ライフサイエンスコーパス: bacillary

1 cular proliferative tissue response known as bacillary angiomatosis (BA) and bacillary peliosis (BP)

2 Bacillary angiomatosis (BA), one of the many clinical ma

3 Bacillary angiomatosis and bacillary peliosis are vascul

4 al serious sequelae of infections, including bacillary angiomatosis and bacillary peliosis.

5 Bartonella henselae, the causative agent of bacillary angiomatosis and cat scratch disease, also rec

6 trench fever and a cause of endocarditis and bacillary angiomatosis in humans, has the highest report

7 fever, chronic bacteremia, endocarditis, and bacillary angiomatosis in humans.

8 A case of bacillary angiomatosis infection presenting as a skin no

9 The bacillary angiomatosis responded to 6 months of therapy

10 cterium associated with cat scratch disease, bacillary angiomatosis, and bacillary peliosis.

11 onella henselae, the agents of trench fever, bacillary angiomatosis, and parenchymal peliosis, and in

12 ases such as trench fever, endocarditis, and bacillary angiomatosis, B. quintana must survive and rep

13 duals with B. hensalae infection may develop bacillary angiomatosis, bacillary peliosis, and relapsin

14 y life threatening to the patient, including bacillary angiomatosis, bacillary peliosis, and verruga

15 e and B. quintana, have been associated with bacillary angiomatosis, but culture and speciation are d

16 istology of splenic biopsy was suggestive of bacillary angiomatosis, but immunohistochemistry ruled o

17 nt of trench fever and an etiologic agent of bacillary angiomatosis, has an extraordinarily high hemi

18 vidual, B. henselae-induced angiogenesis, or bacillary angiomatosis, is characterized by vascular pro

19 serious human infections globally, including bacillary angiomatosis, Oroya fever, trench fever, and e

20 limaea) henselae causes cat-scratch disease, bacillary angiomatosis, peliosis hepatis, and fever in h

21 exposures was administered to patients with bacillary angiomatosis-peliosis and to 96 matched contro

22 Of the 49 patients with bacillary angiomatosis-peliosis, 26 (53 percent) were in

23 ae and B. quintana, the organisms that cause bacillary angiomatosis-peliosis, are associated with dif

24 and who had clinical lesions consistent with bacillary angiomatosis-peliosis.

25 ifestations include conjunctival and retinal bacillary angiomatosis.

26 al pathogen, causing cat scratch disease and bacillary angiomatosis.

27 to cause endocarditis, peliosis hepatis, and bacillary angiomatosis.

28 roliferative tumors, a disease process named bacillary angiomatosis.

29 cat scratch disease, bacillary peliosis, and bacillary angiomatosis.

30 rculosis is not invariable and may depend on bacillary as well as host factors.

31 with unique biological adaptations, namely, bacillary band and stichosome, found only in whipworms a

32 l features, including stichocytes and paired bacillary bands.

33 proficient littermates resulted in increased bacillary burden and excessive pulmonary inflammation ch

34 tection results increased progressively with bacillary burden and immunosuppression.

35 e time of BCG vaccination also increased the bacillary burden and reduced T cell responses after chal

36 aracterized by a moderately increased tissue bacillary burden and severe pulmonic histopathological d

37 ain, improved survival, a 2.5 log lower lung bacillary burden at 35 days post-infection, and reduced

38 erferon (IFN) gamma responses in controlling bacillary burden in human immunodeficiency virus (HIV)-a

39 significantly reduced the lipid content and bacillary burden in Mtb-infected macrophages.

40 ose in well-nourished controls, although the bacillary burden in the malnourished animals continued t

41 serodiagnostic proteins for the spectrum of bacillary burden in tuberculosis.

42 ole-cell lysate were associated with reduced bacillary burden on sputum smear grade, days to culture

43 relations between human responses and sputum bacillary burden were assessed by quantile and hurdle re

44 hich may provide a dynamic measure of sputum bacillary burden when used longitudinally.

45 We observed decreasing bacillary burden with increasing level of immunosuppress

46 sses a spectrum of characteristics-including bacillary burden, clinical severity, and access to care-

47 Pretreatment, measures of bacillary burden, including sputum smear microscopy, cul

48 Children differ from adults in bacillary burden, spectrum of disease, the metabolism an

49 ositive; eight (61.5%) of these 13 had a low bacillary burden, with smear grades scanty or 1+ (1-99 a

50 We find that higher bacillary burden, younger age, larger mean upper arm cir

51 lished disease, is associated with very high bacillary burden.

52 ens are associated with reduced tuberculosis bacillary burden.

53 DRplus is directly related to the specimen's bacillary burden.

54 and reactive protein numbers increased with bacillary burden.

55 atients, and (iii) responses correlated with bacillary burden.

56 on of precursors may be a general feature of bacillary cell wall biogenesis.

57 of rifampicin plasma exposure in optimizing bacillary clearance and improving treatment outcomes, ev

58 ulosis and explored the relationship between bacillary clearance and treatment outcomes.

59 We explored these relationships by modeling bacillary clearance in sputum in adult patients on first

60 IL-2 did not enhance bacillary clearance or improvement in symptoms in human

61 ned high intrapulmonary drug exposure, rapid bacillary clearance, shorter treatment duration and bett

62 lveolar cells was not associated with sputum bacillary clearance.

63 lining fluid was associated with more rapid bacillary clearance.

64 enerally by an inability to achieve complete bacillary clearance.

65 rowth, differential macrophage viability, or bacillary clumping.

66 n vitro activity against the clostridial and bacillary collagenases.

67 ction usually results in the immune-mediated bacillary containment of latent tuberculosis infection (

68 prosy, a clinical form characterized by poor bacillary control due to T helper 2 cells, M2 macrophage

69 In addition, bacillary control was associated with down-regulation of

70 vity was accompanied by a small reduction in bacillary counts, but this did not affect modeling of ba

71 -VEGF therapy resulted in 100% resolution of bacillary detachment and significant decreases in CST an

72 In eyes with bacillary detachment at baseline, the anti-VEGF treatmen

73 VEGF treatment resulted in the resolution of bacillary detachment in 100% of the eyes.

74 Bacillary detachment is an OCT signature that is identif

75 Bacillary detachment was identified in 7.4% (6 of 81) ey

76 olume at baseline compared with eyes without bacillary detachment.

77 higella flexneri causes 270 million cases of bacillary dysentery (blood in stool) worldwide every yea

78 SS to invade human intestinal cells to cause bacillary dysentery (shigellosis) that is responsible fo

79 e Escherichia coli (EIEC), known for causing bacillary dysentery akin to Shigella species, comprises

80 Shigella flexneri causes a severe form of bacillary dysentery also known as shigellosis.

81 iaceae family, causing ~269 million cases of bacillary dysentery and >200,000 deaths each year.

82 for understanding the induction mechanism of bacillary dysentery and for evaluating Shigella vaccine

83 ome serotypes of Escherichia coli that cause bacillary dysentery and hemorrhagic colitis, respectivel

84 nge with virulent S. flexneri 2a can provoke bacillary dysentery and severe pathogenesis in adult mic

85 n and nonhuman primates, Shigella spp. cause bacillary dysentery by invading colon epithelium and pro

86 Shigella flexneri causes bacillary dysentery in humans by invading epithelial cel

87 Shigella species cause bacillary dysentery in humans by invasion, intracellular

88 eri is a gram-negative bacterium that causes bacillary dysentery in humans that is characterized by a

89 lla dysenteriae serotype 1, a major cause of bacillary dysentery in humans, can use heme as a source

90 f Shigella dysenteriae, a causative agent of bacillary dysentery in humans.

91 The laboratory surveillance of bacillary dysentery is based on a standardised Shigella

92 A major challenge in combating bacillary dysentery is the lack of a small-animal model

93 Shigella flexneri causes bacillary dysentery with symptoms resulting from the inf

94 Shigella spp. are the major cause of bacillary dysentery worldwide.

95 Shigellosis (previously bacillary dysentery) was the primary diarrhoeal disease

96 Shigellae cause bacillary dysentery, a bloody form of diarrhoea that aff

97 Shigella spp. are causative agents of bacillary dysentery, a human illness with high global mo

98 xneri is responsible for the endemic form of bacillary dysentery, an acute rectocolitis in humans.

99 Shigella spp. cause shigellosis, also called bacillary dysentery, and invade colonic epithelial cells

100 Shigella flexneri, an etiological agent of bacillary dysentery, causes apoptosis in vitro.

101 esponsible for diseases such as diarrhea and bacillary dysentery, commonly afflicting infants and chi

102 mple, Shigella spp., the causative agents of bacillary dysentery, differ from the closely related com

103 Shigella flexneri, the causative agent of bacillary dysentery, injects invasin proteins through a

104 Shigella, the etiological agent of bacillary dysentery, rapidly kills human monocyte-derive

105 immunity to Shigella, the causative agent of bacillary dysentery, requires several episodes of infect

106 e demonstrate here that a causative agent of bacillary dysentery, Shigella flexneri, uses the type II

107 gella flexneri, the causative agent of human bacillary dysentery, switches off host sumoylation durin

108 Shigella, the leading cause of bacillary dysentery, uses a type III secretion system (T

109 ve bacterial pathogen and causative agent of bacillary dysentery.

110 a bacterial pathogen and causative agent of bacillary dysentery.

111 de of Shigella flexneri 2a, a major cause of bacillary dysentery.

112 ic pathogen that is the predominant cause of bacillary dysentery.

113 at invades the colonic epithelium and causes bacillary dysentery.

114 cultative intracellular organism that causes bacillary dysentery.

115 lining fluid was associated with more rapid bacillary elimination and shorter time to sputum negativ

116 brook agar are used to calculate the rate of bacillary elimination from sputum collected from patient

117 y associated with faster sterilization phase bacillary elimination from the SSCC model (odds ratio [O

118 uid, and alveolar cells as covariates in the bacillary elimination models.

119 Bacillary elimination rates (BER) were estimated using l

120 In Malawi, we modeled bacillary elimination rates (BERs) from sputum cultures

121 chest radiograph was associated with slower bacillary elimination.

122 counts, but this did not affect modeling of bacillary elimination.

123 d abundant intra- and extracellular bacilli, bacillary fragments, and granular antigen-staining in me

124 owever, RPE cells are better able to control bacillary growth and RPE cell survival is greater than t

125 and found this gene to be necessary for full bacillary growth and survival.

126 alpha (TNF-alpha) is required for control of bacillary growth and the protective granulomatous respon

127 h1-mediated cellular immunity and control of bacillary growth at one pole to poor Ag-specific T cell

128 2O2, catalase and peroxidase activities, and bacillary growth rates measured both intracellularly in

129 gulatory and metabolic pathways required for bacillary growth restriction and reactivation.

130 y third day for 15 days, but weight loss and bacillary growth resumed when KGF was withdrawn.

131 ice were infected with low doses of BCG-TNF, bacillary growth was controlled, granulomas were small a

132 oth-based method with microscopic reading of bacillary growth, the microscopic observation drug susce

133 luminal phagocytes to remain permissive for bacillary growth.

134 lippine cohort comprising patients with high bacillary indices (BI; average:4,9), 94%(n = 161) of MB

135 <.0001), antibiotic-resistant Gram-negative bacillary infections (2.5 infections/100 admissions vs.

136 Accuracy was assessed with 158 Gram-negative bacillary isolates, including 134 carbapenemase producer

137 inistration of oral chemotherapy resulted in bacillary killing.

138 -secreting murine TNF-alpha (BCG-TNF) led to bacillary killing.

139 CST, SRF, and total fluid than eyes without bacillary layer detachment (P < 0.05 for each comparison

140 A greater proportion of eyes with bacillary layer detachment had high-exudative volatility

141 The majority of eyes with bacillary layer detachment have high-exudative volatilit

142 retina in 47% (8/17) of cases and tractional bacillary layer detachment in 12% (2/17) of cases.

143 nti-VEGF treatment resulted in resolution of bacillary layer detachment in 97.9% of eyes by week 48.

144 The presence of bacillary layer detachment may provide an important imag

145 ased SRF and SHRM volume; however, eyes with bacillary layer detachment never reached the level of BC

146 herapy resulted in a very high proportion of bacillary layer detachment resolution with significantly

147 Classic bacillary layer detachment was identified in 7.2% (47/65

148 Bacillary layer detachment, an OCT signature representin

149 In eyes with bacillary layer detachment, anti-VEGF treatment improved

150 nd SHRM volume at baseline than eyes without bacillary layer detachment.

151 he level of BCVA improvement as eyes without bacillary layer detachment.

152 49/100) were more likely to have a preserved bacillary layer on SS-OCT with low-amplitude outer retin

153 These had a thickened bacillary layer with high-amplitude ORCs with photorecep

154 rity (intact, patchy or complete loss of the bacillary layer).

155 M. tuberculosis reduced the MBLA-detectable bacillary load (estimated number of CFU [eCFU] per milli

156 her among participants who had higher sputum bacillary load (P < .01).

157 However, mycobacterial sputum bacillary load and clinical characteristics, including a

158 bDeltambtE exhibited a significantly reduced bacillary load and histopathological damage in the organ

159 r Ag-specific T cell immunity with extensive bacillary load and Th2 cytokine-expressing lesions at th

160 ve treatment, had a similar TB-MBLA-measured bacillary load at 8 weeks to those who were smear micros

161 m tuberculosis (1:1) also did not reduce the bacillary load but caused increased expression of tumor

162 r risk of false resistance compared to other bacillary load categories combined (risk ratio: 8.20; 95

163 (BCG-vector) at a low dose led to increased bacillary load in all organs and an extensive granulomat

164 After 2 months, the bacillary load in lungs was reduced from 9.74 log10 at b

165 harmacological inhibition of CBS reduces Mtb bacillary load in mice.

166 uggests M tuberculosis bloodstream infection bacillary load is causally related to outcomes.

167 h in people living with HIV and disseminated bacillary load might be a key driver of disease severity

168 This group had a significantly lower sputum bacillary load relative to correctly classified smear-po

169 ing to TB-MBLA, 19 of these patients cleared bacillary load to zero by week 2 of treatment and remain

170 the proportion of TB detected with very low bacillary load will increase.

171 ionship between cerebrospinal fluid (CSF) TB bacillary load with mortality.

172 e signal to be detected (low Ct value = high bacillary load) and may approximate TB bacillary load.

173 e signal to be detected (low Ct value = high bacillary load) and may approximate tuberculosis (TB) ba

174 with Ct values in the low tertile (ie, high bacillary load) had 57% 2-week mortality-worse than the

175 with Ct values in the low tertile (i.e. high bacillary load) had 57% 2-week mortality; worse than the

176 and number of lung lesions, decreases in the bacillary load, and improvements in survival, compared w

177 ed host-protective immune responses, reduced bacillary load, and increased survival compared with ani

178 n, severe meningeal inflammation, persistent bacillary load, and progressive clinical deterioration.

179 High CSF TB bacillary load, as measured by Xpert Ultra Ct tertile, i

180 dictor of incident Mtb infection than sputum bacillary load, chest radiographic characteristics, or s

181 SIRT2 inhibitor-treated mice display reduced bacillary load, decreased disease pathology and increase

182 TNF neutralization led to increased lung bacillary load, disrupted granuloma architecture with ex

183 mined by immunohistochemistry; intracellular bacillary load, following TLR2 and TLR4 blockade.

184 cavitary TB is associated with higher sputum bacillary load, our findings support the hypothesis that

185 ha-induced inflammation without reducing the bacillary load.

186 accelerated mortality without affecting the bacillary load.

187 A is a good method for measuring the initial bacillary load.

188 tissue granulomatous reaction, and increased bacillary load.

189 e to culture positivity was used to estimate bacillary load.

190 impact of appropriate treatment on cough and bacillary load.

191 high bacillary load) and may approximate TB bacillary load.

192 with aerosol culture status, independent of bacillary load.

193 load) and may approximate tuberculosis (TB) bacillary load.

194 ophagy and in the reduction of intracellular bacillary load.

195 The treatment resulted in increased lung bacillary loads and even further reduced survival.

196 osis had a significant decrease in pulmonary bacillary loads and tissue damage.

197 ive because it improved survival and reduced bacillary loads in spleen whereas clarithromycin and ami

198 nfection with HN878 or W4 resulted in higher bacillary loads in the cerebrospinal fluid and brain, in

199 +) T cells are generated in response to high bacillary loads occurring during tuberculosis.

200 Notably, 89% of 55 patients with very low bacillary loads on their initial Ultra had false RR-TB r

201 IFNAR1), displayed marked elevations in lung bacillary loads, accompanied by widespread pulmonary nec

202 c CD8(+) T cells develop in response to high bacillary loads, as occurs during tuberculosis, and are

203 alues had a positive association with higher bacillary loads.

204 s for pulmonary tuberculosis (PTB) with high bacillary loads.

205 n is largely driven by samples with very low bacillary loads.

206 cin prevented death but had little impact on bacillary loads.

207 d murine macrophages with high intracellular bacillary loads.

208 commended for the treatment of gram-negative bacillary meningitis and neonatal meningitis.

209 derive robust, quantitative descriptions of bacillary morphologies consequent on gene silencing.

210 a is the only species of human origin with a bacillary morphology.

211 characterization of eight strains of another bacillary Neisseria species from human infections.

212 anulomas, the two cell types colocalized and bacillary numbers were substantially lower, suggesting t

213 to a feed-forward loop that causes increased bacillary numbers, greater T cell dysfunction, and progr

214 nse known as bacillary angiomatosis (BA) and bacillary peliosis (BP) in some human hosts.

215 Bacillary angiomatosis and bacillary peliosis are vascular proliferative manifestat

216 Herein, we report the first case of bacillary peliosis hepatis due to systemic Bartonella he

217 Bacillary peliosis hepatis is an uncommon but well recog

218 revious observations in cat scratch disease, bacillary peliosis, and bacillary angiomatosis.

219 nfection may develop bacillary angiomatosis, bacillary peliosis, and relapsing bacteremia with fever

220 e patient, including bacillary angiomatosis, bacillary peliosis, and verruga peruana.

221 ctions, including bacillary angiomatosis and bacillary peliosis.

222 l-lined cystic disease in the liver known as bacillary peliosis.

223 scratch disease, bacillary angiomatosis, and bacillary peliosis.

224 ermination in Mtb, with potential impacts on bacillary persistence, reactivation, and efficiency of t

225 d understanding of the heterogeneity in both bacillary physiology and host immune response that poten

226 Gram-negative bacillary pneumonia is a common and serious illness asso

227 owth ceases because of the depletion of this bacillary population needs to be modified.

228 defined as variabilities in human behavior, bacillary properties, and host physiology that fuel the

229 In contrast, bacillary replication was controlled in live phagocytes.

230 tress protein believed to be involved in the bacillary response to adverse conditions and in non-repl

231 Five weeks after infection with either bacillary strain, the inbred rabbits had significantly l

232 e MTB gene regX3 appears to be essential for bacillary survival during phosphate limitation and in ma

233 , we show here that sigF is not required for bacillary survival under nutrient starvation conditions

234 ient in phosphate-related genes, we assessed bacillary survival under phosphate-limited conditions an

235 peroxides) may be an important mechanism of bacillary survival within the host phagocyte.

236 mpact of aerobic glycolysis on intracellular bacillary survival, demonstrating that infection-induced

237 lammatory IL-10, and increased intracellular bacillary survival.

238 After the inhalation of 220 to 880 bacillary units, all of the rabbits were overtly well un

239 After the inhalation of 3,900 to 5,800 bacillary units, half of the rabbits died of progressive

240 e stages of the IBC pathway, filamentous and bacillary UPEC detach from the biofilm-like IBC, fluxing

241 lls prior to infection resulted in decreased bacillary viability, presumably due to extracellular kil

242 lular CatG expression, resulted in increased bacillary viability.