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1 rains kill these amoebae and leave an intact bacterial lawn.
2 n prior to application to the phage-infected bacterial lawn.
3 icles, as indicated by plaque formation in a bacterial lawn.
4  on these bacteria and form plaques in their bacterial lawns.
5 enter development and display slow growth on bacterial lawns.
6  naturalistic environments including thicker bacterial lawns.
7 trackers including low worm density and thin bacterial lawns.
8  in cell migration, forming large plaques on bacterial lawns.
9           Solitary foragers move slowly on a bacterial lawn and disperse across it, while social fora
10 e atg1-1 mutant, which does not aggregate on bacterial lawns and arrests as loose mounds on nitrocell
11 r primarily clear circular zones of lysis on bacterial lawns at the site of gamma phage inoculation a
12 cidal compounds have been shown to eradicate bacterial lawn biofilms through electroceutical treatmen
13 phagy mutants do not aggregate in plaques on bacterial lawns, but they do proceed further in developm
14 ses of bacteria, form visible lesions within bacterial lawns (called plaques), which are employed ubi
15                         When developing on a bacterial lawn, cells of the mutant strain (dtfA- cells)
16  The DdPPK1 mutant formed smaller plaques on bacterial lawns compared with those of the WT.
17    Furthermore, we find that biofilm-forming bacterial lawns including Bacillus subtilis and Pseudomo
18  bodies that do form when cells are grown on bacterial lawns lack the one- and two-dimensional symmet
19               The carbohydrate is present in bacterial lawns prior to addition of nematodes, indicati
20 , and atg8(-) is more aberrant in plaques on bacterial lawns than on nitrocellulose filters.
21                                In the assay, bacterial lawns were grown on agar plates, harvested wit
22 y evaluation showed improved robustness when bacterial lawns were tested with high- and low-density i