity and quantit

296 ynaptic efficacy are concurrently somata and axon terminals, with the direction of cortical current f

297 Perforant path (PP) axon terminals within the dentate gyrus (DG) contained a

298 Retinal ganglion cell axon terminals within the P and K layers were reconstruc

299 Optogenetic activation of CCK(NTS) axon terminals within the PVH reveal the satiating funct

300 ressed in dendrites, unmyelinated axons, and axon terminals within the STN.