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1 onous neuronal activity after TCL as well as axonal sprouting.
2 he exception of NGF/Adts, failed to increase axonal sprouting.
3 M may also be required for adult cholinergic axonal sprouting.
4  catecholamines) and initiating compensatory axonal sprouting.
5 roliferation and remyelination, and promoted axonal sprouting.
6 p1 in GnRH neurons counteract Sema3A-induced axonal sprouting.
7 l protein that mediates myelin inhibition of axonal sprouting.
8 n the CNS may stabilize the node and prevent axonal sprouting.
9 ing GAP-43 or L1 showed minor enhancement of axonal sprouting.
10 ized aspiration lesions, which do not induce axonal sprouting.
11 t nonhuman primates retains the capacity for axonal sprouting.
12 chondroitin sulfate proteoglycans that limit axonal sprouting after CNS injury.
13  during axonal regeneration, with overlap to axonal sprouting after stroke.
14 Semaphorin 3A (Sema3A), a known inhibitor of axonal sprouting, also alters vascular patterning.
15  to prevent neuronal degeneration, stimulate axonal sprouting and ameliorate behavioral deficits in v
16 ing both GAP-43 and L1 showed more extensive axonal sprouting and axonal growth into the proximal por
17 le (NCAM) expression in motor neurons during axonal sprouting and compensatory reinnervation was expl
18 ther actions, however, such as prevention of axonal sprouting and effects on G-protein-coupled recept
19 nction studies, we found that GDF10 produced axonal sprouting and enhanced functional recovery after
20 uromuscular junction denervation by inducing axonal sprouting and enhancing motor neuron viability.
21 ytoplasmic SRF (SRF-DeltaNLS-GFP) stimulated axonal sprouting and facial nerve regeneration in vivo.
22                                     Aberrant axonal sprouting and failed regeneration of injured axon
23 ondroitin sulfate proteoglycans and improves axonal sprouting and functional recovery after spinal co
24 ved signal mediated by prostacyclin triggers axonal sprouting and functional recovery in a mouse mode
25   Thus, GDF10 is a stroke-induced signal for axonal sprouting and functional recovery.
26 ogo-A with neuronal NgR1 limits regenerative axonal sprouting and functional recovery.
27 ificed two and four weeks after surgery, and axonal sprouting and glial changes were assessed in the
28            In contrast, moderate spontaneous axonal sprouting and induced-sprouting seen under differ
29 l as immune cell infiltration and changes in axonal sprouting and locomotor recovery.
30 in periinfarct cortex and is an inhibitor of axonal sprouting and motor recovery in stroke.
31 act receptor expression may produce aberrant axonal sprouting and neuroaxonal dystrophy.
32  MUs involves two coordinated processes: (i) axonal sprouting and new branching growth from adjacent
33                         A mechanism based on axonal sprouting and occupancy of the vacant synaptic sp
34  expression of GAP43 (P < 0.01), a marker of axonal sprouting and plasticity, in the peri-infarct cor
35         Recovery from stroke is dependent on axonal sprouting and reconnection that occurs during a t
36 ry after stroke; knocking down GDF10 blocked axonal sprouting and reduced recovery.
37 ng nervous system development and facilitate axonal sprouting and regeneration after injury in the ad
38  LRP1 agonists significantly enhance sensory axonal sprouting and regeneration after spinal cord inju
39 CNS) have been implicated in the blocking of axonal sprouting and regeneration following injury.
40 ce that ciliary neurotrophic factor promotes axonal sprouting and regeneration in the periphery raise
41 e that the transplantation of BMSCs enhances axonal sprouting and rewiring into the denervated spinal
42 t 5 and 9 d postlesion, during the period of axonal sprouting and synaptogenesis, there was an increa
43 xperience led to topographically appropriate axonal sprouting and synaptogenesis.
44 et tissues stimulate sympathetic and sensory axonal sprouting and that an absence of p75NTR by sensor
45 ogo-NogoReceptor (NgR) pathway might enhance axonal sprouting and thereby recovery after focal brain
46 y of spared Nf1-/- DRG neurons for increased axonal sprouting, and by non-cell-autonomous contributio
47 ental switch in mTOR dependency for inducing axonal sprouting, and indicate that PTEN deletion in adu
48 B2 (CAErbB2) led to synaptic loss, exuberant axonal sprouting, and lethality at birth.
49 to limit neuroplasticity, activity-dependent axonal sprouting, and recovery in the adult.
50 nt of endogenous neurogenesis, angiogenesis, axonal sprouting, and synaptogenesis in the ischaemic br
51 rons, Schwann cell hyperplasia, and aberrant axonal sprouting around the medulla were observed in NGF
52 in the disease, associated with distal motor axonal sprouting as part of the reinnervation response t
53 pto) during sleep were associated with local axonal sprouting as revealed by the increase of anatomic
54  the SON exhibits a decreased propensity for axonal sprouting as the animal ages, we performed a unil
55 ancement of the MDM2/p53-IGF1R axis enhances axonal sprouting as well as functional recovery after sp
56 hyperalgesia without denervation, and robust axonal sprouting at 5 months after surgery.
57 ratum (s.) oriens of the hippocampus exhibit axonal sprouting beyond their normal territory and inner
58 n on AD-related synaptotoxicity and aberrant axonal sprouting by ablating or overexpressing Fyn in hu
59 licated NGF in the regulation of cholinergic axonal sprouting by intact neurons projecting to the hip
60 se results confirm that post-lesion reactive axonal sprouting can be delayed in the central nervous s
61 he ability to respond to growth factors with axonal sprouting, cell hypertrophy, and activation of fu
62 combination leads to an additive increase in axonal sprouting compared with single treatments.
63 de; with the transcallosal and corticospinal axonal sprouting correlating with functional recovery.
64 s, fiber length as well as the morphology of axonal sprouting, deep within the tissue.
65 25-day-old rat may contribute to the loss of axonal sprouting following axotomy.
66 e 125-day-old rat demonstrated an absence of axonal sprouting following injury.
67 ntage of marked differences in the degree of axonal sprouting from contralateral homotypic cortex aft
68 es in the NL revealed that CH also prevented axonal sprouting from occurring.
69 s with reinnervation of denervated fibres by axonal sprouting from slow fibres.
70                                     Enhanced axonal sprouting from the ipsilesional pyramidal tract i
71 r these proteins in epigenetic regulation of axonal sprouting, growth factor-dependent survival of ne
72     The results showed that estrogen induces axonal sprouting in a brainstem-spinal pathway in the ad
73 Others have shown a decreased propensity for axonal sprouting in an aged brain; therefore, to see if
74                                              Axonal sprouting in cortex adjacent to the infarct is pa
75     Recovery after stroke is associated with axonal sprouting in cortex adjacent to the infarct.
76  expression is increased in association with axonal sprouting in deafferented adult rat hippocampus.
77 ulated during the ovarian cycle and promotes axonal sprouting in hypothalamic neurons secreting gonad
78 ral stem cell development and is a marker of axonal sprouting in mid stages of embryonic development.
79 Stroke induces a unique microenvironment for axonal sprouting in periinfarct cortex, in which growth-
80       Thus, we hypothesized that the lack of axonal sprouting in the 125-day-old rat SON may be due t
81 t contralateral cortical neurons may undergo axonal sprouting in the denervated striatum following a
82                                 In contrast, axonal sprouting in the hippocampus of hAPP mice was una
83 t that endogenous FGF-2 promotes cholinergic axonal sprouting in the injured adult brain.
84  is estrogen dependent, and estrogen induces axonal sprouting in the NRA-lumbosacral pathway.
85           The impairment of kindling-induced axonal sprouting in the null mutants could not be attrib
86 ospinal cell somata coincided with increased axonal sprouting in the spinal cord.
87     Thus, Nogo-A plays a role in restricting axonal sprouting in the young adult CNS after injury.
88 nic or viral blockade of NgR function allows axonal sprouting in vivo.
89  motor system drives significant spontaneous axonal sprouting instead of axon regeneration.
90                CISL induced vasopressinergic axonal sprouting into the external zone of the median em
91 ioral sparing is not clearly understood, but axonal sprouting is a likely candidate.
92 denervated neonatal muscles is deficient and axonal sprouting is absent.
93    Knowing the circuit-level determinants of axonal sprouting is important for repairing motor circui
94 emaining viable motor neurons; however, this axonal sprouting is insufficient to compensate for motor
95  due to dorsal column injuries is related to axonal sprouting is not known.
96 overy process, but the signal that initiates axonal sprouting is not known.
97 connections in areas denervated by a lesion (axonal sprouting) is more widespread than previously tho
98 critical for both proper axonal function and axonal sprouting, is inhibited by stroke and that this i
99 he results suggest that M1 injury results in axonal sprouting near the ischemic injury and the establ
100          The molecular systems that underlie axonal sprouting, neurogenesis, and gliogenesis after st
101 l period of neural development that includes axonal sprouting, neurogenesis, and surges of select neu
102 itic connectivity and contribute to aberrant axonal sprouting observed in AD patients.
103  exuberant neurite outgrowth and hippocampal axonal sprouting observed in knock-in mice expressing FA
104 ged in the denervated striatum suggests that axonal sprouting occurred in response to the lesion.
105                                        Local axonal sprouting occurs, producing an increase in unmyel
106 reviously demonstrated an unexpected, robust axonal sprouting of contralateral corticostriatal neuron
107  exogenous tPA delivery increased poststroke axonal sprouting of corticobulbar and corticospinal trac
108                                              Axonal sprouting of corticospinal and raphespinal fibers
109                                              Axonal sprouting of excitatory neurons is frequently obs
110 tion (ECS) has been shown recently to induce axonal sprouting of granule cells in the rodent hippocam
111              We investigated whether central axonal sprouting of primary afferents spared by the rhiz
112  or all of the cortical hand map; 2) central axonal sprouting of spared primary afferents into the do
113 rate parallel signs of mild degeneration and axonal sprouting of sympathetic innervations in early st
114 perilesional tissue, erythropoietin enhanced axonal sprouting of the contralesional, but not ipsilesi
115 ecovery, perilesional tissue remodelling and axonal sprouting of the corticorubral and corticobulbar
116 sis and viral tracing confirmed compensatory axonal sprouting of the surviving neurons.
117 roke, bilateral innervation occurred through axonal sprouting of the uninjured CRT and CST.
118 001), axonal branching (P < 0.001), terminal axonal sprouting (P < 0.001)] were all present to an inc
119  of cardiac innervation, show alterations in axonal sprouting, process development and distribution o
120 s (TCL) of sensorimotor cortex, which induce axonal sprouting, produced two sequential patterns of lo
121              The regenerative and collateral axonal sprouting rates, blood vessel growth rate and Sch
122  As part of the disease process, an aberrant axonal sprouting response is known to occur near Abeta d
123                                          The axonal sprouting response is mediated by the trkA recept
124 d protein 43 (GAP-43), a molecular marker of axonal sprouting, showed a selective increase in GAP-43
125 n post-stroke angiogenesis, neurogenesis and axonal sprouting suggests a continuum of vascular and ne
126                                              Axonal sprouting that causes reorganization likely takes
127 ing spinal trauma, the limited physiological axonal sprouting that contributes to partial recovery of
128 ynaptic-cellular alterations (e.g., reactive axonal sprouting) that lead to dentate hyperexcitability
129                                              Axonal sprouting thus may provide a mechanism by which t
130 e added TrkB receptor ligands did not induce axonal sprouting to account for increased inhibitory syn
131 ted a trophic response to NGF in the form of axonal sprouting toward the NGF source.
132 ribution is explained by the wide pattern of axonal sprouting triggered by CAErbB2.
133                                         When axonal sprouting was implemented, the seizure threshold
134  perpetual axonal atrophy, degeneration, and axonal sprouting was observed over time, with increasing
135 rned neuronal activity and determine whether axonal sprouting was prevented.
136 s of lesions and in the blockade experiments axonal sprouting was strongly correlated with synchronou
137 n rat cerebellar slice cultures by promoting axonal sprouting with formation of vesicle-filled bouton
138 sters that appears to be caused by excessive axonal sprouting with the formation of new, smaller acet
139        Eight days after NGF/Adts injections, axonal sprouting within the dorsal horn was apparent, an

 
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