ライフサイエンスコーパス: bacterial endotoxin

1 subjects receiving an inflammatory stimulus (bacterial endotoxin).

2 ms responsible for altered responsiveness to bacterial endotoxin.

3 ry--K1 fibroblasts defective in responses to bacterial endotoxin.

4 mportant role in the innate response against bacterial endotoxin.

5 entified as signal-transducing receptors for bacterial endotoxin.

6 mRNA expression in macrophages is induced by bacterial endotoxin.

7 nto the liver, and increased serum levels of bacterial endotoxin.

8 MH) challenged with a shock-inducing dose of bacterial endotoxin.

9 lenged them with lipopolysaccharide (LPS), a bacterial endotoxin.

10 tly by pathogen-associated molecules such as bacterial endotoxin.

11 imuli, such as proinflammatory cytokines and bacterial endotoxin.

12 appaB pathway during both MI and exposure to bacterial endotoxin.

13 (delayed) systemic inflammatory response to bacterial endotoxin.

14 e were exposed to inhaled cigarette smoke or bacterial endotoxin.

15 saccharide to CD14 and host response to this bacterial endotoxin.

16 suppressed systemic inflammation induced by bacterial endotoxins.

17 sensitive pro-inflammatory response towards bacterial endotoxins.

18 n response to distension or stimulation with bacterial endotoxins.

19 d kinases (IRAK) regulates responsiveness to bacterial endotoxins.

20 ns of the following microbiological markers: bacterial endotoxin, 3-hydroxy fatty acids, and muramic

21 Bacterial endotoxin, 3-hydroxy fatty acids, N-acetyl-mur

22 Because bacterial endotoxin activates Kupffer cells, the purpose

23 The treatments explored include bacterial endotoxin adsorption, antipyretics and vasopre

24 Peripheral administration of bacterial endotoxin, an immune stimulant, induces eviden

25 ive stress occurs in animals challenged with bacterial endotoxin and can affect the expression of imp

26 ogroup B to facilitate metabolic labeling of bacterial endotoxin and compared interactions of purifie

27 ogic changes, and reduced systemic levels of bacterial endotoxin and concentrations of Pseudomonas ae

28 ignificantly raised plasma concentrations of bacterial endotoxin and cytokines.

29 inflammation by mediating responses to both bacterial endotoxin and multiple endogenous ligands, inc

30 GPI fraction, which is at least as active as bacterial endotoxin and Mycoplasma lipopeptide and, ther

31 Various biochemical stimuli, such as bacterial endotoxin and the inflammatory cytokines recom

32 h Wnts suppress proinflammatory responses to bacterial endotoxin and to TLR1/2, TLR7, and TLR9 ligand

33 absence of a bacterial backbone, absence of bacterial endotoxins and no contamination by bacterial R

34 he immune system, potentiates the effects of bacterial endotoxin, and increases the lethality of cert

35 ll as in healthy subjects receiving low-dose bacterial endotoxin, and show that these severe stresses

36 and biologically relevant amphiphiles (e.g., bacterial endotoxins), as well as sorting aqueous drople

37 eived bilateral injection of OVD spiked with bacterial endotoxin at 7.0 endotoxin units/ml.

38 ion and provided blood samples to assess two bacterial endotoxin biomarkers, lipopolysaccharide-bindi

39 tor 4, which is best known as a receptor for bacterial endotoxin but is also a poorly understood medi

40 e major producers of IFNgamma in response to bacterial endotoxin but not to interleukin-12, and; (iii

41 opolysaccharide factor (LALF) can neutralize bacterial endotoxin, but its ability to prevent mortalit

42 ypically attribute to bacterial infection or bacterial endotoxins, but infections due to viruses, fun

43 ents a mammalian hepatic defense response to bacterial endotoxin by modulating HDL.

44 cells were able to respond to challenge with bacterial endotoxin by mounting an acute inflammatory re

45 innate defenses that include recognition of bacterial endotoxins by Toll-like receptor 4 (TLR4).

46 Bacterial endotoxin can induce inflammatory and metaboli

47 Lipopolysaccharide (LPS), a potent bacterial endotoxin, can induce SerpinB2 expression via

48 Inflammation in response to bacterial endotoxin challenge impacts physiological func

49 and cardiovascular dynamics in response to a bacterial endotoxin challenge.

50 rabbits to evaluate the ocular reactivity to bacterial endotoxin contained in Dulbecco's phosphate-bu

51 rity and identity, sterile filter integrity, bacterial endotoxin content, sterility, stability) confi

52 No binding of the bacterial endotoxin Cry1A(c) to captured vesicles contai

53 4-dependent cell activation by gram-negative bacterial endotoxins depends on sequential endotoxin-pro

54 We tested whether bacterial endotoxin E. coli lipopolysaccharide (LPS) aff

55 receptor 4 (TLR4), together with MD-2, binds bacterial endotoxins (E) with high affinity, triggering

56 In addition, pretreatment with bacterial endotoxin elicited a reduction in basal O2 con

57 s a critical role in septic shock induced by bacterial endotoxin (endotoxemia).

58 ese enzymes in rats subjected to intraportal bacterial endotoxin exposure (lipopolysaccharide [LPS],

59 erved that animals survived a lethal dose of bacterial endotoxin if they had been previously treated

60 vestigated whether exposure to Gram-negative bacterial endotoxin in early neonatal life can alter neu

61 ed a systemic hyper-inflammatory response to bacterial endotoxin in localized aggressive periodontiti

62 peptidoglycan fragment (muramyl peptide) and bacterial endotoxin in the induction of inflammatory pro

63 sphosphonate enhanced the immune response to bacterial endotoxin in the lung and increased the level

64 liver disease identified increased levels of bacterial endotoxin in the portal circulation, suggestin

65 The constructed system can detect bacterial endotoxins in the range of 0.001-5EU/ml and ba

66 nd the cell line indicate that gram-positive bacterial endotoxins induce hypoxia-inducible factor 1al

67 Our study showed that bacterial endotoxins induced NADPH oxidase activation in

68 Lipopolysaccharide, a bacterial endotoxin, induced secretion of the inflammato

69 iency of HIF-1alpha attenuated gram-positive bacterial endotoxin-induced cellular motility and proinf

70 and display a more pronounced and prolonged bacterial endotoxin-induced febrile response than wild t

71 Therefore, a past history of bacterial endotoxin-induced inflammation can lead to imm

72 ted by TLR4 signaling, such as Gram-negative bacterial endotoxin-induced lung injury and HDM-triggere

73 flammatory effects in a rat acute model of a bacterial endotoxin-induced pulmonary inflammation.

74 ssion: extrapulmonary burn injury suppresses bacterial endotoxin-induced pulmonary neutrophil recruit

75 gnificant therapeutic approach in preventing bacterial endotoxin-induced sepsis and tissue damage.

76 lammation and liver damage as well as during bacterial endotoxin-induced septic shock.

77 ntal protocols that controlled for potential bacterial endotoxin-induced TNF-alpha secretion, the cur

78 inhibition of aldose reductase (AR) prevents bacterial endotoxin-induced uveitis in rats.

79 Lipopolysaccharide (LPS), a bacterial endotoxin, induces inflammation in macrophages

80 Bacterial endotoxin is a potent and predominant microbia

81 Bacterial endotoxin is a potently inflammatory antigen t

82 Gut-derived bacterial endotoxin is an important cofactor in the path

83 In conclusion, we have demonstrated that bacterial endotoxin is internalized and transported to s

84 Bacterial endotoxin is known to induce interferon gamma

85 Bacterial endotoxin levels were high in the serum from V

86 cBAF, and PBAF-in the macrophage response to bacterial endotoxin (lipid A).

87 We found that the internalized bacterial endotoxin lipopolysaccharide (LPS) activated t

88 Bacterial endotoxin lipopolysaccharide (LPS) activation

89 Microbial components such as bacterial endotoxin lipopolysaccharide (LPS) can trigger

90 Here, we show that the proinflammatory bacterial endotoxin lipopolysaccharide (LPS) decreases P

91 traperitoneal injection of the Gram-negative bacterial endotoxin lipopolysaccharide (LPS) elicits a r

92 Peripheral injection of bacterial endotoxin lipopolysaccharide (LPS) induces bra

93 Injection of bacterial endotoxin lipopolysaccharide (LPS) into the hi

94 Gram-negative bacterial endotoxin lipopolysaccharide (LPS) is implicat

95 Because the bacterial endotoxin lipopolysaccharide (LPS) is known to

96 Gram-negative bacterial endotoxin lipopolysaccharide (LPS) triggers th

97 Here, we report that super low dose bacterial endotoxin lipopolysaccharide (LPS), as low as

98 Occludin, and Claudin-3, and exacerbated the bacterial endotoxin lipopolysaccharide (LPS)-induced dam

99 defined as day 18.5 or later) by attenuating bacterial endotoxin lipopolysaccharide (LPS)-induced mat

100 on of inflammatory factors stimulated by the bacterial endotoxin lipopolysaccharide (LPS).

101 receptor-mediated innate immune response to bacterial endotoxin lipopolysaccharide (LPS).

102 r normal conditions and after injection of a bacterial endotoxin lipopolysaccharide (LPS).

103 to protect the liver from injuries upon the bacterial endotoxin lipopolysaccharide (LPS).

104 mposing systemic inflammation induced by the bacterial endotoxin lipopolysaccharide (LPS).

105 ntext of inflammation stress mimicked by the bacterial endotoxin lipopolysaccharide (LPS).

106 to inflammation induced by injection of the bacterial endotoxin lipopolysaccharide (LPS).

107 ia in young and aged mice with a low dose of bacterial endotoxin lipopolysaccharide (LPS, 2.5 mg/kg)

108 inic-polyribocytidilic acid [Poly(I:C)], the bacterial endotoxin lipopolysaccharide, the locally acti

109 NOS-2) is induced following stimulation with bacterial endotoxin (lipopolysaccharide (LPS)) and/or pr

110 Inflammatory stimulants such as bacterial endotoxin (lipopolysaccharide (LPS)) are known

111 nstem can control the diverse responses to a bacterial endotoxin (lipopolysaccharide (LPS)) that pote

112 iators TNFalpha, interleukin (IL)-1beta, and bacterial endotoxin (lipopolysaccharide (LPS)).

113 egulated by pro-inflammatory stimuli such as bacterial endotoxin (lipopolysaccharide (LPS)).

114 Exposure to a nontoxic dose of bacterial endotoxin (lipopolysaccharide [LPS]) potentiat

115 Bacterial endotoxin (lipopolysaccharide [LPS]), a glycol

116 ogenesis of systemic inflammation induced by bacterial endotoxin (lipopolysaccharide [LPS]).

117 Macrophages become activated by bacterial endotoxin (lipopolysaccharide) and other stimu

118 ultures of rat hepatocytes after exposure to bacterial endotoxin (lipopolysaccharide) in a nitric oxi

119 n repetitive exposures to different doses of bacterial endotoxin (lipopolysaccharide) or other stimul

120 Using bacterial endotoxin (lipopolysaccharide) to initiate an

121 Their gut microbiota, bacterial endotoxin (lipopolysaccharide), tight junction

122 systemic TNF-alpha expression in response to bacterial endotoxin (lipopolysaccharide).

123 tem is up-regulated after stimulation with a bacterial endotoxin (lipopolysaccharide).

124 The effects of bacterial endotoxin (lipopolysaccharide, LPS) are amplif

125 that caspase-11, the cytosolic receptor for bacterial endotoxin (lipopolysaccharide: LPS), enhances

126 amus in response to intravenous injection of bacterial endotoxin - lipopolysaccharide (LPS).

127 is influenced by inflammatory Gram-negative bacterial endotoxin [lipopolysaccharide (LPS)], mechanis

128 Here we show that in the presence of bacterial endotoxin, lipopolysaccharide (LPS), alpha-syn

129 cytokines and chemokines is a key feature of bacterial endotoxin, lipopolysaccharide (LPS)-induced in

130 h-old) mice experimentally challenged with a bacterial endotoxin, lipopolysaccharide (LPS, 1.5 mg/kg

131 Bacterial endotoxin, lipopolysaccharide, is a prototypic

132 These events are activated by the bacterial endotoxin, lipopolysaccharide, that is release

133 rmeability and subsequently translocation of bacterial endotoxin-lipopolysaccharide into the blood.

134 The bacterial endotoxins lipopolysaccharides (LPS) are impor

135 Using an intratracheal bacterial endotoxin LPS challenge model, we found that t

136 Finally, ChQ prevents mouse PTBs induced by bacterial endotoxin LPS or progesterone receptor antagon

137 are more sensitive to the lethal effects of bacterial endotoxin LPS, and in the experiments reported

138 4, which recognize viral dsRNA poly(I:C) and bacterial endotoxin LPS, respectively.

139 CD14 is a major receptor for the bacterial endotoxin LPS.

140 tor in the CNS innate immune response to the bacterial endotoxin LPS.

141 Stimulating macrophages with bacterial endotoxin (LPS) activates numerous intracellul

142 Recognition of bacterial endotoxin (LPS) elicits multiple host response

143 ated after intraperitoneal administration of bacterial endotoxin (LPS) in murine lung and kidney, but

144 on of NO in the down-regulation of CYP2B1 by bacterial endotoxin (LPS) in rat hepatocytes cultured on

145 Bacterial endotoxin (LPS) is responsible for much of the

146 ration across endothelial cells activated by bacterial endotoxin (LPS) or IL-1beta (60 and 46%, respe

147 we found that treatment of macrophages with bacterial endotoxin (LPS) or Pseudomonas induced L-PGDS

148 ages are among the most sensitive targets of bacterial endotoxin (LPS), responding to minute amounts

149 reported that increasing core temperature of bacterial endotoxin (LPS)-challenged mice to the normal

150 Bacterial endotoxin (LPS)-induced synthesis of anandamid

151 Bacterial endotoxin (LPS)-mediated sepsis involves sever

152 changes in acute inflammation in response to bacterial endotoxin (LPS).

153 Thus, innate responses to fibrinogen and bacterial endotoxin may converge at the evolutionarily c

154 We previously demonstrated in a bacterial endotoxin mouse model of BPD that disrupting f

155 chemotactic activity during incubation with bacterial endotoxin or aggregated IgG, (b) to mediate th

156 exposed to mediators of inflammation such as bacterial endotoxin or lipopolysaccharide (LPS) in a var

157 Bacterial endotoxin or lipopolysaccharide is the major p

158 ndly altered response when rechallenged with bacterial endotoxin or lipopolysaccharide.

159 ed in media, with or without the addition of bacterial endotoxin or varying molar concentrations of e

160 rophages (AMs) prechallenged with either the bacterial endotoxin or with Pseudomonas aeruginosa and o

161 Bacterial endotoxins or lipopolysaccharides (LPS), cell

162 e-establish intestinal symbiosis, neutralize bacterial endotoxins, or adsorb gut-derived uremic toxin

163 at can be induced by inflammatory cytokines, bacterial endotoxin, osmotic shock, UV radiation, and hy

164 We report that bacterial endotoxin persists in human donor lungs after

165 TLR4, the innate immunity receptor for bacterial endotoxins, plays a pivotal role in the induct

166 -1 cells, using diverse cell stimuli such as bacterial endotoxin, proinflammatory cytokines (IL-1 and

167 ithin the serum, and decreased levels of the bacterial endotoxin protein in the asthmatic state.

168 h had been added one of 5 different doses of bacterial endotoxin ranging from 0.02 to 1.4 endotoxin u

169 eptor 4 (TLR4) is the principal receptor for bacterial endotoxin recognition, and functional variants

170 t mammalian cell activation by Gram-negative bacterial endotoxin requires sequential protein-endotoxi

171 olecular pattern molecules (PAMPs) including bacterial endotoxin, respiratory viruses, and microbial

172 The results indicate that AR mediates the bacterial endotoxin signaling that could damage HLECs by

173 Bacterial endotoxins such as lipopolysaccharides (LPS) a

174 munity as a pattern recognition receptor for bacterial endotoxins, such as LPS.

175 icals was found to pass all requirements for bacterial endotoxin testing as specified in the United S

176 tion of Limulus amebocyte lysate (LAL)-based bacterial endotoxin testing using centrifugal microfluid

177 Volume delivered, pH, sterility, and bacterial endotoxin tests yielded passing results on all

178 g inquiry into the mechanism of responses to bacterial endotoxin, the abundant lipopolysaccharide com

179 influence cytokine production in response to bacterial endotoxin; the high LBP:BPI ratios observed in

180 h kills meningococci and binds to and clears bacterial endotoxin, these being the primary inducers of

181 ng of lipopolysaccharide (LPS, also known as bacterial endotoxin) to human hemoglobin is known to res

182 eripherally with lipopolysaccharide (LPS), a bacterial endotoxin, to induce an inflammatory episode.

183 ated alcohol-induced gut barrier disruption, bacterial endotoxin translocation into the circulation,

184 Septic shock from bacterial endotoxin, triggered by the release of lipopol

185 Our findings demonstrate that flies detect bacterial endotoxins via a gustatory pathway through TRP

186 Lipopolysaccharide (LPS), a bacterial endotoxin, was used to elicit IL-6 production

187 athogenesis of sepsis is mediated in part by bacterial endotoxin, which stimulates macrophages/monocy

188 (NK) cells and displayed hypersensitivity to bacterial endotoxin, with their innate immune cells prod