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1 ed anterograde transport when added to squid axoplasm.
2 located within juxtaparanodal and internodal axoplasm.
3 d in nodal axoplasm compared with internodal axoplasm.
4 hondrial stationary sites in nodal/paranodal axoplasm.
5 re of PAD inhibited anterograde FAT in squid axoplasm.
6 hondrial distribution and transport in nodal axoplasm.
7 /internodal axoplasm than in nodal/paranodal axoplasm.
8 ced by 86% in 1-mo-old P0-CNS juxtaparanodal axoplasm.
9 ected a single approximately 220 kDa band in axoplasm.
10  liposomes and soluble components from squid axoplasm.
11 posomes, isolated axonal vesicles, and whole axoplasm.
12 u hybridization and microaspiration of their axoplasm.
13 ntibodies on organelle transport in extruded axoplasm.
14 er mg of protein 40-fold higher than that of axoplasm.
15 nse core vesicles along the perimeter of the axoplasm.
16 patial distribution of puncta in subcortical axoplasm.
17          Of the total nonmuscle myosin II in axoplasm, 43.2% copurified with organelles in the 15% su
18 er (M) axon was evaluated in isolated M-cell axoplasm after (1) staining with YOYO-1 and (2) inspecti
19                 Our method for isolating CNS axoplasm also represents a new tool to study axon biolog
20 is approximately 235-kDa protein (p235) from axoplasm and demonstrate that it is a myosin, because it
21 entially at the surface boundary of isolated axoplasm and distributed longitudinally at random interv
22 hibit both fast axonal transport in isolated axoplasm and elongation of neuritic processes in intact
23 the 72-kDa radiolabeled band in heat-shocked axoplasm and glial sheath samples comigrated with a band
24  recovery of normal elemental composition in axoplasm and mitochondria of small, medium and large dia
25 utant FUS-induced impairment of FAT in squid axoplasm and of axonal outgrowth in mammalian primary mo
26 lass of unconventional myosins is present in axoplasm and optic lobes.
27 impairing axonal transport in isolated squid axoplasm and primary hippocampal neurons.
28 idence, we conclude that the p196 present in axoplasm and purified from optic lobes is a squid homolo
29 rade, but not retrograde, transport in squid axoplasm and reduced the amount of kinesin bound to MBOs
30   After crush injury, LRP-1 is lost from the axoplasm and substantially upregulated in Schwann cells.
31 kDa, 84 kDa, and 87 kDa appeared in both the axoplasm and the sheath.
32 oteins with reported RNA binding activity in axoplasm, and levels of several change with axon injury
33  mitochondria were readily identified in the axoplasm, and the ultrastructural integrity of Schwann c
34                                  In extruded axoplasm, antibody disruption of kinesin or the dynactin
35  that neurofilament-dependent structuring of axoplasm arises through an "outside-in" signaling cascad
36               This myosin is also present in axoplasm, as determined by two independent criteria.
37 egeneration were prominent in juxtaparanodal axoplasm at 1 mo of age.
38                                The herniated axoplasm became directed back towards the internode, for
39  signals were also distributed in peripheral axoplasm below the matrix.
40  inhibited fast axonal transport in isolated axoplasm by decreasing both the number and velocity of v
41  ions are swept out of the membrane into the axoplasm by hyperpolarization.
42 ort speed was significantly reduced in nodal axoplasm compared with internodal axoplasm.
43 ow-contrast background, showed that isolated axoplasm contained characteristic 25 nm P signals, which
44                 These data indicate that MGA axoplasm contains relatively high levels of constitutive
45 anges included a significant decrease in the axoplasm diameter of myelinated neurons and an increase
46 tation of RhoA and phosphorylated cofilin in axoplasm-enriched samples from injured optic nerve.
47                We have used iTRAQ to compare axoplasm-enriched samples from naive vs injured optic ne
48                                 Furthermore, axoplasm ensheathed by 65% of the CNS incisures examined
49                        Furthermore, purified axoplasm exhibits adenosine-to-inosine activity and can
50  have developed a novel method to enrich for axoplasm from rodent optic nerve and characterised the e
51  fast axonal transport along microtubules in axoplasm from squid giant axons.
52 directly inhibit fast axonal transport using axoplasm from the squid giant axon and suggest that axon
53                            Organelles in the axoplasm from the squid giant axon move along exogenous
54      In vitro motility assays performed with axoplasm from the squid giant axon showed a requirement
55    Vesicle motility assays in isolated squid axoplasm further demonstrated that both mutant merlin an
56                                              Axoplasm, however, contained the constitutively active f
57 otubule packing density), fractional area of axoplasm in the nerve fiber bundle (f), mitochondrial fr
58 atrix, and a confluent volume of subcortical axoplasm integrated through an actin cytoskeleton.
59              Our detergent-free method draws axoplasm into a dehydrated hydrogel of the polymer poly(
60 indings were derived from examination of the axoplasm isolated from myelinated fibers as axoplasmic w
61 th in transgenic mice and, more recently, in axoplasm isolated from squid giant axons.
62 sufficient for activation of this pathway in axoplasms isolated from squid giant axons.
63             Both oAbeta and CK2 treatment of axoplasm led to increased phosphorylation of kinesin-1 l
64          Previous work in the isolated squid axoplasm model demonstrated that several pathogenic form
65 l spacing of plaques around the periphery of axoplasm near the axon-myelin border are likely reasons
66 tive HSP 70s and that, after heat shock, MGA axoplasm obtains inducible HSPs of 72 kDa, 84 kDa, and 8
67 perisynaptic/extrasynaptic membranes and the axoplasm of 13% of excitatory-like, presumably glutamate
68 m and endoneurium, with ultimate delivery to axoplasm of distal peripheral nerves.
69 s, the mutant virus failed to enter into the axoplasm of ganglionic neurons.
70 proximately 1-3% of the total protein in the axoplasm of MGAs.
71  resulting in accumulation of free NE in the axoplasm of sympathetic nerves.
72  without affecting the rate of production of axoplasm or microtubule polymer, and without decreasing
73         Despite the extensive pruning, total axoplasm per neuron increases as axons elongate, thicken
74 ime analysis of vesicle mobility in isolated axoplasms perfused with oAbeta showed bidirectional axon
75                             Western blots of axoplasm probed with an affinity purified antibody to ch
76 t axon extension but does not interfere with axoplasm production.
77 eriments presented here using isolated squid axoplasm reveal inhibition of FAT as a common toxic effe
78       The amount of heat-induced proteins in axoplasm samples was greater after a 2-hour heat shock t
79 re also distributed in a delimited volume of axoplasm, subjacent to the plaque.
80 ficantly larger in juxtaparanodal/internodal axoplasm than in nodal/paranodal axoplasm.
81                                     In squid axoplasm, the M1 peptide dramatically inhibits fast axon
82 hibits FAT in a human cell line and in squid axoplasm through a pathway that involves activation of c
83 e for neurofilament-dependent structuring of axoplasm through intra-axonal crossbridging between adja
84 ning physiological saline; (2) we exposed GA axoplasm to Ca2+-containing salines and observed that me
85 gh spectrin and ank2-L, extend deep into the axoplasm to promote microtubule organization.
86                      To deliver HSV into the axoplasm, viral particles stripped of their envelopes by
87 ffusion coefficient of the moving tubulin in axoplasm was 8.6 micrometer(2)/s compared with only 0.43
88                                              Axoplasm was fractionated through a four-step sucrose gr
89  did form in severed GAs after >99% of their axoplasm was removed by internal perfusion; (3) we exami
90                         Using isolated squid axoplasm, we show that MPP+ produces significant alterat
91 and minus-end vesicle populations from squid axoplasm were isolated from each other by selective extr
92 ired anterograde and retrograde FAT in squid axoplasm, whereas FUS WT had no effect.
93                                 Treatment of axoplasm with antibodies to the p150(Glued) subunit of d
94                    Furthermore, perfusion of axoplasms with active CK2 mimics the inhibitory effects